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biomedical model
all illness can be explained by abnormal somatic processes
Traditional in Western medicine
Assumes that our health + disease is caused by physical + biological processes
People get sick because of physical things like viruses, mutated cells, genes, etc…
Ex: What contributes to heart disease? Plaque in arteries
biopsychosocial model + how it explains health
health is influenced by biological, psychological, and social factors
Something to consider: placebos; they don’t have any medical ingredients, so we cannot just use the biomedical factors to explain something psychological
How significant is the role of health behaviors (e.g., tobacco use, poor diet/physical inactivity) in current major health problems in the U.S.?
these factors make up about 50% of the contribution to US mortality, with tobacco, poor diet, and alcohol, being the most significant
does correlation = causation?
no, correlation is just an association between 2 variables
instead of A causing B, B could cause A
or C (third variable) could cause A and B
how correlation coefficients are interpreted (r = -.04) and positive/negative correlations
the closer you are to 1, the stronger the correlation
r = -.04 would be a not very strong and negative correlation
positive: variables change in same direction
negative: variables change in opposite directions
experiment
study where the independent variable is manipulated
moderators
variable that changes the magnitude and/or direction of the relation between the IV and DV
For example: If I study work experience and salary, I hypothesize that:
Work experience (years) affects salary
Gender identity moderates the above relationship
So, the relationship between years of experience and salary differs between men, women, and nonbinary people
mediators
variable through which the IV is related to the DV
For example: If I study socioeconomic status and childhood reading ability, I hypothesize that parent education level is a mediator
So, socioeconomic status affects reading ability mainly through its influence on parental education levels
internal validity
likelihood that changes in DV were caused by IV
Confounding variable: variable potentially responsible for change in DV, but it is not the IV
Threat to internal validity
external validity
likelihood that same results would be obtained using same study with other people/situations (generalizability)
confounds
variable that’s potentially responsible for the change in the DV, but is not the IV
ex: students who received caffeine may have also slept more or prepared more for the exam than the control group in a caffeine study
quasi-experimental design
comparison of groups without random assignment
What conditions must be met for causality to be tentatively assumed in quasi-experimental design?
Pre Existing groups differ
Study compares groups that were already formed before research started
Ex: you have people who don’t smoke and those who are already smokers
Temporal priority established (time)
Ensuring that the IV came before the DV
Do people smoke first and then develop lung cancer or the other way around?
Dose to response relationship exists
Relationship between amount of exposure to something and the resulting changes in body function/health
Do people who smoke more have higher rates of lung cancer than those who smoke less?
Intervention has an effect
Smokers who quit smoking: is there a change in their outcome (does their risk of lung cancer go down?)
Animal analogs
Animal behaviors used to study similar psychological phenomena in humans (usually done because it would be harmful or unethical to induce in humans)
retrospective design
predictor variables measured after outcome variables already known (looking backwards)
Issue: the outcome already occurred; data collection of the predictor variable is biased
Ex: does stress cause ulcers?
Measure whether or not people have ulcers and then measure stress in the past (in the past 6 months, how many stressful life events have you experienced?)
prospective design
predictor variables assessed in advance of outcome (looking forward in time)
Ex: measure stress first and see if they develop ulcers later
cross-sectional design
collecting data from sample at one point in time
Issue: can’t establish temporal precedence
Does not have to be different age groups; it means that all variables are being measured at the same time (measuring predictor and outcome variables at same time)
Ex: measure people’s stress & whether or not they have ulcers
longitudinal design
collecting data from sample at more than one point in time
Issue: time-consuming
Ex: measure stress first and later (ex: a month) measure if they developed ulcers
meta-analysis
Statistical combo of results of multiple studies
Ex: you want to know if meditation helps sleep: you could read all the studies on meditation and sleep, but if there’s a literature review, someone has compiled different studies and read about them
They pick out the effect size from each individual study and find the average
You could read one meta analysis and find out what’s going on in this field
Average effect size across studies
allostatic load + what contributes to it
repeated allostasis associated w/cumulative physiological cost (e.g. decreased immunity)
Allostasis is great, but that can happen to us too much, taking a toll on us
Results from:
Chronic stressors
Inability to adjust to continuous or recurrent stressors (failure of coping); someone goes to work everyday, stressed because of their boss
Inability to end stress response–traffic/aggressive behaviors of other drivers makes you stressed, then you get to work and you’re still stressed physiologically
What is the autonomic nervous system (including the sympathetic and parasympathetic nervous systems) and how is it affected by stress?
