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define chronic adaptations
are physiological changes which occur in the body as a result of training, specific to the type of training done. Thus, improving performance.
3 types of adaptations (technically 4)
Cardio-vascular (counts as 2)
Muscular
Respiratory
aerobic adaptations 3 sub-categories (technically 4)
cardio & vascular
respiratory
muscular
anaerobic adaptations 1 sub-category
muscular (and neural)
cardio aerobic adaptations (4)
Decreased Resting HR
Increased Stroke volume (SV)
Increased Max Cardiac output (Q)
Increased contractility of the heart
vascular aerobic adaptations (5)
Increased Hemoglobin
Increased capillarization
Increased Blood volume
Decreased Blood pressure
Increased blood supply to heart
respiratory aerobic adaptations (5)
Increased minute ventilation (RR & TV)
Increased TV
Increased V02 Max
Increased Pulmonary Diffusion
Increased ventilatory efficiency
muscular aerobic adaptations (8)
Increased triglyceride stores
Increased glycogen stores
Increased A-v02 Difference
Increased myoglobin content
Increased Mitochondria size & Number
Increased oxidative enzymes
Red fiber hypertrophy
Glycogen sparing
aerobic muscular acronym
F - fuel stores
A - a-v02 diff
M - myoglobin
M - mitochondria
O - oxidative enzymes
S - slow twitch hypertrophy
muscular anaerobic adaptations (8) / not neural
Hypertrophy
Increased contractile filaments
Increased glycolytic enzymes
Increased ATPase / phosphokinase
Increased PC stores
Increased ATP stores
Increased tolerance of H+ ions and buffering
Increased size of connective tissue (tendons)
muscular anaerobic adaptations (4) / neural
Increased motor unit recruitment
Increases rate of activation
Increased preferential recruitment of fast twitch fibers
Increased motor unit coordination
performance link for anerobic adaptations
ALL lead to faster and more forceful muscular contractions meaning you can jump higher, run faster, throw further etc.
define HR
is the number of beats per minute
what is happens to HR / type of adaptation
CARDIAC aerobic adaptation
Resting HR will decrease, due to the fact SV has increased (but resting 02 remains the same)
BUT MAX HR remains the same at 220 – age
what is SV
is the amount of blood pumped out of the left ventricle per beat
what happens to SV / type of adaptation (3)
CARDIAC aerobic adaptation
The left ventricle gets bigger and stronger due to aerobic training
This leads to increased SV
Allows more blood (and thus 02) to be pumped out per heartbeat
what happens to SV at submax intensity
plateaus at 65-70% HRM
what is cardiac output (Q)
the amount of blood pumped out of the left ventricle per minute
what happens to SV after training / type of adaptation
CARDIAC aerobic adaptation
maximal stroke volume gets bigger
relationship between Q, SV, and HR (3)
max SV increases but max HR is the same, still max cardiac output increases
this means more blood is pumped out of the heart per minute, which means more o2 can get to the muscles
meaning we can work aerobically at higher intensities
what is the equation of Q
Q = SV x HR
what happens to Q at rest after aerobic training
at rest Q will remain UNCHANGED, due to bigger stroke volume and lower heart rate
what happens to Q at submax after aerobic training
at sub max activities Q will remain UNCHANGED, due to bigger stroke volume, and lower working HR
what happens to Q at max after aerobic training
at max intensities Q will INCREASE, due to bigger Stroke volume, and max HR remains same
Explain capillarisation & type of adaptation (2)
VASCULAR aerobic adaptation
increased capillarisation to heart, lungs and skeletal muscles (especially slow twitch fibres)
more capillaries
thus increases blood and oxygen supply to muscles / diffusion
aerobic vascular adaptations (4) / type of adaptation
VASCULAR aerobic adaptation
increased capillarisation
Increased blood plasma / blood volume (more blood)
Increased Hemoglobin / blood carrying cell (more 02)
Decreases Systolic blood pressure (at rest) / heart not working as hard due to more effective transportation of blood (SV)
how does aerobic training influence blood pressure (3)
VASCULAR aerobic adaptation
BP at rest is decreased
BP at sub max levels decreased
BP at max levels is unchanged
what is systolic & diastolic BP
Systolic pressure – pressure on the arteries when the heart contractions and blood is pumped out of the heart.
Diastolic pressure – pressure in the arteries when the heart relaxes and the heart fills with blood.
what is ventilation & equation
is the total amount of air breathed in and out per minute.
