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Basic anatomical structures of larynx (epiglottis, hyoid bone, vocal folds when abducted/adducted, cartilages, and muscles, arytenoid etc.)
Study Photo
Abducted
VF open
Adducted
VF closed
How sound is created: the power source, vibrating source, and resonator
Power source: the lungs/ air
Vibrating source: VF are brought together and airflow is applied to them and they vibrate
Resonator: vibrations from the VF move throughout the vocal tract which changes shape different aspects of sound
Vocal tract
pharynx, oral cavity, nasal cavity
Quality
hoarseness
breathiness
strain/ harshness
glottal fry
aphonia
hoarseness
irregular VF vibration, noisy, rough, rapsy, husky
breathiness
air escaping through the vocal folds
strain/ harshness
tension that can be heard when phonation is produced with forcefully adducted vocal folds
glottal fry
cracking or frying sound when a person is phonating near the bottom of their pitch range/ insufficient breath support
aphonia
no voice
Pitch
abnormally high/low
falsetto
diplophonia
montone
falsetto
un-naturally or artificially high-pitched voice or register, especially for males
diplophonia
audible production of two distinct pitches simultaneously
Resonance
hypernasality
hyponasality
cul-de-sac resonance
hypernasality
excessive nasal resonance; too much air is going through the nasal cavity
hyponasality
lack of nasal resonance when nasal consonants are produced
cul-de-sac resonance
resonance is described as hollow; tongue is in a ball in back of mouth
What causes resonance abnormalities?
dysfunction of velopharyngeal junction
Vocal Nodules
organic
benign, callous like
often bilateral
soft & pliabile early on, over time before hard & interfere with VF vibration
occur at the location on the folds where there is the highest impact during phonation
low pitch, raspy, breathy, loss of vocal range
often occurs with heavy voice use & frequent misuse
Polyps
organic
fluid filled lesion
result of a blood vessel rupturing then swelling
typically unilateral
has its own blood supply
cause is thought to be phonotraumatic behaviors
low pitch, rough, breathy, effortful phonation, globus sensation
Sessile Polyps
Raised but flatter bump
Pedunculated Polyps
Stem like, bump hanging off the VF
Reinke’s Edema
organic
buildup of fluid in the superficial layer; most severe form the membranous portion becomes permeated with fluid
result of long-standing trauma or chronic exposure to irritants (smoking)
swelling can become large enough to cause shortness of breath
lowered pitch and varying degrees of hoarseness
Laryngeal Web
organic
congenital
normally VF separate by 10th week of gestation
lack of separation results in webbing: a thin sheet of connective tissue that attaches the two VF
Recurrent Respiratory Papilloma (RRP)
organic
one of the most common benign neoplasm of the larynx
benign wart-like growths often beginning at the anterior portion of the VF and then spreading to the other structures of the larynx
thought to be caused by HPV
hoarse, stridor, aphonia, possibly respiratory distress
Neoplasm
abnormal growth of cells
Laryngectomy
organic
can be total or partial removal of the larynx
trachea and esophagus are seperated
person breathes through stoma in their neck
loss of VF = loss of voice
tracheoesophageal voice prosthesis and/or electrolarynx can allow for oral communication despite absence of larynx
Vocal Fold Paralysis
organic
recurrent branch of the 10th cranial nerve supply for most of the laryngeal muscles
damage anywhere along the vagus nerve can cause VF paralysis or paresis
can be unilateral or bilateral
paralysis can be in the abducted or adducted position
Organic Voice Disorders Structural vs. Neurological
Structural: the physical structure is impaired
Neurological: something in the neurological path is impaired
Muscle Tension Dysphonia
functional
also called hyperfunctional dysphonia
condition of extra work and strain of the laryngeal muscles
can occur as a compensatory mechanism for another underlying problem or can be a learned habit
voice is pressed, strained, may cut out, loss of pitch range, loss of endurance
Aphonia
functional
psychogenic voice disorders can result from psychological suppression of emotion
called conversion disorders because the person is converting emotional conflicts into physical symptoms
VF are structurally normal and they function normally for nonspeech behaviors
person may whisper to produce voice
does not stop until person resolves emotional conflict
may require some sort of therapy
Who is all apart of the voice assessment team?
otolaryngologist/ ENT
SLP
allergist
gastroenterologist
radiologist
Otolaryngologist/ ENT on Voice Assessment Team
Examines ears, nose, sinuses, throat, and vocal mechanism. Provides a diagnosis and prescribes any medications needed to treat the diagnosis, and performs surgery.
