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Cognition
The systematic way in which a person thinks, reasons, and uses language AND/OR:
The mental process of knowing, including aspects such as awareness, perception, reasoning, and judgment
Sensory Compensation
When one sense is lost, other senses often become heightened:
Example: patient is blind, but has great hearing
When patient is ill, they are sometimes unable to compensate
Sensory Reception
Process of receiving data of external and internal environment via the sense
Kinethetic
Awareness of body position and movement
Stereognosis
Sense that perceives solidity of objects and their size, shape, and texture; perceive and identify objects
Functional Sensory Input
Reception — Perception — Reaction
Register that something is hot — perceiving the pain — react by moving hand away from the burner
Reticular Activating System (RAS)
Organizer of incoming stimuli:
Poorly defined network — extends from hypothalamus to medulla
Mediates sensoristasis
Monitors and regulates incoming sensory stimuli
RAS Filtering
Muffling out unimportant/irrelevant stimuli to maintain focus;
determines what you should and should not be focusing on
React to same stimuli differently based on environment
Example: baby crying in public = sympathy, aww cute
VERSUS: baby crying at 3am = annoyed
Sensoristasis
Optimal arousal state that is regulated by RAS
Somnolent
Extreme drowsiness, but will respond normally to stimuli
Normal stimuli = knocking on the door, speaking to them, light touch — they will wake up
Stupor
(Unconscious) Can be aroused by extreme and/or repeated stimuli; unable to express how they are feeling and there could be airway obstruction/dysfunction
Extreme stimuli = raised voice, shaking them, bed movement, etc.
Sans stimuli = patient will return to state
Narcotics/certain drugs = this is the response to overdose
Coma
(Unconscious) Cannot be aroused and does not respond to stimuli
Stimuli = sternal rubbing, extreme stimuli to get ANY response
Concerns: BREATHING
Causes: medically induced, hypotension (poor circulation), extreme bodily trauma, electrolyte abnormalities
Needs immediate intervention — should be top priority
Normal
Alert, aware, and responsive — interacting and conscious
Sensory Deprivation
Reduction in or absence of usual and accustomed stimuli
Patients might develop this to normal hospital commotion and become anxious
Sleep = harder to sleep in hospital (shared space, noise, illness, etc.), causing altered cognition
Sensory Deprivation INTERVENTIONS
Help patient form a routine
Keep mind going, maintain stimulation
Frequent interactions during the day
Medication (sleeping aids, etc.)
If they were taking OTC drugs outside of hospital stay, try to replicate as best you can
Encourage movement
Family visitation/pet therapy
Keep them comfortable (letting them wear their own pajama pants, etc.)
Reorienting clocks
Sensory Overload
One or more of the senses are overloaded; overstimulation, overwhelmed, panicky
Signs/symptoms: agitation, racing thoughts, confusion
Causes: internal (pain, nausea, etc.) stimuli, external (roommate, hospital setting) stimuli, other (overuse of medical jargon)
Sensory Deficit
Impairment, or lack of senses
Impaired vision, hearing, taste, smell, or tactile perception
Can be reversible or permanent
Be sensitive to loss sense — ensure patient is able to understand you by taking proper interventions
Example: olfactory loss — unable to smell gas/smoke or when food has spoiled; taste can also be muffled
Sensory Processing Disorder
Difficulty in the way the brain receives sensory information, or the way the brain organizes and uses that sensory input
Leads to challenges interacting in the environment
Factors Impacting Sensory Function
Age = Gradual degradation
Culture
Personality and Lifestyle
Stress and Illness
Medication = side-effects can cause sense impairment or cognitive alteration
Presbyopia
Age-related vision degradation = a need for reading glasses
40 years and older
Cataracts
Clouding of the eye lens
Seen in 50% of patients over 80 years old
Glaucoma
Increased intraocular pressure — irreversible optic nerve damage
Retinopathy
Non-inflammatory damage to retina — may be caused by uncontrolled diabetes
Macular Degeneration
Loss of visual field in the center of the eye — strong peripheral vision
Presbycusis
Age-related hearing loss — 80 years and older
Could be due to cerumen buildup
Peripheral Neuropathy
Damage to peripheral nervous system — uncontrolled diabetes, shingles, and longterm alcohol usage/severe vitamin deficiencies (these two often go hand-in-hand)
NOT REVERSABLE
CVA - Stroke
Sensation, longterm deficients
Reversible OR permanent damage to the nerves
Face, drooping, swallowing, movement, etc.
