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general description of VF nodules
bilateral bumps at the juncture of the anterior and middle 2/3 of the VFs
true or false: there is considerable variation in VF nodules
true
prevalence of VF nodules in school age (Killic)
30%
VF nodules involves inflammatory mid-membranous generation of what?
the basement membrane zone and superficial layer of lamina propria
Etiology VF nodules
phonotrauma and hyper function from:
talking/shouting
throat clearing
sound effects
singing
VF perceptual ax
breathy
decreased pitch
flat
deteriorates throughout the day
VF visual ax
hourglass closure/open glottal chink (ant/post)
increased mass of TVFs
decreased amplitude and mucosal wave
edema/erythema
true or false: VF nodules only affect closure and pliability
false: they can affect closure, pliability, and symmetry
VF nodule initial management
behavioural voice tx
young clients may need psych support
VF nodule treatment (+vascular lesions)
oral steroids may help
surgery for VF nodules?
if removed surgically w/o any voice treatment, nodules likely to return
Reinke’s edema
chronic diffuse swelling of the superficial lamina propria of the VF
what happens to the lamina propria in RE?
the collagen architecture is disrupted and a thick, gelatinous fluid like material develops in Reinke’s space
true or false: RE is usually unilateral
false: usually bilateral
what does RE often affect?
the anterior 2/3s of glottal margin (vibrating portion) or membrane covering the muscular portion of the folds
true or false: RE is non-cancerous
true
common causes of RE
smoking
chronic vocal hyperfunction
laryngopharyngeal reflux
prolonged exposure to inflammatory stimuli accompanied by abnormal healing
RE effect on closure
VFs may not fully close → breathy or hoarse voice
reduced closure due to large swollen tissue → also lower pitch from mass
RE effect on symmetry
often bilateral, but sometimes more pronounced on one side so vibrations may not be symmetrical
RE effect on pliability
less pliable because of the mass, but variable (some may be more pliable)
Ax for RE
laryngeal exam high priority
4 kinds: edema of one fold, edema of both, edema of one with a lesion on either, edema of both with a lesion on one or both
CAPE-V
Self perception of QoL
voice tx for RE
eliminate cause of problem
promote proper use of voice
is surgery recommended for RE?
often no, not a permanent solution but if behavioural tx not successful surgery may be indicated
what are VF polyps
focal abnormalities within the superficial lamina propria that are slightly deeper than nodules
true or false: VF polyps are often unilateral
true, with a reactive lesion found immediately across from the polyp
what do VF polyps result from?
a single vocal event (like yelling at a concert)
what kinds of polyps are there?
translucent, fibrotic, hyaline, hemorrhagic, or mixed
what increases likelihood of polyps?
smoking
true or false: VF polyps can potentially result from long term vocal abuse/irritation
true
once a small polyp begins….
any continued phonotrauma will irritate it, adding to its growth
VF polyp perceptual findings
hoarseness and breathiness
moderate dysphonia
sessile
a polyp attached to the VF
pedule
a polyp that has a stalk like growth
VF polyp effect on closure
worse glottal closure (incomplete depending on size and location)
VF polyp effect on symmetry
usually unilateral (physical asymmetry and asymmetric closure)
vibration asymmetry
phase delay due to mass
VF polyp effect on pliability
smaller mucosal wave amplitude, magnitude, and persistence
surgical interventions for VF polyps
microflap: lifting flap, removal, flap placed back
epitelial cordotomy: microdissection
most effective followed by vocal rest
improvement for VF polyps
type of polyp associated with improvement (translucent more likely)
smaller more likely to improve
voice tx often provided in combo with surgery
true or false: voice therapy only is not a warranted first course of treatment before surgery for VF polyps
fasle
voice tx for VF polyps
coaching for avoiding vocal abuse
altering pitch and volume
how to breathe to best support speech
how to reduce pressure on the VFs
VF cyst description
benign
usually unilateral
occur on VFs or anywhere on ventricular folds
how do cysts often appear compared to VF nodules?
soft and pliable vs hard and fibrotic
true or false: clients with VF polyps always experience dysphonia
false: it depends on the site of lesion
what cyst location is more prone to dysphonia?
saccular cysts between the false VFs and thyroid cartilage
common causes of cysts
can develop in utero
repetitive phonotruama
glands becoming clogged and mucous becomes trapped
what are the two kinds of cysts?
mucous glands in vocal folds become plugged in the middle portion of the upper lamina propria
cyst fills with keratin in the deep layers of the lamina propria
perceptual ax cysts
pitch instability
breathiness
hoarseness
roughness
airway obstruction
cyst effect on closure
unilateral → one VF is closing completely while one is not
bilateral → hourglass closure or chink
more flexible than nodules → may assist with more closure
cyst effect on symmetry
unilateral → each VF is moving differently which affects amplitude and mucosal wave
typically not symmetrical when they are bilateral
tx for cysts
voice therapy and transoral microlaryngeal surgery
true or false: it is rare for a VF cyst to resolve without surgery
true
voice therapy management for cysts
education on vocal hygiene and reducing vocal abuse
strategies to reduce hyperkinetic behaviours to obtain best vocal fold vibration
laryngeal papilloma is aka
recurrent respiratory papillomatosis (RRP)
laryngeal papilloma description
wart like tumours that grow in the larynx on or around the vocal folds due to a viral infection of epithelial cells
the majority of cases of laryngeal papilloma are caused by what?
