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Temporomandibular (TMJ) Capsular Pattern
Limitation of mouth opening.
Cervical Spine Capsular Pattern
Side flexion and rotation equally limited, then extension.
Glenohumeral (Shoulder) Capsular Pattern (think "the whip" lol)
Lateral rotation (ER) > Abduction > Medial rotation (IR).
Sternoclavicular Joint Capsular Pattern
Pain at end range, especially horizontal adduction and full elevation.
Acromioclavicular Joint Capsular Pattern
Pain at end range, especially horizontal adduction and full elevation.
Ulnohumeral (Elbow) Capsular Pattern
Flexion > Extension.
Radiohumeral Joint Capsular Pattern
Flexion > Extension > Pronation > Supination.
Proximal Radioulnar Joint Capsular Pattern (think turning doorknob)
Supination and pronation equally limited.
Distal Radioulnar Joint Capsular Pattern
Full ROM with pain at extremes of pronation and supination.
Radiocarpal (Wrist) Capsular Pattern
Flexion and extension equally limited.
Carpometacarpal (Thumb) Capsular Pattern
Abduction > Extension.
Thoracic Spine Capsular Pattern
Side flexion and rotation equally limited, then extension.
Lumbar Spine Capsular Pattern
Side flexion and rotation equally limited, then extension.
Hip Capsular Pattern (think karate kick)
Flexion > Abduction > Medial rotation (IR).
Knee Capsular Pattern
Flexion > Extension.
Talocrural (Ankle) Capsular Pattern
Plantarflexion > Dorsiflexion.
Talocalcaneal (Subtalar) Capsular Pattern
Limitation of range of motion.
Axillary Nerve — Roots & Innervation
Roots: C5-C6
Motor: Deltoid, Teres Minor
Sensory: Regimental badge area (lateral shoulder)lower half of deltoid and upper part of long head of triceps
what are the causes of axillary nerve damage and its effects
Axillary nerve can be injured due to:
- Dislocation of shoulder
- Fracture of surgical neck of humerus
► Effects:
- "Square shoulder" from deltoid muscle atrophy
- Sensory loss over lower half of deltoid
Musculocutaneous Nerve — Roots & Innervation
Roots: C5-C7
Motor: Biceps Brachii, Brachialis, Coracobrachialis
Sensory: Lateral forearm
Radial Nerve — Roots & Innervation
Roots: C5-C8,T1
Motor: Triceps, Anconeus, Brachioradialis, Extensor Carpi Radialis Longus/Brevis, Supinator, Extensor Digitorum, Extensor Indicis, Extensor Digiti Minimi, Extensor Carpi Ulnaris, Abductor Pollicis Longus, Extensor Pollicis Longus/Brevis
Sensory: Posterior arm, posterior forearm, dorsal web space between thumb and index finger
Radial Nerve — Clinical Presentation / Sign
Deformity: Wrist Drop (inability to extend wrist and digits)
Common Injury Site: Radial groove of humerus (mid-shaft humeral fracture), compression at Axilla ("Saturday night palsy")
Median Nerve — Roots & Innervation
Roots: C5-8, T1
Motor: Pronator Teres, Flexor Carpi Radialis, Palmaris Longus, Flexor Digitorum Superficialis, Flexor Digitorum Profundus (lateral 1/2), Flexor Pollicis Longus, Pronator Quadratus, LOAF muscles (Lumbricals 1 & 2, Opponens Pollicis, Abductor Pollicis Brevis, Flexor Pollicis Brevis)
Sensory: Palmar surface of thumb, index, middle, and radial half of ring finger
Median Nerve — Clinical Presentation / Sign
Deformity: Ape Hand (at rest; loss of thumb opposition) and Hand of Benediction (when attempting to make a fist)
Common Injury/Entrapment Sites: Carpal Tunnel, Pronator Teres
Anterior Interosseous Nerve (Branch of Median) — Motor & Clinical Sign
Motor: Flexor Pollicis Longus, Pronator Quadratus, FDP (lateral half)
Clinical Sign: Loss of "OK" sign (pincer grasp fails due to lack of DIP/IP flexion in index finger and thumb)
Sensory: NONE (pure motor)
Ulnar Nerve — Roots & Innervation
Roots: C8-T1
Motor: Flexor Carpi Unaris, Flexor Digitorum Profundus (medial 1/2), Hypothenar muscles, Lumbricals 3 & 4, Adductor Pollicis, Dorsal Interossei (DAB), Palmar Interossei (PAD)
Sensory: Ulnar 1.5 digits (ulnar half of ring finger, small finger) palmar and dorsal aspects
Ulnar Nerve — Clinical Presentation / Sign
Cubital Tunnel Syndrome
- Compression at cubital tunnel at elbow
- Paralysis of flexor carpi ulnaris, flexor digitorum profundus (ulnar half), hypothenar
eminence, interossei, and the third and fourth lumbrical muscles
- All sensations affected
- Inability to grip paper
► Guyon's Canal Syndrome
- Sensory changes and progressive weakness of muscles innervated distal to the site
Ulnar Claw hand
- Hyperextension at 4th,5th MCP, Flexion at 4th, 5th IP due to weakness of lumbricals and interossei
Long Thoracic Nerve — Roots, Innervation & Sign
Roots: C5-C7
Motor: Serratus Anterior
Clinical Sign: Scapular Winging (medial border of scapula lifts off thorax, especially with forward arm elevation/pushing)
