APETITE & OBESITY

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PSYC 233

Last updated 5:01 PM on 8/18/26
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8 Terms

1
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Hypothalamus/ARC (Arcurate nucleus)

  • NPY/AgRP (Orexigenic) : Increase hunger, increase food

  • POMC/CART (Anorexigenic) : Increase satiety, decrease food


2
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Ghrelin

In stomach.

Ghrelin increase → NPY/AgRP increase → Food increase

3
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Leptin

By adipose tissue.

  • Inhibit NPY/AgRP and activate POMC/CART → Decrease food


4
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Leptin resistance

In obesity, leptin may be high but less responsive. Make satiety signal less effective.

Leptin present doesnt mean the signal is working effectively.

5
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ob/ob Mice Evidence

  • ones that had no leptin → overate and obese

  • ones with leptin → food intake decreased


6
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Short term satiety

CCK and PPY released from gut after eating → increase satiety → intake decrease.

7
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Homeostatic v Hedonic

Homeostatic: Eating via. energy needs → Hormones/Hypothalamus

Hedonic: Eating via. reward/wanting even when energy need met. VTA → NAcc DA

Eating = Homeostatic control + Hedonic reward



8
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Obesity/Evaluation

  • Eating = Hypothalamus + Hormonal + Reward + Env/Learned cue influences.

  • Too reductionist, leptin resistance shows why

  • Animal work

  • Multiple interacting systems