853-T spine pathos

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56 Terms

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PT diagnosis for TOS

depends on movement pattern

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TOS

refers to compression of neurovascular strcutures in the interscalene triangle

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interscalene triangle 

  • anterior: anterior scalene

  • posterior: middle scalene

  • inferior: medial surface of 1st rib

  • brachial plexus trunk and subclavian artery pass through

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costoclavicular space

  • anterior: middle third of clavicle 

  • posteromedial: by the first rib

  • posterolateral: upper border of scap

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thoraco coraco pectoral space

  • superior: coracoid process

  • anterior: pec minor

  • posterior: ribs 2, 3, 4

  • brachial plexus continues through this space alongside the subclavian artery

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bony abnormalities that could impact the thoracic outlet

  • cervical rib

  • elongated C7 transverse process

  • abnormalities of the 1st rub or clavicle

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soft tissue abnormalities that could impact the thoracic outlet 

  • congenital abnormality of the scalene muscles 

  • hypertrophy of the anterior scalene 

  • brachial plexus passing through the anterior scalene muscle 

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joint impairments that could impact the thoracic outlet

  • clavicle needs to be able to elevate, retract, and spin posteriorly during arm elevation

  • AC joint needs to allow scapular movement 

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symptoms of true nTOS

  • typically “compressor” symptoms, day>night

  • objecitve weakness and/or sensory loss

  • confirmed with positive neurophysiological testing

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symptoms of disputed nTOS

  • “releasor” symptoms, night>day

  • subjective weakness and/or sensory changes

  • neurophysiological tests are normal

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nature of symptoms with TOS

varies with upper or lower brachial plexus involvement 

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aggs for TOS

overhead work, prolonged sitting with forward head posture, and increased thoracic kyphosis

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24 hr pattern for TOS

may awaken at night due to UE paresthesias

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release phenomenon 

  • release of tension and/or compression of the perineurial blood supply to the brachial plexus

  • signals return of normal sensation

  • prognostic indicator of favorable outcome 

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TOS “releasors”

those with symptoms at night due to release phenomenon

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TOS “compressors”

pts with symptoms during the day associated with compression on the thoracic outlet from aggs

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posture for TOS

  • rounded shoulders, forward head, increased thoracic kyphosis

  • also posterior tilt, downward rotation, and/or depression of the scaps 

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integ screen for TOS

  • cyanosis with vTOS

  • paleness with aTOS

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positive cervical rotation lateral flexion tests in TOS patients

  • suggests elevated 1st rib

  • cervical side bendind is limited due to TP of T1 contacting and being blocked by elevated 1st rib

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special tests for nTOS

  • supraclavicular pressure test

  • adson’s test

  • costoclavicular manuever

  • wright’s test

  • cyriax release test

  • roos test

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potential pertinent history for TOS

  • may not report any meaningful history

  • may report more symptomatic with overhead activities 

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potential predisposing factors for TOS

bony abnormalities in the C or upper T spine and/or 1st rib

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diagnosis of TOS is supported when

  • history includes non-radicular symptoms in the neck, shoulder,arm which are worsened by movements and/or position of the neck, arms, and shoulder girdle

  • accompanied by cluster of provocation tests

  • assessment of postural dysfucntions and “container” mobility indicates tension and/or compression on the brachial plexus

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medical management for TOS

radiographs may be used to rule out a cervical rib, long TP of C7, or sequelae of prior clavicle fracture

  • C spine radiographs: rule out spondy or osteophytes 

  • chest x-rays: rule out pancoast tumor

EMGS: may show abnormal results

conservative: botox injections to anterior and middle scalenes to temporarily reduce pain and spasm 

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PT diagnosis for T4 syndrome

usually a pattern of painful thoracic flexion

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definition of T4 syndrome

clinical pattern that involves UE paresthesia and pain w/ or w/o symptoms into the neck and/or head

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pathobiological mechanisms of T4 syndrome

  • hypothesized that segmental spinal nerves that contain afferent fibers from sympathetic nerves are entrapped, or sympathetic fibers are experiencing ischemia

  • hypothesized that sustained or extreme postures tax the arteriolar system of the SNS, causing microtrauma

