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PT diagnosis for TOS
depends on movement pattern
TOS
refers to compression of neurovascular strcutures in the interscalene triangle
interscalene triangle
anterior: anterior scalene
posterior: middle scalene
inferior: medial surface of 1st rib
brachial plexus trunk and subclavian artery pass through
costoclavicular space
anterior: middle third of clavicle
posteromedial: by the first rib
posterolateral: upper border of scap
thoraco coraco pectoral space
superior: coracoid process
anterior: pec minor
posterior: ribs 2, 3, 4
brachial plexus continues through this space alongside the subclavian artery
bony abnormalities that could impact the thoracic outlet
cervical rib
elongated C7 transverse process
abnormalities of the 1st rub or clavicle
soft tissue abnormalities that could impact the thoracic outlet
congenital abnormality of the scalene muscles
hypertrophy of the anterior scalene
brachial plexus passing through the anterior scalene muscle
joint impairments that could impact the thoracic outlet
clavicle needs to be able to elevate, retract, and spin posteriorly during arm elevation
AC joint needs to allow scapular movement
symptoms of true nTOS
typically “compressor” symptoms, day>night
objecitve weakness and/or sensory loss
confirmed with positive neurophysiological testing
symptoms of disputed nTOS
“releasor” symptoms, night>day
subjective weakness and/or sensory changes
neurophysiological tests are normal
nature of symptoms with TOS
varies with upper or lower brachial plexus involvement
aggs for TOS
overhead work, prolonged sitting with forward head posture, and increased thoracic kyphosis
24 hr pattern for TOS
may awaken at night due to UE paresthesias
release phenomenon
release of tension and/or compression of the perineurial blood supply to the brachial plexus
signals return of normal sensation
prognostic indicator of favorable outcome
TOS “releasors”
those with symptoms at night due to release phenomenon
TOS “compressors”
pts with symptoms during the day associated with compression on the thoracic outlet from aggs
posture for TOS
rounded shoulders, forward head, increased thoracic kyphosis
also posterior tilt, downward rotation, and/or depression of the scaps
integ screen for TOS
cyanosis with vTOS
paleness with aTOS
positive cervical rotation lateral flexion tests in TOS patients
suggests elevated 1st rib
cervical side bendind is limited due to TP of T1 contacting and being blocked by elevated 1st rib
special tests for nTOS
supraclavicular pressure test
adson’s test
costoclavicular manuever
wright’s test
cyriax release test
roos test
potential pertinent history for TOS
may not report any meaningful history
may report more symptomatic with overhead activities
potential predisposing factors for TOS
bony abnormalities in the C or upper T spine and/or 1st rib
diagnosis of TOS is supported when
history includes non-radicular symptoms in the neck, shoulder,arm which are worsened by movements and/or position of the neck, arms, and shoulder girdle
accompanied by cluster of provocation tests
assessment of postural dysfucntions and “container” mobility indicates tension and/or compression on the brachial plexus
medical management for TOS
radiographs may be used to rule out a cervical rib, long TP of C7, or sequelae of prior clavicle fracture
C spine radiographs: rule out spondy or osteophytes
chest x-rays: rule out pancoast tumor
EMGS: may show abnormal results
conservative: botox injections to anterior and middle scalenes to temporarily reduce pain and spasm
PT diagnosis for T4 syndrome
usually a pattern of painful thoracic flexion
definition of T4 syndrome
clinical pattern that involves UE paresthesia and pain w/ or w/o symptoms into the neck and/or head
pathobiological mechanisms of T4 syndrome
hypothesized that segmental spinal nerves that contain afferent fibers from sympathetic nerves are entrapped, or sympathetic fibers are experiencing ischemia
hypothesized that sustained or extreme postures tax the arteriolar system of the SNS, causing microtrauma
S+S of T4 syndrome
paresthesia in all 5 fingers, in whole hand, or in forearm + hand
UL or BL
non dermatomal pattern
hands feel hot or cold, arm may feel heavy
hands feel or might be swollen
aches and pains, non dermatomal pattern
combo of neck, upper T and cranial pain w/ or w/o abnormal neuro signs
headaches that present in cap presentation
nature of symptoms for T4 syndrome
pains may be crushing, bursting, or like a tight band
aggs for T4 syndrome
pt dependent but often related to poor posture
eases for T4 syndrome
hot shower
massage/ mobilization
24 hr pattern for T4 syndrome
can start in AM and worsen with activity throughout the day
can be worse at night and affect sleep
objective exam for T4 syndrome
PA accessory mobs at T4 segment reproduces the pt’s symptoms
palpation of rib angles may elicit symptoms
observation of trophic changes in the UEs
positive neural provocation tests of the upper limbs
potential pertinent history for T4 syndrome
often insidious onset but may report:
symptoms onset after start of new job
change of work practice
starting a new hobby
jobs with bending and forward stooping, or sedentary posture
potential predisposing factors for T4 syndrome
sedentary lifestyle and impaired posture
PT management for T4 syndrome
manual therapy directed to T spine
postural edu and re-training
movement reeducation after successful manual therapy interventions
medical management for T4 syndrome
radiograph and EMG studies typically negative
NSAIDs may assist with symptom management
PT diagnosis for thoracic disc pathology
typically a pattern of thoracic flexion
S+S of thoracic disc pathology
may be asymptomatic
when symptomatic, symptoms are often non specific intermittent back pain
may or may not have sensory and/or motor changes
symptom nature for thoracic disc pathology
non specific pain and stiffness
aggs for thoracic disc pathology
coughing, sitting, bearing down
eases for thoracic disc pathology
standing, lying supine
24 hr pattern for thoracic disc pathology-
often stiffness first thing in the morning lasting for approx. 20 minutes
may worsen throughout the day with sitting
objective exam for thoracic disc pathology
impaired spinal ROM
possible positive neural provocation tests
may have neuro signs if disc is compromising a nerve root or the spinal cord
potential pertinent history for thoracic disc pathology
most likely will report insidious onset
could report traumatic event such as fall onto the buttocks
potential predisposing factors for thoracic disc pathology
occupations or activities that require repetitive flexion
increased thoracic kyphosis
PT management for thoracic disc pathology
initial: decrease symptoms and promote healing
encourage active rest: walking
avoid lifting/carrying as able
then, work on improving mobility and movement patterns
emphasize thoracic extension and stabilization exercise
address psotural impairments
medical management for thoracic disc pathology
imaging
radiographs may show spurring or failure at vertebral endplates
MRIs still gold standard
surgery
not common but options are laminectomy or costotransversectomy
PT diagnosis for throacic facet dysfunction
will depend on problematic movement pattern
throacic facet dysfunction
symptoms typically UL and may report stiffness
can result from hypo or hyper mobility of facet
pain with palpation of involved facet
PT management should focus on joint mobility and adressing relavent movement impairments
Indications for surgery for neurogenic TOS
For those with true neurological symptoms
weakness
Wasting of hand intrinsic muscles
Nerve conduction study less than 60m/second
Surgical options for neurogenic TOS
Technique should address the underlying cause of TOS
1st rib resection
Cervical rib resection
Anterior and middle scalenectomy
Remove fibrous bands
Remove callus
Indications for surgery for venous TOS
S+S of venous thrombosis
Surgical options for venous TOS
Thrombolytic therapy to dissolve acute thrombus
best if given within 1 week of symptom onset
Still effective up to 1 month post onset
Indications for surgery for arterial TOS
Potential for upper limb ischemia
Surgical options for arterial TOS
arterial reconstruction
Thrombolytic therapy or balloon thrombolectomy
Distal bypass grafting