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acute
rapid and self-limiting
persistent
long term infection. continual production of pathogen
latent
enter and become dormant for a period of time
transforming
enter and induce rapid growth of the infected cell. can lead to cancer development
cytopathic viruses
cause visible damage to the host cellular tissue
non-cytopathic viruses
do not cause visible damage to the host tissue cellular tissue
m cells
sample intestinal contents to generate protective repsonses
ex: salmonella
viral meningitis
virus infection of the tissue surrounding the brian
viral encephalitis
virus infection of the brain
latent infection
life long infection with dormant states; evades by hiding and not producing infectious agent
chronic infection
long term infection with constant production of the infectious agent
targeted immunosuppression
suppresion of the responses that would lead to their survival
bystander effect
pathogen X turns off the immune response that kills pathogen X and pathogen Y. pathogen Y is able to grow
antigenic variation
antigens can change
antigenic shift
large sudden change in the antigen
antigen drift
gradual mutation of the virus
oncogene
if this is “on” rapid division of abnormal cell soccurs leading to cancer