NPTE: SCI

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Last updated 10:41 PM on 7/6/26
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46 Terms

1
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Anterior cord syndrome

hyperflexion injury

-loss of pain and temperture bilaterally

-loss of motor bilaterally

<p>hyperflexion injury</p><p>-loss of pain and temperture bilaterally </p><p>-loss of motor bilaterally </p>
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Central cord syndrome

aka walking SCI

hyperextension injury

if small lesion: bilateral pain and temp

if large lesion:

-loss of motor and sensory function

-affects motor > sensory

-affects UE > LE and proximal > distal

SAFETY RISKS BUT USUALLY CAN AMBULATE

affected tracts: spinothalamic, corticospinal, dorsal column

<p>aka walking SCI</p><p>hyperextension injury</p><p>if small lesion: bilateral pain and temp</p><p>if large lesion: </p><p>-loss of motor and sensory function </p><p>-affects motor &gt; sensory </p><p>-affects UE &gt; LE and proximal &gt; distal </p><p>SAFETY RISKS BUT USUALLY CAN AMBULATE </p><p>affected tracts: spinothalamic, corticospinal, dorsal column </p>
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Brown-Sequard Syndrome

stab gunshot wound

-ipsilateral motor loss (corticospinal)

-ipsilateral proprioception, light touch, vibration loss (dorsal column)

-contralateral loss of pain and temperature sensation (spinothalamic)

nemonic: brown pott

<p>stab gunshot wound</p><p>-ipsilateral motor loss (corticospinal)</p><p>-ipsilateral proprioception, light touch, vibration loss (dorsal column)</p><p>-contralateral loss of pain and temperature sensation (spinothalamic)</p><p>nemonic: brown pott</p>
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Cauda equina syndrome

injury below L1

-lower extremity motor and sensory loss

-areflexic bowel and bladder and saddle anesthesia

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Posterior Cord Syndrome

causes: RARE iatrogenic aka medical error; compression of posterior spinal artery or (NPTE focuses on medical error) hyperextension injury

-DCML affected ... loss of vibration, proprioception, 2 point discrimination, sterognosis

-motor function is preserved

-sensory ataxia due to DCML involvement

<p>causes: RARE iatrogenic aka medical error; compression of posterior spinal artery or (NPTE focuses on medical error) hyperextension injury</p><p>-DCML affected ... loss of vibration, proprioception, 2 point discrimination, sterognosis</p><p>-motor function is preserved </p><p>-sensory ataxia due to DCML involvement</p>
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Dorsal vs ventral horns vs intermediate

dorsal (posterior): sensory

ventral (anterior): motor

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Spinal shock

transient period of reflex depression and flaccidity; can last several hours to 6. months or more

S/S: flaccid paralysis, areflexia, vasodilation, decreased cardiac output and venous pooling

-abscence of reflex activity impairs autonomic regulation, which results in hypotension and loss of ability to sweat

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What reflex indicates the body is out of spinal shock?

bulbocavernosus S2-4

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What level do you start to see respiratory function issues?

above C4

-above C3-C5 need ventilator because supplies phrenic nerve and assist diaphragm control

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Below ___ patient can normally be weaned from ventilator

C4 (must teach assistive coughing to help; glossopharngeal breathing is used when diaphragm is denervated)

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At ___ and below patient can learn self assisted coughing techniques

C5

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Below ___ patient is likely to have near normal ventilatory respiratory functions

T10

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Respiratory muscles C1-T11

knowt flashcard image
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Autonomic dysreflexia

occurs with injuries at or above T6 --> disruption of sympathetic response

-MEDICAL EMERGENCY

-vasoconstriction occurs leading to hypertension and decreased HR

-increased BP, decreased HR, headache, flushing, profuse sweating, anxiety, constricted pupils, blurred vision, piloerection

-systolic increases by 20-30

-vasoconstriction below level of injury and vasodilation above

<p>occurs with injuries at or above T6 --&gt; disruption of sympathetic response</p><p>-MEDICAL EMERGENCY</p><p>-vasoconstriction occurs leading to hypertension and decreased HR</p><p>-increased BP, decreased HR, headache, flushing, profuse sweating, anxiety, constricted pupils, blurred vision, piloerection</p><p>-systolic increases by 20-30</p><p>-vasoconstriction below level of injury and vasodilation above</p>
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Factors that cause autonomic dysreflexia

-bowel and bladder distension, infection, impaction, catheterization blocked, abdominal binder

-skin breakdown, pressure sores, ingrown toenail, and similar stimuli

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neurogenic shock

occurs with SCI above T6

-S/S: decreased BP, decreased HR, cyanosis, warm and dry extremities, decreased CO, peripheral vasodilation and venous pooling

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Orthostatic hypotension seen with SCI above

T6

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Spastic bladder

seen in injury above S2 sacral segment

-bladder contracts and reflexively empties in response to certain level of filling pressure

-reflex action present

-treat with intermittent cauterization every 3-6 hours

-bladder can be emptied by: manual stimulation or timed voiding schedule

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Flaccid bladder

seen in injury below S2

-no reflex present

-can be emptied by: crede maneuver (manual compression of lower abdomen; valsalva; timed voiding schedule

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Independence level for each spinal cord injury

knowt flashcard image
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Heterotopic ossifications

- Common

- Generally near large joints

- No aggressive stretching!

