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what is an ischaemic stroke
blood supply is blocked to a region of the brain
causes of an ischaemic stroke
thrombus or embolus
factors of a thrombus forming
carotid dissection, heart damage, heart conditions, large artery disease
process of an ischaemic stroke
blood clot forms, moves to L MCA, blocks glucose and oxygen supply, causes cell damage which releases DAMPS causing inflammation, increasing intracranial pressure and causing further damage to surrounding cells
how ischaemic stroke is diagnosed
clinical assessment and scans (CT and MRI)
how will an ischaemic stroke appear on scans
hypodense (black)
how does a haemorrhagic stroke appear on scans
hyperdense (white)
what is the middle of the ischaemic stroke, a cluster of necrotic cells
ischaemic core
what is the outer region of the stroke, cells that may survive
penumbra
what is a PACS
partial anterior circulation stroke, affecting 2/3 categories
what are the 3 categories of anterior stroke impairments
sensorimotor, visual and higher cortical dysfunction
what sensorimotor impairments are associated with L MCAS
right hemiparesis and paraesthesia, mostly in face, arm and hand
what visual impairments are associated with LMCAS
right homonymous hemianopia
what higher cortical dysfunction happens with LMCAS
aphasia and motor praxia
what is aphasia
inability to form language
receptive aphasia
Wernicke’s area is impaired causing a word salad
expressive aphasia
Broca’s area is impaired causing broken speech
what is motor apraxia
difficulty planning and initiating movements
what general impairment in higher cortical dysfunction is associated with LMCAS
issues with language, planning and processing
treatment of ischaemic stroke
thrombectomy, thrombolysis, thrombolystics and anti-coagulants
clinical course of ischaemic stroke
acute, sudden onset with lng healing time and likely becoming chronic
stages of stroke healing
acute (3 weeks), sub-acute (3 weeks to 6 months), chronic (>6 months)
types of stroke recovery
spontaneous biological recovery, behaviour restoration/true recovery, compensation/substitution
spontaneous biological recovery
period of heightened recovery of behaviours early after stroke
behaviour restoration/true recovery
return towards normal patterns of motor control with neural repair
compensation/substitution
learning new approaches compared to pre-stroke behaviour
trunk control
maintaining COM over BOS for functional movements and postural adjustments
movements required for reach, grasp and manipulation
trunk and scapula stability, shoulder flexion, elbow extension, hand dexterity
possible impairments of reach, grasp and manipulation
trunk and scapula fractionation, shoulder pain, shoulder and elbow fractionation, long finger flexor spasticity, loss of wrist and finger fractionation, loss of sensation, loss of ROM of IP joints/wrist extension
what is a synergy
muscles in a limb move all joints in the same direction
what synergy is common in the UL
flexor
what synergy is common in the lower limb
extensor
flexor synergy in the UL
shoulder elevation and retraction
GHJ abduction and external rotation
elbow flexion and supination
wrist flexion
finger and thumb flexion and adduction
general fractionation assessment
initiate movement
movement in synergy
small single joint out of synergy
2 joint control
distal single joint control
3 joint contro
fractionation assessment of upper limb
movement in arm
hand to face then opposite knee
isolated elbow flexion
pronation/supination in 90º elbow flexion/ 90º shoulder flexion and elbow extension
thumb extension/abduction, finger extension
shoulder flexion, elbow extension, wrist/finger/thumb movement
specific upper limb motor treatment
closed chain shoulder adduction, abduction and flexion
add on elbow movements, then wrist
kinaesthesia pathway
dorsal column system
what is kinaesthesia
joint position and movement sense
how to assess kinaesthesia
move the joint and ask whena nd which way
discriminative touch pathway
dorsal column system
what is discriminative touch
light touch
assessing discriminative touch
light touch with a tissue, ask wehen and where
nociceptive touch pathway
spinothalamic tract
assessing nociceptive touch
prick with a sharp object, ask where and when
types of sensation treatment
part of motor treatment, electrical stimulation, sensory input
how is sensory treatment part of motor treatment
increasing sensory awareness and stimulation
how to perform sensory input as sensory treatment
vary the stimulus type and area stimulated to engage the person’s awareness
physiotherapist’s role in stroke rehab
rehab motor and sensory function, relearn functional movements, return to ADLs and PADLs, continuing strengthening and balance training, education, assess functional
short term goals
independent ambulation and PADLs
medium term goals
stairs, return to work, gardening, swimming
long term goals
lifeguarding