manages internal organs without conscious control
2 parts: parasympathetic and sympathetic nervous systems
sympathetic nervous system
mobilizes body to react to stressor/activity
(fight or flight; being chased by something)
Increases heart rate
Inhibits digestion
Opens lungs
Dilates pupils
Inhibits salivation
Increases blood glucose
Stimulates adrenal gland to release catecholamines (epinephrine and norepinephrine)
parasympathetic nervous system
nervous system–restores body to normal state after arousal (rest and digest)
Slows heart rate
Stimulates digestion
Constricts lung passages
Constricts pupils
Promotes salivation
Lowers blood glucose
endocrine system + how it’s affected by stress
regulatory system that secretes hormones
hypothalamus
control pituitary gland
pituitary gland
master gland
Secretes hormones that directly influence other endocrine glands
adrenal glands
above kidneys
Adrenal medulla (inner part of adrenal gland; responsible for fight or flight/epinephrine/norepinephrine) and adrenal cortex (outer layer of adrenal gland; produces hormones like cortisol/blood pressure regulating hormones)
SAM system (what they release + speed/duration of response)
activated → adrenal medulla releases catecholamines–epinephrine and norepinephrine (I have a project to finish today ASAP)
quick, short-term
HPA system (what they release + speed/duration of response)
activated → adrenal cortex secretes cortisol (Cortisol; Cortex)
slow, more long-term
What is the Social Readjustment Rating Scale (Holmes & Rahe, 1967)?
list of positive/negative events and how stressful they would be based on the scale (ex: marriage/divorce/death of a spouse were the highest stress/inducing ones)
What is the daily hassles approach to conceptualizing stress?
minor annoying events which require some degree of adjustment
What does research show regarding the relation between daily hassles and health (DeLongis et al., 1982)?
Daily hassles -> negative cumulative effects on health
perceived stress approach to conceptualizing stress and what research shows regarding the relation between perceived stress and health (Young et al., 2004)?
appraisals of life situations as unpredictable/overwhelming (how we interpret the things that happen to us)
Perceived stress scale (Cohen et. al)
Negative correlation between perceived stress and health outcomes
indirect effect model
stress leads to unhealthy behaviors in attempt to cope w/stress
Stress -> unhealthy behavior -> physiological changes -> disease
Some people smoke/drink more when stressed, causing poor health outcomes
direct effect model
direct link between stress & physiological changes leading to disease
Stress -> physiological reactions -> disease
diathesis-stress model
Diathesis = predisposition to disease
Stress = environment
Condition doesn’t develop w/o both diathesis + stress
Cannon’s fight-or-flight response, what physical changes are involved, how he made his initial discovery of ties between psychological processes and digestive processes
physiological stress response—preparation for mobilization; includes:
sweating
dilated pupils
increased heart & breathing rate
tensed + swollen muscles
used x-rays; when a cat in his experiment felt frightened/distressed, its stomach movements would stop
What is Selye’s general adaptation syndrome?
What are the 3 stages in the stress response?