HPV types 6 and 11
LP effect on closure
improper closure due to increased mass
LP effect on symmetry
can be unilateral or bilateral → asymmetric closure and vibration
LP effect on pliability
more/less pliable depending on the density and placement
LP perceptual ax
breathy vocal quality
hoarseness (main symptom)
chronic coughing
LP visual ax
tumor (masses)
edema
true or false: LP treatment is curative
false: considered palliative
true or false: LP Treatment is often surgical
true, due to the resilience of HPV
other tx for LP
laser therapy (no general anesthesia)
antiviral meds
HPV and mumps vax
voice therapy to avoice excessive strain
true or false: you are not able to restore full function with only behavioural management in LP
true
laryngeal web description
congenital or acquired condition characterized by a layer of tissue connecting the VFs
a laryngeal web grows across the ___ in the anterior to posterior direction
glottis
laryngeal web sometimes called a
synechia
what kind of sound does a LW often cause?
high pitched and rough
LW congenital causes
glottal membrane failing to separate in embryonic development
may be associated with velocardiofacial syndrome
LW acquired causes
often results from long term intubation or post VF surgery
can be seen after laryngeal infection of external trauma to the VFs
can occur due to the proximity of the VFs
LW effect on closure
inhibits normal VF vibration, limits VF opening
LW effect on symmetry
web grows symetrically and is surgically removed, maintaining symmetry
LW effect on pliability
webbing limits VFs’ ability to vibrate freely
intensity of the webbing determines how pliable VFs are
LW vocal quality
weak
high pitched, rough sound
inhalatory stridor (congential)
SOB
high pitched/abnormal cry in babies
hoarse
wheezing
LW congenital ax
genetic testing for chromosome 22q deletion
weak cry and aphonia since birth
true or false: if LW is congential it requires surgery immediately
true
LW management
dilation: small balloon in airway to widen or break web
surgery: opening into larynx to cut through the web → cover the VFs to make sure the web does not grow back then coverings removed after 6-8weeks
vocal rest needed as long as covering is in place
may require temporary trach
variable recovery time
MTD description
a spectrum of disturbed VF behaviour caused by increased tension of the (para)laryngeal musculature
MTD etiologies
vocal misuse/overuse
psychological/personality disorders
compensatory vocal habits due to LPR, URI, and/or underlying pathology
maybe neurologic (motor control disorder → improperly process what they hear)
MTD perceptual ax
effortful voicing
vocal fatigue
odynophonia
MTD visual
excessive glottic and supraglottic medial contraction
A-P contraction of supraglottal muscles
decreased vibratory amplitude
psychogenic VF bowing
variety of glottic gaps
behavioural tx for MTD
biofeedback
relaxation
resonant voice tx
circumlaryngeal massage
manual therapy techniques
accent method
semi occluded vocal tract exercises
MTD respiratory patterns
shallow clavicular and accessory muscle breathing
limited abdominal excursion at rest and during vocalization tasks
increased breath-holding patterns
Ventricular phonation description
use of the FVFs during phonation either instead of or along with the TVFs
two causative conditions for ventricular phonation
non-compensatory: habitual, psycho-emotional, ideopathic
compensatory: appropriate compensation for profound true VF dysfunction
ventricular phonation perceptual ax
low pitch
reduced pitch variability
diplophonia
hoarse and breathy quality
ventricular phonation visual ax
looks normal with abduction
ventricular movement/compression during adductory tasks
hypertrophy of ventricular folds
what do you do first in ventricular phonation management?
assess the function and capability of the TVFs
ventricular phonation tx
- if TVFs are capable:
behavioural voice tx
biofeedback
relaxation
inspiratory phonation
labial constriction
- psychotherapy
- pharmacological (botox)
- surgical (excision or laser surgery)
puberphonia description
voice doesn’t drop in adolescent males during/after puberty despite anatomical changes
Puberphonia psychological cause?
maybe difficulty transitioning into adulthood
puberphonia physical cause?
hearing impairment
immature laryngeal maturation
poor neuromuscular coordination
endocrine disorder
neurological condition
puberphonia perceptual Ax
high pitch
breathy
puberphonia visual ax
incomplete glottal closure
increased pitch/length of folds
decreased amplitude and mucosal wave
in some cases hyperfunction (not all!!)
management for puberphonia
if no identifiable physical/organic cause, behavioural tx
techniques for puberphonia
cough → hum, all voiced word (record for feedback)
light pressure on thyroid cartilage
transitioning reflexive pitch to volitional pitch (principles of motor learning)