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Burn
an injury to the skin or other tissue of the body caused by heat, chemicals, electric current, or radiation
Resulting effects if burns
influenced by the temperature and type of burning agent, duration of contact, and type of tissue that is injured
Pts with a burn injury have a multitude of problems
difficulty maintaining an airway and adequate circulation, affecting perfusion
fluid and electrolyte imbalances are common
give attention to preventing malnutrition and infection
Nursing assessment with burns/ injury from explosions
ABCS
lungs sounds
external injury and recognition of possible internal injury
Common sources of burn injuries: Kitchen and bathroom
microwaved food
steam, hot grease or liquids from cooking
hot water heaters set at 140 degrees F or higher
Common sources of burn injuries: General household
heat lamps
fireplaces
open space heaters
radiators
Common sources of burn injuries: Occupational Hazards
tar
cement
chemicals
hot metals
steam pipes
combustable fluids
fertilizers, pesticides
electricity from power lines
sparks from live electric sources
Thermal burns
caused by a flame, flash, scald, or contact with hot objects
severity depends on temperature of the burning agent and duration of contact time
scald injuries often occur in bathrooms and kitchens
flash, flames or contact burns can occur while, cooking, smoking, or burning things
Chemical burns
result of contact with
acids
bathroom cleaners, rust removers, acidifiers for home swimming pools
alkalis
oven cleaners, lye, wet cement, fertilizers
organic compounds
phenols(chemical disinfectants)
petroleum products
Electrical burns
result from intense heat generated from an electric current
direct damage to nerves and vessels, causing tissue anoxia and death, can occur
severity depends on the amount of voltage, tissue resistance, current, pathways, surface area in contact with the current, and length of time that the current flow was sustained
tissue densities offer varius amounts of resistance to electric current
fat and bone offer the most resistance
nerves and blood vessels offer the least resistance
electrical current passes through vital organs (brain, heart, kidney) produces more life-threatening sequelae than that which passes through other tissues
Smoke and Inhalation injuries
breathing noxious chemicals or hot air can damage the respiratory tract
major predictor of mortality in burn patients
rapid initial and ongoing assessment is critical
airway comprise and pulmonary edema can quickly develop within hours of injury
What are the three types of smoke and inhalation injuries that can occur
upper airway injury
lower airway injury
metabolic asphyxiation
Metabolic Asphyxiation
most deaths at a fire scene are the result of inhaling certain smoke elements, primarily carbon monoxide or hydrogen cyanide
oxygen delivery to or consumption by tissues is impaired resulting in hypoxia
Upper airway Injury
resulting from an inhalation injury to the mouth, oropharynx, and/or larynx
mucosal burns of the oropharynx and larynx are manifested by redness, blistering, and edema
the swelling can be massive and the onset rapid
Burns to the neck and chest may make breathing more difficult bc of the burn eschar
becomes tight and constricting
Lower airway injury
an inhalation injury to the trachea, bronchioles and alveoli is usually caused by breathing in toxic chemicals or smoke
tissue damage is related to the duration of exposure to toxic fumes or smoke
carefully asses the pt for facial burns, singed nasal hair, hoarseness, painful swallowing, darkened oral and nasal membranes, carbonaceous sputum(burned saliva), history of being burned in an enclosed space, and clothing burns around the neck and chest
pulmonary edema may not appear until 12 to 48 hours after the burn
then it may manifests as ARDS
Classification of Burn Injury: Extent of Injury
Total Body Surface area(TBSA) determines morbidity and mortality
The quickest way to initially calculate the percentage of TBSA is utilizing the Rule of Nines
Extent of Burns
Lund-Browder chart and rule of nines
First degree burns= to a sunburn, are not included when calculating TBSA
The Lund-Browder chart is considered more accurate bc it considers the pts age in proportion to the relative body-area size
Rule of Nines is often used for initial assessment of bur n pts bc it is easy to remember
For irregular-or odd-shaped burns, the pts hand is 1%TBSA
Location of Burn
the location of the burn injury influences the severity
burns to the face and neck and circumferential burns to the chest or back may interfere with breathing bc of obstruction from edema or leathery, devitalized burn tissue(eschar)
