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ADHD
distinguished by persistent patterns of inattention, hyperactivity and or impulsivity
most are diagnosed in childhood; 50% of these children continue with symptoms in adulthood
affects 1-5% of school aged children
3-5x more likely in males
Cause of ADHD
Not typically known
possibly genetic and environmental
contributing factors: low birth weight, maternal smoking, maternal alcohol consumption, child abuse or neglect, exposure to lead
Neuroscience of ADHD
Reduced amount of norepinephrine (neurotransmitter)
• Regions of the brain involved in ADHD
• Frontal cortex
• Limited norepinephrine causes decreased attention, organization and executive function
• Limbic system
• Deficiency causes restlessness, inattention and emotional volatility
• Basal ganglia
• deficiency causes inattention and impulsivity
• Reticular activating system
• Deficiency causes inattention, impulsivity and hyperactivity
reduced volume of prefrontal cortex, caudate and putnam of basal ganglia and cerebellum
less blood flow to frontal lobe, temporal lobe, parietal lobe, basal ganglia, and cerebellum
Identification of ADHD
Symptoms must be identified
must be presented before age 12
consistent cross least 2 settings
must occur for at least 6 months
must impact academic, vocational and/or social functioning
neuroimaging may show “soft” signs
IDs of ADHD
Family/medical history
Continuous performance tests
Parent Assessment
Teacher/School assessment
Symptoms of ADHD
Decreased executive functioning, organization and time management
Inattention
Hyperactivity/impulsivity - Six or more symptoms of hyperactivity-impulsivity for children up to age 16, or five or more for adolescents 17 and older and adults; symptoms of hyperactivity-impulsivity have been present for at least 6 months to an extent that is disruptive and inappropriate for the person’s developmental level:
• Fidgets
• Leaves seat
• Runs or climbs inappropriately
• unable to play quietly with sedentary activities
• Often on the go
• Talks excessively
• Blurts out answers
• Difficulty waiting their turn
• Often interrupts
Three types
combined presentation
predominantly inattentive presentation
predominantly hyperactive/impulsive presentation
Co Morbidities
Mood Disorders
Anxiety Disorders
Substance Abuse
Conduct Disorder
Oppositional Defiant Disorder
LD
Autism
Cognitive Characteristics of Individuals with ADHD
Self regulation difficulty
memory
self directed speech
control of emotions and motivation
reconstitution or planning
Social and Emotional Characteristics
Self esteem
social functions
Occupations and Activities affected by ADHD
Education
Social Participation
Work
Medical Management
• CNS Stimulants most effective – Ritalin and Concerta (helps 70-80%) – increases the availability of dopamine and norepinephrine at synapses
• May have side effects
• Loss of appetite
• Difficulty sleeping
• Headaches
• Irritability
• Selective norepinephrine reuptake inhibitors such as Strattera – non-stimulant
• Not usually as effective as stimulants
• School personal must not discuss medication as recommendation!!
OT Management of ADHD
• Environmental accommodations
• Decrease visual and auditory distractions
• Allow for breaks
• Allow for fidgets or movement cushions
• Music
• Scents
Behavior Management
Clear expectations
• Concise instructions
• Break down activities and expectations
• Structured routines
• Positive reinforcement
• Parent education and training
• Individual therapy
• Family therapy
• Low-Involvement Strategies
• Eye contact
• Move closer to him/her
• Place hand on his/her shoulder
• Private signal
• Give him/her a break with a chore