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Mannitol
a diuretic used to decrease ICP
Beta blockers
primarily used to decrease HR but also decreases BP (decrease afterload)
Atropine
used for symptomatic bradycardia to increase the HR
Amiodarone
an antiarrhythmic used for afib, ventricular arrhythmias
Epinephrine
vasoconstrictor used to increase BP
Norepinephrine
vasopressor used to treat hypotension, usually in septic shock
Nitroprusside
vasodilator used to treat hypertensive emergency to rapidly decrease bp (decrease afterload)
Clonidine
decrease bp/hr (decrease afterload)
CUS
Concerned, Uncomfortable, Safety concern 'I am concerned about this in pt A, I am uncomfortable, and this could be a safety issue.'
Feedback
Info provided to improve individual/overall team performance
Task assistance
team members foster a climate where assistance will be actively sought/offered
Asystole
s/sx: pulseless, pt is dead; Nursing care: run CPR/ACLS. This is not a shockable rhythm.
Vtach
s/sx: chest pain, SOB, palpitations, dizziness; WITH pulse: cardiovert; WITHOUT pulse: defibrillate
1st Degree Heart Block
PR interval is longer than 0.20 seconds. Monitor- usually no tx
2nd Degree Type 1 Heart Block
PR interval gets progressively longer until a QRS is dropped (p wave is not followed by qrs). Tx: treat underlying, pace, monitor
2nd Degree Type 2 Heart Block
Some p waves are not followed by QRS. More serious than type 1. Tx: O2, pace
3rd Degree Heart Block
P waves/QRS or atria/ventricles are independently operating. P waves will be 6-10, QRS will be 2-4/wide and wacky. This is life threatening and needs CPR/ACLS and pacing
AICD
electronic device placed in chest to monitor for irregular rhythms and shock accordingly
Pacemaker failures
Sense: spike is present but heart still does whatever; Capture: no QRS at all (spike is present); Fire: no spikes at all (ekg looks like underlying rhythm)
Cardiac Tamponade
s/sx: Beck's triad: hypotension, JVD, muffled heart sounds; Tachycardia; SOB; Chest pain
Preload
Volume in the heart at the end of diastole (filling); WAYS TO MEASURE: Central Venous Pressure (CVP) 2-8: measures RIGHT side; IF CVP IS HIGH: FLUID OVERLOAD- jvd, edema; IF CVP IS LOW: FLUID DEFICIT; Wedge: 4-15: Measures LEFT VENTRICLE preload; IF WEDGE IS HIGH: FLUID OVERLOAD- crackles, DOE; IF WEDGE IS LOW: FLUID DEFICIT; Treatment to decrease preload: use diuretics
Afterload
Resistance the heart must face in order to pump blood out to the body; SVR(left side) and PVR(right side) measures; HIGH SVR: too much resistance, give vasodilators like nitro/nitroprusside, -pril. Sx seen: bounding pulses/htn; LOW SVR: too dilated. Use vasopressors. Sx: may be shock- dec LOC
Contractility
'Squeeze': strength of contraction from heart; INC contractility: harder squeeze, blood gets further- bounding pulse; DEC contractility: soft squeeze, blood does not get far- thready pulse
Hypertensive EMERGENCY
>180/120; Organ damage!!!!!