these burns may indicate possible smoke or inhalation injury
Classification of Burn Depth: Superficial (first degree)
destruction of epidermis only
healing time:
3-5 days
wound characteristics
pink or red, dry, painful
Classification of Burn Depth: Superficial partial thickness (second degree)
destruction of epidermis and some dermis
healing time
7-10 day
wound characteristics
moist, pink or mottled red; very painful; blisters; blanches briskly with pressure
Classification of Burn Depth: Deep partial-thickness (second degree)
destruction of epidermis and most of dermis; some skin appendages remain
healing time
2-4 weeks
wound characteristics
pale, mottled, pearly red/white; moist or somewhat dry, typically less painful; blanching decreased and prolonged; difficult to distinguish from full-thickness injury
Classification of Burn Depth: Full-thickness (third degree)
destruction of epidermis, dermis, and underlying subcutaneous tissue
healing time
does not heal, requires skin grafting
wound characteristics
thick, leathery eschar, dry; white, cherry-red, or brown-black; painless; does not blanch with pressure; thrombosed blood vessels
Classification of Burn Depth: Full thickness (fourth degree)
involves underlying fat, fascia, muscle, tendon, and/or bone
healing time
does not heal; may require amputation or extensive debridement
wound characteristics
black, charred, thick, leathery eschar may be present; bone, tendon, or muscle may be visible
Burn management
Initial management of the burn has a dramatic impact on the long0term outcome of the pt
Caring for the pt with a burn injury will require fluid resuscitation, early excision and closure of the wound, tissue debridement and healing, respiratory support, metabolic demand management, and intense microbial surveillance and infection control practices
watch for an assess for Abuse and Neglect in vulnerable populations
Fluid therapy
obtaining IV access is critical for fluid resuscitation and drug administration
at leats 2 large-bore IVs must be in place for pts with burns that ate 15% TBSA or more
it is critical to obtain IV access that can handle large volume of fluid
for pts with burns greater than 30% TBSA, consider a central line for fluid and drug administration and blood sampling
an arterial line is often placed is frequent ABGs or invasive BP monitoring is needed
assess the extent of the burn wound using a standerized tool
then use a standardized formula to estimate the pts fluid resuscitation
parkland formula
fluid replacement is achieved with crystalloid solutions(LR), colloids(albumin), or a combination of the two
IV saline is usually started during the prehospital phase
assess for the adequacy of fluid resuscitations horuly using clinical parameters
UOP most often used parameter, and cardiac parameters
Wound care
one a patent airway, effective circulation, and adequate fluid replacement have been achieved, priority is given to care of burn wounds
you can perform cleansing and gentle debridement(using scissor and forceps) on a shower chart, can take place in a regular shower, or on a pts bed or stretcher
pts find the first wound care to be both physically and mentally demanding
infection can cause further tissue injury and possible sepsis
two approaches to burn wound treatment
open method
limited to facial burns
multiple dressing changes
when the pts open burn wounds are exposed, always wear personal protective equipment (PPE) (e.g., disposable hat, masks, gown, gloves)
Respiratory System
vulnerable to 2 types of injuries
upper and lower airway burns
pt mat beed a fiberoptic bronchoscopy and carboxyhemoglobin blood levels to confirm a suspected inhalation injury
Cardiovascular system
dysrhythmias and hypovolemic shock
Inflammation and healing
burn injury to tissues and vessels cause coagulation necrosis
Immunologic Changes
burn injury challenges the body’s immune system
Clinical manifestations
the pts with severe burns is likely to be in shock from hypovolemia
Endocrine system
watch for transient increases in the pt’s blood glucose levels bc of stress-mediated cortisol and catecholamine release
Sign and symptoms of infection
hypothermia or hyperthermia
increased HR/RR
Decreased BP
decreased UOP
early s/sx
chills
malaise
loss of appetite
Burn management: Fluid resuscitation
critical intervention for burn management
The parkland formula is one of the most widely used nurn resuscitation fluid formulas
Monitor and report abnormal Lab values
sodium and potassium
increased BUN
Increased Lactate
anaerobic metabolism
Glucose
