Psychiatry Comat/Boards II

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Alcohol Withdrawal Symptoms

Physical and psychological symptoms that occur when a person with alcohol dependence suddenly reduces or stops drinking. Symptoms can include tremors, anxiety, hallucinations, and seizures.

<p>Physical and psychological symptoms that occur when a person with alcohol dependence suddenly reduces or stops drinking. Symptoms can include tremors, anxiety, hallucinations, and seizures. </p>
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Charles Bonnet Syndrome

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Paraphillic Disorders

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Rett’s Syndrome

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Parkinson’s Disease Drugs

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Delusional Disorder

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MDD + Psychosis Tx

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Antipyschotic Side effects Commonly tested

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NMS

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Malignant Hypererthymia

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postpartum

Post Partum Psychosis: if mom should be admitted to hospital but can see baby occasionally under close supervision

<p>Post Partum Psychosis: if mom should be admitted to hospital but can see baby occasionally under close supervision</p>
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Autism Spectrum Disorder

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EEG findings in Neuropsychiatric Disorders

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Multi-Axial System of Classification

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<p>Lithium Toxicity Ex.</p>

Lithium Toxicity Ex.

can be caused by an acute overdose on lithium or more chronically from decreased renal perfusion (leading to decreased lithium clearance) via dehydration, thiazide diuretics, NSAIDs, ace inhibitors. Symptoms: GI: N, V, diarrhea, lethargy, confusion, agitation, ataxia, tremors, seizures. Tx: IV hydration, hemodialysis in severe causes

<p>can be caused by an acute overdose on lithium or more chronically from decreased renal perfusion (leading to decreased lithium clearance) via dehydration, thiazide diuretics, NSAIDs, ace inhibitors. Symptoms: GI: N, V, diarrhea, lethargy, confusion, agitation, ataxia, tremors, seizures. Tx: IV hydration, hemodialysis in severe causes</p>
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Sleep Cycle

MDD can also be related to hyperactivity-less sleep —> increased cortisol levels. Increased REM is associated with increased acetylcoline. Dopamine has no effect of REM sleep but does decrease total sleep time. NE: decreases total sleep time and REM sleep. Increased serotonin results in prolonged total sleep time and delta wave sleep and is not associated with increased REM. Alcohol will decrease the amount of REM sleep & decreased sleep latency and increased sleep fragmentation.

<p>MDD can also be related to hyperactivity-less sleep —&gt; increased cortisol levels. Increased REM is associated with increased acetylcoline. Dopamine has no effect of REM sleep but does decrease total sleep time. NE: decreases total sleep time and REM sleep. Increased serotonin results in prolonged total sleep time and delta wave sleep and is not associated with increased REM. Alcohol will decrease the amount of REM sleep &amp; decreased sleep latency and increased sleep fragmentation. </p>
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Brain Dead

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Ganser Syndrome

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Kleptomania

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Acute Stress Disorder

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Adjustment Disorder

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<p>MDD Adjuctive Treatment</p>

MDD Adjuctive Treatment

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BZD withdrawl

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Amphetamine Intoxication

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Acute Dystonia

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<p>Paradoxical Agitation via BZD</p>

Paradoxical Agitation via BZD

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Extra-Pyramidal Effects of Anti-psychotics and treatment options

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<p>Low levels of 5-HIAA ? </p>

Low levels of 5-HIAA ?

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Cognitive Impairment in Elderly Patients

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Therapy Modalities

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Reactive Attachment Disorder

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Postpartum Blues, Depression, Psychosis

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Withdrawl Symptoms

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Narcolepsy

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Synthetic Cathiones

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<p>Dopamine Pathways</p>

Dopamine Pathways

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Key Defense Mechanisms

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Medication Induced Depression

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Delirieum Tremens

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Pt. has a neck mass do you bx or thyroid studies or Iodine uptake ?

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Stages of Behavioral Change

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Treatment for Fronto-Temporal Dementia ?

SSRIs may help manage behavioral symptoms.

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<p>Anti-NMDA Receptor Encephalitis</p>

Anti-NMDA Receptor Encephalitis

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<p>Opioid Withdrawal</p>

Opioid Withdrawal

Patients who are trying to get off opiods-but need acute pain managment, can also be treated with NSAIDs because they are a different Moa-like Ketorolac or ibuprofen, which help alleviate pain without affecting the opioid receptors. Symptoms of withdrawal include anxiety, agitation, and flu-like symptoms. But if a patient who is on chronic opiods and is in severe pain from a severe injury, don’t hold back the opiods.

