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consciousness
awareness of ourselves and environment
sense of emotions, sensations and choices
Cognitive neuroscience
interdisciplinary study of the brain activity linked with our mental processes
Dual processing
information simultaneously processed on separate conscious and unconscious tracks
where does parallel processing happen
unconscious mind
parallel processing
processing multiple aspects of a stimulus or problem simultaneously
where does sequential processing happen
conscious mind
sequential processing
processing one aspect of a stimulus or problem at a time
new info and problem solving
sleep
natural loss of consciousness
Circadian Rhythm
biological clock and regular bodily rhythms that occur on a 24 hour cycle
Alpha waves
relatively slow brain waves of a relaxed, awake state
when does alpha happen
before you sleep
Stage 1 of sleep cycle
irregular brain waves
slowed breathing
resembling hallucinations
hypnagogic sensations
hypnagogic sensations
Body may jerk or may have feeling of floating weightlessly or feeling of falling
Stage 2 of sleep cycle
relax more deeply, abt 20 min
sleep spindles - rapid rhythmic brainwave
Stage 3 sleep cycle
deep sleep stage
delta waves
hard for you be awake
30 min
delta waves
large slow brain waves associated with deep sleep
what is NREM sleep
stage 1, 2 and 3
what happens after stage 3 of sleep cycle
you go back to stage 2 of sleep cylce and then go into 10 min of REM
REM sleep
“rapid eye movement”
brain waves rapid like stage one
20-25% total sleep
how is rem sleep diff than stage 1
heart rate rises
breathing becomes rapid
eyes move rapidly
dreams
Paradoxical Sleep
REM = “paradoxical sleep”
Brain is highly active
Body stays calm/inactive
how long does a sleep cycle last
abt 90 min
what happens to motor cortex in REM
motor cortex active
BRAINSTEM blocks movement —> temporary paralysis
suprachiasmatic nucleus
cell cluster in the hypothalamus that causes pineal gland to decrease its production of melatonin in the morning and increase in the evening
melatonin
sleep inducing hormone
what does sleep protect from
from evolutionary perspective, keeps safe from dangerous periods of wandering in the dark
what does sleep help us recuperate
restore and repair brain tissue, gives neurons a rest
how does sleep make memories
restores and builds our fading memories from the day, strengths neural connections
how does sleep feed creative thinking
become more insightful after sleep, solve problems easier
how does sleep play role in growth process
during sleep pituitary gland releases a growth hormone
sleep loss can be correlated with
Depression and irritability
Depressed immune system
Suppressed immune system to fight of infections
Increased weight gain
Increases ghrelin and decreases leptin
Makes you look older
high blood pressure, impaired memory, and more accidents
insomnia
recurring problems in falling or staying asleep
cant fall asleep or always waking up during sleep
chronic tiredness, depression and hypertension (high blood pressure)
narcolepsy
uncontrollable and sudden attacks of sleepiness
falls asleep at random
lasts short times likes 5 min
accompanied by loss of muscle tension, dangerous to engage in actions like driving
sleep apnea
stopping of breathing during sleep and repeated momentary awakenings
can happen over 100 times
usually NOT aware have it OR waking up in the night
increased tiredness, obesity, and depression
sleepwalking
complex motor activity in stage 3 of sleep
not harmful and usually in children
Rem sleep behavior disorder
sleep disorder in which normal REM paralysis does not happen but
twitching
talking
kicking
occurs, acting out ones dream
dreams
sequence of images, emotions, thoughts passing through a sleeping person’s mind
why do we dream
file away memories —> help sift, sort and fix the day’s experience in our memory
devolp and preserve neural pathways —> believed that it helps devolp neuron and neural network
make sense of neural static
reflect cognitive development —> see dreams as a part of brain maturation and cognitive development
activation synthesis theory
random neural activity and our brain attempts to make sense of it when we dream
sensation
process by which our sensory receptors and nervous system receive and represent stimulus energies from our environment
perception
process of organizing and interpreting sensory information, enabling us to recognize meaningful objects and events
bottom up processing
using our sensory recpetors and works up to the brains higher levels of processing by integrating the sensory information
top down processing
information process guided by higher level mental processes, as when we construct perceptions drawing on our experience and expectations
transduction
converts stimulus energy into neural impulses
receive sensory input
transform it into neural impulses
send it to the brain
Psychophysics
physical properties of stimuli (like intensity) relate to our psychological experience of them
Thresholds
minimum level of stimulus intensity required to be consciously aware of the stimulus
absolute threshold
minimum stimulation needed to detect a particular stimulus 50% of time
subliminal
stimulus you can not detect 50% of the time
signal detection theory
-detect faint stimulus in a background of other stimulation
says there is no fixed absolute threshold
detection depends on factors like experience, expectations, motivation and alertness
difference threshold
minimum difference between two stimuli needed to notice a change 50% of the time
small changes are more easily at low intensities than high ones
40—>45 more noticeable 90—>95
Weber’s law
principle that two stimuli must differ by a constant proportion (percentage) not constant amount
lights differ by 8% and weights 2% to notice change
sensory adaptation
constant stimulation causes your sensitivity to decrease
Example: bad smell fades after a few minutes
Helps us ignore unimportant background stimuli and focus on changes in our environment.
