Lecture 10: Common Disorders of the Equine Hind Limb

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Last updated 10:25 PM on 8/19/26
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40 Terms

1
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cervical spine OA

  • caused by trauma, unnatural head carriage, OC

  • diagnosed by stiff neck, ± lame or neurologic, radiographs

  • treatment → inject facets with corticosteroid, acupuncture, mesotherapy

  • prognosis is fair


2
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<p>what is this?</p>

what is this?

cervical spine OA

3
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impinging dorsal spinous processes

  • kissing spines

  • caused by trauma, poor fitting saddle

  • diagnosed by pain on palpation, reduced performance, radiographs

  • treatment → check saddle fit, inject area with corticosteroids, surgical resection

  • prognosis depends on severity


4
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<p>what is this?</p>

what is this?

impinging dorsal spinous processes aka kissing spines

5
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general back muscle pain

  • caused by underlying hind limb lameness, poor saddle fit, poor riding, trauma

  • diagnosed by pain on palpation, reduced performance

  • treatment → check saddle fit, inject area with corticosteroids, acupuncture, rest

  • prognosis is good once address underlying cause


6
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sacroiliac disease

  • arthritis

  • subluxation of SI joint = hunter’s bump

  • caused by trauma, inappropriate use

  • diagnosed by clinical signs, acute → lame, can feel crepitus by rectal palpation

  • treatment is rest and inject SI joint with corticosteroids

  • prognosis is good


7
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<p>what is this?</p>

what is this?

hunter’s bump from sacroiliac disease

8
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what are the structures involved with causing hind limb lameness in the horse ranked from most common to least common?

hock → below hock → stifle → above stifle

9
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metatarsal 2 and 4 (splint bones) fractures

  • caused by kick injury

  • diagnosed by radiographs, observing wound

  • treatment → ostectomy (segmental vs complete)

  • prognosis is excellent


10
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<p>what is this?</p>

what is this?

splint bone fracture

11
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hock lameness gait characteristics

  • drags toes

  • fails to “track up” on lame leg

  • lame leg adducts most

  • rider feels difference in their connection

  • reduced performance


12
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joint palpation for hock lameness

  • often none

  • bone spavin → hard swelling

  • bog spavin → effusion


13
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diagnosis of hock lameness

  • flexion tests

  • blocks

  • radiographs

  • nuclear scintigraphy

  • CT


14
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what is the most common hind end lameness diagnosed in performance horses?

hock OA

15
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what joints are affected by hock OA?

distal intertarsal and tarsometatarsal joints

16
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hock OA

  • caused by athletic use or genetics

  • diagnosed by + upper limb flexion, blocks to distal hock joints, radiographs

  • treatment → intra-articular corticosteroids, shoeing, facilitated ankyloses or arthrodesis

  • prognosis is good


17
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<p>what joints are affected in this radiograph?</p>

what joints are affected in this radiograph?

distal intertarsal and tarsometatarsal

18
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tibiotarsal effusion (bog spavin)

  • caused by OCD, synovitis, OA (rare), blood (hemoarthrosis), infection (septic arthritis)

  • diagnosed by effusion in TT joint, radiographs, ultrasound, diagnostic arthroscopy

  • treatment → drain joint and inject corticosteroids, arthroscopy

  • prognosis is good


19
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hock OC location from most to least common

  • distal intermediate ridge of the tibia (DIRT)

  • trochlear ridge (lateral > medial)

  • medial malleolus


20
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<p>what view is this?</p>

what view is this?

DLPMO

21
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plantar ligament desmitis (curb)

  • caused by strain on plantar aspect of hock, sickle hocked horse

  • diagnosed by characteristic swelling, mild to moderate lameness, blocks, ultrasound

  • treatment → usually just need rest

  • prognosis is good, will always have a curb swelling


22
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what breed is overrepresented for plantar ligament desmitis?

standardbreds

23
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what is this?

plantar ligament desmitis

24
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calcaneal bursitis (capped hock)

  • caused by repeated trauma → stall kicker

  • diagnosed by characteristic swelling, generally not lame

  • treatment → drain and inject corticosteroids

  • prognosis is excellent for function, poor for cosmesis


25
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<p>what is this?</p>

what is this?

calcaneal bursitis

26
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septic calcaneal bursitis

  • caused by wound over calcaneus (common area to be kicked)

  • diagnosed by being very lame, pressurize bursa, contrast radiographs, ultrasound

  • treatment → flush bursa, regional limb perfusion, may require opening bursa and resecting part of calcaneus

  • prognosis is guarded (50/50)


27
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<p>what is this?</p>

what is this?

septic calcaneal bursitis

28
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tarsal sheath tenosynovitis (thoroughpin)

  • caused by wear and tear, straight hocks

  • diagnosed by characteristic swelling, usually not lame

  • treatment → usually none

  • prognosis is excellent for athletic use, guarded for cosmesis


29
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<p>what is this?</p>

what is this?

tarsal sheath tenosynovitis

30
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ruptured peroneus tertius

  • caused by trauma, full limb hind limb casts

  • diagnosed by ability to extend the hock with a flexed stifle, lameness and swelling, tarsus extends more than usual at the trot

  • treatment → avulsions near stifle, stall rest

  • prognosis is good, poor if it involves stifle joint


31
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superficial digital flexor or gastrocnemius muscle/tendon rupture

  • caused by trauma

  • diagnosed by clinical signs, ultrasound

  • treatment → long term splinting and rest

  • prognosis is fair to poor, good if it heals


32
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<p>what is this?</p>

what is this?

superficial digital flexor or gastrocnemius muscle/tendon rupture

33
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fibrotic myopathy

  • fibrosis ± ossification of the semitendinosus muscle

  • caused by trauma

  • diagnosed by characteristic movement (most evident at walk), palpable hardening in semitendinosus mm

  • treatment → semiten ± semimem tenotomy

  • prognosis is fair for success of treatment


34
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gait characteristics of stifle lameness

  • jumps off lame leg in weight bearing phase → quicker, short step

  • drags toe because leaves leg out behind

  • increased lameness when limb is on outside of circle


35
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stifle lameness

  • clinical signs → joint effusion (femoropatellar pouch easily palpable), hind end muscle atrophy with chronic problem

  • diagnosed by flexion test of upper limb and caudal extension, blocks, radiographs, ultrasound, arthroscopy, nuclear scintigraphy


36
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stifle OC

  • most common breeds are WB, TB, QH

  • lesions → medial femoral cysts, trochlear ridge OCD

  • diagnosed by effusion, lameness when starts training, radiographs

  • treatments → surgery → wait until 1 yr

  • prognosis depends on severity and success of surgery, leads to OA


37
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<p>what is this?</p>

what is this?

medial trochlear ridge OCD on stifle

38
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<p>what is this?</p>

what is this?

stifle cyst lesion from stifle OC

39
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stifle OA

  • caused by underlying OC, trauma, athletic use

  • diagnosed by blocks to the stifle

  • treatment → intra-articular corticosteroids

  • prognosis is fair - high in motion, complicated joint


40
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upward fixation of patella

  • inability to release stay apparatus → patella does not disengage from the medial trochlea of femur

  • caused by weak quadriceps, straight legs

  • diagnosed by stifle being locked in extension (severe) or horse catches their stifle (mild)

  • treatment → immediate = back horse up, exercise strengthen quadriceps, surgery = medial patellar ligament splitting or blistering, medial patellar ligament desmotomy