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norm temp
98.6-99.3 degrees F
norm HR/PR
60-100 bpm
norm RR
12-20 rpm
norm BP
120/80 mmHg
norm SpO2
98%
taking vitals are a quick window into…
general health screening, systems review (cardiopulm, neurological, thermoregulatory, NMS pain), baseline for comparision pre- & post-activity, tolerance of mobility/exercise/treatment, specific populations, diagnostic/prognostic clues
a ___ of VSs is more useful than a single value
trend
VSs in rehab
temp, HR/PR, RR, BP, SpO2, pain, gait speed
VSs don’t exist in a ___, consider influences such as …
vacuum; pt demographics, behavior/lifestyle, situation
body temp
heat production (shiver) & loss (sweat); reflects a heat-balance problem
fever temp
>=100.4 degrees F
the ___ of measurement can change the number, such as …
route; oral, rectal, axillary, tympanic, temporal sites (not interchangeable)
pulse deficit
simultaneous difference in HR at apical & PR at a peripheral site, which should be the same; indirect marker of LV contraction & circulatory response, arrythmia
tachycardia
>100 bpm
bradycardia
<60 bpm
central/cardiac pulse sites & use
carotid artery, apical auscultation; use when peripheral pulse is difficult to palpate, to confirm rhythm, to prioritize centrla circulation
UE pulse sites & uses
radial artery, brachial artery; use for routine measurement or BP in infants
LE pulse sites & uses
femoral artery, popliteal artery, dorsalis pedis artery, post tibial artery; use to assess regional perfusion & compare sides
pulse rate technique
count beats for 60 sec when irregular or when precision matters; count beats for 30 sec*2 for regular rhythm & regular visitors
pulse rhythm technique
count beats for full 60 sec if irregular & consider apical assessment/telemetry/medical follow-up depending on context
pulse amplitude/force scale (O’Sullivan scale)
4+ = full/bounding, can’t be obliterated
3+ = normal/expected intensity
2+ = weak/difficult to palpate, may be obliterated
1+ = weak/diminished intensity, thready/comes & goes
0 = absent, not palpable
HR should ___ w/ ___ dynamic workload
rise; incr
HRmax estimate can guide ___, but can be lowered w/ ___ at a given submax workload
intensity; training
factors that modify HR response
age, fitness, medications (beta-blockers), autonomic dysfunction, temp, dehydration, pain/anxiety, illness, arrhythmia
concerning factors that modify HR response
failure to rise when expected, disproportionate tachycardia, symptomatic bradycardia, new irregularity, slow recovery, symptoms out of proportion
when measuring routine rate, use ___ artery
radial
when measuring irregular rhythm, use ___ artery for full minute
apical
when to measuring BP, use ___ artery
brachial
when measuring distal perfusion, use ___ arteries
dorsalis pedis, post tibial
when measuring side-to-side vascular question, compare ___
bilaterally
arterial insufficiency tests
capillary refill, dependent rubor/elevation, handheld doppler ultrasound, ankle-brachial index (ABI)
venous insufficiency/DVT tests
venous filling/leg elevation, medical referral, Well’s Criteria; Homan’s sign no longer reliable
Well’s criteria (3-level)
used to test venous insufficiency/DVT; swelling, paralysis/paresis, cancer, activity
high risk > 2 → urgent diagnostic pathway for prox leg vein ultrasound
mod risk = 1-2 → DVT not ruled out, refer for diagnostic work
low risk < 1 → D-dimer pathway, follow-up
ventilation
air moving in & out of lungs
pulmonary gas exchange
O2/CO2 cross thru alveolar-capillary membrane; dependent on ventilation & perfusion
transport
O2 carried by hemoglobin; dependent on blood flow
bradypnea
<10 breaths/min
tachypnea
>24 breaths/min
resting HR:RR ratio
4:1
when counting RR, do it ___ so the pt doesn’t change their breathing when they know you are counting
quietly
breathing is influenced by …
chemistry, position, activity, emotion, disease, consciousness
PaO2
partial pressure of oxygen dissolved in arterial plasma; measured from arterial blood gas (ABG), reflects oxygen tension & not Hgb saturation directly
SaO2
