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bereavement
situation in which loved one has died
grief
natural response to the loss of a loved one, associated with physical, emotional, and cognitive symptoms, evolved over time eventually the person is able to adapt and integrate the loss and reengage with life (process of mourning)
What is mourning influenced by?
cultural/religious/social rituals, behaviors, and practices
acute grief/uncomplicated bereavement
-time-limited, unique/individualized process
-involves social, religious, cultural norms
-severity depends on suddenness and circumstances of death, relationship to the deceased
Clinical manifestations of acute grief
intense focused thoughts/memories of deceased accompanied by sadness and yearning (hallmark), emotional pain (sorrow, remorse, frustration, anger, guilt, anxiety), somatic physical distress, loss of interest and feeling disconnected (social withdrawal)
timing of acute grief
begin within first 2 months of loss, starts to resolve/re-engage by 6-12 months
Grief treatment
reassurance, self-care, emotional support through family and friends, support groups, regular check-ins with validation of grief experience and monitoring for signs of function impairment, persistent inability to adapt, or suicidal ideation
prolonged grief disorder
pervasive, intense, and functionally impairing symptoms after the loss
What does incidence of prolonged grief disorder depend on?
severity and relationship of death
Risk factors for prolonged grief disorder
sudden/violent death, older age, females at greater risk, low socioeconomic status, low educational level, affected by racism/inequity/disparities, psychiatric history, military personnel, bereaved multiple times or prior traumatic loss, inadequate social support
According to DSM-5, to diagnose prolonged grief disorder the grief response exceeds ___-___ months
6-12
Treatment for prolonged grief disorder
self-care, psychotherapy (grief-focused is 1st line), support groups, medications if underlying co-morbidities
Trauma
exposure to actual or threatened death, serious injury, or sexual violence
What are ways in which a person can experience trauma?
directly experiencing the event, witnessing it in person as it occurs to others, learning it happened to a close family member or friend, repeated indirect exposure to details of traumatic events (1st responders, military)
Stressors for children/teens (adjustment disorder)
family problems/fighting within family, problems in school, sexuality or identity issues
Stressors for adults (adjustment disorder)
death of a loved one, divorce/relationship problems, person or loved one getting a serious illness, moving to a new home or city, change in financial situation/money problems, problems at work
Adjustment disorder clinical manifestations
depressed mood, anxiety, aggression/vandalism, unable to do usual tasks, trouble concentrating or thinking clearly, sleep disturbances/ bad dreams/ insomnia, acting recklessly (drinking, drugs, affairs), suicidal thoughts
Timing of adjustment disorder
Within 3 months of identifiable stressor, lasts less than 6 months
Diagnosis of adjustment disorder is categorized by major _______ symptom
associated
Treatment for adjustment disorder
psychotherapy (CBT)/ counseling is 1st line, self-care, screen for suicidal ideation and substance abuse
Reactive attachment disorderr
severe neglect, repeated caregiver changes, or institutional rearing during early childhood
reactive attachment disorder etiology
pathogenic caregiving environment
Clinical manifestations of reactive attachment disorder
significant social and emotional impairment, pervasive absence of attachment behaviors (fail to see or accept comfort), marked withdrawal or hypervigilance in social situations, limited positive affect unexplained irritability, sadness, or fearfulness during nonthreatening interactions
timing of reactive attachment disorder
symptoms must be evident before age 5 years
What does reactive attachment disorder have increased risks for?
comorbid psychiatric and psychosocial problems in adolescents, mood disorders, behavioral problems, suicidal ideation, and substance abuse
treatment for reactive attachment disorder
provide stable and nurturing environment, early ID And intervention are critical
acute stress disorder
exposure to actual or threatened death, serious injury, or sexual violence
Clinical manifestations of acute stress disorder
intrusive thoughts/memories, hyperarousal/hypervigilance, avoidance of trauma reminders, depressed mood, dissociation
Timing of acute stress disorder
occurring within 3 days to 1 month following trauma, greater than 1 month is PTSD
Acute stress disorder treatment
trauma focused CBT is first line, ensure safety (SI), self care, meds if comorbid condition
Post-traumatic stress disorder risk factors
adverse childhood experiences (ACEs), less education, lower socioeconomic status, personal/family psychiatric history, females more at risk, poor social support, intentional trauma more risk than unintentional trauma, physical injury as part of trauma event
Clinical manifestations of PTSD
traumatic event, flashbacks (hallmark), hyperarousal/hypervigilance, avoidance of environments or situations that remind them of trauma, depressive symptoms
Timing for PTSD
symptoms greater than 1 month
PTSD diagnosis criteria
patient older than age 6, disturbance causes clinically significant distress or impairment in social or occupational functioning, disturbance not attributable to substance abuse or other medical/psychological condition
Subtypes and complex PTSD
recurrent severe trauma, dissociative identity disorder
Treatment goals of PTSD
-maintain safety while reducing distressing symptoms, avoidant behaviors, and anxiety
-improve adaptive and psychosocial functioning to lessen risk of relapse
Treatment for PTSD
trauma focused psychotherapy is 1st line (CBT, exposure-based therapy, eye movement desensitization and reprocessing- EMDR), self care, SSRI if comorbidities, alpha 1 blocker (prazosin) given before bed
Prognosis of PTSD
the sooner the therapy the better but not beneficial to begin immediately after trauma, 1/3 recovered after 1 year, 1/3 still symptomatic at 10 years