Suprascapular Nerve — Roots & Innervation
Roots: C5-C6
Motor: Supraspinatus, Infraspinatus
Common Entrapment Site: Suprascapular notch or spinoglenoid notch
subscapular nerve-roots and innervation
roots:C5-C6
Motor supply
► Teres Major (Lower Subscapular Nerve)
► Subscapularis
lateral pectoral nerve
C5-C7
motor supply: pec major and clavicular fibers
medial pectoral nerve
C8, T1
motor supply: pec minor
Thoracodorsal Nerve — Roots & Innervation
Roots: C6-C8
Motor: Latissimus Dorsi
dorsal scapular nerve
C5
motor supply: rhomboid major, minor and levator scapulae
nerve to subclavius
C5-C6
motor supply:
subclavius
spinal accessory nerve motor supply
traps, and SCM
Femoral Nerve — Roots & Innervation
Roots: L2-L4
Motor: Iliopsoas, Pectineus, Sartorius, Quadriceps Femoris (Rectus Femoris, Vastus Lateralis/Intermedius/Medialis)
Sensory: Anterior thigh, medial lower leg and ankle via Saphenous Nerve
Femoral Nerve — Clinical Presentation
Functional Deficit: Weakness in hip flexion and knee extension
Gait Deviation: Knee buckling during stance phase or excessive forward trunk lean/hand-on-thigh compensation
Obturator Nerve — Roots, Innervation & Deficit
Roots: L2-L4
Motor: Adductor Longus, Adductor Brevis, Adductor Magnus (anterior part), Gracilis, Obturator Externus
Sensory: Medial thigh
Deficit: Difficulty with leg adduction and scissoring gait compensations
Superior Gluteal Nerve — Roots, Innervation & Sign
Roots: L4-S1
Motor: Gluteus Medius, Gluteus Minimus, Tensor Fasciae Latae (TFL)
Clinical Sign: Trendelenburg Sign / Gait (contralateral pelvis drops during single-leg stance on affected side)
Inferior Gluteal Nerve — Roots, Innervation & Deficit
Roots: L5-S2
Motor: Gluteus Maximus
Deficit/Gait: Gluteus Maximus Lurch (posterior trunk lean at heel strike/initial contact to keep center of mass posterior to hip)
nerve to piriformis
L5-S2
motor supply: piriformis
nerve to quadratus femoris
Nerve Roots: L4-S1
Motor supply
► Gemellus inferior
► Quadratus Femoris
nerve to obturator internus
Nerve Roots: L5-S2
Motor supply
► Gemellus superior
► Obturator Internus
Sciatic Nerve — Roots & Innervation
Roots: L4-S3
Motor: Hamstrings (Semitendinosus, Semimembranosus, Biceps Femoris), Adductor Magnus (posterior part)
Branches: Splits into Tibial and Common Peroneal (Fibular) nerves at the popliteal fossa
Tibial Nerve — Roots & Innervation
Roots: L4-S3
Motor: Gastrocnemius, Soleus, Plantaris, Popliteus, Tibialis Posterior, Flexor Digitorum Longus, Flexor Hallucis Longus
Sensory: Sole of the foot via Medial and Lateral Plantar nerves
Tibial Nerve — Clinical Presentation
Deficit: Loss of plantarflexion and toe flexion, loss of sensation on plantar foot
Gait: Lack of push-off / terminal stance phase
Entrapment Site: Tarsal Tunnel (behind medial malleolus)
Common Peroneal (Fibular) Nerve — Roots & Divisions
Roots: L4-S2
Divisions: Deep Peroneal Nerve, Superficial Peroneal Nerve
Common Injury Site: Fibular head / neck (e.g., from tight casting, lateral leg trauma)
Deep Peroneal (Fibular) Nerve — Innervation & Presentation
Motor: Tibialis Anterior, Extensor Digitorum Longus, Extensor Hallucis Longus, Peroneus (Fibularis) Tertius, Extensor Digitorum Brevis
Sensory: Web space between 1st and 2nd toes
Deformity/Gait: Foot Drop, High Steppage Gait, Slap Foot
Superficial Peroneal (Fibular) Nerve — Innervation & Presentation
Motor: Peroneus (Fibularis) Longus, Peroneus (Fibularis) Brevis
Sensory: Anterolateral lower leg and dorsum of foot (except 1st web space)
Deficit: Loss of foot eversion; varus deformity risk
Lateral Femoral Cutaneous Nerve — Roots & Presentation
Roots: L2-L3
Motor: NONE (purely sensory)
Sensory: Anterolateral thigh
Condition: Meralgia Paresthetica (compression under inguinal ligament due to tight clothing, pregnancy, obesity)
Sural Nerve — Roots, Type & Innervation
Roots: S1-S2
Type: Purely sensory (formed by branches from Tibial and Common Peroneal nerves)
Sensory: Posterolateral calf, lateral foot, lateral ankle
Scapular Elevation — Muscles
Upper trapezius, levator scapulae, rhomboids
Scapular Protraction — Muscles
Serratus anterior, pectoralis minor and major
Scapular Depression — Muscles
Serratus anterior, pectoralis minor and major, lower trapezius, latissimus dorsi
Scapular Retraction — Muscles
Trapezius, rhomboids
Scapular Downward Rotation — Muscles
Levator scapula, rhomboids, pectoralis minor
Scapular Upward Rotation — Muscles
Serratus anterior, trapezius (upper and lower fibers)
Neuropraxia (Sunderland 1°) — Pathology & Causes
Segmental demyelination from mild ischemia due to nerve compression or traction; action potential is blocked locally.
