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Exam 1 NURS 300
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Layers of the skin
Epidermis and dermis (separated by the dermal-epidermal junction membrane)
Function: thermoregulation, protection from pathogens
Epidermis
Top layer
Several layers
Flattened, dead, keratinized skin cells
Function: protect underlying cells/tissues from dehydration, prevents entrance of certain chemicals, allows for evaporation of water off of skin, permits absorptions
Dermis
Inner layer
Contains mostly connective tissue
Collagen, blood vessels, and nerves
Function: tensile strength, mechanical support, protection of underlying bones, muscles, organs
Types of wounds
Partial-thickness, full-thickness, primary intention, secondary intention
Partial-thickness
Damage to epidermis, may go into dermis
Heals through epithelialization
Full-thickness
Damage that extends beyond two layers of skin (into subQ tissue, may even see bone)
Heals from deeper to higher areas
Primary intention
Surgical incision that is sutured or stapled
Implications: heals through epithelialization, minimal scar formation
Secondary intention
Surgical wounds that have contamination or tissue loss, pressure injuries, or burns
Heals using granulation tissue formation, wound contracting, and epithelialization
Wound healing complications
Hemorrhage, infection, dehiscence, evisceration
Hemorrhage
Lots of bleeding, not normal
May indicate dislodged surgical suture, infection, etc
Signs of Infection
Erythema
Increased wound drainage (thick, color, odor)
Periwound warmth, edema, pain
WBC may be increased
Possible presence of fever
Dehiscence
Partial or full separation of wound layers
Risk: obesity, poor nutrition, infection, underlying disease
Serosanguineous drainage could be an indication (but not factual evidence of dehiscence)
Evisceration
Total separation of wound layers
Emergent
Keep inside tissues moist
Risk factors to pressure ulcers
DM, incontinence, impaired sensory perception, older adults, spinal cord injury, long term homes/community care, hospice
Decreased mobility (unable to reposition themselves) or perception of pain is major risk factor
Stage 1 pressure ulcer
intact skin, but localized non-blanchable erythema
Stage 2 pressure ulcer
partial-thickness skin loss
exposed dermis
(no presence of eschar, slough, granulation)
Stage 3 pressure ulcer
full-thickness skin loss
adipose is visible
slough, eschar, and granulation may be visible
Stage 4 pressure ulcer
full-thickness skin and tissue loss
exposed palpable fascia, muscle, tendon, cartilage, or bone
slough and eschar may be visible
tunneling often occurs
Unstageable pressure injury
Concealed full-thickness skin and tissue loss
Typically the extent can’t be determined due to eschar or slough covering it
Once removed, stage 3 or 4 pressure injury may be revealed
Dry, intact eschar should not be removed
Deep Tissue Pressure Injury (DTPI)
Intact or non-intact non-blanchable skin
Deep red/maroon/purple
Results from intense prolonged pressure
May resolve without skin loss or evolve rapidly
Risk assessment
Braden’s Scale
Norton’s Scale
Measurement of pressure injuries
Width (with disposable ruler)
Height (with disposable ruler)
Depth (with sterile cotton tip)
Undermining and land mining (with sterile cotton tip)
Documentation of pressure injuries
Location
Size (width, height, depth, undermining, tunneling)
Color
Drainage
Type of wound bed (granulation, slough, eschar)
Pt reports of pain and discomfort
Types of drainage
Serous, purulent, serosanguineous, sanguineous
Serous
normal drainage
Purulent
Could be indicative of infection
Serosanguineous
Not totally indicative of a problem, but need to keep and eye on it
Sanguineous
Can be common during inflammatory phase
After this stage, may be indicative of damaged capillaries or trauma
Large amounts can be a cause for concern
Heat therapies
warm compress (musculoskeletal injury), warm soaks (promote blood flow), hot packs
Cold therapies
Cold packs, cold, moist and dry compress (soft tissue injury), cold soak (decreases blood flow), ice bags or collars