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Overload
A system must be exercised at a level that it is not accustomed to for adaptation to occur
Specificity
exercise training is specific to the muscles involved ( can't train one leg and expect the other to be just as strong)
Reversibility
The fitness gained during overload are quickly lost when training is stopped
End Diastolic Volume (EDV)
preload (how much blood the heart can take in)
Afterload (TPR)
How much pressure is needed to push blood out of the heart
Cardiac Contractility
the strength of contractions of the heart
Stroke Volume
the amount of blood ejected from the heart with each beat
Hyperplasia
an organ getting larger due to an increase in cells
VO2 max
measure of the maximal capacity of the body to transport and use oxygen during dynamic exercise that uses large muscle groups
a-v O2
How much oxygen is removed from the blood flow and used by tissue
1 - RM
Maximum load that can be moved through a full range of motion
F.I.T.T
Frequency intensity time type
Basic principals of training
FITT
What factors in endurance training cause an increase in VO2 max
Increase in maximal cardiac output and A-VO2
What are the guidelines for increasing endurance training that increases VO2 max
training for 20-60 minutes 3 or more times per week at an intensity > 50% V02 Max
What population sees the largest increase in VO2 Max
Untrained individuals with good genetics
Rate change in VO2 max
15-20% during endurance training
What factors increase stroke volume
End diastolic volume, cardiac contractile, afterload
What factors a-v O2 difference
Increased number of capillaries and increased number of mitochondria
How does an increase in mitochondria influence energy substrait utilization, lactate, H+ concentration
Increases the amount of ADP in which causes a stimulus for more ATP to meet the demands of the system reduces the production of lactate and h+
How does training influence acid-base balance
since an increase in mitochondria decreases the amount of lactate and H+ in the cells blood stays at a higher PH
How does training influence antioxidant enzymes
increases the number of endogenous antioxidants in trained muscles
Effect of exercise on the production of specific proteins and the time frame of peak production
More mRNA is produced and sent where it creates more proteins the peak is 4-8 hours after exercising
Primary signals for muscle adaptation during exercise
mechanical stretch, calcium increases, elevated free radicals, and increase in phosphate/muscle energy levels
secondary signals for muscle adaptation during exercise
AMPK (signals energy production pathways), P38 (signals for mitochondrial biogeneses), PCG 1-alpha (formation of capillaries and mitochondrial biogenisis), CaMK (increases calcium levels), calciuneurin calciuneurin (muscle fiber growth and regeneration), IGF-I/Akt/mTOR (promotes muscle protein synthesis), NFkB (promotes antioxidant enzymes)
muscle adaptations to training
Decreased utilization of plasma glucose, increase in fat metabolism, increased muscle antioxidant capacity
Effect of training on mitochondria
Increases he number of mitochondria
Effect of training on capillary number
Capillary number increases
Mitochondrial adaptations to endurance training and what is the result of these adaptations
increased number of mitochondria, increased signaling for ATP, reduction in lactate and H+ Ions
Transfer of training effect for endurance and resistance training
the responses of the cardiac, pulmonary, and sympathetic nervous system are more dependent of the trained state of the muscle than their own adaptations
The effects of detraining on VO2 max
decrease in maximal cardiac output and amount of oxygen extracted from the blood. Initial decrease in the first 12 days (stroke volume), day 24-84 (a-VO2). 4 weeks of re training to make up for 1 week of not training.
The effects of detraining on strength
due majorly to changes in the nervous system, can maintain strenght for 12 weeks with one workout a week,
What adaptations result in muscle strength gains during short term training
Increased recruitment of motor neurons, alter motor neuron firing rates, and enhance motor units synchronization
What adaptations result in muscle strength gains during long term training
Increase in muscle size
How can endurance training affect strength training
Harder to recruit motor neurons, low muscle glycogen content, over training, depressed protein synthesis
Atherosclerosis
hardening and narrowing of the arteries
Degenerative Disease
result of degenerative cell changes affecting tissues or organs
Web of causation
epidemiological model establishing the cause for diseases
Metabolic Syndrome
increased blood pressure, high blood sugar, excess body fat around the waist, and abnormal cholesterol or triglyceride levels
Epidemiology
study of diseases
Infectious diseases
disease caused by microorganisms invading the tissue
Primary risk Factor for CVD
age, gender, family history, race
Secondary risk for CVD
high blood pressure, diabetes, obesity, smoking physical inactivity, stress
How does exercise help prevent and control disease
improves glucose tolerance, increases breaking of clots, and reducing blood pressure
Development and progress of heart disease
Heart tissue becomes inflamed and recruits white blood cells (leukocytes), adhesion molecules bind to leukocytes, which encourages monocytes to enter the intima and mature into macrophages that ingest LDL cholesterol, all the cholesterol terns them into foam cells which multiply and grow, smooth muscle cells grow over this thickening and hardening the walls of the arteries but are eventually ruptured by an inflammatory response by the foam cells.
