RCP 370 Ch. 7 Assessment of the Cardiovascular System

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29 Terms

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P wave

Depolarization of the atria

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QRS complex

Depolarization of the ventricles

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T wave

ventricular repolarization

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how to calculate regular rhythm

- Count the number of large boxes on the ECG strip between two QRS complexes. Then 300 divided by large boxes (300/boxes).

- Count the wave and time by 10

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Regular heart rhythm

300

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Assessing HR boxes

- each small box is 0.2

- 3 sec = 15 boxes

- 6 sec = 30 boxes

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Sinus Bradycardia

- <60 bpm

- Common causes: weakened or damaged SA node, hypoxemia, increased ICPs, OSA.

- Severe cases: decreased CO, BP, and tissue hypoxia.

- poor capillary refill, cold, clammy and depressed sensorium.

- Treatment: Atropine and oxygen

<p>- &lt;60 bpm</p><p>- Common causes: weakened or damaged SA node, hypoxemia, increased ICPs, OSA.</p><p>- Severe cases: decreased CO, BP, and tissue hypoxia.</p><p>- poor capillary refill, cold, clammy and depressed sensorium. </p><p>- Treatment: Atropine and oxygen</p>
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Sinus Tachycardia

- >100 bpm

- Common causes: severe anemia, hyperthermia, hemorrhage, pain, fever, anxiety, sympathomimetic or parasympathetic Drug Administration.

- Treatment: treat underlying cause, hypoxemia = administer oxygen therapy.

<p>- &gt;100 bpm</p><p>- Common causes: severe anemia, hyperthermia, hemorrhage, pain, fever, anxiety, sympathomimetic or parasympathetic Drug Administration.</p><p>- Treatment: treat underlying cause, hypoxemia = administer oxygen therapy.</p>
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Atrial Flutter

Normal P wave are absent

and replaced by two or more

regular saw-tooth waves,

atria fires rapidly.

- Normal QRS complex

- Atrial rate is usually constant,

250-350 bpm

- Ventricular rate is in the

normal range

- Causes: hypoxemia, damage

to essay node and congestive

heart failure

- Treatment: Digoxin, beta

blockers, calcium channel

blockers.

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Atrial Fibrillation

- Atrial contraction are disorganized (quivering).

- No visible P wave

- Atrial rate ranges from 350-700 bpm

- Atria is clot

- Causes: hypoxemia, damage to SA node also seen in OSA.

- Treatment: digoxin, beta blockers, calcium channel blockers, anticoagulants and thrombolytics.

<p>- Atrial contraction are disorganized (quivering).</p><p>- No visible P wave</p><p>- Atrial rate ranges from 350-700 bpm</p><p>- Atria is clot</p><p>- Causes: hypoxemia, damage to SA node also seen in OSA.</p><p>- Treatment: digoxin, beta blockers, calcium channel blockers, anticoagulants and thrombolytics.</p>
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Premature Ventricular Contraction (PVC)

- The QRS complex is wide, bizarre, and unlike the normal QRS complex Is not preceded by a P wave

- The regular heart rate is altered by a PVC. May be very irregular when there are many PVCs

- Common causes – Myocardial disease, hypoxemia, acidemia, hypokalemia, CHF. Also noted during theophylline, alpha stimulant and beta agonist toxicity.

- Treatment: oxygen, lidocaine

<p>- The QRS complex is wide, bizarre, and unlike the normal QRS complex Is not preceded by a P wave</p><p>- The regular heart rate is altered by a PVC. May be very irregular when there are many PVCs</p><p>- Common causes – Myocardial disease, hypoxemia, acidemia, hypokalemia, CHF. Also noted during theophylline, alpha stimulant and beta agonist toxicity. </p><p>- Treatment: oxygen, lidocaine</p>
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Bigeminal PVC

PVC after a normal heartbeat

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Trigeminal PVC

PVC after q 2 heartbeats

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Ventricular tachycardia (V-tach)

- P wave is not seen

- QRS complex = wide and bizarre

- Ventricular rate ranges from 150 to 250 bpm

- Blood pressure & LOC is often decreased during ventricular tachycardia -> medical emergency

- With pulse -> cardioversion, lidocaine, amiodarone

- Without a pulse -> defib, compressions, epinephrine & amiodarone

<p>- P wave is not seen</p><p>- QRS complex = wide and bizarre</p><p>- Ventricular rate ranges from 150 to 250 bpm</p><p>- Blood pressure &amp; LOC is often decreased during ventricular tachycardia -&gt; medical emergency</p><p>- With pulse -&gt; cardioversion, lidocaine, amiodarone</p><p>- Without a pulse -&gt; defib, compressions, epinephrine &amp; amiodarone</p>
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Ventricular Fibrillation

- Chaotic electrical activity and cardiac activity

- Ventricles quiver

- There is no perfusion beat-producing rhythm

- The is no cardiac output or blood pressure.

- The patient will die in minutes without treatment – CODE

BLUE

- Treatment: Defibrillation and CPR until ROSC

<p>- Chaotic electrical activity and cardiac activity</p><p>- Ventricles quiver</p><p>- There is no perfusion beat-producing rhythm</p><p>- The is no cardiac output or blood pressure.</p><p>- The patient will die in minutes without treatment – CODE</p><p>BLUE</p><p>- Treatment: Defibrillation and CPR until ROSC</p>
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Asystole (Cardiac Standstill)

- The complete absence of electrical and mechanical activity

- Cardiac output stops, and the blood pressure falls to zero.

- The ECG tracing appears as a flat line

- Treatment: CPR and ACLS

<p>- The complete absence of electrical and mechanical activity</p><p>- Cardiac output stops, and the blood pressure falls to zero. </p><p>- The ECG tracing appears as a flat line</p><p>- Treatment: CPR and ACLS</p>
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Most Common Causes Of Cardiac Arrest

- Ventricular fibrillation

- Asystole

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Hemodynamics

The forces that influence the circulation of blood

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Invasive Cardiovascular Monitoring Assessments

- Invasive monitoring = assessment and treatment of critically ill patients.

(1) intracardiac pressures and flows via a pulmonary artery catheter

(2) arterial pressure via an arterial catheter (systemic) (3) central venous pressure via a central venous catheter

(4) coronary artery pathology (e.g., the use of the procedure coronary angiography

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Systemic Arterial Catheter

The most commonly used mode of invasive hemodynamic monitoring

• Referred to as an a-line

• More accurate than cuff pressures

• Measures:

- Continuous systolic, diastolic, and mean arterial blood pressure

Fluctuations in blood pressure

- Data for guidance of therapy decisions for hypotension or hypertension ABG blood draws

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CVP

2-6 mmHg

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PAP (pulmonary artery pressure)

- 15-35/5-15

- 25/10

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PCWP (pulmonary capillary wedge pressure)

4-12 mmHg

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Cardiac Output (CO)

4-8 L/min

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Stroke Volume

40-80 mL

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Stroke volume index

40 ± mL/beat/m2

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Cardiac Index

3.0 ± 0.5L/min/m2

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Pulmonary vascular resistance

50-150 dynes × sec × cm-5

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Systemic vascular

resistance

800-1500 dynes × sec × cm−5