In what 2 ways was he incorrect?
body’s generalized attempt to defend itself against stressors
3 stages:
Alarm (fight/flight)–immediate impact; body mobilizes to respond to stress (high physiological arousal)
Resistance (allostasis)–body keeps trying to respond to stressor–high physiological arousal (but not as high as in alarm)
Exhaustion –physical resources (including stress hormones) are depleted
He wasn’t entirely correct–
Our stress response varies depending on the nature of the stressor–it’s not general
our physical resources aren’t depleted
What is cardiovascular reactivity?
reaction to stress
What is the size of the relation between social support and health (relative to other known predictors of health such as smoking; Holt-Lunstad & Smith, 2012)?
social support has a greater than/equal effect on mortality rate as smoking
direct effects hypothesis
social ties provide protection during stressful & non-stressful times
buffering hypothesis
social ties provide protection against effects of stress
potential mechanisms in the relation between social support and health (e.g., cognitive appraisal)
Cognitive appraisal– having other people around helps us cope with our stress/perceive our situations
Health behaviors–social support improves our health by improving our healthful behaviors
People exercise together, “let’s try to prepare more healthful meals,” having other people support us through quitting smoking is very helpful OR people do drugs with their friends
Adherence– people we live with remind us to do things like go to appointments, take medicine
Psychoneuroimmunological pathways– how our psychological state impacts what’s going on in our nervous system -> immune system
Social support help to minimize our physiological response to stress
tend-and-befriend hypothesis
Does social support protect us physiologically in the face of stress?
Females’ responses to stress may be different than males
Successful stress responses should involve protection of offspring
Tending–taking care of offspring
Befriending–affiliating w/other people; during stressful times, some people, primarily females, will affiliate w/other people
Langer and Rodin’s (1976) and Rodin and Langer’s (1977) studies?
Langer & Rodin, 1976:
Control in a residential setting (nursing home)
Greater choices to nursing-home residents -> improvement in health 3 weeks later
Randomly selected 2 floors in a nursing home; If they gave the residents more control over their lives, would that affect their wellbeing?
Responsibility-induced (more control): you decide what to do with your furniture/make sure you water your own plants
Comparison condition: told the same things, but no control (giving them plants and nurses will water it)
Findings:
If they had increased responsibility/control/choice, they were happier/more active/sociable, compared to those in the control group who did not have control
Rodin & Langer, 1977
Follow up to the previous study (18 months later)
Those in responsibility-induced condition had significantly more happy/actively interested
Were half as likely to die if they had control
hardiness and its 3 components
Commitment (extent to which people are emotionally invested in what they do; ex: a student who is really interested in the content of what they’re learning/how it applies to situations versus someone who just memorizes the content for exams)
Control (people with high control think that what they do matters, as opposed to feeling helpless)
Challenge (the way we think about stressful/difficult things in our lives; ex: seeing traffic as 10 minutes of your life you’ll never get back)
explanatory style
how we habitually explain the causes & future outcomes/patterns regarding bad things that happen to us
health behavior model
a. Personality -> health-related behavior -> health
b. Childhood conscientiousness -> lowest smoking & alcohol use in adulthood (Friedman et al, 1995; Hampson et al, 2006)
someone is more conscientious, so they smoke & drink less, leading to better health outcomes
stress moderation model
Personality moderates the effect of stress on health
Ex: 2 people were in a bad traffic jam in the morning: a high hostile person is getting red in the face, heart rate and temperature is rising; a low hostile person is fine, heart rate is normal, sitting
big 5 personality traits/which is related to health behaviors/longevity
Extraversion (outgoing), agreeableness (how well you get along w/others), conscientiousness (how responsible you are, on-time), neuroticism (worry, concern, emotionally unstable), openness (creative, trying new things)
Conscientiousness
Lower mortality risk with higher conscientiousness (Friedman et al., 1993; Turiano et al., 2013)
Mediators?
