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what is the origin of shoulder elbow hand and wrist disorders most commonly
the cervical spineee
Cervical n roots are vulnerable to injury bc they are horizontal
C1 and C2 subluxations can lead to weak what
weak atlantooccipital ligaments
where does the vertebral artery enter thru
C6 TVP foramen
(and ascends beneath the atlantooccip ligament)
what is cervical instability
traumatic injury worsened with upright position but relieved by head support
ADI <3mm adults is normal and <5mm is normal for kids
cervical radiculopathy is what
nonspecific neck pain that radiates to one arm
facet syndrome/muscle strain/ligament sprain is what
neck pain that is worse with movement
cervical myelopathy is what
bilateral neck pain into upper extremities, sometimes including balance issues
upper cross syndrome is what
pain with specific posture changes, (fixed w other postures)
AROM flexion should be what
60 degrees
AROM extension should be what
75 degrees
AROM lateral flex should be what
45 degrees
AROM rotation should be what
80 degrees
make sure pt is supine and only use one inclinometer for this one :)
rust sign
pt spontaneously grasps their head with both hands to take weight off their cervicals.
due to severe pain, RA, fractures, severe subluxations
(check ADI immediatelyyyy)

myelopathy is due to
cord injury!! always pathological (spurs, central disc herniations, tumors, dislocations)
myelopathy S/s
UMN findings! hyperreflexia, spastic extremities, babinski sign, bowel/bladder issues
Lhermitte sign
pt seated, dr passively flexes their head.. cervical myelopathy IF shooting shock like pain into LE/UE

Libman sign
dr puts thumb pressure on pts mastoid processes until pt is too uncomfortable
used to determine pt pain threshold

IVF encroachment is
displacement/irritation of the nucleus, cartilage, apophyseal fragments, annular tissue fragments beyond IVD space
--> obstructed foramina which can irritate or compress the nerve root
MC IVF to be encroached
C6 and C7
MC cuase of IVF encroachment
lateral canal stenosis (osteophytes or ligmentum flavum hypertrophy
cervical disc herniations
s/s IVF encroachment
neck pain and distal paresthesia in a DERMATOMAL distribution
weak muscles supplied by the NR
decreased DTR
muscle fasciculations
worse radiating pain w movments
bakody sign
pt seated putting palm on top of their own head
+= radiating pain DECREASES meaning cervical n root compression BECAUSE elevated suprascapular nerve relieves traction of lower trunk of brachial plexus

what is a better test, spurling or bakody?
BAKODY. no bopping pts head so less pain, and is just as reliable
foraminal compression test
pt rotates head from side to side
dr pushes down on head at neutral and at both rotated sides
+=pain reproduced
LOCAL= foraminal encroachment
RADICULAR= nerve root compression
jackson compression
pt is seated and rotates head. then pt laterally bends and dr presses there and at neutral
+=increased pain.. SOL, subluxation, edema, DDD, tumor, disc herniation

if you perform jacksons compression and find local contralateral pain, what is your diagnosis
muscle strainnnnn
if ipsilateral, can be IVF encroachment (local) or nerve root involvement if radiates
spurling test
pt is seated and rotates head, then laterally flexes and dr pushes down, then laterally flexes and extend head with push
no pain? then dr bops in neutral position
no pain? then dr bops them with lat flex/extended position
+=radicular pain-NR compression
+=local pain=facet
(not for muscle)

distraction test
pt seated, doc does upward pressure to lift pts head
+= relief of radiculat symtoms
= NR compression, facet capsulitis, IVF encroachment
increased pain? muscle spasm

valsalva maneuver
have pt bear down abdominally
+= radiculat pain increased bc increased intrathecal pressure
NR compression by SOL, disc herniation, tumor, osteophyte

dejerine sign
pt coughs, sneezes, strains, defectates
+= increased radicular s/s bc mechanical obstruction of spinal fluid flow
SOL causes nerve root compression, disc nernations -> IVD spinal cord tumor or compression fractures

do intervertebral veins have valves?
NAHHHHRRRR
pressure increases and blocks venous flow which distends veins and forces the dura toward the spinal cord which causes pain w dejerine sign
how should dejerine positive pts cough/sneeze
with knees bent and leaned against a wall
an unexpected absence of dejerine after having it positive previously is a sign of
fragmentation of a disc
naffzinger test
pt seated while dr holds pressure over both jugular veins for 30-40seconds, pt then coughs deeply
+= pain along NR distribution or local pain = sprain/strain
SOL in spinal column, mC spinal meningioma
***CONTRAINDICATED FOR GERIATRICS*******A
swallow test
pt is seated and swallows water
+= difficulty swallowing or pain
SOL, disc protrusion, tumors, osteophytes anteriorly, lig sprains, muscle strains, fractures
what are brachial plexus lesions caused by
MC traction injuries on motorcycles when neck laterally flexes away from the involved side (+ ipsilateral shoulder depression)
ORRRR
weak deltoid, supra/infraspinatus, biceps
brachial plexus tension test
pt holds hands behind head and dr pulls elbows back while pt flexes head
+= radicular pain= NR involvement

