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Auditory Nerve (CN VIII):
Sends auditory info to brainstem
Cochlear Nucleus:
First brainstem synapse; processes timing/intensity
Superior Olivary Complex:
Localizes sound via binaural input
Lateral Lemniscus:
Signal relay to midbrain
Inferior Colliculus:
Sound integration, reflexive responses
Medial Geniculate Body:
Thalamic relay to cortex
Auditory Cortex (Temporal Lobe):
Final auditory processing
Acoustic Neuroma/Vestibular Schwannoma:
Unilateral, slow-growing tumor; diagnosed via MRI; treated with surgery or radiation
Auditory Neuropathy Spectrum Disorder (ANSD):
Normal OAE, abnormal ABR; managed with amplification or CIs
Auditory Processing Disorder (APD):
Difficulty processing auditory info; diagnosed through behavioral tests; treated with therapy and strategies
Age-related Central Decline:
Affects complex processing; managed with training and amplification
Tinnitus
Definition: Perception of sound with no external source
Causes: Hearing loss, noise exposure, neurological
Mechanism: Hyperactivity in central pathways
Treatment: Sound therapy, CBT, counseling
Hyperacusis
Definition: Heightened sensitivity to sound
Causes: Cochlear damage, migraine, neurological
Treatment: Sound desensitization, therapy
Vestibular System
Anatomy: Semicircular canals, utricle, saccule
Disorders: BPPV, Meniere’s, vestibular neuritis
Symptoms: Vertigo, imbalance, nausea
Tests: ENG/VNG, calorics, rotary chair
Treatment: Vestibular rehab, repositioning maneuvers
Hearing Aids
Function: Amplify sound
Components: Microphone, amplifier, receiver, battery
Candidates: Mild-profound HL
Benefits: Improved speech understanding, access to sound
CROS/BAHA/Bone Conduction/OTC
CROS: For unilateral deafness
BAHA: Conductive or SSD
OTC: Mild-moderate HL
Implantable Devices
BAHA, Middle Ear Implants, Cochlear Implants
Components (CI): External processor, internal electrode
Candidates (CI): Bilateral severe-profound SNHL, limited HA benefit
Hearing Assistive Technology (HAT)
Types: FM systems, alerting devices, captioned phones
Use: Enhance hearing in specific settings (e.g., classrooms)
Goals of Amplification
Improve audibility
Support communication
Reduce listening effort
Acoustic feedback =
Whistling from leakage
Gain =
Output – Input
Distortion =
Sound degradation
Frequency Response =
Range of amplified Hz
Output limiting =
Max dB output
ITC
In the ear - (Mild-Moderate HL)
RIC / receiver in the ear canal
(Mild-Severe HL)
BTE/ behind the ear
(Mild-Profound HL, used in adults & children)
WHO ICF Model for DHH:
Anatomical structures:
Cochlea, auditory nerve
WHO ICF Model for DHH:
Activity limitation:
Difficulty understanding speech
WHO ICF Model for DHH: Participation restriction:
Avoidance of social events
WHO ICF Model for DHH
Personal factors:
Age, motivation, coping style
WHO ICF Model for DHH:
Environmental factors:
Family support, accessibility
Counseling & Aural Rehab
Communication strategies: e.g., face the person, reduce background noise
Consider individual’s preferred communication style
Use visual modalities (sign language, speechreading)
Deaf Culture & Multiculturalism
Terminology: Person-first vs. identity-first language
Modality: ASL, cued speech, oralism
Hearing Aid Components
a. Battery = Power source
b. Microphone = Picks up/converts sound
c. Receiver = Converts electrical to acoustic signal
d. Amplifier = Boosts signal strength
Vertigo
a. Causes: BPPV, Meniere’s, vestibular neuritis
b. Treatment: Repositioning, rehab, meds
Cochlear Implant Candidacy
✅ B) A person with moderately-severe to profound SNHL in both ears not benefiting from HAS
Difference: BAHA vs. CI
BAHA: Bone conduction; used for conductive loss or SSD
CI: Stimulates auditory nerve directly; used for severe SNHL
Q: What are symptoms of an acoustic neuroma?
A: Unilateral tinnitus, progressive hearing loss, poor word recognition.
Q: What is Auditory Neuropathy Spectrum Disorder (ANSD)?
A: Bilateral SNHL with present OAEs and abnormal ABR.
Q: What is Auditory Processing Disorder (APD)?
A: Normal hearing with poor speech-in-noise recognition and difficulty with auditory tasks.
Q: What causes tinnitus?
A: Hearing loss, noise exposure, neurological conditions.
Q: How is tinnitus treated?
A: Sound therapy, CBT, and counseling.
Q: What is hyperacusis?
Over-sensitivity to sound; can cause fear or annoyance.
Q: What are vestibular system components?
A: Semicircular canals, utricle, saccule.
What are common vestibular tests?
A: ENG/VNG, calorics, rotary chair.
What does a cochlear implant do?
A: Bypasses damaged cochlea to directly stimulate the auditory nerve.
What is the difference between a BAHA and a CI?
A: BAHA uses bone conduction for conductive/SSD; CI stimulates auditory nerve for SNHL.
Q: What is acoustic feedback in a hearing aid?
A: Whistling from sound leakage
Q: What does gain refer to in hearing aids?
A: Amplification level (output minus input
What does frequency response describe?
A: The range of frequencies a hearing aid amplifies.
Q: What does output limiting in hearing aids do?
A: Limits the maximum volume in dB.
Q: What is the WHO ICF model used for?
A: To assess hearing loss impact: structure, activity, participation, personal/environmental factors.
Which is preferred: ‘Deaf person’ or ‘person who is deaf’?
A: Use ‘Deaf person’ when referring to cultural identity; ‘person who is deaf’ in clinical contexts.