Neurogenic Communication Disorders

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24 Terms

1
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RHD (right hemisphere damage)

  • visuospatial deficits/visual neglect

  • anosognosia

  • prosodic, inferencing, and discourse deficits

  • sustained & selective attentions deficits

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frontal lobe damage

  • executive function deficits (planning, problem solving, reasoning)

  • memory loss, consciousness, impulse control

  • motor planning candor programming

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parietal lobe damage

  • sensory deficits

  • difficulty reading/writing, spatial relationships

  • mathematical deficits

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temporal lobe damage

  • deficits in auditory perception/sensation/integration

  • categorization difficulties, memory & recognition deficits

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Broca’s Aphasia

  • nonfluent

  • intact comprehension

  • impaired repetition

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Wernicke’s Aphasia

  • fluent

  • impaired comprehension

  • impaired repetition

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Transcortical Motor Aphasia

  • Nonfluent

  • intact comprehension

  • intact repetition

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Transcortical Sensory Aphasia

  • Fluent

  • Impaired comprehension

  • Intact repetition

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Conduction Aphasia

  • Fluent

  • Intact comprehension

  • Impaired repetition

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Mixed Transcortical Aphasia

  • Nonfluent

  • Impaired comprehension

  • Intact repetition

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Global Aphasia

  • Nonfluent

  • Impaired comprehension

  • Impaired repetition

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Anomic Aphasia

  • only deficit is word retrieval

  • Fluent

  • Intact comprehension

  • Intact repetition

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Broca’s Aphasia site of damage

left frontal lobe in the posterior inferior frontal gyrus (left hemisphere)

<p>left frontal lobe in the posterior inferior frontal gyrus (left hemisphere)</p>
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Wernicke’s Aphasia site of damage

left posterior superior temporal gyrus

<p>left posterior superior temporal gyrus</p>
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Transcortical Motor Aphasia site of damage

supplementary motor cortex (anterior to Broca’s area)

<p>supplementary motor cortex (anterior to Broca’s area) </p>
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Transcortical Sensory Aphasia site of damage

posterior to wernicke’s area at the temporo-occipital parietal junction

<p>posterior to wernicke’s area at the temporo-occipital parietal junction </p>
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Conduction Aphasia site of damage

supramarginal gyrus of parietal lobe (superior to wernicke’s area)

<p>supramarginal gyrus of parietal lobe (superior to wernicke’s area)</p>
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Global Aphasia site of damage

damage to multiple areas & can vary

<p>damage to multiple areas &amp; can vary </p>
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Flaccid Dysarthria

  • lower motor neuron

  • Primary deficit is weakness

  • hypernasality, nasal emissions, imprecise consonants, breathy, wet, hoarse, monopitch/loud, slow and slurred DDKs, tongue fasciculations

  • etiologies: stroke, myasthenia gravis,

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Spastic Dysarthria

  • Bilateral UMN

  • strain-strangled voice, harsh, low pitch, monopitch, excess & equal stress, imprecise consonants

  • etiologies: CP, bilateral strokes, TBI

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Ataxic Dysarthria

  • Cerebellum

  • Primary deficit is incoordination

  • slow & slurred speech, irregular articulatory breakdowns, distorted vowels, excess & equal stress, prolonged phonemes

  • etiologies: cerebellar stroke/injury/atrophy

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Hypokinetic Dysarthria

  • Basal ganglia (dopamine depletion; extrapyramidal tract)

  • Primary deficit is rigidity and decreased ROM

  • rapid rate/short rushes of speech, DDKs fast & imprecise, reduced loudness, reduced stress

  • etiologies: Parkinson’s

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Hyperkinetic Dysarthria

  • Basal Ganglia (excess dopamine; extrapyramidal)

  • Primary deficit is involuntary movement

  • involuntary movements at rest, voice stoppages, prolonged phonemes, variable rate, inappropriate silences

  • etiologies: Huntington’s

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Unliateral UMN

  • Unilateral UMN

  • Primary deficit is weakness, incoordination, spasticity

  • unilateral facial weakness, harsh voice, articulatory imprecision, slow rate

  • etiologies: Unilateral CVA