chest trauma pt 1

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Last updated 4:52 AM on 7/20/26
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32 Terms

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Epidemiology of Thoracic Trauma

High Mortality and Severe Complications. Thoracic trauma accounts directly for 20% to 25% of all trauma deaths, and serves as a major contributing factor in up to 50% of remaining trauma fatalities.

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Respiratory Threat

4 of the top 10 clinical complications are respiratory (Pneumonia, DVT/PE, unplanned extubation, and ARDS).

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Classifications of Thoracic Trauma

Categorized as blunt (sudden positive compression force) or penetrating (foreign object structural breach).

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21% of Total Trauma Cases

Based on national database trauma metrics representing over 183,000 patients.

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Blunt Chest Trauma Mechanisms

Deceleration and Direct Impacts.

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Primary Etiology of Blunt Thoracic Trauma

Motor vehicle crashes (steering wheel impact, seat belt restraint), severe falls, and high-velocity bicycle impacts are the primary etiology of blunt thoracic trauma.

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Pathological Results of Blunt Chest Trauma

Chest wall fractures, dislocations, diaphragmatic barotrauma, pleuropulmonary injury, and great vessel rupture.

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Blunt Trauma Pathophysiology

HHC.

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HHC

Hypoxemia, Hypovolemia, Cardiac Failure.

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Hypoxemia

Arising from severe airway disruption, lung parenchymal lacerations, mechanical chest wall collapse, massive hemorrhage, and acute pneumothorax.

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Hypovolemia

Triggered by massive system fluid and blood loss from great vessels, acute cardiac chamber rupture, or rapid accumulation within the hemothorax.

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Cardiac Failure

Resulting from pericardial tamponade, blunt myocardial contusion, or highly elevated intrathoracic pressure restricting ventricular fill.

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Effects of Hypoxemia, Hypovolemia, and Cardiac Failure

These conditions cause impaired ventilation-perfusion mismatch, potentially driving acute kidney injury, shock, and system failure.

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Rib and Sternal Fractures

Patterns of Bony Thoracic Trauma.

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Ribs 4 through 10

The most commonly fractured bony structures in blunt trauma; typically managed conservatively with supportive measures.

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Ribs 1 through 3

Rare fractures due to heavy musculature; carry high mortality due to immediate risk of subclavian vessel laceration.

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Lower Rib Fractures

High clinical suspicion for laceration of underlying highly vascular solid organs (spleen and liver).

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Sternal Trauma

Primarily caused by steering wheel impacts; highly associated with underlying myocardial contusion.

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Pathological Cycle of Splinting

PTS-PD.

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PTS-PD

Primary Trauma & Splinting followed by Pulmonary Deterioration.

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Primary Trauma & Splinting

Severe pain, muscle spasm, and localized point tenderness over fracture sites cause the patient to "splint" the chest.

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Effect of Primary Trauma & Splinting

The patient breathes in an ultra-shallow, rapid manner, completely avoiding deep breaths, coughing, or physiological sighs to avoid pain.

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Pulmonary Deterioration

Suppression of the cough reflex and shallow chest excursion leads directly to alveolar collapse (atelectasis) and secretion retention.

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Complications of Pulmonary Deterioration

Retained secretions develop into localized pneumonitis and progressive hypoxemia, which can trigger complete respiratory failure.

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Flail Chest

Occurs when three or more adjacent ribs are fractured at two or more distinct anatomical sites, producing a free-floating chest wall segment.

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Free-Floating Segment Mechanics

Occurs when three or more adjacent ribs are fractured at two or more distinct anatomical sites, producing a free-floating chest wall segment.

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Inspiration in Flail Chest

During inspiration, positive atmospheric pressure pulls the detached flail segment inward, restricting lung expansion.

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Expiration in Flail Chest

On expiration, positive intrathoracic pressure pushes the segment outward, blocking efficient exhalation.

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Systemic Cardiopulmonary Decline

This abnormal, paradoxical movement (pendelluft) results in a severe increase in anatomical dead space and a critical reduction in alveolar ventilation.

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Pendelluft

Abnormal, paradoxical movement that results in a severe increase in anatomical dead space and a critical reduction in alveolar ventilation.

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Mediastinum Movement in Flail Chest

The continuous swinging motion of the mediastinum compresses the heart, lowering venous return and cardiac output.

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Effects of Flail Chest

Hypoxemia, respiratory acidosis, and shock.