SLP on Voice Assessment Team
Examines the functionality of the VF and vocal tract. Provides insight to how the larynx is functioning to produce voice. Designs a treatment plan to include direct therapy and/or behavioral modifications.
Allergist on Voice Assessment Team
Evaluate and treat allergies, asthmas, and immunologic disorders.
Gastroenterologist on Voice Assessment Team
Evaluate and treat digestive orders which may be impacting the voice. (ex. GERD)
Radiologist on Voice Assessment Team
Diagnose abnormalities via CT, MRI, and PET imaging.
Acoustic Assessment
allows SLP to capture data on irregularities in the vocal signal & compare to norms
can measure stability of the VF vibration and amount of turbulence or noise in the acoustic signal
Relative Average Perturbation (RAP)
can get from acoustic assessment
also known as jitter
represents the average frequency (pitch fluctuations) from one vibratory cycle to the other
Shimmer
can get from acoustic assessment
represents the average amplitude (loudness) fluctuations from one vibratory cycle to the other
Voice Turbulence Index (VTI)
can get from acoustic assessment
measure of inharmonic energy (noise) which correlates incomplete glottis closure
Noise to Harmonic Ratio (NHR)
can get from acoustic assessment
represents the ratio of spectral noise to harmonic energy
relative level of noise increases when VF vibration becomes irregular or VF fail to completely close
Aerodynamic Assessment
measures the airflow and pressure related to speech and phonation
allows us to understand how the respiratory system is interacting
Visual Assessment
videostroboscopy
VF vibrate 80-300 times per SECOND in average adults; this is seen as a blur on traditional endoscopy
flashing light (called stroboscopy) along with laryngeal microphone allows synchronization to make the vibration appear to be happening in slow motion
this allows SLPs to view functional abnormalities
Transoral Videostroboscopy
Rigid
Transnasal Videostroboscopy
flexible
Perceptual Assessment
how does the voice sound?
subjective measure, but the CAPE-V allows us to make it slightly more standardized
Self-Reported Outcome Tools Assessment
measures patients perception of the problem
repeatable for pre- and post- treatment measures
Indirect Intervention for Vocal Abuse & Misuse
Education: help the patient understand what they may be doing to have caused the damage
Vocal Health Strategies
don’t strain the voice
hydrate
don’t smoke
be cautious of caffeine and alcohol intake
avoid excess coughing and clearing throat
prioritize rest
What is physiologic voice therapy?
strives to balance the three subsystems of voice production
does not directly work on isolated voice symptoms
most approaches can be used with a varie
Physiologic Voice Therapy: Vocal Function Exercises
Engages the larynx without any tension, improves physiological frequency range.
Physiologic Voice Therapy: Resonant Voice Therapy
Actively directing airflow up and forward to the nose and face in order to maximize resonance. Allowing more “bang for your buck” and minimizing strain.
Physiologic Voice Therapy: Flow Phonation
Facilitating increased airflow, ease of phonation, and forward focused resonance. Achieving phonation with minimal TVF pressing.
Physiologic Voice Therapy: PhoRTE
Strengthens the respiratory drive and VF closure to combat muscle loss due to aging.
Physiologic Voice Therapy: LSVT or SPEAK OUT!
Intensive exercise treatments developed for patients with Parkinson’s Disease.
Symptomatic Voice Therapy
focuses on modification of vocal symptoms or perceptual voice components
addresses voice improvement through direct symptom modification
Symptomatic Voice Therapy: Semi-Occluded Vocal Tract (SOVT) Exercises
Phonating through a narrowing at any point along the vocal tract in order to maximize interaction between vocal fold vibration and the vocal tract.
Symptomatic Voice Therapy: Confidential Voice
Reduce laryngeal tension and hyperfunction and increase airflow.