Consciousness
Awareness and responsiveness; requires intact RAS
Attention
Required to acquire and express information; allows you to focus on SOME stimuli and ignore others
Memory
Allows you to retain SOME information and forget others;
Linked to the senses — more likely to remember if memory is connected to more senses
Learning
What we are attempting to learn, must be meaningful and linked to previous knowledge — build on concepts to gain more understanding, etc.
Communication
Verbal and non-verbal
Normal Cognition
Baseline is individual for each patient;
bCAM Assessment
Brief Confusion Assessment Method
Polypharmacy
May lead to drug interactions that cause cognitive alterations
Beers Criteria
List of medications that should not be used (or should be used with extreme caution) in adults over the age of 65
Could cause cognitive changes and dangerous affects
Expressive Aphasia (Broca’s Aphasia)
Inability to express words one wants to say — verbal or written
Limited speech; slow or takes great effort; reduced grammar, poor articulation
Person knows what they want to say but can’t find the words
Frustrating to the patient
Receptive Aphasia (Wernicke’s Aphasia)
Difficulty understanding verbal or written words
Impaired auditory comprehension and feedback
Dysarthria
Motor speech disorder related to stroke or brain injury
Speech is slurred or garbled, slow, soft
Delirium
Acute confusional state; typically sudden onset
REVERSABLE
Caused by: infections, metabolic disorders, chemical imbalance, etc.
Hyperactive Delirium = patient is VERY confused and active
Dementia
Generalized impairment of intellectual functioning; interferes with social and occupational functioning
Gradual onset that is progressive
IRREVERSIBLE
Alzheimer’s Disease, Diffuse Lewy Body Disease, Frontopemporal/Vascular, etc.
Anosognosia
Not aware of their illness or condition due to damage or changes to the brains frontal lobe (the part of the brain that is responsible for our self image)
Aphasia
Difficulty with or complete loss of all aspects of language caused by damage to the left side of the brain (the part of the brain responsible for language)
Agnosia Definition
Loss of recognition and inability to process sensory information
Apraxia Definition
The loss of purposeful or skilled physical movement even if you understand the instructions
Amnesia Definition
Loss of memory and/or the ability to form new memories
Altered Percpetion
Loss of visual perception and the inability to interpret sensory information
Apathy
Individual has a loss of interest or concern for everyday things, for example a lack of personal hygiene
7 A’s of Dementia
Progressive symptoms of dementia:
Anosognosia
Aphasia
Agnosia
Apraxia
Amnesia
Altered Perception
Apathy
Agnosia Types
Auditory: inability to recognize or differentiate between sounds, like you aren’t able to recognize familiar voices
Tactile: inability to recognize or identify objects by touch
Visual: inability to recognize objects or individuals when seeing them
Apraxia Types
Buccofacial: unable to carry out facial movements on command
Limb (kinetic): inability to make precise movements with an arm or leg
Ideomotor: inability to make appropriate movements in response to verbal commands
Ideational: inability to make multiple sequential movement, for example getting yourself dressed
Verbal: difficulty coordinating mouth movements
Constructional: inability to draw or construct simple figures
Oculomotor: difficulty moving eyes on command
Amnesia Types
Anterograde: inability to form new memories
Retrograde: loss of previously made memories
Transient global amnesia: fluctuating confusion or agitation over the course of several hours
Depression
Mental decline that is very rapid where individuals may struggle to do things for themselves and will often go into social isolation
Reversible condition
May be mistaken for dementia