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Types of TBI injuries
Severe TBI: Open head or closed head injury
Non-traumatic: drug overdose, chronic substance use, carbon monoxide, toxins, anoxia (depletion of O2)
Immediate medical interventions after TBI
Reduce inc intracranial pressure (ICP) monitor for >40; Hypotension/hypoxia monitored
Surgical interventions (remove object; evacuate hematoma; bone flap to reduce pressure)
Decorticate posturing (decorticate rigidity) & location of trauma
UE in a spastic flexed position, internal rotation (IR) and adducted
LE spastic extended, IR and adducted
Location: injury in cerebral hemis, internal capsule, above the superior colliculus
Decerebrate posturing (decerebrate rigidity) & location of trauma
UE & LE in extension, adduction and IR, wrist and fingers in flexion
location: Lesion below the superior colliculus-brainstem region
Retrograde amnesia
cannot remember events prior to injury
Anterograde amnesia
cannot form memories or store/retrieve information after injury
Post-traumatic amnesia (PTA)
following injury where patient is confused and seems unable to store recall new info
length of PTA is predictor of prognosis
Secondary medical issues related to TBI
WB status may interfere w/participation of rehab
pulmonary restrictions
Decubiticus Ulcers (from prolonged pressure of skin)
SCI+TBI (30% of cases)
OT rehab focus in TBI cases
Splinting/casting UE; neuromuscular re-edu; cognitive training; ADL participation; transfers; w/c mobility; equipment training/needs; home eval; caregiver training
Glasgow Coma scale & scoring
designed to assess severity of coma and impaired consciousness
3-8: severe TBI
9-12: mod TBI
13-15: mild TBI
Glasgow scoring aspects
Eye-opening response: 1 (NR), 2 (R to pain), 3 (R to speech), 4 (spontaneous R)
Motor Response: 1 (NR), 2 (extensor response--Decerebrate posturing), 3 (abnormal flexion--Decorticate posturing), 4 (withdraws), 5 (localize), 6 (obey)
Verbal response: 1 (NR), 2 (random sound), 3 (inappropriate words), 4 (confused convo), 5 (oriented)
overall score (E+M+V)
lowest 3 - highest 15
Galveston orientation and amnesia test GOAT
measures cognitive level of patients post-injury-so realistic recovery plan can be communicated
low GOAT = longer PTA period
Longer PTA found in patients w/diffuse/bilateral brain injury
RLA I
Total A: No Response;
lack of awareness (orientation), no wakefulness period (in coma); coma rarely lasts 3-4wks unless induced
TBI Clinical signs and symptoms
Autonomic fxns: vital signs
Consciousness: level of arousal, cognition, length of coma
Motor fxns: reflexes etc.
Pupillary response: Depth of coma-pathological signs of coma
Ocular movement: If cranial nerves has been impacted--Abnormalities in pupil size, shape and light
RLA II
Total A: persistent vegetative state (PVS);
No awareness of self/env
No automatic motor response/localizing
Incontinence bowel and bladder
Positive signs: sleep/wake cycles; Brainstem, autonomic fxns: gag/swallow, random vocalizations / movements
RLA III
Total A: minimally conscious state
Some awareness w/wakefulness
Follows commands, gestures/Verbal response to Qs
Crying, laughing, smiling/relevant stimuli
Visual tracking
reaching/holds objects
W/C positioning goals for RLA I-III
Prevent deformity, tone normalization; pressure management, promote fxn; inc sitting tolerance; provide proper body mechanics; enhance respiratory fxns
RLA I-III other interventions
Dynamic head positioning device;
spasticity management (normalize tone)--neuromuscular blocks that can be used in addition to casting
When do you cast
When splinting has failed to control severe tone/contracture
ROM is dec and prolonged stretches necessary
consequences of lack of intervention
Contractures
Limited head/trunk control
Inc caregiver assistance
Limited participation in daily occupations
RLA IV
max A: confused/agitated
Alert and often heightened level of activity
Absent short-term memory
May exhibit aggressive bx/flight bx
RLA V
Max A:confused, inappropriate, non-agitated
Alert not agitated
Not oriented to person/place/ time
Unable to learn new material
Can respond to simple commands; can converse on social automatic level briefly; consistent with external cues
Verbalizations about present events often inappropriate and confused
RLA VI
mod A: confused, appropriate
Inconsistently oriented to person/place/time
Remote memory more accessible than recent memory
Able to use assistive memory devices max A
Show carryover of previously learned tasks (self-care)
Unaware of impairments, disabilities, safety risks
RLA VII
min A for ADLs; automatic & appropriate