Burn management: Resuscitative phase
begins at the time of injury and continues for approximately 48 hours until the massive fluid and protein shift have stabilized
main concerns are the onset of hypovolemic shock and edema formation
ends when fluid mobilization and diuresis begin
Burn management: Acute phase
begins with the onset of diuresis at approx. 48-72 hours after injury and continues until wound closure occurs
this phase typically occurs in a burn center and may last for weeks or months
Burn management: Pathophysiology
a healing burn injury causes many pathophysiologic changes in the body
diuresis from fluid mobilization occurs, and the pt is less edematous
bowl sounds return
depth of burn wounds may be more apparent as they “declare” themselves as partial or full thickness
pathophysiology-Fluid and Electrolyte Shifts
the greatest initial threat to a pt with a major burn is hypovolemic shock
it is caused by a massive shift of fluids out of the blood vessels bc of increased cap permeability can begin as early as 20 minutes after the burn
Burn management: Nursing care
focuses on the promotion of wounds healing
the prevention of infections and complication
provision of psychosocial support
Rehabilitation phase
the formal rehabilitation phase begins when the pts wounds have nearly healed and they are engaging on some level of self care
Primary Goals in the rehabilitative phase
to improve function and range of motion
ti minimize scarring and contractures
restore pts ability to participate in society and return to an established
Emergency Management of Thermal Burns: Etiology
hot liquids or solids
flash flame
open flame
steam
hot surface
Ultraviolet rays
Emergency Management of Thermal Burns: Assessment findings: Partial Thickness (superficial; first degree) Burn
redness
pain
moderate to severe tenderness
minimal edema
blanching with pressure
Emergency Management of Thermal Burns: Assessment findings: Partial-thickness(deep; second-degree) Burns
moist beds, blisters
mottled white, pink to cherry-red
hypersensitive to touch of air
moderate to severe pain
blanching with pressure
Emergency Management of Thermal Burns: Assessment findings: Full-thickness(third and fourth-degree) Burns
dry
leathery eschar
waxy white, dark brown, or charred apperance
strong bum odor
impaired sensation when touched
absence of pain with severe pain in surrounding tissues
lack of blanching with pressure
Emergency Management of Thermal Burns: Initial
If unresponsive, assess ABC
If responsive, monitor ABC
Stabilize cervical spine
Assess for inhalation injury
Provide supplemental O2 therapy PRN
Anticipate ET intubation and mechanical ventilation with circumferential full-thickness burns to the neck and chest or large TBSA burn
Monitor vital signs, LOC, respiratory status, O2sat, and heart rhythm
Remove nonadherent clothing, shoes, watches jewelry, glasses or contacts
Cover any concurrent thermal burns with dry dressings or clean sheet
Establish 2 IV access
Begin fluid replacement
Insert cath
elevate limbs above heart to decrease edema
Give Iv analgesia and assess the effectiveness
Identify and treat other associated injuries
fractures, injuries
Emergency Management of Thermal Burns: Ongoing Monitoring
Monitor ABC
Monitor vital signs, O2sat, heart rhythm, and LOC
Monitor pain
Monitor UOP
Emergency Management of Inhalation Injury: Etiology
exposure of respiratory tract to intense heat or flames
inhalation of noxious chemicals, smoke, or CO
Emergency Management of Inhalation Injury: Assessment Findings
History of being trapped in an enclosed space, being in an explosion, ir having clothing catch fire
rapid, shallow resps
increasing hoarseness
coughing
singed nasal or facial hair
smoky breath
carbonaceous sputum
productive cough with black, grey, or bloody sputum
irritation of upper respiratory or burning pain in throat or chest
difficulty swallowing
cherry-red skin color
restlessness, anxiety
altered mental status; coma, confusion
decreased O2 sat
dysrhythmias
Emergency Management of Inhalation Injury: Interventions: Initial
If unresponsive, assess circulation, airway, and breathing.
If responsive, monitor airway, breathing, and circulation.
Stabilize cervical spine.
Assess for concurrent thermal burn.
Provide 100% humidified O2.
Anticipate endotracheal intubation and mechanical ventilation with significant inhalation injury.
Monitor vital signs, level of consciousness, O2 saturation, and heart rhythm.
Remove nonadherent clothing, jewelry, glasses, or contact lenses (if face was exposed).
Establish IV access with two large-bore catheters if burn >15% TBSA.
Begin fluid replacement.
