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breast feeding definition
process of feeding mother’s breast milk to her infant either directly from breast or expressing milk and bottle feeding it to the infant
timeline for milk production- hormones
progesterone
estrogen
prolactin
oxytocin
timeline for milk production- placenta delivery
event triggers release of prolactin and oxytocin
prolactin stimulates milk production
oxytocin triggers milk ejection
timeline for milk production- mammary development
in pregnancy, the mammary glands grow and mature
estrogen and progesterone affect this
timeline for milk production- breast stimulation
infant sucking stimulates the anterior pituitary gland, releases oxytocin and prolactin
milk is produced and ejection occurs
phases of breast milk
colostrum
transitional milk
mature milk
→ fore milk
→ hind milk
colstrum
develops during pregnancy and last days after pregnancy
yellow, thick, small amount
nutrient dense: low fat, high protein, many vitamins and antibodies
helps prevent jaundice
transition milk
produced after colostrum milk until around 2 weeks postpartum
quantity increases, changes in appearance
contains more fat, sugar, vitamins, calories
mature milk
thinner and waterier
fore milk- beginning of feeding, more water and lactose
hind milk- end of feeding, higher fat content
golden hour
baby is alert and ready to fed within 1-2 hours after birth
post delivery recover skin to skin contact on mother’s chest
baby will root and seek breast
recovery sleep
early hunger cues
mouth open, licking lips, rooting
mid hunger cues
eating hands, more active and wiggling
late hunger cues
crying, agitated
newborn nutrition needs
100-110 kcal/kg/day
feed on demand, about every 2-3 hours
advantages of breastfeeding
immunologic properties
easy digestions
content varies per need
helps with attachement
reduces risk of SIDS
Free
better neurodevelopment
disadvantages of breastfeeding
medications pass through breastmilk
HIV transmission
limits partner involement
employment and childcare
maybe stressful and painfull
signs of successful breasfeeding
nursing at least 8 times in 24 hrs
adequate latch
6-8 wet diapers daily
3+ stools daily
infant weight gain
baby is satisfied after a feeding
mastitis
inflammation of mammary gland, infection related to impaired skin intergrity and milk stasis
flu like symptoms, and red tender breasts, hot to the touch, treat with NSAIDs and antibiotcs
engorgement
distention and swelling of breast tissue, breast become hard and tender
may occur during let down or ineffective emptying of breast milk
treat using massage, hand expression, heat/cold packs, NSAIDs
signs of infant dehydration
depressed fontanelles
rapid pulse and respirations
low grade temp
few wet diapers, dark urine
prolonged capillary refill
dry skin/mucosa
sunken eyes
lethargy
preparing breast milk
can be given at room temp
don’t use microwave
feed semi upright and burp regularly
discard leftover after 1 hr
suppressing lactation
absence of stimulation eventually decreases supply, most women experience engorgment, leakage, and discomfort before lactation ceases
snug bra 24 hr
avoid breast stimulation
do not restrict fluid intake
reduce salt intake
take mild analgesics to reduce discomfort
use cool compress or ice packs
make take 5-7 days
APGAR score
A-appearance (color)
P- pulse (heart rate)
G- grimance (reflex irritability)
A- activity (muscle tone)
R- respirations (respiratory effort)
vital signs newborn
temp- 97.7 - 99.5
HR- 110-160 murmurs
RR- 30-60 bpm
BP- 50-70 systolic, 30-45 diastolic
immediate newborn care
clear airway
thermoregulation
vitamin K
erythomycin
Hep B
first feeding
parent newborn attachment
newborn distress
retractions
nasal flaring
grunting
facial grimacing
cyanosis
abdominal distension
vomiting
jitteriness (glucose less than 40)
neonatal transition
increase cardiac output and contractility
surfactant release and promotion of pulmonary fluid clearance
thermoregulation and cold stress
reflex breathing triggers
low blood oxygen levels
increase blood carbon dioxide levels
low blood pH
temperature changes
drying
newborn respiratory
normal- 30-60 bpm
periodic breathing lasting 5-15 seconds
abnormal- apnea lasting more than 20 seconds
fetal cardiac shunts
ductus venosus
foramen ovale
ductus arteriosus
neonate/infant heat loss
convection- exposure to cool air
radiation- exposure to cool objects near infant
evaporation- wet body surface
conduction- cold table surface, scale, hands
infant cold stress
lethargy, hypotonia, can lead to apnea and respiratory distress
infant face/eyes/ears
symmetry is important
eyelids are edematous during first few days of life
mouth should be pink
unconjugated (indirect) bilirubin
breakdown of hemoglobin from RBCs, fat soluble
not an excretable form and a potential toxin
conjugated (direct) bilirubin
converted version of yellow indirect bilirubin, water soluble pigment
