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Medication teaching for benzodiazepines, including which neurotransmitter it affects
Benzodiazepines
Neurotransmitter affected: GABA (gamma-aminobutyric acid) — enhances its effect to produce a calming effect.
Teaching:
Short-term use only (risk of dependence)
Avoid alcohol and CNS depressants
Do not stop abruptly (risk of withdrawal)
May cause drowsiness, dizziness, confusion
Identify the 4 levels of anxiety,
o describe what occurs in each stage and
o interventions effective at each stage
Mild: special attention; increased sensory stimulation; motivational - Encourage learning, problem-solving
Moderate: feeling something is definitely wrong; nervousness/agitation; difficulty concentrating; able to be redirected - Use calm communication, help focus
Severe: trouble thinking and reasoning; tightened muscles; increased vital signs; restless, irritable, angry - Stay with client, use simple directions
Panic: fight, flight, or freeze response; increased vital signs; enlarged pupils; cognitive processes focusing on defense - Ensure safety, stay calm, reduce stimuli
Role of defense mechanisms in anxiety
Purpose: Unconscious strategies to reduce anxiety.
Examples:
Denial: Refusing to accept reality
Repression: Unconsciously blocking out painful or anxiety-provoking thoughts, memories, or feelings.
Regression: Reverting to behaviors characteristic of an earlier developmental stage when faced with stress or anxiety.
Sublimination: Channeling unacceptable impulses or desires into socially acceptable or constructive activities.
Example: Someone with aggressive urges becomes a professional boxer or takes up intense exercise.
Projection: Attributing one’s feelings to others
Displacement: Redirecting emotions to a safer target
Rationalization: Justifying behaviors with logical reasons
Best combination of treatment modalities for anxiety
Medication with therapy
Cognitive techniques: identifying and facing anxiety-producing situations
correct thoughts, substitute more balanced thoughts
Homework assignments
Behavioral therapy: Systematic desensitization
Medication:
SSRIS: GAD, Panic, Phobia, OCD, BDD, Trichotillomania
SNRIS: GAD, Panic
Buspirone: GAD
Hydroxyzine: as needed for: GAD exacerbation, anxiety attack, panic attack
Clonidine: Management/prevention of anxiety
Benzodiazepine: Extreme anxiety/panic as needed, Risk for addiction/dependence
Combination of:
Cognitive Behavioral Therapy (CBT)
Medication (SSRIs, SNRIs, benzodiazepines short-term)
Relaxation techniques (breathing, mindfulness)
Therapy for anxiety
CBT: Most effective (identify and change negative thought patterns and behaviors)
Exposure therapy: Especially for phobias
Treatments for phobias
Systematic desensitization (pairing relaxation techniques with gradual exposure to the feared object or situation)
Flooding (less common – exposure to greatest fear all at once)
CBT
Homework
Be able to identify and differentiate obsessions from compulsions
OBSESSION = THOUGHTS
COMPULSION = BEHAVIORS
Characteristics of OCD, including preferred treatment settings (hospital inpatient or clinic outpatient/group therapy)
Obsessions/Compulsions
Thoughts or behaviors that interfere with personal, social, and/or occupational functioning
The person realizes that their thoughts/behaviors are unreasonable, but they cannot stop/control them
OCD relieves anxiety when situations producing anxiety cannot be avoided
Mild to Moderate OCD:
✅ Outpatient therapy is preferred
✅ Group therapy can be helpful for support and shared strategies
✅ CBT with Exposure and Response Prevention (ERP) is the gold standard
Severe OCD or when safety is a concern:
✅ Inpatient hospitalization may be necessary
Especially if the person is unable to function, is suicidal, or has co-occurring disorders
Etiology of OCD
Biological Factors:
Neurotransmitter imbalance, especially low serotonin levels.
Abnormal activity in brain circuits involving the orbitofrontal cortex, anterior cingulate cortex, and basal ganglia.
Genetic predisposition: Higher risk if a first-degree relative has OCD.
Psychological Factors:
Cognitive distortions (e.g., overestimating threats, perfectionism).
Learned behaviors reinforced by anxiety relief (e.g., compulsions reduce distress temporarily).
Environmental Factors:
Childhood trauma or abuse, Stressful life events.
In rare cases, infections (e.g., PANDAS in children).
Goals/Outcomes of OCD treatment
The primary goals are to reduce the frequency, intensity, and distress caused by obsessions and compulsions, and to improve daily functioning.
Identify characteristics of body dysmorphic disorder (BDD)
Exaggerated belief body is deformed or defective
Person aware beliefs are exaggerated
Usually have another mental disorder (depression common)
Visit plastic surgeons and dermatologists
Definition and characteristics of post-traumatic stress disorder (when does it occur in relation to “event” that caused it, what behaviors are typical?)
Disturbing pattern of behavior demonstrated by someone who has experienced, witnessed, or been
confronted with traumatic event. Event posted actual or threatened death or serious injury.
Responses
o Intense fear
o Helplessness
o Terror
Present for at least 1 month
Interferes with life functions
May occur years later· Differentiate acute stress disorder from PTSD
Differentiate acute stress disorder from PTSD
Acute stress disorder occurs 3 days to 4 weeks after trauma
Acute stress disorder resolves by 4 weeks
How to respond to a patient experiencing a flashback
Focus the client in the present, grounding techniques
Teaching for family members of survivors of trauma (promoting self-esteem)
It may take years for the patient to reach their goals
Validate their feelings: “It’s okay to feel this way.” Avoid judgment or pressure: Don’t say “You should be over this by now.” Celebrate small victories: Acknowledge progress, no matter how small.