Health behaviors (conscientiousness might predict lower smoking and drinking, producing longer life)
type A personality
characterized as competitive, hurried, hostile, and tense
Having a hostile personality drove the association between Type A and heart disease
Hostility associated w/increased risk of heart disease & death
internality vs externality
internality–making an internal attribution for bad outcomes; that bad thing happened because of me; self blame when we do not deserve that blame
externality–this job wasn’t made for me, this situation doesn’t match me well
stability vs instability
stability–does this type of thing always happen to me?; stability over time; “Oh this always happens to me time and time again/it will never get better,” “I’ll never get a job”
instability–it was just this one time; “This one time I didn’t get the job”
globality vs specificity
globality–our thoughts about this leaking into other areas of our life; we think, “my life is bad”) specificity–”this area of my life is not going well”)
problem-focused coping
addressing the source of our stress
Ex: Prof’s friend is not satisfied with her job, but she got a professional resume consultant, looking for new jobs
emotion-focused coping
not addressing the source of our stress, but our emotions about it
Ex: prof’s friend is doing emotion-focused coping; talking about it, seeking social support, having a good cry
approach coping
changing conditions/expressing emotions (taking emotions head on)
Expressive writing, positive
avoidance coping
ignoring/denying the problem
Abusing alcohol, sleeping, excessive video games
It can be okay for a short term stressor (ex: if you have a root canal appointment, it might be better to not think about it)
relation between religiosity and health (McCullough et al., 2000)
religiosity associated with better health, lower mortality risk (McCullough et al., 2000)
Moderator: public v. private religious involvement (public = going to services/activities; private = praying on your own); the public involvement had a greater effect
relation between expressive writing and subsequent health outcomes (Pennebaker & Frattaroli)
Write about trauma (experimental) or superficial topic (placebo control; Pennebaker & Beall, 1986)
Outcomes:
Reduced # of doctors visits
Enhanced immune functioning
Lower BP and heart rate
Better self-rated health
Mean effect size
d = .47 (r = .23) (Smyth, 1998) medium effect size (.2 = small, .5 = medium, >.8 = large)
d = .15 (r = .075) (Frattaroli, 2006) small/extra small effect size
Moderators where people benefited more from expressive writing: physical health problems, history of trauma, disclose at home, previously undisclosed
What is the relation between gratitude and health? What were the results of Emmons and McCullough’s (2003) study?
gratitude leads to better health
3 conditions: gratitude, hassles, events
Gratitude -> psychological and physical well-being
Thinking about what we’re grateful for helps us continue to savor the good things in our lives
For 10 weeks, once a week, the gratitude condition wrote about one thing they were grateful for
Hassles–told to think about things that were hassles
Events–neutrals; events that they experienced
mindfulness and health outcomes of mindfulness
focus on and awareness of current thoughts; accept & acknowledge the present
Health outcomes:
Stronger immune functioning
Lower blood pressure
Less pain
how humor is related to health
Humor -> health
Potential mechanisms:
Physiological changes in the systems of the body–laughter lowers cortisol levels, improves respiration
Inducing positive emotional states
Moderating adverse effects of stress on health–humor is a good way of coping w/stress
Increasing social support
self-affirmation
Positive affirmation of values–we are a person who enjoys ___ and has good friends, and is a kind friend, etc…
Lowers physiological response to stress (lower epinephrine)
Participants wrote about important personal values before exam -> lower physiological response to stress (Sherman et., 2009)
self-affirmation was most helpful to those w/high psychological vulnerability
temptation bundling
pairing immediately satisfying activities with those that require more effort but provide long term benefits (Milkman et al., 2014)
Ex: exercise; “I think I should exercise because it will lower my chances of developing heart disease; I should watch a show (and only when I’m exercising) while I do it”
friction
Make unwanted behavior less rewarding by adding friction–external barriers (e.g effort) that make performing the behavior more difficult (e.g Wood 2019)
Ex: I’m trying to reduce blood sugar: I can make it inconvenient to eat them; Instead of having the pack of cookies out, I could put them on a high shelf where it is harder to get them
Make desired behavior easier by removing friction
Ex: Why don't more of us eat fruits/veggies?
If I were to want a watermelon, I would have to wash, cut it, etc…
Solution: get a watermelon, cut it up, put it in a container, put it in your fridge for next time
health belief model and its 6 components
4 factors influence participation in preventive health behaviors:
Perceived susceptibility–beliefs about the degree of health threat
Should I get a COVID booster? Am I susceptible to getting it?