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Fasciculus cuneatus

ascending tract; sensory tract for trunk, neck, and UE proprioception, vibration, two-point discrimination, and graphesthesia

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Fasciculus gracilis

ascending tract; sensory tract for trunk and LE proprioception, vibration, two-point discrimination, and graphesthesia

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Spinocerebellar tract (dorsal)

ascending tract; sensory tract that ascends to the cerebellum for ipsilateral (I/L) subconscious proprioception, tension in muscles, joint sense, posture of the trunk and LEs

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Spinocerebellar tract (ventral)

ascending Tract; sensory tract that ascends to the cerebellum, with some fibers crossing and subsequently recrossing at the level of the pons for I/L subconscious proprioception; tension in muscles; joint sense; and posture of trunk, UEs, and LEs

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Spinothalamic tract anterior

ascending tract; sensory tract for light touch and pressure

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Spinothalamic tract (lateral)

ascending tract; sensory tract for pain and temperature sensation

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Corticospinal tract anterior

descending tract; pyramidal motor tract responsible for I/L voluntary, discrete, and skilled movements

Note: Damage to corticospinal (pyramidal) tracts results in a positive Babinski sign, absent superficial abdominal and cremasteric reflexes, and the loss of fine motor or skilled voluntary movement

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Corticospinal tract lateral

descending tract; pyramidal motor tract responsible for C/L voluntary fine movement

Note: Damage to corticospinal (pyramidal) tracts results in a positive Babinski sign, absent superficial abdominal and cremasteric reflexes, and the loss of fine motor or skilled voluntary movement

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Where does the spinal cord end?

L1 and then goes into conus medullaris then to cauda equina (we do lumbar punctures at L3-4 to avoid spinal cord injury)

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Dorsal column medial leminscus (DCML)

-Proprioception

-vibration

-graphesthesia

-barognosis

-stereognosis

-2-pt discrimination

-kinesthesia

-fine touch

(poor val got GBS 2x, keep fighting)

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Anterolateral system

Anterior STT: Crude touch ("did you feel it")

Lateral STT: Pain and temperature

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Corticospinal tract

movements

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Which tracts are ascending vs descending tracts

ascending: sensory tracts (DCML and anterolateral system)

descending: corticospinal

<p>ascending: sensory tracts (DCML and anterolateral system)</p><p>descending: corticospinal</p>
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Conus medullaris vs cauda equina

conus medullariS = symmetrical

cauda equinA = asymmetrical

<p>conus medullariS = symmetrical </p><p>cauda equinA = asymmetrical </p>
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Complete vs incomplete SCI

complete: No sensory or motor function in the lowest sacral segments (S4 and S5)

Incomplete: Motor and/or sensory function below the neurological level including sensory and/or motor function at S4 and S5

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ASIA SCI assessment segmental level muscle groups C5-S1

knowt flashcard image
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Motor level steps

1. Lowest Level at which strength is at least

3/5

2. All levels above being 5/5

Scored for each side, overall score is last

normal for both

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Sensory level steps

Defined as the level where sensory

function is normal on both sides of the

body.

Easy Steps:

1. Lowest level where you have "2's"

2. All above levels being "2's"

(0 is no sensation; 1 is present/impaired, 2 is normal)

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Neurological level

CAUDAL most level with NORMAL

sensory and motor function on

BOTH sides of the body.

Easy Steps:

1. Find Motor and Sensory level

2. Pick the higher one

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ASIA A-E scale

ASIA C: more than half have a grade less than 3/5

<p>ASIA C: more than half have a grade less than 3/5</p>
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Autonomic dysreflexia/hyperreflexia causes and signs/symptoms

• At or above T6

• Noxious stimuli below level of lesion

• Rise in systolic blood pressure of 20 to 30 mmHg is diagnostic of an episode of autonomic dysreflexia

• More common in chronic stage (3-6 months after injury); can be seen in acute too

• More common with complete SCI

<p>• At or above T6</p><p>• Noxious stimuli below level of lesion</p><p>• Rise in systolic blood pressure of 20 to 30 mmHg is diagnostic of an episode of autonomic dysreflexia</p><p>• More common in chronic stage (3-6 months after injury); can be seen in acute too</p><p>• More common with complete SCI</p>
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What to do with autonomic dysreflexia

• SIT UP and LOWER LEGS

• Remove painful stimuli:

̶Loosen clothing, abdominal binder

̶CHECK BLADDER distension: Unclamp catheter, drain it

• Monitor vitals throughout: If still no change, medical/nursing

assistance > meds to lower BP (Nifedipine, nitrates, and captopril)

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Functional level C1-L3 transfers vs WC/ambulation ability

look at chart with spinal cord injury functions

<p>look at chart with spinal cord injury functions</p>
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Diaphragm innervation

C3,4,5 keep the diaphragm alive

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C4 can be off ventilator if they do

glossopharyngeal breathing