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common diagnoses in birth-2
developmental dysplasia, septic arthritis
common diagnoses in 2-12yrs
transient synovitis, coxa vara, legg-calve-perthese
common diagnoses in 8-17yrs
Slipped Capital Femoral Epiphysis (SCFE)
18-young adult
Osteochondritis dessicans, strains, stress fractures, femoroacetabular impingement and labral pathology, osteitis pubis, apophyseal injuries
20-40 years
RA
30-50 years
Osteonecrosis/ avascular necrosis
>50 years
DJD, trochanteric bursitis, hip fracture
Common Areas of Referral
lumbar spine, TL junction, SIJ, knee, Viscera
Factors Related to Fall Risk
Berg Balance Scale, Dynamic gait (DGI), Falls efficacy scale (self perception tests), History of imbalance, Type of AD used
Harris Hip Score
4 sections: Pain, Function, Deformity, ROM
Max score=100 (indicates no pain/dysfunction) (higher score the better)
MCID= 7-9 pts
Lower Extremity Function Scale (LEFS)
Rates difficulty performing 20 activities
Max score= 80 (no difficulties)
MDC & MCID= 9 pts
Developmental Dysplasia
Normal bone development of acetabulum and/or femoral head is disrupted by subluxation or dislocation
Causes: genetic, hormonal, mechanical
If improperly treated, can result in degenerative arthritis as an adult
Treatment for Developmental Dysplasia
Maintain favorable position until acetabulum and femoral head develop enough to decrease chance of further involvement
Transient Synovitis
Non-specific inflammation of synovium which is self limiting
Presents with flexion/abduction/lateral rotation posture, low grade fever
Treatment for Transient Synovitis
Bed-rest with limited weight bearing, heat, massage, NSAIDs
If pain persists: Check for Legg-Calve-Perthes
Septic Arthritis
Acute and rapidly progressing infection caused by pyogenic bacteria in the hip
Can progress to femoral head necrosis within 24 hours
Treatment for Septic Arthritis
Joint aspiration, drainage, antibiotics, hip spica, surgery
Legg-Calve Perthes Disease (LCPD)
Avascular necrosis (osteonecrosis) of capital femoral epiphysis caused by compromise of blood supply to ossification centers
2-12 years (most common 6-7 years)
Treatment for LCPD
Proximal varus osteotomy (6-10 y/o), decrease stress on hip, joint mobs & ROM, strengthening exercises, gait training
Slipped Capital Femoral Epiphyses (SCFE)
10-17 y/o for boys & 12-14 y/o for girls. Typically after a growth spurt
Growth plate is strained and epiphyses “slips” off.
Insidious onset, Treat as an emergency
Treatment and Rehab for SCFE
Surgery is treatment of choice, or skeletal maturity
Post op: Strengthening LE, increase ROM, gait training, balance/propriception, muscular endurance
No permanent disability if treated quickly
Type 1 Salter-Harris Fracture
Through growth plate
Type 2 Salter-Harris Fracture
Through growth plate and metaphysis
Type 3 Salter-Harris Fracture
Through growth plate and epiphysis
Type 4 Salter-Harris Fracture
Through all 3 elements
Type 5 Salter-Harris Fracture
Crush injury of growth plate
Avascular necrosis (AVN)
30-50 y/o
Impairment of blood to femoral head causing tissue death after 12 hours
Occurs bilaterally if not traumatic
Treating AVN
Conservative: strengthening above and below joint, limiting stress through joint with AD and pt education, pain relief
Surgery: Core decompression, Total hip, trochanteric osteotomy, vascular fibular graft
Osteoarthritis (OA)
>50 y/o insideous onset
Progressive deterioration of articular cartilage, overgrowth of periarticular bone
Idiopathic or Traumatic
Pt has decreased acetabular and femoral neck anteversion
Treatment for OA
Rest, NSAIDS, ROM/Stretching/Strengthening, STM & Joint mobs, activity modification/AD/aquatics (unload joint)
Femoroacetabular Impingement (FAI)
Abnormal or early contact between bony prominences of the acetabulum and femur
Young <50 y/o Insidious onset
Pincer lesion
FAI classification, a deep acetabular socket with excessive coverage of femoral head
