Lower Motor Neurons Disease

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25 Terms

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Upper Motor Neuron Tracts
Which part of the Motor System delivers signals to spinal interneurons and LMNs?
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Lower Motor Neurons
Which part of the Motor system delivers signals directly to the skeletal muscles and elicits contraction of muscle fibers?
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Top down ( Brain to Muscle)
Voluntary movements are controlled from _____ __to__ _____ .
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Axial and Proximal muscles
The Medial aspect of the anterior horn controls …
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Distal Muscles
The Lateral aspect of the anterior horn controls…
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Extensor Muscles
The Anterior aspect of the anterior horn controls…
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Flexor Muscles
The Posterior aspect of the anterior horn controls…
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ALS (amyotrophic lateral sclerosis)
Which disorder affects the upper motor neurons that arises from the cortex and brainstem as well as cranial/spinal lower motor neurons?
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Lower Motor Neuron Disease
The following clinical findings are consistent with

* weakness
* flaccid paralysis of involved muscles
* muscle atrophy
* degeneration of muscle fibers over time
* histological-reaction (degeneration 10-14 days of injury)
* reflexes of the involved muscles are diminished/absent
* No abnormal reflexes are obtainable
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It is when the distal portion of the nerve and the mylin following a lesion is reabsorbed.
What is Wallerian Degeneration?
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The time it takes to build enough ACH to send an AP
What is distal latency on an EMG?
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Onset Latency and Peak Latency
What are the 2 ways that latency is measured (Sensory Nerve AP)?
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The time measured from the stimulus to the initial deflection from baseline
What is Onset Latency?
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The time measured from the stimulus to the peak of the spike
What is Peak Latency?
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It will appear normal b/c you are still testing the healthy portion of the axon ( normal DL and CV)
If the conduction block is **proximal** to both simulation sites what do you expect to see in the CMAPs?
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Barely any signal will go through due to the “break” in the pathway so DL will be increased and CMAP amplitude will decrease
If the conduction block is **distal to both** simulation sites what do you expect to see in the CMAPs?
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The DL and distal CMAP will be normal but the proximal CMAP will show decreased amplitude
If the conduction block is **between both** simulation sites what do you expect to see in the CAMPs?
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Slows down the time as well as possibly no recovery
Once the protective layer of the nerve (endoneurium and perineurium) is damaged how does it affect the level of recovery?
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Decreased amplitude of both distal and proximal CMAP
When it comes to pure Axonal Loss, what would we see in the EMG?
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Increased distal latency and slow conduction velocity
When it comes to pure Demyelination, what would we see in the EMG?
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If the nerve is viable or not
What is one guiding factor we can determine looking at an EMG?
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Stretch Injury, Lacerations, Compression and Repetitive stress injury
What are some of the major causes of (local)Peripheral Neuropathies?
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Cancers, Infections, Inherited Neuropathies (Guillain-Barre), Diabetes (2), Kidney disorders (high levels of ammonia), Autoimmune diseases, Vascular disease, Vitamin Deficiencies and Alcoholism
What are some of the major causes of (systemic )Peripheral Neuropathies?
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UMN injury
If you see signs of hyper-reflexive that usually correlates to UMN or LMN injury
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LMN injury
If you see signs of hypo-reflexive that usually correlates to UMN or LMN injury