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soft tissue injuries
sprains
strains
dislocations
subluxations
sprain
abnormal twisting
stretching of joint or spinal musculature
injury to ligaments
can’t see ligaments on XRAY.. might need MRI
commonly in the ankle or knee
classified in grades
grade 1 = stretching/small tears
grade 2 = larger tears
grade 3 = complete tear
strains
muscle, fascial sheath, or tendon
stretched or torn
can palpate a torn muscle
very painful
firm but extremely tender
dislocations
a complete separation of bones in a joint
most clear sign → deformity
presentation: orthopedic emergency, chance of severe vascular damage
complications: avascular necrosis → dislocation resulted in blood supply cut off to joints, compartment syndrome
treatment: closed reduction, surgical open reduction, immobilization
nursing interventions: PERFUSION ASSESSMENTS, pain relief, support of joint, protection of joint, restrict motion/gentle ROM, rehabilitation
sublaxations
a partial or incomplete dislocation where joint surfaces remain partially in contact, often slipping back into place
bones in joint aren’t aligned properly but still in contact
presentation: partial or incomplete displacement of joint, less severe
treatment: closed reduction, surgical open reduction, immobilization but with less healing time
nursing interventions: PERFUSION ASSESSMENTS, pain relief, support of joint, protection of joint, restrict motion/gentle ROM, rehabilitation
sprains/strains
strain = muscular tissue
sprain = ligaments
clinical manifestations
pain
edema → swelling
decrease in function/weakness
ecchymosis
spasms
diagnostics
x-ray, MRI to r/o fracture or joint damage
complications
joint structure is unstable → tearing of ligaments that hold bones together
avulsion fracture → ligament has disconnect some of the bone from itself, so if ligament is pulled so severely, it can pull some of the bone away
hemoarthrosis → blood in the joints, can cause dysfunction and pain
long-term instability
interventions
prevention: stretching, warming up, strength training, balance training
treatment: stopping the activity, RICE (24-48 hours)
rest, ice, compression, elevate
pain relief → NSAIDs/acetaminophen, don’t give narcs
heat & movement > 48 hours
repetitive strain injuries (RSI)
catch-all term for symptoms and signs located in the neck, upper back, shoulder, arm, elbow, hand, wrist, and fingers
risks:
repetitive activities
high-intensity activity for long time w/o rest
poor posture/awkward position
computer use
overtraining for sports
interventions:
correct precipitating factor
modify behvaior
pain management: NSAIDs, heat/cold application
rest
PT
ergonomics
lifestyle modifications
carpal tunnel syndrome
etiology: compression of median nerve
most common neuropathy of UE
risk factors:
female → hormone imbalances & smaller carpal tunnels
cancer → fluid shifts + hormone imbalances
RA → inflammation and swelling around carpal tunnel
ganglion cysts → little cysts that live on the joints, cause compression of median nerve
DM → impacts perfusion, impacts neurovascular process
PVD → impacts perfusion, impacts neurovascular process
continous wrist movement
assessment:
weakness
burning pain
numbness
impaired sensation
tinel’s sign → tap on median nerve. positive response = sensation of tingling over the median nerve & shooting/tingling pain
phalen’s sign → allow wrist to fall freely and maintain position longer than 60 seconds. if compressed carpal tunnel → tingling of median nerve. see atrophy of thumb muscle and overall hand dysfunction
interventions:
prevention: good ergonomics
TX: relieve cause
early: stop activity, modification, hand splint
later: cortisone injection for anti-inflammatory to decr inflammation around carpal tunnel, PTanti-inflammatory
>6 months: carpal tunnel release Sx.. done if symptoms are lasting more than 6 months or if there is significant impairment to median nerve. open release surgery.