Perceived severity–beliefs about consequences of the illness
How bad would it be if I got COVID?
Perceived benefit of behavior change–beliefs that engaging in the behavior will reduce the threat of the illness
“The COVID booster is highly effective, it will protect me from contracting it”
Perceived barriers–beliefs about obstacles to engaging in a behavior
“What’s stopping me from getting a COVID booster? The pain? Side effects?”
Later, additional concepts were added to this model:
Cues to action–any type of reminder about a potential health problem/behavior
Ex: your doctor sends you a reminder to get your booster, your friend gets COVID, you have a dream about the last time you had COVID
Self-efficacy–a person’s belief that they will be able to execute a behavior
theory of planned behavior and its 4 components
Behavioral intentions: a person’s commitment to performing the behavior
Intentions are determined by:
Attitudes–person’s feelings about engaging in a particular behavior
I’m considering being physically active; what are my attitudes?
I think about how it may prevent heart disease, etc…
Subjective norms–people’s beliefs about whether important others would support them in changing their behavior and beliefs about what other people are doing
Will people in my life be supportive of me doing this? Will my family criticize me?
Are my peers physically active while I’m not?
Perceived behavioral control–the extent to which a person believes they will be able to engage in a behavior
Same as self-efficacy
implementation intentions
Implementation intentions–specific plans for when, where, and how one will engage w/ a behavior
Implementation intention example: I am going to replace eating frosted flakes for breakfast with oatmeal
You’re much more likely to follow through with implementation intentions than goal intentions
transtheoretical model and its stages
Behavior change is a complex process
Changes aren’t always linear → spiral change
Includes relapse
6 stages:
Precontemplation– no intentions to change problem behavior
Before they’re thinking about making a change (ex: someone who is smoking and not thinking about quitting; rationalizing why they’re not quitting)
Contemplation– beginning to consider changing behavior
Ex: smoker starts to realize they’re worried about health outcomes from smoking; information-seeking
Preparation– commitment to change behavior
Ex: smoker is still smoking, but makes a public commitment to quitting (public commitment makes you more likely to follow through); gathering plans for how to quit (setting a date, looking at different techniques)
Action– start engaging in a new behavior
First 6 months of the behavior change; when the behavior change is new
Maintenance– change is sustained over time
Termination– no longer much risk of relapse
Ex: someone who quit smoking is not tempted at all to go back
most important component across various models of health behavior
behavior intent
primary prevention
taking measures to prevent illness
Meant to prevent disease from occurring at all (sunscreen, vaccines, healthful diet, physical activity)
secondary prevention
detecting/treating illness at an early stage as a way of reducing the illness’ potential effects
Catch the disease at an early stage (screenings; mammograms, blood scans, giving yourself insulin to manage diabetes)
tertiary prevention
actions taken to minimize/slow the damage caused by an illness that has developed past an early stage
Disease has already developed; advanced illnesses
for what types of health behaviors (detection vs. prevention) are loss- and gain-framed messages most effective?
Screening/detection: risky–we’re scared something’s wrong with us (better paired w/loss-framed)
Any test; mammogram, blood test
Prevention: not risky–doesn’t make people scared
Health promotion; getting vaccinated, eating fruits/veggies (better paired w/gain-framed)
How effective are fear-based interventions (e.g., Leventhal et al., 1965) and what factors contribute to their effectiveness?
When they work:
when there is a moderate level of fear
a specific strategy is provided for change
ex: Leventhal 1965—tetanus shots
fear & strategy was most likely to lead to behavior change (told the tetanus shot was very effective & gave them a map of campus and appt times)
fear only (graphic images of people w/tetanus): hardly anyone got the shot
focus on short-term consequences (ex: not being able to get a date)
image of having a particular condition (“that could be me”)
What are behavioral nudges? What were the results of Dai et al.’s (2021) study on behavioral nudges and Covid-19 vaccinations?
