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aneurysm
visible, prominent pulsation or dialation
diastole
ventricular relaxation, ventricular filling from atria.
erythema
redness of skin
murmur
harsh, blowing sounds caused by disruption of blood flow
myocardial ischemia
oxygen needs heightened so heart has to work harder
systole
ventricular contraction, blood leaves the ventricles to go to the lungs or body.
what is the pacemaker of the heart
sinoatrial node (SA node)
how many times is the sinoatrial node set to discharge per minute
60-100
if the heart rate is initiated in the atrioventricular node, how fast would the heart beat per minute
60
if the heart rate is initiated in the bundle branches, how fast would the heart beat per minute
30
what is the term used to describe a sudden droop in blood pressure that may occur when an older client changes position from lying or sitting to standing
orthostatic hypotension
what two cultural groups are at the highest risk for hypertension
african americans, spanish (mexicans/cuban/filipino)
dullness when percussing over the heart is
an abnormal finding that could indicate an enlarged heart.
the third heart sound (S3) is heard
immediately after S2.
S3= kentucky
the fourth heart sound (S4) is heard
immediately before S1
S4= there are 4 e's in tenessee
what is happening when S1 is heard?
tricuspid and mitral (AV) valves are closing. begins systole
what is happening when S2 is heard?
aortic and pulmonic (semilunar) valves are closing. end of systole and beginning of diastole.
when would the nurse hear S3
when AV (tricuspid & mitral) valves open, blood flow into ventricles may vibrate during mid-diastole, termed ventricular gallop.
when would the nurse hear S4
before S1, termed atrial gallop.
what is the significance of S3
may indicate heart failure or fluid overload.
what is the significance of S4
may indicate hypertension or ventricular hypertrophy
at erb's point you can hear
S1 and S2 equally
at the aortic and pulmonic valves you can hear
S2
at the mitral valve you can hear
S1
(PMI)
aortic stenosis
murmur at aortic area. narrowing of the aortic valve. may be congenital or caused by atherosclerosis
mitral stenosis
murmur heard at the apical area with the client in the left lateral position. possible causes is rheumatic fever or cardiac infection.
ventricular hypertrophy
occurs due to pumping against high pressures
myocardial ishcemia
oxygen needs of the myocardium are not met as the heart works harder. may be caused by plaque or blood clot. need to assess client for type of pain and associated factors including nausea, epigastric pain, jaw or left shoulder pain.
pulmonary stenosis
narrowing of the opening between the pulmonary artery and the right ventricle. may have murmur at pulmonic area and a thrill in the left second and third intercostal space.
congestive heart failure
caused by hearts inability to pump effectively
infective endocarditis
may see splinter hemorrhage of nail beds. caused by a bacterial infection to the lining of the heart chanbers.
tricuspid stenosis
narrowing of the tricuspid valve. murmur over the tricuspid area
aortic regurgitation
back flow of blood from the aorta into the left ventricle. murmur with the client leaning forward. may result in shortness of breath and fatigue.
left sided heart failure
results in pulmonary congestion, shortness of breath, and orthopnea. crackles may be heard on auscultation.
cor pulmonale
complication of untreated heart failure. results in JVD, peripheral edema, fluid retention and weight gain. may have clubbing.
mitral regurgitation
back flow of blood from left ventricle into left atrium. murmur at apex transmitted to left axillae.
pulmonary edema
fluid accumulation can cause severe shortness of breath, pink frothy sputum, coarse crackles that do not clear with cough, sense of doom.
myocardia infarction
complete disruption of blood flow and oxygen to the myocardial tissue. may lead to death of cardiac tissue. symptoms include chest pressure, tightness, squeezing and shortness of breath. may radiate or have referred pain to the left neck, jaw or shoulder.
pericardial friction rub
occurs due to inflammation of pericardial sac. ask client to hold breath to assess as compared to pleural issues.
blood flow pathway
vena cava, right atrium, tricuspid valve, right ventricle, pulmonic valve, pulmonic artery, lungs for oxygenation.
pulmonic veins, left atrium, mitral valve, left ventricle, aortic valve, aorta and to the body.
preload
(stretch) end diastolic filling pressure
afterload
(squeeze) amount of resistance the ventricles have to overcome to eject blood in systole
blood pressure is
cardiac output times peripheral vascular resistance.
during a physical assessment the patient should
start in upright position then proceed to supine with HOB at 45
when inspecting the precordium (anterior chest), abdomen, legs and skeletal structure:
note color, temperature of skin and presence of hair. observe precordium for any abnormal pulsation, pull, lifts or heaves.
the jugular vein may be visible
when lying flat, should disappear when sitting.
jugular vein distention (JVD) presents in
cor pulmonale (right heart failure)
where should you palpate for PMI
mitral valve over apex
myocardial ischemia or acute coronary syndrome
chest pain (angina), shortness of breath, diaphoresis. pain may be heavy, tight, squeezing, pressure. pain may be referred or radiate to jaw, shoulder, epigastric region. will result in myocardial infarction if not treated.
heart failure
progressive weakening of heart as a pump. usually begins on left side. results in pulmonary congestion and shortness of breath. may progress to cor pulmonale, which results in JVD, peripheral edema, fluid retention and weight gain.
