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Patients with delayed coronary reperfusion following ST-elevation MI (eg, >3 hours from symptom onset) are at increased risk of developing
PIP.
Pleuritic chest pain (↓ when sitting) ± fever
Pericardial friction rub (highly specific)
ECG: diffuse ST-segment elevation & PR-segment depression
Echocardiography: pericardial effusion
Peri infarction peri carditis
milk-alkali syndrome should include measurement of serum ——— which would be expected to be suppressed. Additional findings can include ————- due to intestinal binding of phosphate by calcium carbonate, and ————- due to decreased renal reabsorption of magnesium.
parathyroid hormone (PTH), hypophosphatemia, hypomagnesemia
Nausea, vomiting, constipation
Polyuria, polydipsia
Neuropsychiatric symptoms
MILK ALKALI SYN
isotonic saline followed by furosimide
milk alkali syn
Mammography & ultrasonography
Biopsy ± excision
intraductal papilloma
Anti-GQ1b antibodies are highly sensitive for
Miller Fischer Syndrome (but may be present in other GBS variants with predominant ophthalmoplegia).
GI illness followed by a symmetric descending weakness (as opposed to the lower-extremity weakness in this patient), and even though ophthalmoplegia may occur, facial weakness, dysphagia, and dysarthria are typical
Botulinum toxin causes