excreted by kidneys and in feces
jaundice
benign, normal variation
intervention is to increase number of breastfeedings, warmth, phototherapy, strict I&Os
hyperbilirubin
untreated can cause kernicterus (brain damage)
interventions- monitor VS and I&Os, phototherapy with eyemask
* extremely high levels transfer to NICU for transfusion
infant urination
within first 24hr
6-8 wet diapers a day
meconium stool
first stool, black tarry
causes of high risk newborns
maternal age (>18, <40)
maternal obesity
low socialeconomic status
lack of prenatal care
smoking
substance use
maternal disease
psychological stress
level 1 newborn
well newborn nursery, healthy term newborns, basic stabilization
level 2 newborn
special care nursery, late preterm or midly ill infants
level 3 newborn
NICU, premature/critically il, advanced technology and specialist
level 4 newborn
regional NICU, highest acuity, surgical care and sub specialities on site
NICU team
neonatologist
neonatal nurse practioner
pharmacist
OT/PT/Speech
neonatal nurse
Respiratory therapist
dietian/lactation
social worker/case manager
Training NICU nurse
unit orientation
neonatal resuscitation program
S.T.A.B.L.E (suagr, temperature, airway, blood pressure, lab work)
NICU - emergencies
1- anticipate
2- assign roles
3- check equipment
4- simulate and drill
5- debrief
micro preemie
<26 weeks
preterm
<37 weeks
late preterm
34-36 6/7 weeks
term baby
38-40 weeks
post term
>42 weeks
birth weight AGA
10th - 90th percentile
2,500 - 4,000 g
birth weight LGA
> 90 percentile
>4,000 g
birth weight SGA
< 10th percentile
<2,500 g
high risk condition- respiratory
transient tachypnea of the newborn
respiratory distress syndrome - surfactant deficiency
meconium aspiration syndrom
bronchopulmonary dysplasia- chronic lung disease
high risk condition- GI
necrotizing enterocolitis - bowel inflammation/necrosis (emergency)
high risk condition- neurological
intraventricular hemorrhage (IVH) - bleeding in the brain (preterm)
high risk conditions- other
hyperbilirubinemia - elevated biliribin, phototherapy
hypoglycemia- infant of diabetic mother, small of gestational age
sepsis
apnea of prematurity
transient tachypnea of the newborn
cause- retained lung fluid, common after C section or LGA
signs- tachypnea, grunting, mild retractions soon after birth
management- O2, resolves in 24-72 hrs
respiratory distress syndrome
cause- surfactant deficiency in premature lungs
signs- grunting, nasal flaring, retractions, cyanosis
management- surfactant, CPAP, O2, supportive care
Meconium aspiration syndrome
cause- meconium aspirated into airways, post term infants
signs- respiratory distress, barrel chest, stained skin/nails
management- airway clearence, oxygen/ventilation, antibiotics
bronchopulmonary dysplasia
cause- chronic lung injury from prolonged ventilation/oxygen trauma
signs- ongoing oxygen need beyond 28 days, retractions, poor growth
management- gentle ventilation, nutrition, diuretics, slow weaning
intraventricular hemorrhage
cause- bleeding in fragile germinal matrix vessels, risk rises with prematurity
signs- apnea, lethargy, bulging fontanelle, falling hematocrit
management- supportive care, minimize handling, serial head ultrasounds
hypoxic ischemic encephalopathy
cause- brain injury from hypoxia/ischemia, often birth related
signs- altered tone, seizures, poor feeding, depressed reflexes
management- theraputic hypothermia, seizure and supportive care
hydrocephalus
cause- excess cerebrospinal fluid accumlation increasing intracranial pressure
signs- rapid head growth, bulging fontanelle, sunset eyes
management- serial imaging, ventriculoperitoneal shunt if needed
necrotizing enterocolitis
cause- intestinal inflammation/necrosis- true neonatal emergency
signs- distended abdomen, bloody stools, temp instability, feeding intolerance
management- stop feedings, nasogastric decompression, antibiotics, possible surgery
feeding intolerance
cause- immanture motility and digestion, common in premature infants
signs- large residuals, abdomial distension, emesis, poor weight gain
management- adjust volume/rate, trophic feeds, monitor, rule out necrotizing enterocolotis
infant thermoregulation
neutral thermal environment
incubator for preterm infants
warm hands, hats, blankets to reduce heat loss
infant positioning
neutral head position, midling alignment
flexed limbs with boundaries/nesting
promotes normal musculoskeletal developmental
infant family centered care
open visitation
included in rounds
breastfeeding/pumping
parental support
skin to skin care
infant stress cues
color changes- mottling, pallor, dusky
gaze aversion
hiccupping/yawing
splayed fingers/arching
response: pause care, dim lights, reduce noise, cluster care
infant care discharge planning
begin early
teach the family
CPR training
follow up
high risk pregnancy factors
genetic/chromosomal conditions
cardiovascular distress
infection
diabetes
illicit drug use
smoking, alcohol, substance use