why do eyes take in light energy
to convert neural signals
wavelength
distance between peaks —> determines color
amplitude
height of the wave which determines intensity
cornea
Clear outer layer
Protects the eye
Bends light to start focusing the image
pupil
Dark, adjustable opening where light enters
Works like a camera aperture—more or less light depending on conditions
iris
Colored ring of muscle around pupil
Controls pupil size
Dilates (opens) in low light or when excited
Constricts (closes) in bright light or when calm
Helps regulate how much light reaches the retina so images aren’t too bright or too dim
lens
Behind the pupil
Focuses light onto the retina
Changes shape to keep images clear
accommodation
lens changes shape to focus near vs. far objects on the retina
retina
Light-sensitive inner surface of the eye
Contains rods and cones (receptor cells)
First place where neural processing of visual info begins
rods
outer retina
Sense black, white, gray
Work best in low light
night vision + peripheral vision
cones
Concentrated in fovea (center of retina)
Detect color and fine detail
Work best in daylight / bright conditions
bipolar and ganglion cells
Light signal travels to bipolar cells, then to ganglion cells
blind spot
Where the optic nerve leaves the eye
No rods/cones there → no vision in that tiny point
Retina to Visual Cortex
Each part of the retina sends visual info to a matching spot in the visual cortex (occipital lobe)
Optic Chiasm
Optic nerves cross at the optic chiasm (X-shaped structure)
Visual Info Crossing
half the info from each eye goes to the opposite side of the brain
optic nerve formation
Ganglion cell axons bundle together
These bundled axons form the optic nerve
Optic Nerve Function
Optic nerve = carries neural messages from the eye to the brain
brain pathway
Visual info travels to the thalamus
Then sent to the visual cortex (occipital lobe)
Young-Helmholtz Trichromatic Theory
Retina has 3 color receptors: red, green, blue.
All colors are seen by combining activity from those three receptors.
Yellow is perceived when red + green receptors are activated together
when does colorblindness happen
when one receptor type is missing or not working.
flaws in Trichromatic Theory
People missing red + green receptors (red–green colorblind) can still see yellow, which shouldn’t happen if yellow is only a mix of red + green.
Yellow looks like a pure, unique color—not like a blended one (unlike purple, which clearly feels like red + blue mixed).
Opponent-Process Theory
Color vision is controlled by opposing pairs:
Red ↔ Green
Yellow ↔ Blue
White ↔ Black
one color in a pair is activated, the other is suppressed
afterimages
Stare at green → green sensor tire → when image is gone, the opponent (red) shows up.
Same idea for black→white and yellow→blue
Two stages of current understanding of color vision
Stage 1: Trichromatic
Retina has red, green, and blue cones.
Each responds in different amounts to light wavelengths.
Stage 2: Opponent-Process
Cone signals are sent to opponent-process cells (red–green, yellow–blue, white–black) for final color perception
feature detection
Nerve cells in the occipital lobe (visual cortex)
Respond to specific features: shape, line, angle, movement
processing complex patterns
Feature detectors send info to other areas of the visual cortex
Allows the brain to recognize more complex patterns
parallel processing
The brain processes multiple features at once (not one at a time).
visual scene gets split into color, motion, form, and depth simultaneously.
All pieces processed together —> one complete perception.
Audition
the sense or act of hearing
ear transforms the vibrating air into nerve impulses which our brain decodes as sound
amplitude in sound
loudness; height of sound wave determines how loud
frequency
length of the sound wave, which determines pitch
pitch
tone of a sound in high or low
how is sound measured
decibels (dB)
0 dB = absolute threshold for hearing.
Every +10 dB = 10× increase in sound intensity
Outer ear
auditory canal that funnels sound waves to the eardrum and middle ear
Includes the pinna (your ear)
Middle Ear
Eardrum – membrane that vibrates when sound waves go into ear
three tiny bones in the middle ear known as ossicles
vibrate and trigger the cochlea in the inner ear
Inner Ear
Innermost part of the ear.
Includes the cochlea, semicircular canals, and vestibular sacs.
Cochlea
Snail-shaped, fluid-filled tube in the inner ear.
Sound waves move the fluid → triggers nerve impulses sent to the brain
How Sound Becomes Neural Signals
Sound waves vibrate the fluid in the cochlea.
This makes the basilar membrane ripple → bends hair cells.
Hair cell movement creates neural impulses.
These impulses travel through the auditory nerve → thalamus → auditory cortex in the temporal lobe.
hearing loss
Damage to cochlea, hair cells, or auditory neurons
Can hear sounds but have trouble understanding speech
Causes: disease, aging, loud music
what are devices for hearing loss
Cochlear implant: converts sound → electrical signals → stimulates auditory nerve via electrodes in cochlea
condition hearing loss
Hearing loss caused by damage to the eardrum or ossicles
Loss of ability for these to vibrate and reach cochlea
loudness
Softer sounds → only a few hair cells
Louder sounds → more hair cells also respond
depends on the number of activated hair cells
place theory
theory that links the pitch we heart with place where the cochlea’s membrane is stimulated
frequency theory
theory that rate of nerve impulses traveling up the auditory nerve matches the frequency of tone
volley theory
neural cells can alternate firing. By firing in rapid succesion → achieved combined frequency above 1000 waves per second
how can we determine location of sound
Sounds strike one ear sooner and more intensely than the other, allowing us to locate the sound
Touch
sensitivity to pressure on the skin
Somatosensation
general term for four classes of tactile sensations
Touch
Pressure
Cold
Pain