What are the characteristics of flaccid dysarthria related myasthenia gravis?
muscle weakness and fatigue that can recover with rest, affecdts voluntary muscles of the body, deterioration in articulation, dysphonia, poor loudness, hypernasality, continues until client is unintellible.
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What is spastic dysarthria caused by?
Bilateral UMN lesions (damage to the motor cortex and/or its descending projections), stroke, MS, TBI, MND, cerebral palsy etc.
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Damage to UMN results in an imbalance between what?
excitatory and inhibitory inpot to lower motor neurons
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What are the clinical signs of spastic dysarthria?
weakness, spasticity (hypertonia), slowness of movement, reduced range of movement, no muscle atrophy, pseudobulbar effect, bilateral facial palsy, bilateral hemiparesis
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Unilateral Upper Motor Neuron Dysarthria (UUMN) symptoms present where?
physical signs and symptoms present on the ipsilateral side (opposite side)
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\ The upper face is innervated ______, the lower face is innervated ------
bilaterally, unilaterally
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Ataxic dysarthria is associated with damage where?
In the cerebellum/cerebellar control circuit
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What are the causes of ataxic dysarthria?
Demyelination (MS), stroke, trauma, tumour
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What is ataxia?
uncoordinated, ‘broken’ movements
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What are the impacts of ataxic dysarthria on speech?
imprecise articulation, slurrig, stress is excess and equal, irregular AMRs
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problems with cognitive linguistic processing
Aphasia
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sensorimotor planning
Apraxia of Speech (AOS)
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neuromuscular execution
Dysarthria
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What are the two main functions that are affected by AOS?