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Double-Blind
Exp. where neither the participant or the experimenter are aware of which condition people are assigned to (drug studies)
Single-Blind
only participant blind - used if experimenter can't be blind (gender, age, etc)
Skews
created by outliers
Neg skew
mean is to the left (neg side), mode is to the right
Pos skew
mean is to the right
Hawthorne effect
ppl change behavior when watched
Social desirability
ppl lie to look good
Wording effects
how you frame the question can impact your answers
CENTRAL NS
Brain and spinal cord
PERIPHERAL NS
Rest of the NS - relays to Central NS
Somatic NS
Voluntary movement, has sensory and motor neurons
Autonomic NS
Involuntary organs (heart, lungs, etc) - contains the:
Sympathetic NS
fight/flight (generally activates - exception digestion)
Parasympathetic NS
rest / digest (generally inhibits - exception digestion)
NEURON
Basic cell of the NS
Dendrites
Receive incoming NTs
Axon
AP travels down this
Myelin Sheath
speeds up AP down axon, protects axon
Synapse
gap b/w neurons
SENSORY neurons
receive sense signals from environ.-send signal to brain
MOTOR neurons
signals to move - send signals from brain
Interneurons
cells in spinal cord /brain responsible for reflex arc
Reflex arc
important stimuli skips the brain and routes through the spinal cord for immediate reactions (hand on a hot flame)
GLIA
support cells - give nutrients and clean up around neurons
Depolarization
charge of neuron briefly switches from neg to pos. - triggers the AP
Threshold of depolarization
stimulus strength must reach this point to start the AP
All or nothing principle
stimulus must trigger the AP past its threshold, but does not increase the intensity or speed of the response (flush the toilet)
Refractory period
neuron must rest and reset before it can send another AP (toilet resets)
Acetylcholine (ACh)
Memory and movement -in hippocampus, assoc. w/ Alzheimer's
Norepinephrine
sympathetic NS - too little assoc. w/ depression
Endorphins
decrease pain
GABA
Major inhibitory NT
Glutamate
Major excitatory NT (glutes excite you!)
Agonist
drug that mimics a NT
Antagonist
drug that blocks a NT
Reuptake
Unused NTs are taken back up into the sending neuron.(antidepressants cause reuptake inhibition (block reuptake) - treatment for depression)
Cerebellum
movement, balance, coordination, procedural memory (walking a tightrope balancing a bell)
Brainstem / Medulla
vital organs (HR, BP, breathing)
Reticular activating system
alertness, arousal, sleep, eye movement
Cerebral Cortex
outer portion of the brain - higher order thought processes - includes limbic system, lobes, corpus callosum
Limbic System
Amygdala
emotions, fear
BRAIN PLASTICITY
Brain changes via damage and through experience
ENDOCRINE SYSTEM
sends hormones throughout the body
Pituitary Gland
Controlled by hypothalamus. release growth hormones
BRAIN RESEARCH
EEG
shows broad brain activity - not specific - electrical output
fMRI
show brain activity in specific regions, measures oxygen
Lesion
destruction of brain tissue
Hippocampus
episodic and semantic memory (if you saw a hippo on campus you'd remember it!)
Hypothalamus
Reward/pleasure center, eating behaviors - link to endocrine system, homeostasis
Thalamus
relay center for all but smell
Pituitary gland
talks w/ endocrine sys and hypothalamus - release hormones
Occipital Lobe
vision
Frontal Lobe
decision making, planning, judgment, movement, personality, language, executive function - includes the:
Prefrontal cortex
front of frontal lobe - executive function
Motor Cortex
back of frontal lobe - map of our motor receptors - controls skeletal movement
Parietal Lobe
sensations and touch - controls association areas - includes:
Somatosensory Cortex
map of our touch receptors
Temporal Lobe
hearing and face recognition, language
Association areas
receive input from multiple areas / lobes to integrate info
Left hemisphere only
damage to these results in aphasia (damaged speech)
Broca's Area
Inability to produce speech (Broca - Broken speech)
Wernicke's Area
can't comprehend speech (Wernicke's what?)
Corpus Callosum
bundle of nerves that connects the 2 hemispheres - sometimes severed in patients with severe seizures - leads to "split-brain patients"
Circadian Rhythms
24ish hour biological clock of Body temp & sleep
Disrupting it
makes your internal clock get out of sync (jet lag and shift work do this)
Beta Waves
awake (you betta be awake for the exam)
Alpha Waves
high amp., drowsy
NREM (non REM) stages
NREM 1
light sleep, has hypnagogic sensations (falling feeling)
NREM 2
bursts of sleep spindles
NREM 3 Delta waves
Deep sleep
Rapid Eye Movement (REM)
dreaming, cognitive processing
Consolidation
storage of memories
Restoration
helps regenerate the immune system and restore energy
Transduction
convert external stimulus info into APs
Absolute Threshold
detection of signal 50% of time (is it there)
Just noticeable difference
can tell the difference b/w a stronger and weaker stimulus or two similar things (coke vs pepsi, did it get stronger?)
WEBER'S LAW
two stimuli must differ by a constant minimum proportion.(the stronger thing, the more you have to add to tell the difference)
Synesthesia
"disorder" where your senses blend (see sounds, etc)
Sensory Adaptation
diminished sensitivity as a result of constant stimulation (ex. nose blindness)- sensory receptors respond less (get tired)
VISUAL SYSTEM
Lens
focuses light on retina
Retina
contains photoreceptors (rods/cones/ ganglion cells)
Fovea
area of best vision (cones here)
Rods
black/white, dark adaptation
way more rods than cones
located along sides of retina
Cones
color, bright light (red, green, blue) (only in the fovea)
Dichromatism
missing 1 cone
Monochromatism
only have rods
Accommodation
lens changes curvature to focus images on retina
Nearsightedness
better vision near
Farsightedness
better vision far
THEORIES OF COLOR VISION
Trichromatic
three cones for receiving color
Blue
short waves
Green
medium waves
Red
long waves