RR x TV = Ve
what is respiratory rate
is number of breaths per minute.
changes in RR after training / type of adaptation (2)
RESPIRATORY aerobic adaptation
At rest / submax – lower RR
At maximal activity - same RR
what is tidal volume (TV)
total amount of air breathed in and out per breath.
changes in TV after training / type of adaptation
RESPIRATORY aerobic adaptation
increases due to increased strength of respiratory muscles thus you can take in more 02 per breath.
relationship between RR and TV after aerobic training
Despite RR staying the same, the increase in TV allows for overall increased Ventilation (as Ve = RR x TV)
2 changes that allow for increased pulmonary diffusion
an increase in size and surface area of alveoli
a greater alveolar-capillary interface, meaning more capillaries and alveoli are touching
what does increased pulmonary diffusion after training allow for
allows for more 02 to to move from the alveoli into the blood stream, thus allowing for increased efficiency and faster rate of aerobic ATP resynthesis.
what is happens to fuel stores after training / type of adaptation (3)
MUSCULAR aerobic adaptation
there is an increase in glycogen and triglycerides in muscles.
additionally an increase in PC and glycogen stores
More stores = more fuel to metabolise into ATP
what is happens to number & size of mitochondria after training / type of adaptation (2)
MUSCULAR aerobic adaptation
increased Number and size of mitochondria
So, the more sites for Aerobic ATP resynthesis = more Aerobic ATP resynthesis can occur!
what is ventilatory efficiency
after aerobic training the diaphragm and intercostals require less oxygen to help us breathe thus becoming more efficient.
as a result more 02 can go to muscles to produce aerobic ATP.
what is / does a mitochondria do
are the site of aerobic ATP resynthesis
what is happens to no. of myoglobin after training / type of adaptation (3)
MUSCULAR aerobic adaptation
increased amount of myoglobin in the muscles.
which attracts 02 from bloodstream, into the muscle then to the mitochondria
thus, faster rate and greater amount of aerobic ATP resynthesis
what is a-V02 difference & what does it represent
is the difference of oxygen concentration of blood in the arteries compared to the veins
it represents the amount of oxygen being extracted by the muscles
what is happens to a-V02 difference after training / type of adaptation
MUSCULAR aerobic adaptation
a-v02 difference increases with aerobic training, meaning more oxygen is being extracted by the muscles.
what is a-V02 difference classified as
A-v02 difference can be muscular or vascular adaptations.
but we go with muscular.
a-v02 at rest
stays the same
a-v02 during exercise
increases
equation for a-V02 difference
02 concentration of blood in arteries - 02 concentration of blood in veins
what is happens to no. of enzymes after training / type of adaptation
MUSCULAR aerobic adaptation
they increase
what’s does an enzyme do / in particular an oxidative enzyme
speed up chemical reactions
Oxidative enzymes speed up any reactions involving 02 / and speed up the breakdown of glycogen and triglycerides to produce aerobic ATP faster.
what does an oxidative enzyme cause
an increased RATE of aerobic ATP resynthesis, thus can work at higher aerobic intensities.
what are anerobic enzymes / what they do (2 types given)
creatine kinase and glycolytic enzymes are anaerobic enzymes that break fuels down without O2, to produce ATP faster
what is glycogen sparing
an improved ability to oxidize a higher percentage of fat, therefore conserving their glycogen stores / anaerobic ATP production.
These stores can then be used later in the event such as a sprint to the finish line or an intense hill climb.
aerobic adaptations lead to
Increased V02 max
Increased LIP
define V02 max
maximum amount of 02 that can be uptaken, transported and utilized per minute
what is happens to muscle size after training / 2 factors / type of adaptation (2)
MUSCULAR anerobic adaptation
there is an increase in cross-sectional area is due to:
Increased size of muscle fibers (can’t grow more)
Increased contractile protein filaments (actin & myosin)
what training can bring about increased muscle size
Resistance training
what is happens to fuel stores / type of adaptation
MUSCULAR anaerobic adaptation
More stores of PC in the muscles means you can resynthesis ATP at the fastest rate for a longer period of time.
what is happens to anerobic enzymes after training / type of adaptation / the 3 types
they increase
MUSCULAR anaerobic adaptation
ATPase helps to break down ATP quicker
Creatine kinase breaks down PC faster
Glycolytic enzymes help to break down glycogen quicker.
All which allow for quicker rate of ATP resynthesis.