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S+S of T4 syndrome 

  • paresthesia in all 5 fingers, in whole hand, or in forearm + hand

    • UL or BL

    • non dermatomal pattern

  • hands feel hot or cold, arm may feel heavy

  • hands feel or might be swollen

  • aches and pains, non dermatomal pattern

  • combo of neck, upper T and cranial pain w/ or w/o abnormal neuro signs 

  • headaches that present in cap presentation

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nature of symptoms for T4 syndrome

pains may be crushing, bursting, or like a tight band 

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aggs for T4 syndrome

pt dependent but often related to poor posture

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eases for T4 syndrome

hot shower

massage/ mobilization

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24 hr pattern for T4 syndrome

  • can start in AM and worsen with activity throughout the day

  • can be worse at night and affect sleep

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objective exam for T4 syndrome

  • PA accessory mobs at T4 segment reproduces the pt’s symptoms

  • palpation of rib angles may elicit symptoms

  • observation of trophic changes in the UEs

  • positive neural provocation tests of the upper limbs

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potential pertinent history for T4 syndrome

often insidious onset but may report:

  • symptoms onset after start of new job

  • change of work practice

  • starting a new hobby

  • jobs with bending and forward stooping, or sedentary posture

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potential predisposing factors for T4 syndrome

sedentary lifestyle and impaired posture

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PT management for T4 syndrome

  • manual therapy directed to T spine

  • postural edu and re-training

  • movement reeducation after successful manual therapy interventions

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medical management for T4 syndrome

  • radiograph and EMG studies typically negative

  • NSAIDs may assist with symptom management

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PT diagnosis for thoracic disc pathology 

typically a pattern of thoracic flexion

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S+S of thoracic disc pathology

  • may be asymptomatic

  • when symptomatic, symptoms are often non specific intermittent back pain

  • may or may not have sensory and/or motor changes

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symptom nature for thoracic disc pathology

non specific pain and stiffness

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aggs for thoracic disc pathology

coughing, sitting, bearing down

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eases for thoracic disc pathology

standing, lying supine

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24 hr pattern for thoracic disc pathology-

  • often stiffness first thing in the morning lasting for approx. 20 minutes

  • may worsen throughout the day with sitting

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objective exam for thoracic disc pathology

  • impaired spinal ROM

  • possible positive neural provocation tests

  • may have neuro signs if disc is compromising a nerve root or the spinal cord

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potential pertinent history for thoracic disc pathology

  • most likely will report insidious onset

  • could report traumatic event such as fall onto the buttocks

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potential predisposing factors for thoracic disc pathology

  • occupations or activities that require repetitive flexion

  • increased thoracic kyphosis

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PT management for thoracic disc pathology

initial: decrease symptoms and promote healing

  • encourage active rest: walking

  • avoid lifting/carrying as able

then, work on improving mobility and movement patterns

  • emphasize thoracic extension and stabilization exercise

  • address psotural impairments

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medical management for thoracic disc pathology

imaging

  • radiographs may show spurring or failure at vertebral endplates

  • MRIs still gold standard

surgery

  • not common but options are laminectomy or costotransversectomy

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PT diagnosis for throacic facet dysfunction

will depend on problematic movement pattern

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throacic facet dysfunction

  • symptoms typically UL and may report stiffness

  • can result from hypo or hyper mobility of facet

  • pain with palpation of involved facet

  • PT management should focus on joint mobility and adressing relavent movement impairments

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Indications for surgery for neurogenic TOS

For those with true neurological symptoms

  • weakness

  • Wasting of hand intrinsic muscles

  • Nerve conduction study less than 60m/second

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Surgical options for neurogenic TOS

Technique should address the underlying cause of TOS

  • 1st rib resection

  • Cervical rib resection

  • Anterior and middle scalenectomy

  • Remove fibrous bands

  • Remove callus

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Indications for surgery for venous TOS

S+S of venous thrombosis

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Surgical options for venous TOS

Thrombolytic therapy to dissolve acute thrombus

  • best if given within 1 week of symptom onset

  • Still effective up to 1 month post onset

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Indications for surgery for arterial TOS

Potential for upper limb ischemia

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Surgical options for arterial TOS

  • arterial reconstruction

  • Thrombolytic therapy or balloon thrombolectomy

  • Distal bypass grafting