Exam Review AbPsyc

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84 Terms

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1. Considerations for diagnosing children with disorders

developmental norms, environmental context, symptom duration/severity, and potential trauma history. Symptoms may look different in children than adults.

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Is childhood anxiety like adult anxiety?

It shares features, but in children it often manifests as specific fears (e.g., separation) rather than generalized worry.

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3. Separation Anxiety Disorder

→ An intense fear or anxiety about being apart from major attachment figures. Common in young children.

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Children at risk for anxiety disorders

Shy, behaviorally inhibited children or those with controlling parents are at higher risk.

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Childhood vs. adult depression/bipolar

children may show more irritability than sadness

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. Causal factors for childhood depression

→ Genetics, negative life events, parental depression, and cognitive distortions.

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Oppositional Defiant Disorder (ODD)

A pattern of angry/irritable mood, argumentative behavior, or vindictiveness

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Conduct Disorder (CD)

A repetitive and persistent pattern of behavior violating societal norms or others' rights.

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. Relation of ODD, CD, and Antisocial Personality Disorder

ODD may develop into CD, which can progress to Antisocial Personality Disorder in adulthood

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Neurodevelopmental disorders

Disorders with early onset that affect brain development and functioning (e.g., ADHD, ASD).

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ADHD (Attention-Deficit/Hyperactivity Disorder)

Characterized by inattention, hyperactivity, and impulsivity.

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Brain abnormality in ADHD

Delayed cortical development and abnormalities in the prefrontal cortex and dopamine systems

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Autism Spectrum Disorder (ASD)

Characterized by deficits in social communication and restricted, repetitive behaviors.

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Earliest diagnosis of ASD

Signs can be detected as early as 12–18 months.

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Four common deficits in ASD

Social reciprocity, nonverbal communication, relationships, and repetitive behaviors

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Tic

sudden, rapid movement or vocalization

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. Tourette’s

multiple motor and at least one vocal tic.

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Tourette’s: biological and psychological basis

Linked to genetic factors, dopamine dysfunction, and environmental stressors.

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Specific learning disorders

Difficulties in learning key academic skills, such as reading or math, not due to IQ or instruction quality

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Brain abnormality in dyslexia

Reduced activation in left hemisphere language areas, especially the temporoparietal region.

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Intellectual Disability Disorder (IDD)

Significant limitations in intellectual functioning and adaptive behavior starting before age 18

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Causes of IDD

Genetic conditions (e.g., Down syndrome), prenatal factors, birth complications, and environmental deprivation

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Levels of IDD severity

→ Mild: some support needed. Moderate: more support, basic skills. Severe: extensive supervision. Profound: lifelong care.

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: Delirium

Acute confusion, fluctuating awareness, disorganized thinking.

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Stupor

Unresponsive but not unconscious

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Alertness

Normal awareness and response to stimuli.

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Mild vs. Major Neurocognitive Disorders

Mild: Modest cognitive decline; independence mostly intact.

Major: Significant decline that interferes with independence.

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Parkinson’s Disease

A movement disorder caused by dopamine loss in the substantia nigra; may lead to cognitive decline or dementia over time.

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Alzheimer’s Disease — Early Damage

Initially damages the hippocampus, affecting memory formation.

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: Alzheimer’s — Late-Onset Associations

Linked to APOE-e4 gene and aging; more common in older adults.

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3 Major Brain Abnormalities in Alzheimer’s

Amyloid plaques, Neurofibrillary tangles, Neuronal loss

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vascular Dementia

Caused by strokes or restricted blood flow to the brain, leading to brain cell death.

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Amnestic Disorder

Severe memory loss (mainly short-term); other functions remain intact.

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Korsakoff’s Syndrome

Caused by thiamine deficiency (often from alcohol); memory loss and confabulation; partially reversible with treatment

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Traumatic Brain Injuries (TBIs

Brain injuries categorized as closed-head or penetrating; both can cause amnesia, personality changes, or cognitive issues

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Retrograde vs. Anterograde Amnesia

Retrograde: Loss of past memories.

Anterograde: Inability to form new memories.

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schizophrenia and its hallmark symptom?

A chronic psychotic disorder with a hallmark symptom of psychosis—loss of contact with reality, often with delusions or hallucinations.

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When does schizophrenia usually develop and in whom?

Typically late teens to early 30s; earlier in males than females.

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What are delusions and common types?

Fixed false beliefs (not based in reality). Common types: persecutory, grandiose, referential, somatic.

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What are hallucinations? Most common type?

Sensory perceptions without external stimulus. Most common: auditory—hearing voices.

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Disorganized speech examples

Tangentiality, derailment, incoherence (“word salad”).

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Positive vs. Negative Symptoms

Positive: Added experiences (hallucinations, delusions).

Negative: Losses (flat affect, alogia, avolition, anhedonia)

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Schizophreniform vs. Schizoaffective vs. Delusional vs. Brief Psychotic Disorder

Schizophreniform: Like schizophrenia but 1–6 months.

Schizoaffective: Schizophrenia + mood disorder.

Delusional: Non-bizarre delusions only.

Brief psychotic: <1 month psychotic symptoms.

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Visual and auditory deficits in schizophrenia

Trouble filtering sounds, tracking movement, or distinguishing relevant stimuli

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Cognitive and social deficits

Poor working memory, attention, social skills, and interpreting facial expressions or social cues.

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: Other brain abnormalities in schizophrenia

Reduced gray matter, abnormal prefrontal cortex activity, less brain connectivity.

