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NURS 314 Exam 2
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S1 heart sound
First sound
“Lub”
closure of mitral and tricuspid valve
beginning of systole
Best heard at apex (5th intercostal space)
S2 heart sound
Second sound
“Dub”
closure of aortic and pulmonic valves
end of systole/beginning of diastole
best heard at base of heart (2nd intercostal space)
Splitting of S2
pulmonic valve closes slightly later than aortic
Aortic auscultation site
2nd right intercostal space
Pulmonic auscultation site
2nd left intercostal space
Erb’s Point auscultation site
3rd left intercostal space
Tricuspid auscultation site
4th left intercostal space
Mitral (apical) auscultation site
5th left intercostal space, midclavicular line
Use diaphragm of stethoscope for
higher-pitched sounds (S1, S2, pericardial friction rubs)
Use bell of stethoscope for
Lower-pitched sounds (S3, S4, murmurs)
Auscultation
Listen for rate, rhythm, extra sounds
Listen in Z pattern across all areas
Normal heart rate
60-100bpm
< 60bpm = bradycardia
> 100bpm = tachycardia
S3 heart sounds
ventricular gallop
normal in young adults and athletes, pathological in older adults (suggest CHF)
S4 heart sounds
Atrial gallop
can be normal in elderly, but can indicate stiff ventricle (HTN)
Thrills
palpable vibration
suggest turbulent blood flow from significant murmur
Heaves
visible or palpable lifting of chest wall
indicates ventricular hypertrophy
left heave: left ventricular hypertrophy (felt at apex)
right heave: right ventricular hypertrophy (felt at sternal border)
Murmur
sound produced by turbulent blood flow across valves or within heart chambers
Stenosis
narrowed valve impeding forward flow (e.g. aortic stenosis)
Regurgitation
valve doesn’t close properly, allows backflow (e.g. mitral regurgitation)
Septal defects
abnormal opening
Increased flow rate
fever, anemia, pregnancy (can cause physiological murmurs)
Characteristics of murmurs
timing (systolic/diastolic), location (where its heard), radiation, pitch and quality (blowing, harsh, rumbling), grade (I [barely audible] to VI [heard without stethoscope]), shape (crescendo, decrescendo, holosystolic)
Modifiable risk factors for heart disease
HTN, hyperlipidemia, smoking, obesity, sedentary, DM, poor diet, excessive alcohol consumption, stress
Non-modifiable risk factors for heart disease
age (increases with age), gender (males or menopausal women), family history (first-degree relatives with disease), ethnicity (African Americans at higher risk for HTN and CV disease)