Symptomatic Voice Therapy: Auditory Masking
Utilizes the lombard effect to have client communicate with a louder volume.
Alaryngeal Speech
A form of communication used by those who get a laryngectomy. It is communication produced without the larynx.
Electrolarynx
A device that can be used to provide an external sound source to create voice. The head of the device is place firmly on the side of the neck, under the chin, or on the cheek. The patient articulates normally. Devices permit for variations in volume and pitch through switch controls.
Tracheoesophageal Voice Prosthesis
Provides an internal sound source for alaryngeal speech. One-way valve crafted from medically high-grade silicone. When inserted, prevents aspiration of food/ liquid and permits pulmonary air to enter the esophagus when the stoma is occluded while the patient exhales. Needs changed every 6 wks-6 mo.
Esophageal Speech
This utilizes the paitients remaining anatomical structures to provide an internal source for sound generation. The patients are trained to place air into the esophagus and then expel the air while vibrating surround tissues. Fluent esophageal speech requires rapid intake and release of air from the esophagus.
What is dementia?
acquired syndrome consisting of a decline in memory and other related functions
umbrella term to group all diseases involving memory loss
symptoms emerge slowly and worsen over time
What causes dementia?
Cortical dementias: Alzheimer’s Disease & Frontotemporal Dementia
Subcortical Dementias: Parkinson’s & Huntington’s Disease
Mixed Dementias: Vascular Dementia & Lewy Body Dementia
What are some dementia symptoms?
alterations in behavior or personality
co-occurring movement disorders and sensory disturbances
memory loss
difficulty communicating
inability to learn or remember new information
difficulty with planning and organizing
difficulty with coordination and motor functions
personality changes
inability to reason
inappropriate behavior
paranoia
agitation
hallucinations
How can Alzheimers Dementia be managed?
no medications can cure
there are recent FDA approved meds to reduce amyloid deposits
medications to reduce or control cognitive and behavioral symptoms
monitoring diet and fluid intake to prevent dehydration & malnutrition
tranquilizers & antidepressants prescribed to control combativeness or aggression to lessen depression
What are characteristics of memory deficits in Alzheimer’s Dementia?
impaired episodic (recent events)
impaired working (verbal & visuospatial span tasks)
semantic memory only in severe pt’s
procedural memory typically good
long-term memory remains good in early stages
phonology & syntax are relatively preserved in early stages
Parkinson’s Disease
subcortical dementia
in the later stages, some paitients become demented
they demonstrate impaired orientation, language, and loss of ability to function in ADL’s
How is cognition and communication impacted in the early stages of Alzheimer’s Dementia?
difficulty remembering recent events
phonology, syntax, articulation and voice quality are well preserved
mild word-retrieval problems and subtle comprehension difficulties may appear
client can usually recognize difficulties and repair
functional reading comprehension
adequate conversationalists: observe conversational conventions such as turn-taking and eye contact
How is cognition and communication impacted in the middle stages of Alzheimer’s Dementia?
communication impairments become more obvious
more frequent word-retrieval failures and individuals success in repairing them declines
sentence fragments and ungrammatical sentences begin to appear
more passive conversationalists: allow others to pick topic, tone & content
comprehension of nonliteral material is very impaired
How is cognition and communication impacted in the late stages of Alzheimer’s Dementia?
can’t read & write
comprehension of spoken materials is limited to simple & familiar conversation
speech is primarily single words & sentence fragments which are devoid of meaning & robot like
syntax breaks down and stereotypic utterances
neologisms
unaware of errors
some pt become mute or echolalic
What are the goals of dementia treatment?
Lessening advancing effects of the disease on the life of the person with dementia and their caregivers/ family members. Minimizing disruptive effects, ensuring safety, and providing support and direction.
