Critical Care Medications and Nursing Management

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86 Terms

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Mannitol

a diuretic used to decrease ICP

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Beta blockers

primarily used to decrease HR but also decreases BP (decrease afterload)

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Atropine

used for symptomatic bradycardia to increase the HR

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Amiodarone

an antiarrhythmic used for afib, ventricular arrhythmias

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Epinephrine

vasoconstrictor used to increase BP

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Norepinephrine

vasopressor used to treat hypotension, usually in septic shock

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Nitroprusside

vasodilator used to treat hypertensive emergency to rapidly decrease bp (decrease afterload)

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Clonidine

decrease bp/hr (decrease afterload)

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CUS

Concerned, Uncomfortable, Safety concern 'I am concerned about this in pt A, I am uncomfortable, and this could be a safety issue.'

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Feedback

Info provided to improve individual/overall team performance

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Task assistance

team members foster a climate where assistance will be actively sought/offered

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Asystole

s/sx: pulseless, pt is dead; Nursing care: run CPR/ACLS. This is not a shockable rhythm.

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Vtach

s/sx: chest pain, SOB, palpitations, dizziness; WITH pulse: cardiovert; WITHOUT pulse: defibrillate

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1st Degree Heart Block

PR interval is longer than 0.20 seconds. Monitor- usually no tx

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2nd Degree Type 1 Heart Block

PR interval gets progressively longer until a QRS is dropped (p wave is not followed by qrs). Tx: treat underlying, pace, monitor

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2nd Degree Type 2 Heart Block

Some p waves are not followed by QRS. More serious than type 1. Tx: O2, pace

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3rd Degree Heart Block

P waves/QRS or atria/ventricles are independently operating. P waves will be 6-10, QRS will be 2-4/wide and wacky. This is life threatening and needs CPR/ACLS and pacing

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AICD

electronic device placed in chest to monitor for irregular rhythms and shock accordingly

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Pacemaker failures

Sense: spike is present but heart still does whatever; Capture: no QRS at all (spike is present); Fire: no spikes at all (ekg looks like underlying rhythm)

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Cardiac Tamponade

s/sx: Beck's triad: hypotension, JVD, muffled heart sounds; Tachycardia; SOB; Chest pain

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Preload

Volume in the heart at the end of diastole (filling); WAYS TO MEASURE: Central Venous Pressure (CVP) 2-8: measures RIGHT side; IF CVP IS HIGH: FLUID OVERLOAD- jvd, edema; IF CVP IS LOW: FLUID DEFICIT; Wedge: 4-15: Measures LEFT VENTRICLE preload; IF WEDGE IS HIGH: FLUID OVERLOAD- crackles, DOE; IF WEDGE IS LOW: FLUID DEFICIT; Treatment to decrease preload: use diuretics

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Afterload

Resistance the heart must face in order to pump blood out to the body; SVR(left side) and PVR(right side) measures; HIGH SVR: too much resistance, give vasodilators like nitro/nitroprusside, -pril. Sx seen: bounding pulses/htn; LOW SVR: too dilated. Use vasopressors. Sx: may be shock- dec LOC

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Contractility

'Squeeze': strength of contraction from heart; INC contractility: harder squeeze, blood gets further- bounding pulse; DEC contractility: soft squeeze, blood does not get far- thready pulse

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Hypertensive EMERGENCY

>180/120; Organ damage!!!!!

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Hypertensive URGENCY

>180/120

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Stroke

No organ damage present

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Asymptomatic

May be asymptomatic

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CM

Severe headache, chest pain, N/V

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Tx for Stroke

ICU stay- IV antihypertensive- nitroprusside, nitro, nicardipine

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Follow-up Treatment

Outpatient, oral antihypertensives (clonidine)

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Abdominal Aneurysm

Dilation of the vessel

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Abdominal Aneurysm Symptoms

Often asymptomatic, abdominal pulsation

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Abdominal Dissection

Burst of vessel

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Classic signs of AAA

Sudden pain in abdomen, syncope, hypotension

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Abdominal Dissection Treatment

Life threatening unless there is emergent repair

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cardiogenic Shock

Heart suddenly cannot pump enough blood to meet the body's needs. Most commonly caused by severe MI

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Shock Diagnosis

SBP of <90 for longer than 30 min, Inc wedge of >15, Decreased Cardiac Index <2.2

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Shock Confirmation

Confirm w EKG, echo, cardiac enzymes, CXR (cardiomegaly)

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Shock Treatment

Drug therapy: Inotropes (dobutamine, digoxin), vasopressors (norepi), vasodilators (nitroprusside/nitroglycerin)