<p>Patients who are trying to get off opiods-but need acute pain managment, can also be treated with NSAIDs because they are a different Moa-like Ketorolac or ibuprofen, which help alleviate pain without affecting the opioid receptors. Symptoms of withdrawal include anxiety, agitation, and flu-like symptoms. But if a patient who is on chronic opiods and is in severe pain from a severe injury, don’t hold back the opiods. </p>
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<p>Eating disorders + Tx. </p>

Eating disorders + Tx.

anorexia patients can make themselves throw up-main differentating factor is the BMI, that is what makes it anorexia

<p>anorexia patients can make themselves throw up-main differentating factor is the BMI, that is what makes it anorexia </p>
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<p>Clonazipine</p>

Clonazipine

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<p>Schizophrenia or BD I </p>

Schizophrenia or BD I

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PD Pyschosis

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<p>Schizo.</p>

Schizo.

Family therapy and CBT + social skills training are good therapy options

<p>Family therapy and CBT + social skills training are good therapy options</p>
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<p>Somatic Symptom and Related Disorders</p>

Somatic Symptom and Related Disorders

Somatic symptom disorder: when the primary complaint is pain related-medications like SNRIs and TCAs can be useful

<p>Somatic symptom disorder: when the primary complaint is pain related-medications like SNRIs and TCAs can be useful</p>
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<p>REM Sleep Behavior Disorder</p>

REM Sleep Behavior Disorder

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Anti-Depressant Discontinuation Syndrome

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Alcohols effect on Neurotransmitters ?

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Airirpoziles effects ?

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Cotard Syndrome

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Capgrass Syndrome

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Fergoli Syndrome

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Bipolar Disorder in Pregnancy

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Dopamine Pathways

Reward Pathway: Dopamine increase in the VTA & Nucleus Acumbens & PFC. Associated with Cannibas-desire/craving for something. This pathway increases pleaseure and reward and encourages the desire for pleasure and reward behaviors. Different then physical dependence though,

<p>Reward Pathway: Dopamine increase in the VTA &amp; Nucleus Acumbens &amp; PFC. Associated with Cannibas-desire/craving for something. This pathway increases pleaseure and reward and encourages the desire for pleasure and reward behaviors. Different then physical dependence though, </p>
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<p>PTSD</p>

PTSD

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<p>Dissociative Amnesia Ex</p>

Dissociative Amnesia Ex

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<p>Disorganized Speech (with Schizophrenia) </p>

Disorganized Speech (with Schizophrenia)

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Bipolar with Rapid Cycling

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<p>Fragile X</p>

Fragile X

Fetal-Alcohol Syndrome

<p>Fetal-Alcohol Syndrome</p>
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<p>Epileptics and AE: </p>

Epileptics and AE:

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Cloparmine in OCD?

First Line is still Fluoxetiene in terms of meds.

<p>First Line is still Fluoxetiene in terms of meds. </p>
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Brain Antaomy/Phsyiology ?

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Substances & MOA

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<p>Opiods vs BZD ? </p>

Opiods vs BZD ?

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More info on Anti-Pyschotics

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Pseudodementia vs Dementia

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Hypertensive Drugs associated with Depression ?

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PMD & PDD

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<p>Bipolar Disorder &amp; Pregnancy </p>

Bipolar Disorder & Pregnancy

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<p>Cannibus and Psychosis? </p>

Cannibus and Psychosis?

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Serotonin Medication withdrawal

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<p>Specific Phobia or Social Anxiety ? </p>

Specific Phobia or Social Anxiety ?

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<p>Therapy Modalities Part II</p>

Therapy Modalities Part II

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Part III Therapy Modalities (same question as Part II)

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<p>Cognitive Theory ? </p>

Cognitive Theory ?

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Idiopathic Hypersomulance

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<p>Alzehiemer’s Vs Vascular Dementia? Part I</p>

Alzehiemer’s Vs Vascular Dementia? Part I

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<p>Answer Part II</p>

Answer Part II

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<p>Frontal Temporal Dementia </p>

Frontal Temporal Dementia

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Beta Pleated Sheets ?

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Histopathological findings associated with different Dementias ?

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Anxiety Meds in the Elderly ?

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Motivational Interviewing

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