Behavioral nudge–interventions that aim to promote positive behaviors w/o limiting choice (ex: putting fruit at the entrance of the dining halls)
Intervention to increase Covid vaccine behavior (Dai et al., 2021)
Sent invitations to schedule vaccine appts to patients
IV1; randomized to receive text message reminder to schedule appt and link to scheduling website (vs. no text message) associated with higher rates of getting the vaccine than no message
IV2: addressed motivation w/message creating feelings of psychological ownership (“the vaccine has just been made available for you”; “claim your dose”) vs. no message increasing ownership; ownership was more effective than non-ownership
IV3: video (challenging misconceptions about vaccine, increasing vaccine interventions) vs. no video
What is the nicotine regulation model? (and Strasser et al.’s study)
an extension of the fixed-effect model: nicotine level must be above body’s set-point for nicotine to provide pleasurable effects
Smokers smoked lower-nicotine cigarettes (Strasser et al., 2007)
# and size of puffs measured
Compensation for lower nicotine
People took more and bigger puffs of lower nicotine cigarettes to reach the set-point of nicotine
affect-regulation model and supporting studies (McEwen et al., 2008; Schachter et al., 1977)
People smoke to increase positive affect or decrease negative affect
Positive affect–smoke to enhance pleasure (smoking after a delicious meal, which enhances the pleasure)
Negative affect–smoke to cope w/anxiety, stress, tension
Schacter et al., 1977: Smokers exposed to high/low stress (strong/mild shock)
Smoking measured
High stress → more puffs
High stress–given electric shocks that would increase in intensity until participant couldn’t take it anymore
Low stress–mild shocks
McEwen et al., 2008—among a list of reasons, relief from stress was the #1 reason why smokers smoked
What are important aspects of social influence programs to prevent smoking (e.g., highlight peer attitudes)?
Targeted at high-risk group (adolescents)
Provide education about short-term negative effects of smoking
Highlight peer attitudes against smoking
Use role models
What are self-management strategies (e.g., stimulus control)?
Stimulus control–identify stimuli that make you want to smoke and eliminate/avoid them
Response substitution–when you have to urge to smoke, do something else instead
Stress management–people use smoking as a coping mechanism, so they need to come up with others
Contingency contracting–contract we make w/someone that is contingent upon us continuing not to smoke
Identify trusted member of social network and give them $$$
Goal: not smoke for the next 6 months; if I smoke even 1 cigarette in the next 6 months, the friend keeps the money
alcohol myopia & results of MacDonald’s et al. study
behavior influenced by most salient (but not most important/relevant) cues
When we’re intoxicated, we can only focus on cues that are immediately in front of us; being cognitively limited because of alcohol (social cues, informational cues)
Macdonald et al., 1995:
Does alcohol affect attitudes, intentions, and moral obligations toward risky behaviors?
Sober and intoxicated participants were assessed for attitudes, intentions, & moral obligations related to drinking & driving
Questions presented in contingent/noncontingent manner
Non contingent: “I would drive while intoxicated”
Contingent: “If I only had a short distance to drive, I would drive while intoxicated”
Intoxicated participants–higher intentions to drink & drive when asked contingent question than when asked non-contingent question
tension-reduction theory
theory that people drink to cope w/negative emotions
What is pluralistic ignorance? What was Prentice and Miller’s (1993) study?
assumption that our own attitudes & beliefs differ from others’ (despite our behavior being the same)
“You and I are behaving the same way, acting like everything is fine, but in private, we both think people are drinking too much”
I think that you think it’s more fine than you actually think, and that idea is reciprocal
Students believe that there is too much alcohol use on campus, but believe other students approve of the amount of alcohol use (Prentice & Miller, 1993)
We misperceive norms
aspects of intervention programs to prevent alcohol abuse
focuses on people at risk
Provide info about consequences (usually not as effective; college students know how harmful alcohol is)
Skills training–provide strategies for decreasing alcohol (ex: how to drink in moderation; they’re given alcohol, BAC measured, taught how many drinks it takes to get to a certain BAC)
Challenge expectations about alcohol use (giving real/placebo alcohol) –people think they need alcohol to be sociable/have fun, but that is only what they think (ex: participants are taught that they are the funny ones and alcohol is not the thing causing that)
Social influence–challenge perceptions about others alcohol use (give info about peers’ alcohol use) –we inflate what others are doing
antabuse
specifically helps people avoid alcohol; causes violent illness if you drank alcohol while on it
what is problematic about using BMI to predict obesity?
