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Why pulse pressures would be low
bleeding, aoritc stenosis, HF
BASH
why pulse pressure would be high
AFAR
atherosclerosis, fever, aoritic regurgitation
which VS is the first to alter in deterioration
RR
movement of blood. Pulmonary circulation vs systemic
from the body it goes to the right atrium to right vent to pulmonary artery to lungs to pulmonary vein to left atrium to left vent to body
pulmonary circulation: movement of blood from the right heart to the lungs
systemic circulation: movement of blood from left heart out to body
VQ
Ventilation: movement of air to alveoli
Perfusion: blood to alveoli.
They need to be balance, 1.0
high vq= high ventilation, low perfusion (ex: PE stops blood flow)
low vq= low ventilation, high perfusion (ex: atelectasis)
labs for gas exchange
increased creatinine kinase and decreased potassium, decreased hgb, hct, rbc
malignant hyperthermia early signs and treatment
skeletal muscle rigidity, tachycardia
dantrolene + cooling
DIC
disseminated intravascular coagulation
a bunch of little clots occuring with urosepsis
DIC = death is coming
Uncomplicated UTI treatment
trimethoprion/sulfamethoxazone - after culture
Complicated UTI treatment
ciprofloxacin or nitrofurantoin - both fluoroquinolones (bactericidal antibiotics)
patient may also be prescribes bladder analgesics like phenazopyridine/pyridium - may turn urine orange - only use for a few days because it could mask worsening symptoms
nephrotoxic meds
metformin, contrast dye, lithium, cocaine, heroin, analgesics, diuretics
CM of nephrotoxicity
oliguria, dark urine, edema, hypertension, dry itchy skin, confusion
dialysis disequilibrium syndrome
rapid change in ECF composition leading to high osmotic gradient in brain leading to cerebral edema