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Intestinal (Bowel) Perforation: Background
Definition: Rupture in the wall of the gastrointestinal tract, allowing enteric contents to leak into the peritoneal cavity.
Also referred to as intestinal or bowel perforation.
It is a life-threatening condition.
Intestinal (Bowel) Perforation: S/SX
Sudden onset
Acute abdominal pain or cramping
Nausea
Vomiting
Chills
Fever
Shoulder pain
Abdominal bloating
GI Bleed Classification: Overt (Visible) Bleeding
Blood is visible in vomitus (emesis) or stool.
Hematemesis: Vomiting bright red blood or "coffee ground" material.
Melena: Black, tarry stool from digested blood—classically from an upper GI source (e.g., esophagus, stomach, duodenum).
Hematochezia: Bright red blood per rectum, commonly from a lower GI source (e.g., colon, rectum).
Nursing implication: Easily detectable; always demands immediate assessment and stabilization.
GI Bleed Classification: Occult (Hidden) Bleeding
Blood loss is not visible to the naked eye—detected only by laboratory testing (e.g., stool guaiac, occult blood testing).
Often stems from slow, small-volume bleeding (ulcers, malignancies, inflammation).
Subtle clinical signs: Fatigue, pallor, iron-deficiency anemia, or a positive stool blood test.
Nursing implication: Requires vigilance for subtle/anemic symptoms; often detected during routine labs or workup for anemia.
Small Bowel Bleeds
These bleeds originate in your:
jejunum (middle section of your small intestine).
Ileum (last section of your small intestine).
Peptic Ulcer Disease
Upper GI Bleed Cause
Most common cause; ulcers in stomach or duodenum may erode blood vessels.
Gastric/Duodenal ulcer & erosion
Upper GI Bleed Cause
Due to H. pylori, NSAIDs, drugs, stress
Esophageal Varices
Upper GI Bleed Cause
Dilated veins in the esophagus, commonly from portal hypertension due to liver cirrhosis; often life-threatening when ruptured.
Neoplasms or Vascular Lesions (rare)
Upper GI Bleed Cause
Tumors or tangled blood vessels that rupture
Mallory-Weiss Tear
Upper GI Bleed Cause
Linear mucosal laceration at the gastroesophageal junction; typically follows vomiting or retching.
Laceration of distal esophagus from forceful vomiting/coughing
Gastritis/Erosive Esophagitis
Upper GI Bleed Cause
Inflammation or erosion from infection (e.g., H. pylori), NSAIDs, alcohol, or severe GERD.
Tissue inflammation from severe reflux or alcohol/irritants can erode and bleed
Diverticulosis/Diverticulitis
Lower GI Bleed Cause
Outpouchings (diverticula) in the colon wall can bleed, or become inflamed/infected (diverticulitis).
Colorectal Cancer or Polyps
Lower GI Bleed Cause
Benign or malignant growths in the colon or rectum; bleeding may be chronic and occult or brisk and visible.
Infectious Colitis
Lower GI Bleed Cause
Fever, tenesmus, abdominal pain, loose/bloody stools; often C. difficile post-antibiotics
Intestinal Ischemia (acute/chronic)
Lower GI Bleed Cause
Decreased blood flow to bowel → pain, nausea, bleeding
Diverticular Hemorrhage
Lower GI Bleed Cause
Top cause of brisk hematochezia (massive bright red bleeding); not always with diverticulitis
Inflammatory Bowel Disease (IBD)
Lower GI Bleed Cause
Includes ulcerative colitis and Crohn’s disease; inflammation damages the intestinal lining, leading to bleeding.
Especially ulcerative colitis—chronic mucosal inflammation, sunken ulcers, bleeding
Hemorrhoids or Anal Fissures
Lower GI Bleed Cause
Hemorrhoids: swollen rectal veins causing painless bright red bleeding
Anal fissures: tears in anal canal leading to painful, bright red blood per rectum
Swollen rectal veins; painless bright red bleeding
Inflammatory Bowel Disease (IBD)
is an umbrella term for two chronic, relapsing inflammatory diseases:
Ulcerative colitis (UC): Recurrent, inflammatory, ulcerative disorder affecting the colon and rectum.
Crohn’s disease (regional enteritis): Acute or chronic inflammatory disorder that can affect any GI segment (mouth to anus), characterized by "skip lesions."
Takeaway
IBD is chronic and relapsing.
Nursing priorities: Manage inflammation, maintain nutrition and hydration, and provide psychosocial support for lifelong adaptation and coping.
IBD: Epidemiology & Risk Factors
More common in industrialized countries.
Incidence is higher among teenagers.
May have a hereditary (genetic) tendency.
Strong positive correlation: Smoking and oral contraceptive use increase risk.
Pathology & Colonoscopy Findings
IBD = Inflammation + Damage: This means IBD causes visible, structural changes seen on colonoscopy.
IBS = Irritation + Sensitivity: In contrast, IBS is functional, with normal-appearing tissue on colonoscopy.
Key Distinction:
Both IBD and IBS cause chronic bowel symptoms, but only IBD results in tissue injury and systemic effects (fever, weight loss, anemia).
Nursing Care Focus: UC and Chron’s
Managing inflammation and pain
Preventing dehydration and malnutrition
Supporting coping and quality of life
Help patients adapt to a lifelong condition
Address body image, anxiety, and social impacts
Postoperative and Procedural Considerations for GI Patients
General GI Surgery: Early ambulation, pain control, monitor for return of bowel function, wound/drain checks, prevent pneumonia/DVT. Alert for signs of peritonitis/obstruction.
Ostomy: Check stoma color/size/moisture, protect skin, fit pouch properly, empty at 1/3–1/2 full, teach patient. Report abnormal stoma findings (dusky, black, active bleeding).
Endoscopic/Radiologic Procedures: NPO 8 hrs, bowel prep for lower GI imaging, monitor for perforation, patient teaching about stool changes or contrast.
Tube Placement (PEG/PEJ/G-tube): External bolster slightly loose, confirm placement before feeding, flush tube routinely, skin/peristomal care, monitor for infection, leakage, granulation/fungal rash.