Hypertensive URGENCY
>180/120
Stroke
No organ damage present
Asymptomatic
May be asymptomatic
CM
Severe headache, chest pain, N/V
Tx for Stroke
ICU stay- IV antihypertensive- nitroprusside, nitro, nicardipine
Follow-up Treatment
Outpatient, oral antihypertensives (clonidine)
Abdominal Aneurysm
Dilation of the vessel
Abdominal Aneurysm Symptoms
Often asymptomatic, abdominal pulsation
Abdominal Dissection
Burst of vessel
Classic signs of AAA
Sudden pain in abdomen, syncope, hypotension
Abdominal Dissection Treatment
Life threatening unless there is emergent repair
cardiogenic Shock
Heart suddenly cannot pump enough blood to meet the body's needs. Most commonly caused by severe MI
Shock Diagnosis
SBP of <90 for longer than 30 min, Inc wedge of >15, Decreased Cardiac Index <2.2
Shock Confirmation
Confirm w EKG, echo, cardiac enzymes, CXR (cardiomegaly)
Shock Treatment
Drug therapy: Inotropes (dobutamine, digoxin), vasopressors (norepi), vasodilators (nitroprusside/nitroglycerin)
Invasive Monitoring Equipment
Intra-aortic balloon pump + Impella Vascular Assist Device
Shock Procedures
PCI, CABG
Sepsis Risk Factors
SIRS, infection, trauma, major surgery, acute pancreatitis, burns
Sepsis Symptoms
SIRS criteria consists of low bp, fast hr/rr, and abnormal wbc. May also present w fever/chills, sweating, altered mental status
Sepsis Treatment
FLUID RESUSCITATION using NS or LR at least 30 ml/kg
Sepsis Antibiotics Administration
Adm abx asap after identifying possible sepsis. Ideally within 1 hr
Hypovolemic Shock Symptoms
Increased hr, decreased bp dizziness, oliguria, pallor, weak pulses
Hypovolemic Shock Treatment
Stop the loss and replace volume
Neurogenic Shock Symptoms
Decreased hr/bp, hypothermia, oliguria, decreased loc, all pressures decreased
Neurogenic Shock Treatment
Treat cause, maintain c-spine, restore vascular volume using vasopressors/fluids, optimize o2 delivery
Oxygenation Curve
High CADET: face RIGHT (right releases, meaning hgb lets go of o2 easily)
Normal Hypoxemia
80-100
Mild Hypoxemia
60-75
Moderate Hypoxemia
45-59
Severe Hypoxemia
<45
Intubation Criteria
Hypercapnia/hypoxia, failure to protect airway
Nursing Care for Intubated Patients
Aseptic practices, HOB at least 30 degrees, oral care frequently, turn pt frequently to prevent pressure sores/dvt
Tension Pneumothorax Symptoms
Increased pleural pressure, lung sounds absent on affected side, chest pain, air hunger
Tension Pneumothorax Treatment
Doctor decompresses trapped air then places a chest tube
Open Chest Injury
Penetrating chest wall injury that sucks air in/out.
Flail Chest
Two rib fractures in two or more places.
Acute Respiratory Distress Syndrome (ARDS)
Life threatening with severe inflammation and fluid buildup in alveoli.
ARDS Symptoms
Dyspnea, restlessness, cyanosis, accessory muscle use, increasing hypoxemia (O2 does not work - refractory).
P/F Ratio
A ratio of <250 indicates severe hypoxemia in ARDS.
Pulmonary Artery Wedge Pressure
A value of <17 indicates complications in ARDS.
Chest X-Ray in ARDS
Shows ground glass appearance.
Shunt Calculations
PaO2 (given) / FiO2.
Normal Shunt Value
Normal is 350-450.
FiO2 Calculation
Every 1 L is 3% added to 21% (room air).
Neuro Trauma Mechanisms
Acceleration, deceleration, and coup-contrecoup injuries.
Focal Brain Injury
Localized to one area.
Diffuse Brain Injury
Affects many areas of the brain, causing widespread damage.
Basil Skull Fracture Symptoms
Battle sign, raccoon eyes, rhinorrhea/otorrhea (CSF leakage).
General Skull Fracture Symptoms
Decreased LOC, deformity of skull, unequal pupils, abnormal posturing.
Diffuse Axonal Injury Symptoms
Severe headache, decreased LOC, meningeal signs: nuchal rigidity, photophobia.
Increased ICP Treatment
Mannitol (diuretic), increase HOB, cluster care.
Cushing's Triad
Hypertension, bradycardia, irregular respirations (Cheyne-Stokes).
Spinal Cord Injury Symptoms
Loss of movement/sensation lower than point of injury.
Autonomic Dysreflexia
Occurs in patients above T6, causing extreme headache and hypertension.
Abdominal Compartment Syndrome
Occurs when intra-abdominal pressure rises to a level that impairs organ functioning.
Hepatic Failure Symptoms
INR >1.5, encephalopathy with no history of cirrhosis <26 weeks.
GI Bleed Symptoms
Occult blood, hematemesis, hematochezia, melena, anemia, decreased BP/increased HR.
Acute Pancreatitis Symptoms
Severe abdominal pain, Cullen's sign, Grey Turner's sign, SIRS, nausea/vomiting, decreased LOC.
Appendicitis Symptoms
Fever, rebound tenderness, Rovsing's sign (palpate LLQ, pain in RLQ).
Palliative Care
An interdisciplinary approach to relieve suffering/improve quality of life.
DNR
Do not resuscitate; treat but do not continue if heart stops.
Death Definition
Total irreversible failure of cardiorespiratory system/irreversible loss of all brain function.