Associated pathologies: Coxa prounda, retroversion of acetabulum, anterior rotation of pelvis in the transverse plane
More common in middle aged active women
CAM lesion
FAI classification, lesion that reduce the medial proximal femoral angle
Associated pathologies: Coxa vara, developmetnal dysplasia, Legg-Calve Perthes disease, SCFE
2nd most common
Mixed lesion
FAI classification, results in an abrasion of the labrum, labral degeneration, ossification of acetabular rim and deepening acetabulum
Most common FAI
FAI treatment
If cause is overuse and no structural deformity: non operative, conservative treatment
Early operative prevents OA
Hip Labral Dysfunction
Compromised seal from tear causing higher stress across the joint, causing joint deterioration
Treatment of labral injury
Conservative: Rest and WB restrictions, NSAIDs and pain meds, manual therapy and joint distraction, movement modification
Operative: Similar to meniscal tears
Anterior hip dislocations
Forced abduction —> femoral neck or trochanter impinges against superior dome of acetabulum —> head out through tear in anterior capsule
Considered an emergency as its associated with arterial injuries or venous thrombosis, neuro status must be documented
Posterior hip dislocation
Occurs with hip/knee in flexion —> limb shortening, adduction and medial rotation
Should rule out femoral head/shaft fracture, and sciatic nerve injury
Fractures
Intracapsular: Rare in kids but if seen is serious, worst prognosis
Extracapsular: >swelling
Immediate groin pain, pain with movement, tender anterior to femoral neck, shortened limb, held in lateral rotation, ecchymosis
Muscle Strains
Caused by stresses placed on muscles especially during eccentric contractions
Commonly at musculotendinous junction of 2 joint and fast twitch muscles
Grade 1 strain
Min tissue disruption, min inflammation, strong and painful, full ROM but pain with stretch
Grade 2 strain
Some fiber disruption, decreased Strength and ROM, severe pain especially with stretching
Grade 3 strain
Complete rupture of muscle fibers, max weakness, no pain with stretching, palpable/visible defect
Strain Treatment
PRICE, NSAIDs, crutches, ROM, isometric, modalities for pain and swelling
Late Stage: endurance and coordination activities to return to sport
Bursitis
Secondary to direct trauma, repetitive activity, or inflammatory arthritis
Insidious onset
Most common of Hip: Trochanteric, Iliopectineal, Ischiogluteal
Bursitis Presentation
Achiness or tenderness over lateral posterior trochanter and thigh, snapping, pain with climbing stairs, lying on side, stretch of glute max, resisted abduction, extension and lateral rotation
Treatment for bursitis
PRICE, NSAIDs, stretching tight structures, strengthening to decrease muscle imbalances
Contusions
Direct blow to anterior or lateral thigh, dull ache over ant thigh with swelling, antalgic gait, difficulty flx/ ext knee, positive SLR and difficulty contracting quads
Grade 1 Contusion
Min discomfort, local tenderness, lack of gait disturbance, painless knee motion
Grade 2 Contusion
Swelling, tender muscle mass, antalgic gait, knee ROM <90, difficulty climbing stairs or getting up from chair
Grade 3 Contusion
Extreme pain/ swelling, knee ROM <45 ,limp, effusion into ipsilateral knee, AD needed
Treatment for Contusions: Phase 1
RICE, NWB with AD
Treatment for Contusions: Phase 2
Restore motion, progress WBAT as swelling resolves, active flexion/extension, gentle PROM
Treatment for Contusions: Phase 3
Functional rehab once ROM increases and WB has progressed, progress strengthening as pain allows
Myositis Ossificans
Complication of contusion of quadriceps causing development of heterotopic bone in muscle
Myositis Ossification Treatment
Rest and crutches, gentle ROM as pain resolves, submax isometrics once allowed, NSAIDs and steroids to decrease swelling
Traumatic Hip Instability
Dislocation/subluxation