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Main neurotransmitter in schizophrenia

Dopamine

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Expressed Emotion (EE) and relapse

High EE (criticism, hostility, overinvolvement) in families increases relapse risk.

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Medications for schizophrenia

Antipsychotics (typical and atypical); target dopamine to reduce symptoms.

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Estrogen’s role in schizophrenia

May have a protective effect, possibly explaining later onset in females.

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Four phases of human sexual arousal

Desire

Excitement (arousal)

Orgasm

Resolution

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Male Hypoactive Sexual Desire Disorder

Low or absent sexual thoughts/fantasies and desire for sex, causing distress; may be linked to stress, depression, or hormone levels.

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Erectile Disorder

Persistent inability to get or maintain an erection; often caused by anxiety, stress, cardiovascular issues, or substance use.

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Premature Ejaculation

Ejaculation within one minute of penetration, earlier than desired; common and often linked to anxiety or low control.

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Delayed Ejaculation Disorder

Marked delay or absence of ejaculation despite adequate stimulation; may stem from psychological or medical issues.

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Female Sexual Interest/Arousal Disorder

Lack of sexual interest, thoughts, or response; causes include stress, relationship issues, hormonal changes, or trauma

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Issue with Stage Model for Women

Women may not experience arousal linearly; emotional connection and context often matter more than physical stages.

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Genito-Pelvic Pain/Penetration Disorder

Persistent pain or fear during intercourse, or tensing of pelvic floor muscles; may be caused by trauma, anxiety, or medical issues.

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Female Orgasmic Disorder

Delay or absence of orgasm; causes include guilt, anxiety, lack of knowledge, or relationship issues

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Gender Dysphoria

Distress from a mismatch between assigned gender and experienced gender. Not all children persist into adulthood

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Paraphilic Disorders vs. Paraphilias

Paraphilias: Unusual sexual interests.

Paraphilic Disorders: Cause harm or distress to self/others.

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Voyeuristic Disorder

Intense arousal from watching unsuspecting people undress or have sex; can be criminal if acted on.

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Exhibitionistic Disorder

Recurrent urge to expose genitals to unsuspecting strangers; related to thrill-seeking and sometimes linked to sexual offenses

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Sexual Sadism Disorder

Arousal from inflicting pain or humiliation on others; a disorder if non-consensual or causes distress. Linked to aggressive or antisocial traits

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Sexual Masochism Disorder

Arousal from being humiliated or hurt; only a disorder if it causes significant distress or impairment. May not involve actual sadists.

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Fetishism

Sexual arousal from non-living objects or body parts. May develop from early associations or conditioning.

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Transvestic Disorder

Arousal from cross-dressing; may involve autogynephilia (arousal at the thought of being a woman), but not always.

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Pedophilic Disorder

Attraction to prepubescent children; causes may include brain abnormalities, early trauma, or low arousal to adults.

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Incest

Sexual relations between close relatives. Most avoid it due to instinctive aversion and social taboos (Westermarck effect).

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Substance-related disorders

A group of conditions involving the misuse of alcohol, drugs, or other substances that lead to significant impairment or distress, including health problems, disability, and failure to meet responsibilities.

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Substance abuse vs. dependence vs. tolerance vs. withdrawal

Abuse: Repeated use despite negative consequences.

Dependence: Physiological and/or psychological reliance on a substance.

Tolerance: Needing more of a substance to achieve the same effect.

Withdrawal: Physical/mental symptoms after reducing or stopping use

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Alcohol Use Disorder (AUD)

A chronic relapsing brain disease involving a strong craving for alcohol, continued use despite problems, and inability to control use.

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Mesocorticolimbic Pathway (MCLP)

The brain’s “reward pathway” involving dopamine release; it reinforces behaviors like drug use by associating them with pleasure.

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Is AUD genetic?

Yes—there is a genetic predisposition. People with a family history of AUD are at higher risk.

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Why is opium addictive?

It mimics endorphins and produces intense euphoria, quickly leading to tolerance and dependence.

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Endorphins

Natural neurotransmitters that reduce pain and enhance pleasure; opiates hijack this system.

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Dopamine Theory of Addiction

Suggests substances cause surges in dopamine, reinforcing use because it feels rewarding.

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Reward Deficiency Syndrome

Some people have underactive reward systems and use substances to “feel normal” or experience pleasure.

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Depressants vs. Stimulants

Depressants (alcohol, barbiturates): Slow brain activity.

Stimulants (cocaine, meth): Increase alertness and energy by speeding up the CNS.

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Cocaine, Methamphetamine, Caffeine, Nicotine Effects

Cocaine: Euphoria, confidence, energy.

Meth: Long highs, aggression, paranoia; damages brain over time.

Caffeine: Alertness, jitters.

Nicotine: Calming + stimulating effects; highly addictive.

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Effects of LSD, Ecstasy (MDMA), Marijuana

LSD: Hallucinations, distorted time.

Ecstasy: Empathy, warmth, sensory enhancement.

Marijuana: Euphoria, relaxation, altered perception.

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Side effects of LSD, Ecstasy, Marijuana

LSD: Anxiety, psychosis, flashbacks.

Ecstasy: Depression, dehydration, memory issues.

Marijuana: Impaired attention/memory, anxiety.

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Pathological Gambling

A behavioral addiction involving persistent, recurring gambling despite negative life consequences.

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How does pathological gambling develop?

Likely through a mix of impulsivity, reward-seeking behavior, and poor decision-making related to brain reward systems.