CH61: The Patient with a Cardiovascular Disease (LECTURE)

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23 Terms

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Learning objectives

• Identify the cardiovascular conditions that may be encountered in patients seeking oral health care

• Discuss the etiology, symptoms, and risk factors associated with cardiovascular conditions

• Discuss the impact of cardiovascular diseases on the oral cavity and their relationship to oral health

• Plan dental hygiene treatment modifications for the patient with cardiovascular disease

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Why this matters

Cardiovascular disease = #1 cause of death in the U.S.

Oral inflammation systemic inflammation

Dental hygienists often first to identify risk factors

Our goal: keep hearts safe during care + support prevention

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Cardiovascular system review

Heart + vessels work with lungs to deliver oxygen

Pump deoxygenated blood →lungs; oxygenated →body

Both systems interdependent —disease in one affects the other

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Modifiable risk factors  

90 % of MI & stroke risk is preventable

Change be changed: tobacco use, hypertension, hyperlipidemia, diabetes, obesity, stress, inactivity, poor diet

Non-modifiable: age, sex, family history

Dental team role = identify and reinforce healthy behaviors

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Prevention and lifestyle counseling

Encourage tobacco cessation, exercise, diet changes

Link oral and cardiovascular health

Refer to physician if risk signs appear

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Infective endocarditis

Bacterial infection of endocardium or heart valves →vegetations

Caused by: Streptococcus viridans, Staphylococcus aureus

Risk ↑ with bacteremia during subgingival instrumentation

Prevention = excellent oral hygiene + appropriate antibiotic prophylaxis

Who Needs Premedication?

Only for patients at highest risk of adverse outcomes from this

◦ Prosthetic valves or material used for valve repair

◦ Previous infective endocarditis

◦ Certain congenital heart defects

◦ Unrepaired cyanotic CHD (shunts/conduits)

◦ Repaired CHD with residual defect near prosthetic patch/device

◦ Cardiac transplant recipients with abnormal valves

Clinical Management for at risk pts

  • Review medical history each visit –confirm cardiac status

  • Ask: “Has your cardiologist recommended antibiotic premed?”

  • Delay elective care if unclear or unstable

  • Reinforce biofilm control & document antibiotic use

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Fetal circulation and development 

Fetal heart is completely developed by week 9 of gestation

Ductus arteriosus connects pulmonary artery →aorta (bypasses fetal lungs)

Normally closes shortly after birth

If it stays open →patent ductus arteriosus (PDA)

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Congenital heart defects and dental implications

Ventricular septal defect (VSD) = most common congenital heart defect

Other examples: atrial septal defect (ASD) and PDA

May occur with Trisomy 21 and other genetic syndromes

Surgically repaired early in life (may involve prosthetic device or patch)

Premedication indicated for 6 months post-repair or if residual defect remains

  • If unsure whether premed is still needed, always consult with the cardiologist!

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Rheumatic heart disease

From Group A strep →rheumatic fever →valve damage

Preventable by early antibiotic therapy for strep throat

Usually no premed unless other AHA indications exist

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Mitral valve prolapse

Valve between left atrium and left ventricle doesn’t close properly →slight backflow of blood

May produce a click or murmur, often benign

Most cases asymptomatic

No premedication needed unless other qualifying cardiac condition exists

If patient uncertain, verify with physician before treatment

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Heart murmurs and valve disorders

Murmur = sound from turbulent valve flow (not disease itself)

May indicate MVP or rheumatic scarring

Requires medical evaluation if new or symptomatic

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Hypertension

“Silent killer” → major MI/stroke risk

Risk factors: age, obesity, diet, stress

Proceed if controlled; stress reduction, epi ≤ 0.04 mg, monitor BP every visit

≥180/110 + symptoms (severe dyspnea, headache)→ activate EMS

<p><span><strong><span>“Silent killer” → major MI/stroke risk </span></strong></span></p><p><span><strong><span>Risk factors: age, obesity, diet, stress </span></strong></span></p><p><span><strong><span>Proceed if controlled; stress reduction, epi ≤ 0.04 mg, monitor BP every visit </span></strong></span></p><p><span><strong><span>≥180/110 + symptoms (severe dyspnea, headache)→ activate EMS</span></strong></span></p>
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Ischemic heart disease

Reduced blood flow → reduced oxygen to tissue → pain or tissue death

Includes angina, MI, and heart failure

Risk factors: atherosclerosis (*main cause), smoking, hypertension

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Angina pectoris

Chest pain due to temporary ischemia

Stable →predictable triggers (stress, exercise); relieved by rest/nitro

Unstable →unpredictable; may precede MI →urgent referral

Limit stress and chair time; always have nitro & oxygen accessible

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Myocardial infarction

Complete coronary blockage → tissue death

Symptoms: chest pressure, nausea, sweating, shortness of breath, fatigue

Not relieved by rest or nitroglycerin

Emergency response: terminate treatment, sit upright, oxygen, call 911

Do not perform chest compressions unless unconscious & no pulse

Post Care:

Delay elective dental care 4-6 weeks, only after cardiology clearance

◦ Older “6 months” rule no longer currrent

Use stress-reduction techniques and monitor vitals

Keep oxygen and nitroglycerin available during appointments

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Heart failure (CHF) 

Heart can’t pump adequately

Left sided: pulmonary congestion →shortness of breath, fatigue

Right sided: systemic congestion →swelling in feet/ankles, distended neck veins

Both sides: fluid in lungs and legs

Semi-supine or upright; short appointments; limit aerosols

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Ischemic and circulatory conditions at a glance

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Cardiac arrhythmias and devices

Abnormal rhythm or rate →may have pacemaker or ICD

Can be symptomatic or asymptomatic

Most do not require treatment

If they do:

◦ Drug therapy

◦ Lifestyle modification

◦ Medical procedures

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Cardiac surgery and devices

CABG: bypass surgery; no premed after healing (~6 weeks)

Coronary stent: on antiplatelets →mild bleeding; don’t stop without MD approval

Pacemaker/ICD: avoid electromagnetic interference (ultrasonic cords, magnets)

Document device type + placement date

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Antithrombotic therapy

Warfarin, DOACs, Aspirin, Clopidogrel ↑bleeding risk

Do not discontinue without physician clearance

Use local hemostatic measures (pressure, sutures, hemostatic agents)

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Appointment planning and instrumentation

Review history & BP each visit

Stress-reduction protocol (quiet room, short appointments)

Semi-supine if dyspneic

Use HVE; avoid ultrasonic if pacemaker unshielded or respiratory distress present

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Patient education and collaboration

Emphasize daily biofilm control →↓bacteremia risk

Reinforce modifiable risk factors (quit smoking, diet, activity)

Encourage compliance with meds & follow-ups

Coordinate with medical providers as needed

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Key takeaways

Identify cardiac risk →modify care appropriately

4–6 weeks post-MI before elective care (when cleared)

AHA premed = high-risk only

90 % of CVD risk modifiable —DH impact matters