The role of inflammation in the development of chronic disease
visceral fat causes the secretion of more inflammatory cytokines
Modifiable risk factors for coronary heart disease
Smoking, sedentary lifestyle, obesity, hypertension, diabetes, dyslipidemia, high cholesterol, stress
Uncontrollable risk factors for coronary heart disease
Age, family history, gender, race
Metabolic Syndrome and its causes
abdominal obesity, high blood pressure, high fasting glucose, hypertriglyceridemia (insulin resistance) (hypertention) (SNS activity) (obesity)
Treatments for metabolic syndrome. (inventions for diabetes and CV disease)
physical activity, healthy diet, beta blockers, insulin pump, pacemaker
Insulin-Resistance Hypertension hypothesis
Hypertension causes insulin resistance. Hypertension causes a decrease in the small blood vessels in the in the muscle leading to a reduction in the the delivery of glucose and insulin
Physical activity
Any muscular activity
Physical fitness
attributes that people have or develop that relate the the ability to perform physical activity
Exercise
a subset of physical activity that it planned with the goal of improving physical fitness
Target Heart Rate
the heart rate values needed to have effective CRF training
Karvonen formula/Heart Rate Reserve
1. Max HR - resting HR to obtain HRR
2. take 60%-80% of HRR
3. add each HRR to resting HR
ACSM guidelines for strength
2-3 sessions per week, 8-10 exercises of 8-12 reps at 60-80% 1RM
ACSM guidelines for flexibility
10-30sec, 2-4 times. 2 or more days a week
FITT
Frequency Intensity Time Type
ACSM guidelines for cardiorespiratory fitness
3-5 times a week, at 50-80% MHR, for 30-60 min
Recommended progression sequencing of physical activity
walk before running. start at a level that is comfortable for the client
Static stretching
Stretching without movement
Ballistic Stretching
Stretching with rough jerky movements
Dynamic stretching
Stretching through a rage of movement
proprioceptive neuromuscular facilitation (PNF)
Tensing a muscle then stretching it so that the golgi tendon helps relax the muscle for a better stretch
why is proper dosing of exercise important
So that the client receives the benefits of the exercise without being injured
Why is it important to increase a sedentary persons activity by small amounts
To avoid injury and because all of their base stats will be low
Why is important to monitor heart rate frequency during exercise in heat humidity and altitude
A clients heart could have to work harder in these conditions which could be too much for them if they are out of shape or elderly
What are the benefits of cardiovascular exercise
decrease the risk of cardiovascular disease
Ketosis
metabolic acidosis caused by an accumulation of ketone bodies due to a lack of insulin
COPD
Chronic obstructive pulmonary disease causes a reduction of airflow
Myocardial Infarction
Loss of ventricular muscle resulting in compromised ventricular function (usually on beta blockers) (st depression)
Arrhythmia
Irregular heart rhythms
Osteoporosis
Mow bone density most prominent in older women due to the loss of estrogen
Bulimia
an eating disorder associated with binging and puring
Anorexia Nervosa
an eating disorder associated with severely under consuming calories
Immumotherapy
Stimulating an immune response to get the body used to the thing that it is triggered by
Asthma
Chronic inflammation of the airways
Type 1 Diabetes
Diabetes that someone is born with
Type 2 Diabetes
Diabetes that is developed later in life
Insulin Shock
When too much insulin is produced or taken in which can put a person in a diabetic coma (hypoglycemia)
Amenorrhea
Absence of ones period
Dysnmenorrhea
Painful menstration
Valsalva maneuver
Test for COPD blow into a thing as hard as you can
Exercise differences for people with type one and type two diabetes
Type 1 exercise is not as beneficial as it is for those with type 2 diabetes but can reduce some complications associated with type 1 diabetes. Type 2 exercise helps to control blood sugar and is mostly based on aerobic exercise to use up some of the sugar in the blood
Why is it important to control a diabetic's glucose concentration during exercise?
they could easily become hypoglycemic or hyperglycemia during the session
In what scenario(s) should a type 1 diabetic avoid exercise?
if fasting glucose is >250mg/dl if <100mg/dl
What are the reasons a graded exercise test would be recommended for a client?
help in developing the proper exercise prescription
What is the difference between asthma and exercise-induced asthma?
asthma is chronic exercise induced asthma is caused by stimulus while exercising
Asthma drugs
inhaler, chromolyn sodium, beta receptor antagonsits (not too often), albuterol, steroids
What type of exercise(s) is/are most likely to precipitate exercise-induced asthma?
Running ( more strenuous long duration activities)
How can exercise-induced asthma be managed?
reduction in salt intake, increase in omega 3's, increase antioxidant intake
What factors will cause an asthma attack during exercise?
cold air, high intensity, drying of the respiratory trac
How does exercise influence COPD?
it can delay the onset of many of the symptoms
What are the blood pressure readings for a normal, pre-hypertensive, and stage 1 hypertension?
normal <120/<80 pre hypertensive 120-139/80-89 stage 1 hypertension 140/159
What are recommended exercise guidelines for reducing hypertension?
resistance training at 60-80% 1-RM for 30 min as many days as possible and 30-60 min of aerobic exercise at 50-80% vo2 max 3-5 times a week
What are lifestyle recommendations for reducing hypertension?
reduce sodium intake, reduce fat, limit alcohol, stop smoking, eat fruits and veggies
the different stages of cardiac rehabilitation.
phase 1: inpatient exercise program
Phase 2: stretching, exercise, cooldown (light activity)
Phase 3:
What effects would beta-blockers have on a person's physiological response to exercise?
lower heart rate during exercise
What would an exercise prescription for a person with osteoporosis entail?
Weight bearing exercise to increase bone density 3-5 times per week and resistance training 2-3 times per week (30-60 min a day)