fractures
A partial or complete break in a bone
etiology:
traumatic
pathologic → like cancer
fracture classifications
open → open to environment and bone is exposed. traumatic force to cause this. infection risk is higher
closed → not open to the environment
fracture extent
complete
break goes completely through the bone
2 sides are not connected
incomplete
fracture is partly across bone shaft, but bone itself is still intact
fracture, but 2 ends of the bone are still connected
fracture displacement
displaced
two ends of broken bone are separated from each other
out of normal position
more tissue injury than non-displaced
comminuted
fracture with more than 2 fragments
caused by crush injury
fragments are free floating
seen more in severe trauma and falls from great heights
very complicated and requires more healing
non-displaced
bone fragments are in alignment
transverse: bone breaks in a straight line, perpendicular to its long axis
spiral: fracture line coils around the bone like a corkscrew
greenstick: incomplete bone break where bone bends and cracks on one side only. commonly occurs in children due to flexible bones
oblique: diagonal break across a bone’s long axis
stress: occurs in bones that are subjected to repeated stress
clinical manifestations of a fracture
edema
cause: disruption/penetration through skin/soft tissues/bleeding
significance: occlude circulation, nerve damage, compartment syndrome
pain
cause: trauma, spasm, pressure, movement of fracture
significance: reduction in motion
spasm
cause: irritation and protection
significance: displacement
deformity
cause: abnormal position
significance: signals fx, prevent union
ecchymosis
cause: extravasation of blood
significance: distal to injury, resolves
loss of function
cause: disruption
significance: address to preserve ability to restore function
crepitation
cause: crunching of fragments
significance: possible non-union, micromovement = osteogenesis
fracture care goals
REALIGN, IMMOBILIZE, RESTORE FUNCTION
closed reduction
nonsurgical, manual realignment, traction, casting, splints, external fixation, braces
nothing broken thru skin
do the least invasive procedure first due to risks w post-op
open reduction (ORIF)
surgical, internal fixation
disadvantages: risk for infection, anesthesia complications, comorbid disease
fracture healing steps
1. hematoma
bleeding at the site
creates semi-solid clot ~72 hrs after injury
sanguineous drainage = bloody drainage
2. organization into fibrous network
good new blood vessels, osteoblasts to form basis for new bone substance
maintain immobilization so don’t break fibers
3. osteoblasts mobilize, calcium deposits
calcium and magnesium help new bone form
4. callus formation
temporary thickened structure of cartilage and bone forms to stabilize the break
5. remodeling
hard, temporary callus is replaced by strong bone
takes 3 days - 6 months to get through this.. comply w immobilization and instructions/limitations
fracture cast healing
materials
plaster
cheaper, easier to mold
fiberglass
lightweight, stronger, water resistant, preferred
poly/cotton
3-D printed
allow for ADLs
variations
short arm:
stable wrist and metacarpal
long arm:
stable forearm/elbow, unstable wrist
body jacket:
stable spine
monitor for cast syndrome (small bowel obstruction)
compression of mesenteric artery cuts off blood flow to bowel
severe pain, pressure, N/V to abdomen
hip spica:
pelvis and one or both legs
long leg:
tibia, femur
short leg:
foot, ankle
cylinder:
knee or lower leg
jones dressing/jones cast dressing:
sterile foot dressing
fracture patient education
do
elevate first 48 hours → decr swelling
watch for 6 P’s
apply ice in plastic bag
if gets wet → dry completely with a hair dryer on low
hair dryer cold and diphenhydramine for itching
move joints above/below cast
do NOT
get cast wet (unless waterproof)
insert items into or remove parts
bear weight for 48 hours
stick anything into the cast (between cast and skin)
other
pain meds:
muscle relaxants
NSAIDs
opioids
other meds (open fracture)
tetanus and diptheria
cephalosporins
nutrition
options for mobility
no smoking
what are the 6 P’s?
pain
paresthesia
paralysis
nerves can’t transmit impulses
pallor
poor capillary refill
pulselessness
pulse is extremely weak
use doppler
poikilothermia
tissue is cold
traction
pulling force to affected area; counter-traction pulls in opposite direction
pressure to distal to align with proximal
also helps with healing → hematoma is forming and if can’t take to surgery soon, use this to start helping put bone in right area to promote healing
purpose:
prevent/reduce pain, spasms
immobilize area
fx reduction
treat pathology
types of traction:
skin traction
skeletal traction
external fixation
skin traction
short-term treatment
tape, boots, splints
maintains alignment
assists in reduction
diminishes muscle spasms
weights typically 5 or 10 pounds except pelvis
ex) buck’s traction