Oriented to person/place/time
Inc attention & able to work for 30 min on familiar tasks
Min supervision for new learning
Initiates & carries out self-care/household tasks but may have limited memory of events
Unrealistic planning for future; overestimates abilities
Unable to think about consequences
Unaware of others needs/feelings
RLA VIII
stand by A (SBA)/supervision: purposeful & appropriate
Oriented to person/place/time
Attention has inc to 60 min for familiar task
Recalls past events and integrate recent events
Initiates and carries out steps for familiar person, household, community, work, leisure routines and can modify a plan when needed w/ min A
Requires no A once a new task/activity is learned
Needs assistance to make corrective measures when plans needs substantial alterations
Thinks about consequences; irritable/depressed
Acknowledges others needs/feelings
CN I & Tests
Olfactory; sensory fxn
Test ability to detect odor (does not need to ID it)
Test w/common orders (coffee/orange etc)
Do not use noxious odors
CN II & Tests
Optic; Sensory fxn
Visual acuity and visual fields
Asses w/eye chart--Test for visual cuts (peripheral vision)
Bitemporal hemianopsia
loss of temporal half of vision in each eye due to damage to optic chiasm
Homonymous Hemianopsia
visual defects involving either two right or two left halves of visual field of both eye (lose vision of same side on both eyes)
CN III
oculomotor; motor fxn
constricts pupil
CN IV
trochlear motor fxn
moves eyes
CN VI
abducens; motor fxn
moves eyes
CN III, IV, VI tests
tracking/pursuits, convergence, pupil reactivity
CN V & Tests
Trigeminal; Both
Motor: chewing
Sensory: light touch on face, cl must localize touch on both sides of face, compares sides for diff in sensation
CN VII & Tests
facial Nerve; Motor & Sensory fxn
Motor: facial muscles of expression--Ask cl to smile or other expression to inspect facial droop or asymmetry
Also checks for motor planning
Sensory: taste on tip of tongue, salivation
CN VIII
Vestibulocochlear; Sensory: both branches
Cochlear: ability to hear
Test: rub fingers by ear canal w/eyes closed
Test if they know which side the sound is coming from
Vestibular: detects info about position of head/body for balance, equilibrium
Status labyrinth: from utricle and saccule
Linear motion, position of head when static
Dynamic labyrinth: semicircular canals
Movement of head, initial speed of movement
Monocular vision loss
loss of vision in one eye
central scotoma
blind spot in the center of the visual field surrounded by an area of normal vision
CN IX
glossopharyngeal; Both
Sensory for taste in various parts of tongue
Motor for swallow to elevate the palate & propel the bolus chewed food
initiates swallowing and gag reflex
Nystagmus
involuntary, rapid, repetitive eye movements
Two phases:
Slower phase/movement-mediated by brainstem
Rapid phase w/re-fixation back to midline-mediated by cortex
CN X
vagus; Both
sensory fxns
Somatic: sensations felt on skin/muscles
Visceral: sensations felt in organs
Motor fxn: stimulates muscles in pharynx, larynx, soft palate (near roof of mouth), stimulates muscles in heart (helps lower Resting HR); stimulates involuntary contractions of digestive tract (i.e. esophagus, stomach, intestines)--allows food to move through tract
CN XI
Spinal Accessory; Motor Fxn
Allows you to turn head side to side & elevate shoulders
CN XII
Hypoglossal; Motor fxn
Allows tongue mobility (pushing tongue from side to side inside mouth to collect bolus chewed food
Hyperreflexia
Exaggerated reflex response
SC movement disorders
spasticity & four components
Hypertonia, Hyperreflexia, clonus (spastic involuntary contraction; induced by sustained stretching of spastic muscles), degrees of paralysis
Brain stem movement disorders
-Locked-in syndrome: Loss of voluntary motor control
-Decerebrate rigidity
-Associated reactions: inc in tone of parts not involved in particular movement (i.e. when grabbing object in R hand the L hand will do grasping motion)
Basal Ganglia movement disorders
Athetosis: involuntary, continuous, arrhythmic movement disorder
Parkinsons disease: resting tremors; affects timing & rhythm of gait and movements (difficulty stopping movement once they started)
Cerebellar level movement disorders
Ataxia: seen as clumsiness or uncertainty of movement (c/ walk with wide gait)
Dysmetria: difficulties w/ calibration
Dysdiadochokinesia: inability to perform rapidly alternating movement ( cant pronate-> supinate or tap fingers twice w/thumb)
intention tremors: tremors inc in rate/duration when picking up small objects
asthenia: excessive weakness/fatigue in voluntary muscles