What is the SLPs role in dementia treatment?
assess cognitive communication skills to determine strengths and weaknesses
maintain and facilitate strengths in their cognition and communication
educate and help family and caretakers to facilitate communication, be aware of safety issues, and maximize QOL
Candidacy for Dementia Treatment
those who:
show intent to communicate
demonstrate cognitive-linguistic strengths around which to structure a treatment program
respond to cues and stimulation
follow simple directions
exhibit recent/ significant change in status
Cognitive Stimulation for Dementia PTs
picture & object describing
story recall
category naming
problem solving
word association
Environmental Modifications for Dementia PTs
schedule of routine activities
items used in an activity are kept together
checklist for complex procedures posted where the procedure is to be accomplished
Memory Aids for Dementia PTs
electronic organizers with built in alarms
pocket-sized checklists for to-dos
written cues: notes, labels, lists, signs
organizational cues: planners, medication boxes
Rationale and how to use memory books/wallets/ and visual reminders
PT can captilize on spared recognition/ long-term memory
may contain personal info such as family members
may contain ADL’s
reminder cards to remember appts & who they are
Rationale and goals of Montessori based treatment for dementia
uses everyday stimuli and activity to facilitate action, memory and communication
capitalizes on preserved procedural memory abilities of PWD
reduces demands on episodic & working memory
capitalizes on life experience
Global Deterioration Scale (GDS)
no cognitive decline
age associated memory impairment
mild cognitive impairment
mild dementia: decreased knowledge of current and recent events, forgetting some of personal history, difficulty managing personal finances
moderate dementia: unable to recall familiar address or names of family members, not oriented to time or place
moderately severe dementia: forgetting names of very close family members, unaware of year or season, occasionally incontinent, delusional, sleep disturbance
loss of ability to speak, walk, and feed self, inconitnent, brain no longer c
Mini Mental State Examination (MMSE)
Quick, 30 min test assessing cognitive impairment. Assesses domains such as orientation, memory, attention, language and visuospatial skills.
What can cause dysphagia & who can it occur to?
all ages
various conditions such as CVA, TBI, cancer, tumor, PD, ALS, AD
symptoms may be acute or worsen over time
can be sensory, motor, cognitive or a combination
Anatomy and functions of the upper aerodigestive tract
PICTURE
Aerodigestive Tract
oral cavity
nasal cavity
pharynx
larynx
cervical esophagus
Functions of the Aerodigestive Tract
respiration: airway open
phonation: airway open, VF approximated
swallowing: airway closed
Valves in Upper Aerodigestive Tract
Lips: closed to keep food in mouth during chewing and swallowing
Tongue: makes contact with the roof of the mouth or it can maintain a varying degree of space between itself and the hard palate to manipulate and propel the foods/ liquids
Velum: closes during swallow to prevent food from entering nose
Larynx: closes during swallow to prevent the entry of food and liquid into the airway
Upper Esophageal Sphincter: normally closed except when it opens to allow the food/ liquid to enter the esophagus
Phases of swallow: what happens at each phase and what can go wrong at each phase
oral preparation: forming of bolus
oral transport: pushing bolus back
pharyngeal phase: bolus in pharynx
esophageal phase: bolus in esophagus
Aspiration
food into airway below vocal folds
Penetration
food into airway entrance but not below true vocal folds
Regurgitation/ Backflow
food comes back from esophagus into pharynx or from pharynx into nasal cavity
Residue
food remains in mouth or pharynx after swallow
Clinical Symptoms of Dysphagia
difficulty manipulating food
pocketing of food on one side of the mouth
food or liquid coming out of the nose
choking on food and medications
coughing during or right after eating and drinking
extra effort needed to chew and swallow
weight loss
Consequences of Dysphagia
pneumonia
malnutrition
dehydration
death
social isolation
reduced QOL
Bedside clinical swallow assessment: purposes, what it looks like, strengths and limitations
Purpose:
What it looks like: medical history, pt is given liquid and solid trials of different consistencies
Strengths:
Limitations:
Video Fluoroscopic Swallow Study (VFSS)
modified barium study
real time x-ray
movement of bolus can be imaged in real time
later or anterior-posterior plane
various foods given to pt during swallow study
goal is to define PTs swallow disorder
Fiberoptic Endoscopic Evaluation of Swallowing (FEES)
flexible fiberoptic tube goes through the nose
difficult to observe oral stages of swallow
other specialized techniques also used, but not as frequently and not for complete evaluation of swallow
Principles of Dysphagia Management
Eliminate the symptoms of the swallowing disorder without necessarily changing swallowing physiology.