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Invasive Monitoring Equipment

Intra-aortic balloon pump + Impella Vascular Assist Device

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Shock Procedures

PCI, CABG

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Sepsis Risk Factors

SIRS, infection, trauma, major surgery, acute pancreatitis, burns

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Sepsis Symptoms

SIRS criteria consists of low bp, fast hr/rr, and abnormal wbc. May also present w fever/chills, sweating, altered mental status

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Sepsis Treatment

FLUID RESUSCITATION using NS or LR at least 30 ml/kg

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Sepsis Antibiotics Administration

Adm abx asap after identifying possible sepsis. Ideally within 1 hr

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Hypovolemic Shock Symptoms

Increased hr, decreased bp dizziness, oliguria, pallor, weak pulses

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Hypovolemic Shock Treatment

Stop the loss and replace volume

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Neurogenic Shock Symptoms

Decreased hr/bp, hypothermia, oliguria, decreased loc, all pressures decreased

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Neurogenic Shock Treatment

Treat cause, maintain c-spine, restore vascular volume using vasopressors/fluids, optimize o2 delivery

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Oxygenation Curve

High CADET: face RIGHT (right releases, meaning hgb lets go of o2 easily)

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Normal Hypoxemia

80-100

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Mild Hypoxemia

60-75

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Moderate Hypoxemia

45-59

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Severe Hypoxemia

<45

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Intubation Criteria

Hypercapnia/hypoxia, failure to protect airway

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Nursing Care for Intubated Patients

Aseptic practices, HOB at least 30 degrees, oral care frequently, turn pt frequently to prevent pressure sores/dvt

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Tension Pneumothorax Symptoms

Increased pleural pressure, lung sounds absent on affected side, chest pain, air hunger

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Tension Pneumothorax Treatment

Doctor decompresses trapped air then places a chest tube

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Open Chest Injury

Penetrating chest wall injury that sucks air in/out.

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Flail Chest

Two rib fractures in two or more places.

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Acute Respiratory Distress Syndrome (ARDS)

Life threatening with severe inflammation and fluid buildup in alveoli.

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ARDS Symptoms

Dyspnea, restlessness, cyanosis, accessory muscle use, increasing hypoxemia (O2 does not work - refractory).

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P/F Ratio

A ratio of <250 indicates severe hypoxemia in ARDS.

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Pulmonary Artery Wedge Pressure

A value of <17 indicates complications in ARDS.

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Chest X-Ray in ARDS

Shows ground glass appearance.

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Shunt Calculations

PaO2 (given) / FiO2.

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Normal Shunt Value

Normal is 350-450.

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FiO2 Calculation

Every 1 L is 3% added to 21% (room air).

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Neuro Trauma Mechanisms

Acceleration, deceleration, and coup-contrecoup injuries.

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Focal Brain Injury

Localized to one area.

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Diffuse Brain Injury

Affects many areas of the brain, causing widespread damage.

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Basil Skull Fracture Symptoms

Battle sign, raccoon eyes, rhinorrhea/otorrhea (CSF leakage).

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General Skull Fracture Symptoms

Decreased LOC, deformity of skull, unequal pupils, abnormal posturing.

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Diffuse Axonal Injury Symptoms

Severe headache, decreased LOC, meningeal signs: nuchal rigidity, photophobia.

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Increased ICP Treatment

Mannitol (diuretic), increase HOB, cluster care.

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Cushing's Triad

Hypertension, bradycardia, irregular respirations (Cheyne-Stokes).

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Spinal Cord Injury Symptoms

Loss of movement/sensation lower than point of injury.

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Autonomic Dysreflexia

Occurs in patients above T6, causing extreme headache and hypertension.

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Abdominal Compartment Syndrome

Occurs when intra-abdominal pressure rises to a level that impairs organ functioning.

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Hepatic Failure Symptoms

INR >1.5, encephalopathy with no history of cirrhosis <26 weeks.

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GI Bleed Symptoms

Occult blood, hematemesis, hematochezia, melena, anemia, decreased BP/increased HR.

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Acute Pancreatitis Symptoms

Severe abdominal pain, Cullen's sign, Grey Turner's sign, SIRS, nausea/vomiting, decreased LOC.

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Appendicitis Symptoms

Fever, rebound tenderness, Rovsing's sign (palpate LLQ, pain in RLQ).

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Palliative Care

An interdisciplinary approach to relieve suffering/improve quality of life.

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DNR

Do not resuscitate; treat but do not continue if heart stops.

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Death Definition

Total irreversible failure of cardiorespiratory system/irreversible loss of all brain function.