dependent on positioning & alignment of patient - laying in bed of sitting up in chair
must keep traction forces on at all times → only time to remove is in life threatening/limb threatening situation
no weights touching floor → must hang freely
weight must move freely thru pulleys → no knots
skeletal traction
longer treatment → maybe person can’t go get surgery for several weeks
treats fractures, contractures (extreme tightening of muscle)
wire/pin inserted into bone
immobilizes
weights = 5 to 45 pounds
attaching weight to wires/pins
can hold much more weight and be in place longer
disadvantages:
infection, consequences of immobility
external fixation
metal pins & wires inserted into bones
attached to external rods
provides stabilization while fracture heals
used for complex fractures, bony defects, limb lengthening
increased risk for infection
requires pin care
hydrogen peroxide and gauze
complications of healing - infection
high incidence
devitalized/contaminated tissue is growth medium
anaerobic bacteria
leads to increased cost/risk for morbidity
treatment:
aggressive surgical debridement
IV antibiotics for 7 days-6 weeks-6 months
warmth, fever, swelling, purulent drainage, bright red erythema
complications of healing - union problems
bones have not healed properly together
if person is at high risk for this, surgeon may not operate
nonunion (pseudoarthritis)
malunion
causes of delayed union and nonunion
poor blood supply
insufficient nutrients
insufficient immobilization
metabolic disorders: DM, PVD
smoking
complications of healing - compartment syndrome
can be caused by fractures, severe trauma, casts that are too tight, tight dressings
medical emergency
etiology:
internal
hemorrhage
swelling
fluid
external
tight dressings
tight casts
manifestations
6 P’s
pain
pallor
pulselessness
paresthesia
poikilothermia
interventions
do not elevate limb, keep at heart level → want to promote blood flow to the area
no cold packs → don’t wanna decr blood supply
remove or loosen bandages, split cast → don’t wanna worsen blood flow
monitor BP, prevent hypotension
O2 to maximize saturation
urgent surgical consult (fasciotomy)
cut into fascia and release pressure → allow room for swelling to occur
releases pressure on nerves and blood vessels
allows tissues to be perfused
high risk of infection → use antibiotics
assess for neurovascular intactness → get baseline and check frequently
complications of healing - VTE
common causes
surgery → venous stasis causes clots and blood can’t go back to heart..
clot can travel to heart and lungs to be deadly
fracture of hip, pelvis, long bones
multiple trauma
immobility → muscles aren’t contracting → venous stasis, so no blood going back to heart
prior VTE
venous stasis
hypercoagulable state
could be from a med
endothelial injury
during surgery → vessels are cut
S&S
thigh/calf pain or tenderness
UNILATERAL EDEMA
warm to touch
red streaks/discoloration
general interventions:
prevent and treat
heparin/enoxaparin
early immobilization of fracture
early ambulation
compression stocking
SCDs - sequential compression devices
complications of healing - myositis ossificans
calcification in soft tissue
new bone forms in muscle tissue after injury - healing process occurs in wrong place
usually from trauma
tx:
ROM exercises to loosen area and maintain functionality
strengthening
ESWL (extracorporeal shockwave lithotripsy)
using shock waves to break apart bone tissue
sx:
pain with movement
swelling
limited ROM
hard palpable lump in affected muscle/tissue
complications of healing - fat embolism
embolisms can travel to heart and lungs and cause major issues
release of fat globules that break off and enter bloodstream
emboli = moving
thrombus = stationary
once thrombus is broken off and starts moving, it becomes an emboli
long bone break (tibia, femur, ribs, pelvis) leads to release of marrow
when breaks of long bones → incr risk of fat globules being released into the bloodstream
fat drop becomes stuck in small vessels → ischemia occurs at distal capillaries
hormonal changes caused by trauma can stimulate release of free fatty acids
can mimic other complications like pulmonary embolism
can happen in young and old people
factor in many fracture deaths
risks:
total joint replacement
spinal fusion
liposuction
crush injuries
bone marrow transplan
common MS causes:
multiple fx
fx of long bones
hip replacement
S&S
24-48 hours after fx
LOC changes - poor O2 exchange
confusion
restlessness
dyspnea
respiratory depression signs bc fat doesn’t belong in pulmonary vasculature of lungs
tachypnea
tachycardia
fever
petechiae
microhemorrhages → fat globules have landed in capillaries and caused breakage
hypoxia
interventions
prevention is key
early stabilization of long bones
patient education at D/C
promote lung expansion
oxygen IVF
monitor respirations
TCBD, repositioning
possible intubation & mechanical ventilation
complications of healing - osteomyelitis
