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How was ADHD viewed in the early 1900s?
It was believed to be caused by poor “inhibitory volition” and “defective moral control.”
What historical event influenced early ideas about ADHD as a brain disorder?
The encephalitis epidemic of 1917–1918, which led to the concept of “brain-injured child syndrome.”
What is the current understanding of the core difficulty in ADHD?
Problems in self-regulation and behavioral inhibition.
What is the duration requirement for ADHD symptoms?
Symptoms must persist for at least 6 months.
By what age must ADHD symptoms appear?
Symptoms must be present before age 12, with evidence of onset before age 7.
In how many settings must symptoms be present?
Two or more settings (e.g., home and school).
What must symptoms cause to qualify for diagnosis?
Clear functional impairment or interference with developmentally appropriate functioning.
How many inattention symptoms must be shown for diagnosis?
Children: 6 or more
Adults: 5 or more
Give 3 examples of inattention symptoms.
Easily distracted
Avoids tasks needing sustained effort
Often forgetful in daily activities
How many hyperactivity-impulsivity symptoms are required?
Children: 6 or more
Adults: 5 or more
Give 3 examples of hyperactivity-impulsivity symptoms.
Talks excessively
Blurts out answers
Fidgets or squirms
What are the subtypes of ADHD?
Predominantly Hyperactive-Impulsive
Predominantly Inattentive
Combined Type
Can subtypes of ADHD change over time?
Yes, individuals may shift subtypes as they mature.
What are some associated issues with ADHD?
Learning problems
Executive function deficits
Low self-worth or inflated ego
Language impairments
Sleep problems
Risky behavior
What is the prevalence of ADHD in children and adults?
2–10% in children
1–6% in adults
What comorbidities are commonly seen with ADHD?
MDD, OCD
Mood and anxiety disorders
Learning and communication disorders
Tic and coordination disorders
How heritable is ADHD?
70–90% genetic contribution; higher concordance in MZ twins than DZ.
Which genes are linked to ADHD?
Dopamine transporter gene (DAT) and dopamine receptor gene (DRD4).
Name a few prenatal risks for ADHD.
Smoking during pregnancy
Birth complications
Early developmental issues
What brain structures show differences in ADHD?
Smaller corpus callosum, right frontal lobe, basal ganglia, and cerebellum
What neurotransmitter is primarily involved in ADHD?
Dopamine.
What are the main treatments for ADHD?
Medication
Parent Management Training (PMT)
Educational intervention
Combined/intensive interventions
Additional supports
What medications are commonly used for ADHD?
Stimulants (e.g., Adderall, Ritalin, Vyvanse)
Non-stimulants (e.g., Strattera)
Antidepressants (e.g., Wellbutrin)
Blood pressure meds (e.g., Clonidine, Guanfacine)
What does Parent Management Training (PMT) include?
Behavior modification
Environmental structuring
Reducing conflict
What are examples of educational interventions?
Daily report cards
Visual schedules
Classroom structure to support attention
What are examples of additional interventions for ADHD?
Counseling
Support groups
Caution against controversial treatments