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5 Ps
passenger, passageway, parts, powers, psyche
passenger
fetal head, fetal attitude, fetal lie, fetal presentation
fetal head elements that allow movement thru birth canal
fontanelles - have 2 (anterior and posterior - posterior closes after 1-2 mo, anterior is later), gaps “soft spots” between skull bones
sutures - fibrous flexible joints connecting skull plates. form to bone by adulthood
fetal attitude
relation of fetal parts to each other. can be vertex (full flexion), sinciput (moderate flexion), brow (partial extension), face (complete extension)
fetal lie
relationship of longitudinal axis of fetus to longitudinal axis of mother
longitudinal = baby’s spine parallel to mother. can be cephalic (head first) or breech (feet first)
transverse = baby’s spine perpendicular to mother, requires cesarean
oblique = diagonal, will change before labor
fetal presentation
fetal body part presenting to maternal pelvis
vertex (head first), breech (feet first), or shoulder first
parts - station
location of baby’s head in relation to invisible line drawn between two ischial spines of pelvis, -4 to +4. negative numbers = baby’s head above station, 0 = at station, positive numbers = baby’s head below station
parts - position
position of fetus relative to anterior, posterior or sides of maternal pelvis
first letter - which side of maternal pelvis is the presenting part on? (L or R)
second letter - which fetal part is presenting? O = occiput, M = mentum, S = sacrum
third letter - orientation of presenting part relative to maternal pelvis anterior (A), posterior (P) or transverse (T)
OA = yay (preferred birthing position); OP = ouch (more difficult/painful birth)
powers - cervix
effacement (thinning of cervical walls) and dilation (enlargement of cervical canal to ~10cm
described by 3 numbers - % effaced, cm dilated, and station
electronic fetal monitoring (EFM)
provides a continuous tracing of fetal heart rate (FHR) allowing its characteristics to observed and evaluated
objectives of EFM
prevent fetal injury that might result from disruption of normal fetal oxygenation during labor
recognize specific features of the FHR that suggest a normal acid/base balance, and identify patterns that are indicative of impending or existing fetal acidemia so that appropriate interventions may be initiated
benefits of EFM
evaluate fetal well-being
useful in diagnostic testing
help evaluation labor progress
risks of EFM
supine postural hypotension
intrauterine perforation and infection (with internal monitoring)
decreased personal interaction (attention paid to machine)
mother unable to ambulate or change position
influences on FHR - utero-placental unit
at term, 10-15% of maternal CO (500-750 mL/min) perfuses the uterus each minute
oxygenated blood from mother is delivered to intervillous space (where maternal-fetal exchange of O2, CO2, nutrients, waste products, and water occur) in placenta via uterine arteries
influences on FHR - oxygenation to fetus
dependent on:
adequate oxygenation of mother
adequate blood flow to placenta
adequate uteroplacental circulation
adequate umbilical circulation (umbilical cord has 2 arteries which carry deoxygenated blood from fetus to mother and 1 vein which carries oxygenated blood to fetus)
fetus’s own innate ability to initiate compensatory mechanisms to regulate FHR
influences on FHR - parasympathetic stimulation
vagal nerve stimulation slows FHR and helps maintain variability (which develops at 28-30 wk gestation)
influences on FHR - sympathetic stimulation
increases FHR (occurs thru release of norepinephrine. may be stimulated during hypoxemia
influences on FHR - fetal mechanisms
CNS, baroreceptors, chemoreceptors, hormonal regulation
fetal/placental reserve
the reserve of O2 available to the fetus to withstand the transient changes in blood flow and oxygen during labor
auscultation of FHR
performed with fetoscope or doppler; determine fetal presentation/position using Leopold’s maneuvers
listen before, during and after a contraction
guidelines vary for labor stage 1/2 and low/high risk pregnancies
category I FHR
normal. strongly predictive of well-oxygenated, nonacidodic fetus with normal acid-base balance
category II FHR
indeterminate. not predictive of abnormal acid=base status, but not I or III. require evaluation and continued monitoring
category III FHR
predictive of abnormal fetal acid-base status and requires prompt evaluation and
FHR pattern interpretation
FHR baseline - 110-160 bpm, what is the variability?
periodic (with contractions) and episodic (without/between cchanges - accelerations and decelerations.