maternal obesity
age <15, >35
placenta previa
placenta implants in the lower uterine segment
painless, bright red vaginal bleeding
risk factors- advanced maternal age, multiple gestation, infertility treatment
management- bedrest, pelvic rest, no vaginal exams, monitor fetal heart tones, c section
placenta abruption
premature separation of a normally implanted placenta from the uterine wall
bleeding may be visible (external) or concealed (internal)
signs- sudden onset pain, rigid uterus, dark red bleeding, fetal distress
risk factors- ama, trauma, short umbilical cord, hypertension (most common)
* emergency
abortion
loss of pregnancy prior to 20 weeks
may be spontaneous (miscarriage) or elective
risk factors- genetics/chromosomal, uterine abnormalities, hormone imbalance
abortion classifications
threatened- bleeding, cervix closed, fetus viable
inevitable/imminent- cervix dilating, loss unavoidable
complete- all products of conception expelled
incomplete- partial expulsion, misoprostol/mifepristone, D&C may be needed
missed- fetal demise without expusion
recurrent pregnancy loss- 3+ consecutive pregnancy lossess
septic- infection of uterine contents, emergeny, life threatening
ectopic pregnancy
implantation outside the uterine cavity (fallopian tube, ovary, peritoneal cavity, or cervix)
interventions- methotrexate stops cell growth and allows the body to to absorb the pregnancy tissue, RhoGAM if Rh(-) for future pregnancies
stillbirth
fetal death after 20 weeks of gestation, 1 in 160 pregnancies
cervical insufficiency
premature, painless dilation of the cervix without labor or contractions
cord prolapse
umbilical cord falls through cervix ahead of the presenting part
emergency intervention- manually hold the presenting part off the cord while client is positioned in trendelenburg or knee chest. deliver via emergency c section
preterm labor
labor 20-37 weeks of gestation
#1 cause of neonatal morbility
bedrest, hydration, tocolytic therapy, corticosteroids, magnesium sulfate
tocolytic drugs
nifedipine
indomethacin- NSAID, <32 weeks only beause it may cause premature closure of the ductus arteriosus
atosiban- oxytocin antagonist
magnesium sulfate
CNS depressant, calcium antagonist- relaxes smooth muscles
mag toxicity signs- respiratory depression, confusion, cardiac arrest
antidote- calcium gluconate IV push
hypertension in pregnancy
chronic- pre exisiting before 20 weeks
gestational- onset after 20 weeks, no proteinuria
preeclampsia- HTN and proteinuria or organ dsyfunction, occurs after 20 weeks
eclampsia- preclampsia and seizures, life threatening emergency
preeclampsia
signs- severe headache, visual disturbances, decreased urine output
mild management- bedrest, low sodium. fetal monitoring
severe management- bedrest, anticonvulsants, antihypertensives, delivery is the only cure
eclampsia
preeclampsia and seizures
management- maintain airway, prevent injury, only cure is to delivery placenta
HELLP syndrome
HTN absent in 10-15% of cases
symptoms- N/V, flu like symptoms, epigastric pain, jaundice
commonly misdaignosed (gastroenteritis, hepatitis)
diabetes in pregnancy
pregnancy increases insulin demand for fetal growth, placenta (endocrine organ) causes insulin resistance
gestational (GDM): onset during pregnancy occurs in up to 10%
may resolve postpartum or progress into type 2
screening- 75g GTT between 24-28 weeks, >120 mg/dL → 3 hr test
fetal effects of diabetes in pregnancy
LGA
poorly controlled insulin dependent
congenital abnormalities
neonatal hypoglycemia
hyperbilirubin
polyhydroamnios
shoulder dystocia risk
respiratory distress
vulnerable populations- adolescent pregnancy
rates declining but still elevated in southern states and ethnic minorities
lack of contraceptive use or knowledge
listen more than you talk, build trust
assess self esteem, mental health, decision making skills
education on nutrition, prenatal care, parenting skills
vulnerable populations- substance use disorder pregnancy
no safe amount during pregnancy
torch infections - other
syphilis, gonorrhea, HIV, Hep B, varicella
group B strep is leading cause of neonatal sepsis
torch infections - rubella
pregnant women cannot receive MMR (live vaccine)
torch infections - cytomegalovirus
risks- mental impairment, hearing/vision loss
torch infections- herpes simplex (HSV)
c section if active outbreak at labor
acyclovir after 36 weeks to prevent outbreak
HIV in pregnancy
modern ART is highly effective, transmission risk is <1% with meds and formula feeding
c section indicated only if high viral load, above 1,000 copies/mL
start AZT or combo ART in1st trimester
newborn- AZT syrup for first 6 weeks of life
cardiovascular disease in pregnancy
increase risk- increased cardiac output in pregnancy strains compromised hearts
anemia reduces O2 delivery to fetus
hyperemesis gravidarum in pregnancy
severe N/V impacting hydration and nutrition (beyound usual morning sickness)