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feen jbhs 2526
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cardiac output
measures the amount of blood ejected by ventricles in one minute
preload
stretching of heart when ventricles are full
afterload
force heart has to overcome to eject blood
heart failure
heart is unable to pump enough blood to meet the body’s needs
ischemia
decrease in bloodflow to an area
infarction
tissue damage because of a decrease in bloodflow
angina
chest pain because of a decrease in oxygen or bloodflow to the heart
cardiomyopathy
heart muscle disease making it hard to pump blood
valve stenosis
stiffening of heart valves
coronary arteries
vessels delivering blood to heart tissue
Stable angina
chest pain worse with activity and relieved with rest. inadequite oxygen to heart muscle during activity. stable plaque in coronary arteries
unstable angina
chest pain with activity and rest. can lead to a heart attack. plaque ruptures and partially blocks coronary arteries.
increased backflow
pulmonary congestion, increased workload on the right heart, leads to right and left heart failure
decreased forward flow
tired, weak, cold extremeties, decreased urine output, dizzy, syncope, altered mental state
hypertrophy
cardiac output decreases, increased growth, valve stenosis/stiffening
stenosis
heart valves stiffen, causing poor forward flow.
SA node
pacemaker of the heart
AV node
responsible for the pause.
Bundle of HIS
accepts impulse and splits off into right and left bundles
purjunkie fibers
carries impulse to ventricles through right and left bundles.
60-100 bpm
normal sinus rhythm
Atrial Fibrillation
quiver in the heart, uneven QRS (uneven squeeze)
Ventricular Fibrillation
abnormal cardiac rhythm, no sinus rhythm, wonky currents.
Sinus Tachycardia
regular wave structure, rapid heart rate
Sinus Bradycardia
regular wave structure, extremely slow heart rate
Asystole
little to no EKG activity, no electrical currents from the heart.
P wave
wave responsible for atrial depolarization
T wave
wave responsible for ventricular repolarization
QRS wave
wave responsible for ventricular depolarization
Electrocardiogram
detects heart patterns and electrical pulse. cannot determine deformities within the heart.
Modifiable risk factors
smoking, diet, lifestyle, obesity, alcoholism
nonmodifiable risk factors
genes, history, age, gender, race