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Disc-Related LBP / Disc Herniation Presentation
• LBP ± unilateral buttock/leg symptoms
• sitting or flexion may aggravate some presentations
• cough/sneeze/strain may reproduce symptoms
• onset may be obvious or seemingly innocuous
• leg symptoms may occur with nerve root involvement
• typically ages 20-60
• PA mobility may be uncomfortable
Disc-Related LBP / Disc Herniation Examination
• repeated movement testing
• centralization/peripheralization
• identify directional preference when present
• SLR/slump when neural involvement suspected
• neuro screen when radiculopathy is suspected
Radicular Pain
• abnormal neural activity from nerve root/DRG
• leg pain may occur without neurologic loss
• may be caused by disc or facet
Radiculopathy
• motor, sensory, and/or reflex deficit
• trend neurologic findings over time
• may or may not occur with radicular pain
Adherent Nerve Root Presentation
• persistent/recurrent radiating leg symptoms
• symptoms with combined spinal + limb loading
• limited tolerance to SLR/slump positions
• may coexist with radicular pain or previous radiculopathy
• typically not acute
• limited flexion and ipsilateral deviation
• pain at end range
• increased motion with repeated motions
• symptoms with contralateral side bending
• SLR (+)
• quadrant test (-)
• may have muscle guarding/TTP
• no difference between sitting, standing, walking
Adherent Nerve Root Examination
• SLR/slump reproduces familiar symptoms
• myotomes, dermatomes, reflexes likely (-)
• structural differentiation changes symptoms
• compare sides and relevant joint positions
• determine whether spinal position alters the neurodynamic response
Active Lumbar Movement-Restriction (Facet Lock) Presentation
• sudden onset with rotation, extension, or awkward movement
• localized, often unilateral LBP
• may feel "stuck" or unable to straighten
• marked guarding and restricted lumbar movement
• usually little to no distal neurologic involvement
• limited flexion or extension
Active Lumbar Movement-Restriction (Facet Lock) Examination
• observe protective posture and movement avoidance
• AROM limited by pain/guarding
• extension/rotation may reproduce familiar symptoms
• localized PA testing may reproduced symptoms
• rule out other causes of an acutely painful lumbar presentation
• quadrant (+)
• deviation into flexion with sidebending towards affected side
• PA stiffness above or below
Facet-Mediated LBP Presentation
• axial/paraspinal pain
• may be aggravated by extension and/or rotation loading
• local or referred symptoms are possible
• typically unilateral
Facet-Mediated LBP Presentation Examination
• reproduce familiar symptoms with relevant loading
• assess movement and functional response
• no single clinical test reliably identifies the facet
Pars Stress Injury / Spondylolysis Presentation
• adolescent / young athlete (wrestling, gymnastics, football, volleyball)
• repetitive extension and rotation exposure
• typically involves MOI
• focal activity-related LBP
• extension loading may reproduce symptoms
• physical tests alone do not rull in diagnosis
• doesn't appear on x-ray for 10-14 days (MRI needed for diagnosis)
• typically unilateral
Isthmic Spondylolisthesis
• younger patient
• associated with pars defect
• repetitive extension/rotation exposure
• commonly L5-S1
• increased lordosis
• palpable divot in spine
• muscle guarding with PA mobility
• TrA/multifidus activation deficits
Degenerative Spondylolisthesis
• older adult
• commonly L4-L5
• may coexist with stenosis/claudication
• increased lordosis
• palpable divot in spine
• muscle guarding with PA mobility
• TrA/multifidus activation deficits
Lumbar Spinal Stenosis / Neurogenic Claudication Typical Patient
• usually older adult (60+)
• degenerative changes common
• may coexist with degenerative spondylolisthesis
Lumbar Spinal Stenosis / Neurogenic Claudication Presentation
• bilateral (central foramen) or unilateral (IV formamen) presentation
• leg symptoms > back symptoms
• buttock/thigh/leg pain, heaviness, paresthesia, or weakness
• worse with standing and walking
• better with sitting, flexion - "shopping cart" sign
• walking tolerance often limited
• limited extension
• extension, ipsilateral sidebending bring on symptoms
• may have flexed posture
Lumbar Spinal Stenosis / Neurogenic Claudication Examination
• walking/standing reproduce familiar symptoms
• flexion/sitting often relieves symptoms
• lumbar extension may reproduce symptoms
• neuro exam may be (-) at rest
• differentiate between neurogenic vs. vascular claudication
• slump/SLR (-)
• may have core weakness
• quadrant test (+)
Lumbar Movement-Control / Hypermobility Typical Patient
• often younger (< 40 years)
• female
• frequently active/athletic
• may have greater generalized mobility
• often reports previous LBP episode or injury
• recurrence may follow increased or novel activity
• increase in activity or new activity
• may be able to crack back on demand
Lumbar Movement-Control / Hypermobility Clinical Pattern
• localized/recurrent LBP
• painful arc with lumbar ROM
• aberrant movement from flexion
• may demonstrate Gower's sign
• sitting, standing, walking often tolerated short-term
• symptoms/guarding may increase with activity
• prone instability test (+)
• may have crease in low back
Lumbar Movement-Control / Hypermobility Examination
• observe flexion and return from flexion
• prone instability test
• PA testing may suggest increased mobility
• assess trunk control during functional loading
• look for meaningful adjacent mobility restrictions
• core assessment
• TrA/multifidus activation
• might have tight hip flexors, lengthened hamstrings
• SLR may be > 90°
• ↑ tone / muscle guarding with PA mobility
Degenerative Lumbar Presentations
• disc degeneration / spondylosis
• facet arthroplasty
• degenerative spondylolisthesis
• some improvement with repeated movements
• limited in multiple ranges
• heat improves pain
• limited PA mobility
Lumbar Overuse / Soft Tissue Presentation (Lower Cross Syndrome) Presentation
• gradual onset with repetitive loading / inadequate recovery
• deconditioning or working beyond current capacity
• movement patterns may repeatedly overload sensitive tissues
• muscle strain may occur suddenly on a background of overuse
• typically bilateral
• diffuse or localized LBP
• usually little or no distal radiation
• muscle guarding / splinting may be prominent
• symptoms may increase with extension or prolonged activity
• walking may be less tolerated; sitting may provide relief
• ↑ lordosis - tight hip flexors, lengthened abdominals, hypertonic paraspinals
• may have core weakness
• muscle guarding with PA mobility
• may be TTP
Lumbar Overuse / Soft Tissue Presentation (Lower Cross Syndrome) Examination
• reproduce familiar symptoms with relevant loading
• assess trunk/hip strength and endurance
• observe movement quality and control
• assess soft tissue sensitivity without over-interpreting trigger points
• recheck joint mobility as guarding decreases