arterial Hgb oxygen saturation; measured on arterial blood; represents proportion of Hgb binding sites carrying oxygen
SpO2
peripheral oxygen saturation estimated by pulse oximetry; fast & useful for trends
good measuremnet technique for pulse oximetry
warm, perfused extremity; minimize motion; allow reading stabilization; treat at rest, activity, recovery; check that displayed pulse matches pt
common limitations of pulse oximetry accuracy
poor circulation/low perfusion; motion; skin pigmentation, thickness, temp; nail polish/artificial nails; tobacco/dyshemoglobinemias
hypoxemia clues from pulse oximetry
dyspnea/tachypnea; restlessness/anxiety/confusion; cyanosis'; tachycardia/dysrrhythmia; fatigue/exercise intolerance; worsening mental status
BP reflects ___ against ___
flow; resistance
arterial pressure = ___*___
CO*vascular resistance
BP technique sources of error
cuff bladder/improper size, arm position, muscle activity, site/side, repeat readings
elevated BP
120-129/<80 bpm
stage 1 HTN BP
130-139/80-89
stage 2 HTN BP
>140/>90
manual BP technique
seated position at quiet rest w/ arm supported at heart level
cuff bladder centered over brachial artery
palpate radial pulse & inflate cuff until pulse disappears → note level
reinflate ~20-30 mmHG above estimated systelic level
deflate ~2-3 mmHg/sec while auscultating brachial artery
read Korotkoff phase 1 (SBP) & phase V (disappearance, DBP)
repeat for unexpected values & average readings when appropriate
contraindications/precautions for manual BP on a limb
vascular access, srugery, injury, lymphedema risk, pt-specific restrictions
in dynamic exercise, ___ rises proportionate to dynamic workload & ___ slightly changes
SBP; DBP
isometric/static exercises can produce substantial ___ in both SBP & DBP
incr
mean arterial pressure (MAP)
avg pressure driving systemic perfusion across cardiac cycle; norm 70-110 mmHg = (SBP + 2 * DBP/3; can turn VSs into physiologic meaning → perfusion of organs depends on adequate pressure
rate-pressure product (RPP)
simple estimate of myocardial work/oxygen demand; HR*SBP; can turn VSs into physiologic meaning → relate symptoms such as angina to myocardial demand
IASP
pain is sensory & emotional, associated w/ actual/potential tissue damage
acute pain
an expected protective response to noxious input/tissue injury; sympathetic & adrenal responses scaled by threat/intensity; closely linked to inflammation/tissue damage; better localized; persists while noxious process is active & should improve w/ recovery
chronic/persistent pain
persists beyong expected tissue healing/becomes a condition in its own right; from acute pain or insidious onset; less proportional to ongoing tissue injury; consider central, peripheral, cognitive, behavioral, emotional, and social factors
cognitive beahvior model of persistent pain
pain experience → thoughts/beliefs → emotion → behaviors → physical consequences → reinforcement of pain → back to start; pain is influenced bby interactions among physical, cognitive, emotional, and behavioral factors
muscle pain descriptors
cramping, dull, aching
peripheral nerve pain descriptors
sharp, bright, lightning-like
bone pain descriptors
deep, naging, dull
vacular pain descriptors
throbbing, pulsing, diffuse
nerve root pain descriptors
sharp, shooting
sympathetic/autonomic pain descriptors
burning, pressure, singing, aching
fracture pain descriptors
sharp, severe, intolerable
quantifying pain & impact
intensity, location, pattern, onset, chronic pain/behavior tools such as fear-avoidance beliefs questionnaire (FABQ) & pain anxiety symptoms scale (PASS)
VS decision pathway
measure → verify → compare → interpret → act
arterial fluid system
delivers O2 & nutrients under pressure; failure → ischemia, claudication, distal tissue loss
venous fluid system
returns blood toward heart; valves & muscle pumps assist; failure → venous HTN, edema, skin change
lymphatic fluid system
returns interstitial fluid & proteins, supports immune function; failure → protein-rich edema, fibrosis, infection risk
integumentary system functions