Atraumatic Hip Instability
global laxity, idiopathic nature, groin pain with rotation on fixed foot, anterior hip pain with OP into hip extension and lateral rotation
Piriformis Syndrome Treatment
Muscle energy techniques, PNF, lateral rotator strengthening, neuromuscular reeducation, steroids, botox, surgical release
Adult Patient History Questions
Traumatic injury, Developmental dysplasias, Childhood hip conditions, Family history of connective tissue disease
Cyriax Capsular Pattern
gross limitation in flexion/abduction/medial rotation, slight limitation of extension, little to no limitation in lateral rotation
Meadows Capsular Pattern
Early arthritis, Painful flexion/adduction/medial rotation, Limited in extension/ adduction/medial rotation
Transient Synovitis: Severe Involvement
Pain in hip; Refuses to walk; All AROM/PROM is limited
Transient Synovitis: Moderate Involvement
Pain in thigh, knee, and hip; Antalgic gait; Limited hip extension and medial rotation
Signs of Septic Arthritis
Irritability, fever and chills, shorter leg, prominent greater trochanter, swollen thigh, hip spasms
Hip in flexion/abduction/lateral rotation, decreased ROM at hip (medial rotation)
Signs of Legg-Calve Perthes Disease (LCPD)
Pain and “achiness” in hip, groin, or knee. Can refer to thigh
Pain in WB, antalgic gait, tender anterior/posterior capsule, decreased ROM, trendelenburg, ipsilateral side shorter
SCFE Presentation
Out toeing, Abduction and lateral rotation with passive hip flexion, Knee pain is initial sx for 46% of patients, tender anterior and lateral hip, adductor spasm, fatigue after walking, limited ROM in flexion/abduction/medial rotation, quad atrophy, antalgic gait, trendelenburg
Early Avascular Necrosis
Pain is mild dull ache or throbbing, intermittent in groin or hip, typically occurs with WB and decreases with rest
Normal ROM and gait
Progressed Avascular Necrosis
Pain moves into inner thigh and eventually becomes constant
Slight limp becomes noticeable in gait
Late Avascular Necrosis
Stiffening of the joint with muscle spasm (iliopsoas, rectus femoris, sartorius)
Glutes and piriformis tender to palpation
Limited abduction and medial rotation
FICAT Stage 0
Xray: normal
MRI: normal
Bone scan: n/a
Clincal sx: none
FICAT Stage 1
Xray: normal or minor osteopaenia
MRI: edema
Bone scan: increased uptake
Clinical sx: pain typically in groin
FICAT Stage 2
Xray: mixed osteopaena &/or sclerosis
MRI: geographic defect
Bone scan: increased uptake
Clinical sx: pain and stiffness
FICAT Stage 3
Xray: crescent sign & eventual cortical collapse
MRI: same as Xray
Bone scan: pain and stiffness
Clinical sx: radiation to knee and limp
FICAT Stage 4
Xray: end stage with evidence of secondary degenerative change
MRI: same as Xray
Bone scan: n/a
Clinical sx: pain and limp
OA presentation
>50 y/o insideous onset
Groin or greater trochanter pain, pain with WB, stiffness after resting and relieved with activity
Capsular pattern: decreased extension/medial rotation/extreme flexion
Idiopathic OA
Occurs in middle age with no known cause (DJD)
Traumatic OA
Occurs in response to injury, deformity, or disease
OA on Xray
Osteophytes
Joint space narrowing
Subchondral sclerosis
FAI Presentation
Presents with unilateral groin and anterolateral hip pain, clicking or locking, decreased ROM on exam, empty end feel with flexion and rotation, c sign, sharp pain with pivoting (especially internal rotation) , ant pinching with sitting
Rehab Considerations After Arthroscopic Decompression
Excessive hip flexion, Abduction, and Medial Rotation
Initially limited, improve over time
Causes of Labral Injuries
Trauma, Hyperextension, FAI, Decreased acetabular anteversion, Decreased femoral neck anteversion
Labral Injury
Females>Males
Gradual hip pain (90% anterior hip or groin pain)
Slight ROM limitations with rotation/hip flexion/adduction/abduction
Sx for >2 years
Major cause of OA in young patients