severe infection: bone, marrow. periosteum
usually see it a few months after surgical intervention
infection in the bone
etiology:
open fracture
prosthetics
periodontal disease, UTI, TB, puncture wounds, infectious pathogens
risk factors:
trauma, open wounds, immunocompromised pts, vascular insufficiency, prosthetic devices
manifestations
acute < 1 month
usually local
bone pain unrelieved by rest, increased with activity, swelling, tenderness, warmth, decreased ROM
chronic > 1 month
local and sytemic
fever, chills, night sweats, nausea, malaise
diagnostics
history & physical
labs
WBCs, ESR, CRP
soft tissue or bone biopsy, blood/wound culture
x-rays, bone scans, MRI/CT
interventions:
acute
IV ABs - long term course, 6 week to 6 months
surgical debridement
AB irrigation
AB acrylic beads implanted in bone
chronic
IV ABs
surgeries
incision/drainage
debridement
sequestrectomy
bone graft
amputation - sometimes the best, safest option
hyperbaric O2
complications of healing - avascular necrosis
common MS causes:
interruption of blood supply to bone by fx → bone death, non healing
most common with long bones (hip, talus, femur at knee joint)
not an infection…
S&S, Dx
pain
decreased sensation
MRI diagnosis
general interventions:
surgery
sometimes bring in new bone
assessment of fracture
compare affected side with contralateral side
assess distal to fracture
vascular assessment
if not neurovascularly attached - could have necrotic tissue
skin color → supposed to be pink
skin temp → supposed to be warm
pulse → supposed to be palpable
cap refill → supposed to have under 3 secs
neuro assessment
movement
sensation
pain
neuro pain not gonna work w opioids
surgery: preoperative
standard pre-op workup
educate regarding immobilization device
activity limitations
assurance of assistance
pain control available
skin preparation
surgery: postoperative
monitor VS
NV checks
paralysis
paresthesia
pulse
cap refill
monitor positioning
turning, moving
monitor for compartment syndrome
monitor for drainage & bleeding
PT/OT
hip fractures
super common in elderly
90% are fall-related
by age 90:
30% of females and 20% of males will have hip fx
30-50% die within a yr due to complications of fx, immobility
potential long-term dsiability
due to decr muscle strength, altered reflexes, and meds that incr fall risk
classification of hip fx
intracapsular
within the joint capsule
higher risk of avascular necrosis
capital
subcapital
transcervical
extracapsular
outside of joint capsule
intertrochanteric
sub trochanteric
femoral neck
also incr risk of avascular necrosis
hip fracture manifestations
external rotation
muscle spasm → give muscle relaxants
shortening of affected extremity
severe pain & tenderness at site
hip fracture treament
temp bucks traction
reduces spasms
surgical repair
allows early mobilization
decreases risk of complications
hip fracture interventions
preoperative
assess co-morbidities
muscle spasm reduction
teach
proper exercise unaffected side
overhead trapeze/handrail use
postoperative
monitor VS, I&O
respiratory
monitor wound damage
assess extremity
hip fracture post-op care
regular assessments:
Vs
Is&Os
respiratory
TCDB → turn cough deep breathe
post surg patient at high risk of pneumonia bc meds depress breathing, not moving around, not breathing deeply
fluid gets caught in lungs
pain
wanna get up and moving
pain meds can help
check dressing/site
assess for complications:
hip dislocation
infection
bleeding
positioning
OOB day of or day after surgery
avoid turning to affected side
overbed trapeze
abductor pillow
do not let leg cross midline
don’t cross legs or bend joints greater than 90 degree angles
physical therapy
placement needs
osteoarthritis pathophysiology
cartilage destruction → wear and tear
joint space narrows
bone-on-bone
crepitation
osteophytes (bone spurs)
joint pain, stiffness, decreased mobility, muscle atrophy
osteoarthritis characteristics
chronic
progressive
degenerative
unilateral, can progress to bilateral
if on one side, body compensates and eventually can affect other side
non-systemic
localized inflammation/pain
shouldn’t be seeing it in places other than the joints
risk factors of osteoarthritis
trauma
inflammation
joint instability
mechanical stress
skeletal deformities
lack of exercise
obesity
aging
drugs
osteoarthritis joints involved
weight-bearing
hips
knees
spine
hands
osteoarthritis physical assessment
morning stiffness that goes away within an hour
joint pain - worsens as day goes on
early: goes away w rest
later: doesn’t go away w rest
acute to chronic pain
muscle spasms
crepitus
deformity (hands): bony growth at joint. debilitating and interferes with mobility
heberden’s nodes
bouchard’s nodes
psychosocial changes r/t pain, disability
osteoarthritis diagnostics
XRAY, MRI, CT
detect joint space narrowing, bony sclerosis, osteophyte formation
bone scan
detects bone growth, regeneration
osteoarthritis assessment