6 neural adaptations of resistance training
C.R.F.P
increased Co-ordination of motor units
increased Recruitment of motor units. (more neural transmissions) increased force of contraction
increased Firing rate of motor unit activation. (faster neural transmissions) increased speed of contraction
increased Preferential recruitment of fast twitch fibers.
what happens to recruitment of motor units / (more neural transmissions) / type of adpatation
NEURAL anaerobic adaptation
it increases
Steps of increased recruitment AND firing rate of motor units
Increased no. of motor units recruited = More force produced = BIGGER contractions = Lift more/throw further
OR
Increased firing rate = More FREQUENT contractions = run faster/ride faster
what happens to synronisation / co-ordination of motor units after training / type of adaptation
NEURAL anaerobic adaptation
training means different motor units can contract together = leading to greater force production
what happens to preferential recruitment of FT fibers / type of adaptation
NEURAL anaerobic adaptation
increases.
we usually recruit slow twitch fibers before fast twitch, but training allows us to recruit more fast twitch fibers early to create more explosive force.
what is lactate tolerance
when muscles continue to resynthesise ATP at a fast rate despite the presence of lactate (hydrogen ions)
what does lactate tolerance allow
allows people to be able to continue to work at high intensities (utilize anaerobic glycolysis) for longer periods of time.
what causes increased lactate tolerance
anaerobic training.
and buffers which reduce shock, and neutralize the acidity of the muscles to allow them to continue to contract
what does H+ do to glycolytic enzymes
H+ limits glycolytic enzymes and slows anaerobic glycolysis significantly
define pulmonary diffusion
the rate at which gases (carbon dioxide and oxygen) can diffuse in and out of the lungs and capillaries
process of pulmonary diffusion
alveoli / lungs have high 02 concentration — which goes into the capillaries to the heart (to muscles)
thus capillaries have high C02 concentration — goes into alveoli to be breathed out
what are other names for fast twitch fibers and characteristics
type 2 / white
fast contraction
more forceful
fatigue faster
what are other names for slow twitch fibers and characteristics
type 1 / red
slow contraction
less forceful
take longer to fatigue
2 measures of v02 max
absolute
relative
define relative v02 max
measures v02 max in ml per kg per minute (ml/kg/min)
define absolute v02 max
measures vo2 max in litres per minute (L/min)
what is the best measure of V02
a relative measure this is because it takes into consideration body weight and how effectively they body ususes the oxygen.
This allows for more accurate readings and more effective comparison against different body types.
FICHT formula
for v02 max
V02 Max = Q x A-v02 diff
lactate tolerance is… (2)
an anerobic adaptation (muscular)
ITS DIFFERENT FROM L.I.P
lip is…. (2)
an aerobic adaptation
ITS DIFFERENT FROM LACTATE TOLERENCE (anerobic)
LIP is FAD explained (LIP IS AEROBIC)
LIP is FAD / AEROBIC
F = FINAL exercise intensity where lactate production equals lactate removal (definition)
A = this means the athlete can work at higher AEROBIC intensity
D = this delays the increasing contribution from the anerobic glycolysis system therefore stopping the rapid accumulation of H+ ions… and the ability to work harder for longer (performance link)
what TWO, & ONLY TWO factors contribute to LIP (& glycogen sparing)
increased size and number of mitochondria
increased oxidative enzymes
how to answer a LIP & contributing factors question
Increased size and number of mitochondria (increased the sites where aerobic ATP production occurs)
Increased oxidative enzymes (speed up chemical reactions thus the production of aerobic ATP can occur more quickly)
All allowing for a greater LIP
All which contribute to faster rate of aerobic ATP production, allowing us to work at higher aerobic intensity
So we can run at aerobic intensity for longer, delay the contribution from anerobic ES.
what is the length of the event for glycogen sparing
2-3 hours.
roughly 90 minutes of glycogen so an event shorted you can just use glycogen as it wont run out
respiratory adaptations (answer link to…)
link to increased uptake of oxygen
cardiovascular adaptations (answer link to…)
transportation of blood and oxygen around the body
muscular adaptations (answer link to…)
utilization of oxygen via the active muscles
anaerobic neural muscular ancromym
CRFP
C – Coordination of motor units ↑
R – Recruitment of motor units ↑ (force)
F – Firing rate of motor units ↑ (speed)
P – Preferential recruitment of fast-twitch fibres ↑
anaerobic muscular ancromym
FLAG C
F – Fast-twitch hypertrophy
L – Lactate tolerance (H+ tolerance and buffering) ↑
A – ATP & PC stores ↑
G – Glycolytic enzymes ↑
C – Increased contractile filaments & connective tissue (more forceful contraction)