barrier, sensation, thermoregulation, immunity & visible evidence; failure → impaired tolerance, healing
wound healing phases
inflammation → proliferation → maturation; phases overlap
things needed for wound healing
oxygen, moisture & cover, nutrition
local limitations of healing
perfusion/ischemia, edema, pressure/shear, bioburden/infection, foreign material/necrosis, repeated trauma, excessive dryness/maceration
systemic limitations of healing
diabetes/neuropathy, nutrition/hydration, smoking, age/fragility, anemia/oxygenation, immune suppression, cardiac/renal/vascular disease
context limitations of healing
med effects, radiation/cancer treatment, adherence & support, off-loading/pressure relief, activity & mobility, compression appropriateness, care environment
arteries
carry blood away from heart at higher pressure; thick, muscular, elastic walls; relatively narrow lumen; no valves; pulse = pressure wave
capillaries
exchange surface b/n blood & tissues; 1-cell thick endothelium; very small lumen; diffusion of oxygen, nutrients, wastes; leukocytes can move into tissues (oncotic pressure)
veins
return blood to the heart at lower pressure; thinner walls; wider lumen; valves limit backflow; muscle pump assists return
peripheral artery disease (PAD)
inadequate arterial inflow due to smoking, DM, HTN, dyslipidemia/hypercholesterolemia, kidney disease, cardiovascular disease, obesity/low activity, older age, trauma, surgery, arterial embolus, hypercoagulable states
arterial insufficiency
dependent w/ rubor of dependency/elevation test; claudication, pain at rest, pain worsened w/ elevation; cool temp w/ pallor elevation; shiny, hairless skin & reduced tissue quality; reduced/asymmetric/absent pulse; distal pain/pressure point wounds; reduced walking endurance → fatigue instead of pain
ankle-brachial index (ABI)
ankle SBP/brachial SBP = norm 1.00-1.40, resting perfusion adequate; guides referral & influences compression, debridement, wound prognosis, exercise safety
chronic venous insufficiency
age, low activity, obesity, pregnancy, long periods of standing/sitting, heredity, prior DVT → edema, skin hyperpigmentation, erythema, skin texture changes, venous ulcers, aching/heaviness, warm temp, pulse present
pressure injury
tissue load exceeds tolerance, shear forces, friction, moisture, malnutrition, impaired perfusion, sensory loss, immobility → ischemia → persistent erythema, nonblanchable redness, firm tissue, tenderness, color change → cell necrosis
pressure injury sites when lying supine
occiput, scapulae, elbows, sacrum/coccyx, heels
pressure injury sites when side-lying
ear, shoulder, ribs, greater trochanter, knees, malleoli
pressure injury sites when sitting
scapulae, spinous processes, sacrum, ischial tuberosities, heels
pressure injury sites when lying prone
cheek/ear, acromion, chest/breast, iliac crest, patella, toes
pressure injury stages
stage 1: skin intact w/ nonblanchable erythema
stage 2: partial-thickness skin loss w/ exposed dermis, wound bed viable, no slough/eschar
stage 3: full-thickness skin loss, visible adipose, granulation, undermining/tunneling
stage 4: full-thickness skin loss, exposed fascia/muscles/tenndon/cartilage/bone
unstageable: full-thickness skin loss w/ obstructive slough/eschar; unconfirmable depth
deep tissue: persistent red/maroon/purple discoloration (blood blister)
lymphedema
reflects impaired lymph transport; developmental abnormal lymphatic vessels/nodes; acquired after cancer treatment, surgery, radiation, trauma, or chronic venous disease; swelling, heaviness, tightness, numbness, fibrosis, recurrent infection risk, impaired would healing, decr ROM & mobility
measuring swelling can be done via tissue quality, ___, ___
girth; volume
diabetic peripheral neuropath can change … function
sensory, motor, autonomic, and vascular
foot screening connects ___ to loading & ___ risk, so we must …
sensation; tissue; inspect skin, sensations, & perfusion/pulses → education
skin descriptors
color, moisture, texture, firmness, temp, elasticity, turgor, asymmetry, contour, shape, hair, nails, lesions