erbs palsy
upper brachial plexus (C5,6,7)
difficult births with forceps or vacuums
waiters tip deformity

klumpkes palsy
lower brachial plexus (C8,T1)
breech births, falling and trying to catch yourself by grabbing something
claw hand

upper limb tension test
pt is supine..
1) depress scapula
2) abduct shoulder
3) supinate forearm, wrist/finger extension
4) shoulder externally rotate
5) elbow extension
6) contralat lat bend
7) ipsilat lat bend
+=pain in first 6 steps OR decreased pain in 7th step

shoulder depression test
pt is seated, dr pushes shoulder down and then pt laterally bends neck away
+= radicular pain
brachial plexitis, ADHESIONS OF DURAL SLEEVES, spinal nerve root involvement

bikele test
abduct shoulder to 90 degrees and extend arm behind
+= resistance or increased radicular pain
brachial plexus neuritis, meningitis
brudzinskis test
pt is supine with straight legs, dr passively flexes head
+=BOTH knees buckle- meningeal irritation

kernig sign
pt supine, dr bends one knee to 90 and then straightens it upward, positive if other knee bends = meningeal irritation

o'donoghue maneuver
pt is seated, do all AROMs actively then passively
+= pain AROM?- muscle strain
+=pain PRROM?-ligament sprain

soto hall test
pt is supine and dr puts hands on pts sternum while passivley flexing them
+= local pain in posterior spine
fracture, subluxation, exostoses, disc lesion, strain/sprain
meningeal irritation if knees buckle!!

mild muscle strain
low grade inflammation and minimal swelling/edema
moderate muscle strain
laceration of fibers, hemorrhaging into surrounding tissues, swelling and edema
severe muscle strain
complete disruption of motor units, tendon pulls apart from bone
mild ligament sprain
only a few fibers severed
moderate ligament sprain
more tearing but not complete
sever ligament sprain
complete tear from attachment
sprain/fracture ligament
ligament attachment pulls piece of bone with it (avulsion)
TOS is what
the neurovascular compression of the brachial plexus or subclavian-axillary vessels as they exit the thoracic cavity and enter the UE

neurologic TOS
LOWER trunk of the brachial plexus
abnormal nerve stretch/compression causes it
MC women with DROOPING shoulders
vascular TOS
subclavian artery and vein
unilateral arm swelling
MC in men
what compresses the brachial plexus
1st rib (floor) can trap brachial plexus btwn other structures

TOS presentation
diffuse arm symptoms, pain, numb, tingling, weak grip strength
worse w overhead activities
TOS is MC in
4th/5th digits (C8,T1)
causes of TOS
trauma, posture, tight scalenes, tight pecs, cervical ribs
allen test
pt seated, pumps hands and squeezes
dr occludes radial and ulnar artery then releases one to see refill <5sec normal
+= >5sec refill= vascular occlusion of rad/uln or TOS if both

what runs btween the scalene muscles
the subclavian artery! it then becomes the axillary artery then becomes the brachial artery which breaks into ulnar and radial arteries at the cubital fossa

if blood supply to the hand is occluded it can cause
compartment syndrome
allen maneuver
palpate radial pulse w pts arm at a 90 degrees up. have pt rotate head away
+= pulse lessens/gone=TOS

costoclavicular maneuver
AKA edens test
pt seated and dr palpates bilateral radial pulse and extends arms behind them and pt flexes head forward
+= decreased pulse - TOS

halstead maneuver
dr palpates radial pulse and applies downward traction on arm, pt extends neck and looks up
+= decreased pulse, TOS

reverse bakody sign
pt seated, puts palm on top of head at elbow level w head
+= pain increases in arm/hand
= TOS due to INTERSCALENE COMPRESSION