Neuropraxia (Sunderland 1°) — Signs & Recovery
Pain, minimal muscle wasting, numbness, and weakness. Recovery occurs within minutes to days.
Axonotmesis (Sunderland 2° & 3°) — Pathology & Causes
Loss of axonal continuity with intact connective tissue; Wallerian degeneration occurs distal to lesion.
Axonotmesis (Sunderland 2° & 3°) — Signs & Recovery
Muscle wasting, complete loss of function, sensation returns before motor. Regenerates 1 mm/day over months.
Neurotmesis (Sunderland 3°, 4°, & 5°) — Pathology & Causes
Complete severance of nerve fiber and connective tissue coverings; caused by severe trauma or avulsion.
Neurotmesis (Sunderland 3°, 4°, & 5°) — Signs & Recovery
Anesthesia (no pain), muscle wasting, complete functional loss. Recovery takes months and requires surgery.
What are the three classifications of peripheral nerve injuries?
Neuropraxia → Axonotmesis → Neurotmesis (least severe to most severe).
Which peripheral nerve injury has the best prognosis?
Neuropraxia.
Which peripheral nerve injury requires surgery?
Neurotmesis.
What structures are damaged in neuropraxia?
None—the nerve remains intact; there is no nerve degeneration.
What causes neuropraxia?
Compression causing ischemia.
Which nerve injury is commonly seen in carpal tunnel syndrome?
Neuropraxia.
What is the prognosis for neuropraxia?
Excellent if compression is removed; recovery occurs within weeks to months.
What structures are damaged in axonotmesis?
Axon, myelin, and varying amounts of connective tissue (endoneurium, perineurium, epineurium).
What commonly causes axonotmesis?
Crush injuries or traction injuries.
What determines prognosis after axonotmesis?
The amount of connective tissue damage.
What is the rate of axonal regeneration?
1-3 mm/day (≈1 inch/month).
What structures are damaged in neurotmesis?
Complete severing of the axon, myelin, and all connective tissue.
What are the clinical findings with neurotmesis?
Complete loss of function requiring surgical repair.
After axonal regeneration, do regenerated axons remyelinate back to preinjury levels?
No.
Why is incomplete remyelination clinically important?
It decreases nerve conduction velocity and impairs movement speed and coordination.
What is collateral sprouting?
Intact axons reinnervate denervated muscle fibers.
What muscle fiber change commonly occurs with collateral sprouting?
Type I fibers often convert to Type II fibers.
Mononeuropathy involves what?
One peripheral nerve.
What is an example of a mononeuropathy?
Carpal tunnel syndrome.
What is mononeuropathy multiplex?
Two or more individual nerves affected without a polyneuropathy pattern.
What common diseases are associated with mononeuropathy multiplex?
Diabetes, renal disease, chronic alcoholism.
What is radiculopathy?
Involvement of nerve root(s).
What is plexopathy?
Involvement of the brachial or lumbosacral plexus.
What are the three biggest NPTE risk factors for polyneuropathy?
Diabetes, renal failure, alcohol abuse.
Name autoimmune diseases associated with polyneuropathy.
Guillain-Barré syndrome, Sjögren syndrome, lupus.
What hereditary disorder commonly causes polyneuropathy?
Charcot-Marie-Tooth disease.
Approximately what percentage of polyneuropathies are idiopathic?
25%.
Segmental demyelination primarily damages what?
Myelin.
Which condition is a classic example of segmental demyelination?
Guillain-Barré syndrome.
Can remyelination occur after segmental demyelination?
Yes, if treated.
Axonal degeneration progresses in what direction?
Distal → proximal.
Which disease commonly causes axonal degeneration?
Alcoholic neuropathy.
Which polyneuropathy affects both myelin and axons?
Diabetic polyneuropathy.
What sensory distribution is typical in polyneuropathy?
Stocking-and-glove pattern (distal → proximal).
Which sensory fibers should always be tested in polyneuropathy?
Small fibers (pain, temperature) and large fibers (proprioception, kinesthesia).
What autonomic signs should be screened for in polyneuropathy?
Dry skin, warmth, edema, orthostatic hypotension, vasodilation/loss of vasomotor tone.