baseline FHR (how to calculate)
mean FHR rounded to increments of 5bpm during a 10 minute window, excluding accelerations, decelerations or marked variability
baseline variabiliy
fluctuations in baseline FHR that are irregular in amplitude and frequency
most important predictor of fetal oxygenation during labor; presence shows interaction between sympathetic/paraympathetic NS and shows that fetus is not in metabolic acidosis
absent - amplitude undetectable
FHR accelerations
visually apparent abrupt increases lasting less than 30 seconds above baseline FHR
15 beats above baseline for 15 sec (or 10×10 for fetus <32 weeks)
predictive of adequate fetal oxygenation and absence of fetal acidemia, often
FHR decelerations
transitory decreases in FHR baseline. may be early, variable, late or prolonged
classified according to shape, timing and duration in relation to the contraction
recurrent = occurring in 50%+ of uterine contractions in a 20 min period
intermitent = occurring in <50% of uterine contractions in a 20 min period
early decelerations
gradual decrease in FHR that mirrors uterine contractions (UC) - nadir (lowest point) aligns with peak/acme of contraction
associated with head compression (from stimulation of fetal vagus nerve)
considered benign - no intervention necessary
variable decelerations
most common deceleration - abrupt decrease in FHR (onset to nadir <30 seconds)
decrease is 15bpm below baseline lasting 15 sec-2 min
may be episodic or periodic
associated with umbilical cord compression or occlusion
late decelerations
gradual decrease in FHR (onset to nadir 30 seconds) below baseline, with nadir occurring after peak of UC
indicates transient or chronic uteroplacental insufficiency - decreased availability of O2
nursing actions for intrauterine resuscitation
objective: improve fetal oxygenation/uteroplacental perfusion while assessing and eliminating the stressor
maternal position change
administer fluid bolus
ad
prolonged deceleration
decrease in FHR 15bpm below baseline lasting 2 min - 10 min from onset to return to baseline. indicates:
profound change in fetal O2
interruption of uteroplacental perfusion
interruption of umbilical blood flow
vagal stimulation
evaluating uterine activity and contractions
essential part of FHR interpretation, must be accurate
factors to evaluate:
frequency
duration
intensity
resting tone
tachysystole = contractions occurring too close together, insufficient fetal oxygenation
studies show that continuous EFM -
is associated with an increase in operative deliveries and c/s
is associated with decrease in neonatal seizures
not associated with lower cerebral palsy rates or neonatal death rates
main function of estrogen during pregnancy
vascularity and vasodilation
enlarges/maintains uterus, develops placenta, prepares breasts for lactation
helps mature fetal organs
main function of progesterone during pregnancy
relaxes smooth muscle and stimulates respiratory function
GTPAL
G - Gravida (total # of pregnancies, regardless of duration, including current)
T - # of term infants born (≥ 37 wks)
P - # of preterm infants (20-37 wks)
A - # of abortions (spontaneous or induced)
L - # of currently living children (note: twins counts as 1 pregnancy, 2 babies)
intrapartum physical assessment
birthing parent VS
uterine assessment - contraction onset/frequency/duration/intensity, palpation and EFM
cervical assessment - dilation, effacement, position, consistency, fetal presenting part, station, membrane status
membrane status - SROM
spontaneous rupture of membrane - amniotic sac breaks on its own
membrane status - AROM
artificial rupture of membrane - provider ruptures amniotic sac to speed labor
membrane status - PROM
premature rupture of membrane (PPROM if occurs before 37 weeks of gestation, high risk)
diagnostics for membrane status
nitrazine swab - test that indicates presence of amniotic fluid
ferning - pattern made by dried/crystallized urine which indicates ruptured membrane
pooling - visible accumulation/collection of amniotic fluid in the vagina
Leopold’s maneuevers
systematic method of abdominal palpation used in obstetrics to determine the position, presentation, and lie of a fetus inside the uterus
FHR devices
external - doppler measures FHR, tocodynameter measures uterine contractions
internal - IUPC (intrauterine pressure catheter) and/or FSE - fetal scalp electrode
principles of care to promote normal birth
1. Labor begins on its own
2. Freedom of movement throughout labor
3. Continuous labor support
4. No routine interventions
5. Non-supine positions for birth
6. No separation of parent/baby after birth and unlimited breastfeeding opportunities
APGAR
newborn health assessed at 1 min and 5 min, then again at 10 min if below 7. all elements assessed on 0-2 scale
A - appearance (skin color)
P - pulse (HR)
G - grimace (reflexes and irritability)
A - activity (muscle tone)
R - respiration (breathing rate/effort)
visceral pain
diffuse/poorly localized, dull and aching pain, felt more during first stage of labor
somatic pain
very localized, sharp and intense pain, felt more during transition phase of first stage and during stage two of labor
neuraxial - standard epidural
injected into epidural space
is a smoother experience with adjustments possible
slower onset, ideal for early labor
neuraxial - combined spinal epidural (CSE)
injected into CSF then epidural space
quick onset plus prolonged dosing option, less motor block
more hypotension and fetal bradycardia
ideal for early and active labor
neuraxial - spinal
injected into CSF only
quick onset, less motor block
more hypotension and fetal bradycardia
ideal for C-sections (quick onset, time limited)
most common adverse effect of neuraxial anesthesia
maternal hypotension which can lead to fetal bradycardia
well-established side effects of epidurals
itching - treat with naloxone
hypotension - treat with vasopressor
longer 1st and 2nd stage of labor
need for forceps or vacuum delivery
fever
common medications in each phase of first stage of labor
latent - sedatives
active - opioids, neuraxial
transition - neuraxial