Substantial % of overweight & obese people healthy, substantial % of normal weight people unhealthy (Tomiyama et al., 2016)
Over 40k participants, were either normal/overweight/obese
Blood pressure, glucose level, insulin resistance measured
There was so much overlap, therefore you can’t reasonably use BMI as a proxy for health; there is a big range of health indicators
More accurate–% of body fat
Distribution of fat (“apples” vs “pears”)
Waist-to-hip ratio
Apple shaped (more abdominal fat is much bigger risk for things like heart disease
Pear shaped–more gluteal fat
set-point theory
Body seeks to maintain certain weight
Consume fewer calories → metabolism slows
Consume more calories → metabolism increases (“that’s too much, let’s burn that off”)
“Normal weight” participants consumed 1000 extra calories per day for 9 weeks (Levine et al., 1999)
Large range in weight gain
Some gained more than 9 pounds, some gained less than 1 pound
internal-external hypothesis
use of external cues rather than internal cues (hunger) for eating
Ex: food taste, smell
Variety
mood regulation theory and Rutters et al. (2009)
Mood regulation/emotional eating/stress eating–food is used to manage moods (stress, anxiety, depression)
Stress → eating in the absence of hunger (Rutters et al., 2009)
All full (they gave them all lunch)
Induced stress (induced by solving math problems that had no solution vs ones that had an easy solution)
Offered snacks to relax
Stress → higher consumption of food
restraint theory
people restrict the amount & type of food they eat in an attempt to lose weight
Do diets work? How do they affect stress levels?
Diets May Fail
Dieting increases stress (Tomiyama et al. 2010)
Participants assigned to restricting
Results:
Restricting → increased cortisol
Monitoring → increased stress
What are some ways of non-adhering?
Failure to keep scheduled appointments
Failure to take full dose
Taking other meds
Failure to take mds at correct intervals
Taking expired meds
Creative/intelligent/rational nonadherence
average rate of nonadherence
25%-50%
causes of nonadherence
Cost
Patient factors
Forgetting
Failure to understand
Not believing in efficacy of treatment
Anxiety, depression, self-efficacy
Environmental factors
Social support
Culture
Treatment factors
Long-term treatment
Complex treatment
Interference w/activities
Aversive side effects
Expected benefits and efficacy of treatment
Physician-patient communication
Clarity of instructions (“take with meals”)
how physician-patient communication is related to adherence (Haskard-Zolnierek and DiMatteo (2009))
Physician communication → better patient adherence
Physician training → better patient adherence
how can adherence be increased?
Association between physician communication & patient adherence–meta-analysis
Clear written materials, tape of visit
Reminders
Simplifying regimens
Physicians’ nonverbal behavior
Increasing positive affect
What did Guéguen et al. (2010) find with regard to physicians’ nonverbal behavior and patient adherence?
Intervention–physician gave instructions and touched or didn’t touch forearm
DVs–touching the patient on the forearm increased adherence significantly and increased more for males than females
# pills left in bottle
Higher perceived competence of doctor
Higher perceived concern (care) of doctor
supplier-induced demand
when health care providers encourage patients to seek more services or treatments than medically necessary
What are Szasz and Hollender’s (1956) 3 models of physician-patient interaction (e.g., activity-passivity)?
Activity-passivity– physician in control, patient passive (very doctor-centered)
Guidance--cooperation–patient has voice, physician makes decisions (patient can give preferences, but physician has final say
Mutual participation–equal partnership
How important is patient history (what the patient says) in making a diagnosis?
56-85% diagnoses can be made on the basis of what the patient says (can be more important than doing tests)