effects of right sided heart failure (cor pulmonale)
JVD and peripheral edema. long term heart failure may have clubbing. need low sodium diet, daily weights.
effects of left sided failure
pulmonary congestion and shortness of breath.
stenotic valves do not:
fully open (narrowing)
regurgant valves do not:
fully close
what are septal defects
openings between the right and left atria or right and left ventricles.
pleuritis
chest pain worse with breathing, shallow respirations, pleural rub. can have patient hold breath to rule out cardiac cause.
apnea
absence of breathing
eupnea
normal, good, unlabored ventilation
dyspnea
difficulty breathing
egophony
patient says "E" during auscultation.
normal sounds like E.
consolidation sounds like A.
bronchophony
patient repeats "99" during auscultation.
normal= muffled.
consolidation= clear, loud.
whispered pectoriloquy
patient whispers "1,2,3"
normal = muffled.
consolidation= clear.
fremitus
a vibration felt by palpitation
crackles
brief, discontinuous, popping lung sounds that are high-pitched, intermittent, nonmusical due to fluid.
fine= crackling.
coarse= bubbling.
may hear crackles in atelectasis.
wheezes
musical high pitched breathing with a rasp or whistling sound. a sign of airway constriction or obstruction.
orthopnea
is the sensation of breathlessness when lying flat, relieved by sitting or standing.
to assess ask patient how many pillows they use to sleep or if they sleep in a recliner.
orthopnea can be caused by congestive heart failure or emphysema.
what is the significance of dullness on percussion?
consolidation.
could indicate fluid, tumor or infection.
the nurse percussions over a patient with pneumonia. percussion over an atelectasis in the lungs would reveal?
dullness
dead space where no gas exchange occurs
trachea and bronchi
which lung has 3 lobes
right
asthma
narrowing of the airways resulting in shortness of breath, weeping on expiration, sometimes inspiration, coughing and labored breathing.
alveoli
tiny sacs-like air spaces in the lung where carbon dioxide and oxygen are exchanged.
emphysema
chronic lung condition where there is permanent damage to alveoli. hyperinflated because air is stuck in alveoli. barrel chest with pursed lip breathing. clubbing may be seen due to chronic hypoxia.
pneumonia
consolidation in the lungs. alveoli fill with fluid, bacteria. patient will have crackle lung sounds.
pneumothorax
trapped air in pleural space. a large part of the lung or complete lung collapse can result in unequal chest expansion. may hear hyper resonance.
surfactant
fluid in alveoli that reduce surface tension of pulmonary fluids. helps prevent alveoli from collapsing.
palptation techniques for respiratory system
tactile fremitus, crepitus (subcutaneous emphysema), lung expansion.
palpitation of tactile fremitus
place hands over the lung fields, have client say "99". assess low, mid and high thorax for symmetry.
increased fremitus with thick secretions in central airways.
increased fremitus with pneumonia.
decreased fremitus as you move away from the midline.
hyperresonance
hyper inflated from emphysema, pneumothorax, or COPD (hyper inflated lung due to emphysema will cause hyper resonance)
documentation of normal findings
resonance over lung tissue and dullness over ribs.
auscultation of the lungs
auscultate the posterior thorax for sounds side to side. listen over bare skin to entire respiratory cycle of inspiration and expiration.
it is normal the feel tactile fremitus the strongest
between the scapulas
rhonici
musical low pitched snoring sounds due to thick bronchial secretions.
stridor
high pitched sound with inspiration and expiration. consider obstruction or swelling from allergic reaction.
cheyne-stokes
deep, rapid breathing followed by periods of apnea.
kussmal
rapid, labored breathing. seen in diabetic ketoacidosis
pleural effusion
fluid in pleural space
kyphosis
exaggerated posterior curvature of the thoracic spine. associated with aging. severe deformity may decrease lung expansion and increase cardiac problems.
scoliosis
lateral curvature and rotation of the thoracic and lumbar spine. may cause distortion of the lung, which results in decreased lung volume.
developmental considerations for pregnant females
difficulty on exertion due to pressure on diaphragm and increase oxygen consumption
developmental considerations for elderly
loss of elasticity of lungs, decreased lung expansion, decrease cough ability
developmental considerations for infants
hyper resonance is normal for infant
the braden scale is
used to assess risk for pressure ulcer development.
each topic rated on a 1 to 4 scale with 1 being very high risk and 4 being very low risk. add total score to determine if client is at risk.
peripheral cyanosis
cyanosis on the fingers and toes due to cold exposure, anxiety, or inadequate circulation.
central cyanosis
cyanosis on the head, neck and truck that usually develops during cardiac arrest or CPR due to inadequate oxygen.
circumoral cyanosis
blue around the mouth of a newborn.
pallor
pale color which may indicate bleeding, anemia or hypotension. consider checking hemoglobin and hematocrit (H&H)
jaundice
yellow color. inspect the sclera, hard palate, mucous membranes and skin.
inspect for petechiae in
buccal areas of the mouth or the conjunctivae as well as lighter areas of the skin such as the abdomen, buttocks.
pallor in dark skin will appear
ashen gray. assess for pallor in dark skinned people by their mucous membranes, lips, tongue or conjunctivae