Roos test
pt is seated and abducts arms to 90 degrees like goal posts
pt holds for one minute then pumps hands for 3min
+= weak/lowered arm or reproduced symptoms-TOS

adson test
palpate radial pulse and have pt turn head TOWARD side and inhale hold for 10sec
+= decreased pulse- TOS bc ANTERIOR SCALENE HYPERTROPHY

modified adson test
palpate radial pulse and have pt turn head AWAY from side and inhale hold for 10sec
+= decreased pulse-TOS bc MIDDLE SCALENE hypertophy (or cervical rib)

hyperabduction maneuver
aka Wrights test
pt seated w arms at sides
palpate radial pulse then abduct arms to 180degrees
+= decreased pulse at DIFFERENT angles
(lower is problematic side)
TOS bc HYPERABDUCTION SYNDROME

superficial reflexes are
motor response to scraping the skin. Either present or absent, asymmetrical is abnormal
they have to ascend to the brain then descend as motor neurons
cremasteric reflex
L1/L2 and ilioinguinal and genitofemoral nerves
stroke inner thigh S->I and ipsilateral testicle should elevate
if not, hydrocele or varicocele

gluteal reflex
L4-S2 and inferior gluteal nerve
stroke skin over glute max and see contraction of muscle
anal wink
S2-S5 and hemorrhoidal nerve
stroke skin of perianal area
contraction of external sphincter= normal
assoc w cauda equina syndrome, SOL
what are pathologic relfexes
superficial and deep reflexes controlled by motor cortex or pyramidal tracts
LMN released/inhibited= primitive responses that are pathological in adults (normal up to 6mo-2yrs)
hoffman sign
corticospinal tract ABOVE C5. flick middle finger down
+= index and thumb come together like Ok sign
=UMN lesion above C5

tromner sign
corticospinal tract above C5
tap VOLAR surface of middle finger upward
+= flexion of fingers and adduction of thumb
UMNL above C5

oppenheim sign
corticospinal tract
stroke anterior tibia S-> I
+=babinski sign-UMNL

babinski sign
corticospinal tract
stroke foot in a 7 shape
+=dorsiflexed big toe and flared other toes
-UMNL

gordons sign
corticospinal tract
squeeze calf
+=babinski, UMNL

rossolimo sign
corticospinal tract
tap ball of foot/tips of toes
+=PLANTARFLEXION of toes - UMNL

chaddock sign
corticospinal tract
lateral malleolus to pinky toe stroke
+=babinski sign-UMNL

schaefer sign
corticospinal tract
squeeze achiles
+=babinski sign-UMNL
pupillary light reflex what is aff what is efferent
aff- Cn2
Eff-Cn3

ciliospinal reflex aff and efferent
aff= cervical pain fibers and CN5
eff-cervical sympathetics
painful side of neck w pinching skin->dilate pupils 1-2mm on painful side
absent? cervical sympathetics problem
HORNER SYNDROME (myosis, ptosis, anhydrosis)

oculocardiac reflex
aff- CN 5
eff- CN 10
press thumbs into eyeballs... HR and BP should decrease
carotid sinus reflex
aff- CN 9
Eff- CN 10
press on carotid sinus
decreased HR and BP
bulbocavernous reflex
S3-S4
stroke/pinch/prick glans of penis
cx of muscle/urethral contraction, anal sphincter cx
muscle reflex dysfunction
decreased reflex and decreased strength
neuromuscular junction dysfunction
stretch reflex decreased and parallel strength loss
peripheral nerve dysfunction
stretch reflex decreases and minimal weakness
nerve root dysfunciton
stress reflexes decreased in proportion to contribution of nerve root
superficial reflexes NOT depressed
cord/brainstem dysfunction
stretch reflexes HYPOactive at level of lesion and HYPERactive below the lesion
cerebellum dysfunction
lesions not associated with stretch reflex changes
not visible normally
basal ganglia dysfuncion
no consistent DTRs or superficial reflex changes
DEMENTIA!
cerebral cortex dysfunction
unilateral diseases affect motor cortex-UMNL
increased muscle stretch reflex
decreased abdominal/cremasteric reflex on CONTRALAT side
emotional expression of reflexes... cry/laugh inappropriatey "PSEUDOBULBAR"
hemicord lesion
AKA brown sequard lesion
1) damage to LATERAL corticospinal tract? = ipsilateral UMN weakness
2) damage to DC?= ipsilateral loss of vibration and joint position
3) damage to ANTERIOR-LATERAL spinothalamic tract?= contralateral loss of pain/temp

transverse cord lesion
partial/complete interruption to ALL motor/sensory pathways BELOW the lesion... weakness/reflex loss
trauma, tumors, MS, transverse myelitis

anterior cord lesion
1) anterolateral pathways (spinothalamic damage)- pain/temp decrease
2) anterior horn cell damage= LMN weakness below lesion
causes- trauma, MS, ANTERIOR SPINAL ARTERY INFARCT!!!!!!!!!!!!,
incontinence common