history
physical
impact on ability to perform ADLs
pain management beliefs, practices
physical exam of joints
compare with contralateral joint
osteoarthritis goals
maintain or improve joint function
not much else u can do to reverse other than joint replacement
use joint protective measures
achieve independence in self care and maintain optimal role function
use drug and non-drug strategies to manage pain
osteoarthritis interventions
medication
ASA
APAP
NSAIDs
topical creams
non-pharm pain mgt
acupuncture
yoga
massage
guided imagery
balance rest/activity
rest during acute inflammation
modify activities to decr joint stress
avoid prolonged standing, kneeling, squatting (knee OA)
diet & exercise
dietary changes PRN
aerobic
ROM
quad strengthening
assistive devices
splinting
support
cane/walker
temperature therapy
ice for inflammation
heat for stiffness
osteoarthritis patient education
pain mgt
body mechanics
corrective use of assistive devices
joint protection and energy conservation
nutrition
weight and stress management
exercise
osteoarthritis support
assure deformity is not usual course of OA
therapy, support groups
community resources - www.arthritis.org
rheumatoid arthritis pathophysiology
chronic, systemic autoimmune disease, inflammation of connective tissue in synovial joints
body attacks own cells
damage caused by inflammation
all ethnic groups, across lifespan, peak 30-50 yo
1.3 million americans
women > men (2-3x)
see higher incidence in women
1. precipitating event (antigen)
2. autoimmune response
3. inflammatory cascade
4. synovial inflammation
5. cartilage and bone damage
6. formation of rheumatoid nodules
7. systemic effects
rheumatoid arthritis S&S
early
nonspecific
joint inflammation
fatigue
aching
morning stiffness - 1 hr or longer
low-grade fever bc immune system active and thinks something is wrong
later
moderate to severe pain
joint softness, swelling
inflammatory process happening
red and swollen all the time
joint tenderness
muscle atrophy → weakness bc avoiding use of joint
systemic S&S
SQ nodules
osteoporosis
vasculitis
sjorgren’s syndrome
immune system attacks moisture-producing glands → causes dry eyes and mouth with more joint pain
joint deformities
boutonniere
swan neck
ulnar deviation
rheumatoid arthritis manifestations
boutonniere deformity
ulnar deviation
swan neck deformity of fingers
extra-articular manifestations
RA is a systemic disease → manifestations that happen outside of the joints
rheumatoid nodules
25% RA
SQ, firm, nontender, usually on extensor surface joints
usually not removed bc they just come back
if in lungs, sclera, poorer prognosis
sjorgrens syndrome
secondary to RA or independent d/o
10-15% RA
diminished lacrimal/salivary secretions
dry mouth, gritty, and burning eyes
decreased tearing and photosensitivity
rheumatoid arthritis diagnostics
HPI and physical findings
labs
WBC > 20k (mostly neutrophils)
+ RF (rheumatoid factor) in 80%
elevated ESR (erythrocyte sedimentation rate), CRP (C-reactive protein)
inflammatory markers
when these are elevated → body is in process of systemic inflammation
ANA (anti-nuclear antibodies)
anti-CCP (anti-cyclic citrullinated peptides)
four of the following:
morning stiffness > 1 hr
swelling in 3+ joints
swelling in hand joints
symmetric joint swelling
erosions on hand x-rays
rheumatoid nodules
serum RF
rheumatoid arthritis interprofessional team goals
prevent deformity
decrease inflammation
pain management
maintain function
increase mobility
rheumatoid arthritis interventions
acute
drug therapy
medication teaching
rest
joint protection
lightweight splints
ROM exercises
occupational therapist
plan care around pain
home care
stress management
balance nutrition
alternate rest with recovery
avoid total bed rest
modify activities to avoid overexertion
avoid flexion position
individualize exercise plan
rheumatoid arthritis meds
NSAIDs & anti-inflammatory
APAP and ibuprofen
DMARDs - disease modifying antirheumatic drugs
non-biologics
methotrexate → decreases progression of joint deformity and erosion
lower risk of toxicity. slows and helps suppress immune system
puts pt at risk of infections
avoid crowds
avoid illness
avoid ETOH
BRMs
modifies specific parts of immune system to help slow it down
most TNF blockers
etanercept (enbrel)
adalimumab (humira)
infliximab (remicade)
flare-ups
corticosteroids
systemic
local: intraarticular steroids
immune suppression → incr risk of infection
NSAIDs
topical analgesics
give immediately and consistently to slow disease progression and decrease risk of joint deformity
DMARDs
lessen joint erosions and deformity
chosen based on RA progression, functional level, lifestyle, cost
methotrexate
fewer sx in days to weeks, lower cost, fewer SEs
bone marrow suppression, hepatotoxicity, iatrogenic birth defects
lab monitoring: CBC, chem panel
given when flare-ups/exacerbations occur
corticosteroids (provide sx relief and immune suppression)