Insert indwelling urinary catheter if burn >15% TBSA.
Elevate burned limb(s) above heart to decrease edema.
Obtain arterial blood gas, carboxyhemoglobin levels, and chest x-ray.
Give IV analgesia and assess effectiveness frequently.
Identify and treat other associated injuries (e.g., fractures, pneumothorax, head injury).
Cover concurrent burned areas with dry dressings or clean sheet.
Anticipate need for fiberoptic bronchoscopy or intubation.
Emergency Management of Inhalation Injury: Interventions: Ongoing Monitoring
Monitor airway, breathing, and circulation.
Monitor vital signs, O2 saturation, heart rhythm, and level of consciousness.
Monitor pain level.
Monitor urine output.
Emergency Management of Electrical Burns: Etiology
electric wires
utility wires
lighting
defibrillator
Emergency Management of Electrical Burns: Assessment Finding
Leathery, white, or charred skin
Burn odor
Loss of consciousness
Impaired touch sensation
Minimal or absent pain
Dysrhythmias
Cardiac arrest
Location of contact points
Diminished peripheral circulation in injured extremity
Thermal burns if clothing ignites
Fractures or dislocations from force of current
Head or neck injury if fall occurred
Depth and extent of wound difficult to visualize.
Assume injury greater than what is seen.
Emergency Management of Electrical Burns: Interventions: Initial
Remove patient from electrical source while protecting rescuer.
If unresponsive, assess circulation, airway, and breathing.
If responsive, monitor airway, breathing, and circulation.
Stabilize cervical spine.
Provide supplemental O2 as needed.
Monitor vital signs, heart rhythm, level of consciousness, respiratory status, and O2 saturation.
Check pulses distal to burns.
Remove nonadherent clothing, shoes, watches, jewelry, glasses or contact lenses (if face was exposed).
Cover burned areas with dry dressings or clean sheet.
Establish IV access with two large-bore catheters if burn >15% TBSA.
Begin fluid replacement.
Obtain arterial blood gas to assess acid-base balance.
Insert indwelling urinary catheter if burn >15% TBSA.
Elevate burned limb(s) above heart to decrease edema.
Give IV analgesia and assess effectiveness frequently.
Identify and treat other associated injuries (e.g., fractures, head injury, thermal burns).
Emergency Management of Electrical Burns: Interventions: Ongoing Monitoring
Monitor airway, breathing, and circulation.
Monitor vital signs, O2 saturation, heart rhythm, level of consciousness, and neurovascular status of injured limbs.
Monitor pain level.
Monitor urine output.
Monitor urine for development of myoglobinuria secondary to muscle breakdown and hemoglobinuria secondary to RBC breakdown.
Anticipate possible administration of NaHCO3 to alkalinize the urine and maintain serum pH >6.0.
Emergency Management of Chemical Burns: Etiology
acids
alkalis
organic compounds
Emergency Management of Chemical Burns: Assessment findings
Burning
Redness, swelling of injured tissue
Degeneration of exposed tissue
Discoloration of injured skin
Localized pain
Edema of surrounding tissue
Tissue destruction continuing for up to 72 hr
Respiratory distress if chemical inhaled
Decreased muscle coordination (if organophosphate)
Paralysis
Emergency Management of Chemical Burns: Interventions: Initial
If unresponsive, assess circulation, airway, and breathing before decontamination procedures.
If responsive, monitor airway, breathing, and circulation.
Stabilize cervical spine.
Provide supplemental O2 as needed.
Brush dry chemical from skin before irrigation.
Remove nonadherent clothing, shoes, watches, jewelry, glasses or contact lenses (if face was exposed).
Flush chemical from wound and surrounding area with copious amounts of saline solution or water.
For chemical burn of the eye(s), flush from inner to outer corner of eye with water or lactated Ringer's (if available).
Cover burned areas with dry dressings or clean sheet.
Establish IV access with two large-bore catheters if burn >15% TBSA.
Begin fluid replacement.
Insert indwelling urinary catheter if burn >15% TBSA.
Elevate burned limb(s) above heart to decrease edema.
Give IV analgesia and assess effectiveness frequently.
Contact poison control center for assistance.
Emergency Management of Chemical Burns: Interventions: Ongoing Monitoring