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3 period of conception to birth
period of the zygote
period of the embryo
period of the fetus
period of the zygote
begins when egg is fertilized in the fallopian tube
period of rapud cell division
ends 2 weeks later when the zygote is implanted in the wall of the uterus
period of the embryo
from 3-8 weeks after conception
body parts are formed during this period (including the ear)
embryo rest in the amnion flled with amniotic fluid
umbilical cord join embryp to placenta
period of the fetus
from 9 weeks after conception to birth
increase in size and system begin to function
age of viability: 22 - 28 weeks
influences on prenatal dvlpmt
teratogens
general risk factors
teratogens
a drug or other substance capable of interfering with the dvlpmt of a fetus, causing birth defect
may be illicit drugs or things that aren’t ok when pregnant
ex: cocaine, alcohol, marijuana, caffeine, environmental hazards (asbestos, toxins in air/water)
teratogens & diseases pass thru placenta & attack fetus (TORCH infections)
how teratogens influence prenatal dvlpmt
not universally harmful or mutual exlusive - may affect the mother but not the unborn child (depends on timing & regularity of exposure)
harm particular structures at specific points in dvlpmt
general risk factors
nutrition: adequate amount of food, protein, vitamins, & minerals
stress: decreases oxygen to fetus & weakens mother’s immune system
mother’s age: neither too young (12-13 yr/o) nor too old (35+)
increased maternal age is a high risk for down’s sydrome
effects of teratogens in weeks 1-2
errors divind zygote and implantation = prenatal death/miscarriage
effects of teratogens in week 3
major congenital anomilies in the C.N.S & heart
effect of teratogegns in week 4-5
major congenital anomalies in the eye & heart
effect of teratogens in week 6
major congential anomalies in the ear, teeth, upper & lower limbs
effect of teratogens in week 7-8
major congenital anomalies in the palate, ear, and external genitalia
effects of teratogen in week 9
functional defects & minor congenital anomalies in the brain, eyes, palate, & teeth
effects of teratogens in week 16
functional defects & minor congenital anomalies in the brain, eyes, teeth, external genitalia
effects of teratogens in week 20-38
functional defects & minor congenital anomalies in the central nervous system
prenatal diagnosis & treatment
diagnosis: ultrasound, amnicentresis (blood test for clincal markers - looks for things like Down’s), chorionic villus sampling can detect physical deformities & genetic disorders
treatement: fetal medicine & genetic engineering are experimental
influences on dvlpmt during child birth
STD/I - active herpes breakout while passing thru the birth canal may cause hearing loss &/or blindness
Get stuck in birth canal
Anoxia/Asphyxia - lack of oxygen to the brain
ex: (miecal cord) - umbilical cord wraps around neck
what does APGAR stand for?
A - activity
P - pulse
G - reflex/grimace
A - appearance/color
R - respiration
APGAR scoring
a score is given at 1 min, 5 min, & 10 min after birth to assess newborn’s health
10 minutes is usually only for babies with problems after birth
*there is no correlation btwn APGAR score and IQ or intelligence success outcomes
APGAR scoring scale
normal: 7-10
some resuscitative measures required: 4-7
immediate resuscitation: 2-3
extremely immediate resuscitation: 0-1
may have failure to thrive
assessing the newborn
APGAR
NBAS - comprehensive assessment of infant
newborn hearing screening
the lower the APGAR score, the longer the delay in screening
4 primary stages of newborn
alert inactivity
waking activity
crying
sleeping - half of the newborns sleep is REM, baby sleeping on back may prevent SIDS
birth complications
anoxia - lack of oxygen: lads to surgical removal of the fetus (C-section)
premature & low birth weight/small for date
prematurity is less serious than low birth rate
low birth weight babies
babies born who weight less than 3 lbs 4 ounces (1500 grams)
only 1.5% of babies are low birth weight
causes of low birth weight
premature birth (less than 37 weeks
intrauterine growth restrictions
problems with the placenta
poor maternal health
birth defects
more African-American babies are born low birth weight
Age - teen mothers, mothers above 35+
multiple births
poor mother’s health
twins/triplets
symptoms of very low birthweight babies
low oxygen lvls
inability to maintian body temp
difficulty feeding & gaining weight
infection
breathing problems (respiratory distress syndrome) - immature lungs
nuerological problems (bleeding in the brain)
gastrointestinal problems
sudden infant death sydrome (SDIS)
long-term problems
cerebral palsy
blindness
hearing loss
intellectual disability
cause of hearing loss (from prenatal-postnatal)
prenatal: conceptin to fetal stage (22-28 weeks or 7 months of prengancy)
teratogens: alcohol, illicit durgs, caffiene
poor nutrition
perinatal: 22 weeks of pregnancy to 7 days after birth
complications while in birth canal: active herpes outbreak, anoxia
postnatal: after 7 days of brith
illness/infection, medication in NICU (IV for myosin, vancomyosin)
high risk factors for infant hearing loss
A) - Anoxia/Aphixia: no O2 to the brain, umbilical cord wrapped around neck
B) - Bacterial Meningitis: inflammation of the sac around spinal cord
C) - Congenital Perinatal Infections: CMV, syphilis, STD (herpes),
D) - Deformity of head/neck: skin tags or pits at/around the ear, craniofacial diff
E) - Elevated bilirubin: jaundice (high risk of auditory neuropathy)
F) - Family History of Hearing Loss: mom/dad, brother/sister, grandma/grandpa, first cousin, niece/nephew
G) - Gram birth weight: less than 3 lbs 4 ounces (1500 grams)
H) - Had a Transfusion: babies in NICU longer than 5 days (preemie, fetal alcohol syndrome, failure to thrive, etc.) or vancomycin drugs through IV