Breakdown of ATP
ATP - A-P-P-P (1-2 secs) , ATPase → ADP - A-P-P + P
ATP-PC system/alactic system
PC →(creatinekinase) P (phosphate) + C (creatine) + 1 ATP yield (2-10 secs)
takes place in muscle sarcoplasm
lactic acid system/glycolytic system
Glycogen → (GPP) glucose → (PFK) pyruvic acid + 2 ATP yield (10 secs - 3 mins) → (LDH) lactic acid
takes place in muscle sarcoplasm
Aerobic glycolysis
glycogen (in presence of o2) → (GPP) glucose → (PFK) →pyruvic acid + 2 ATP yield → (Coenzyme A) acetyl CoA
takes place in the muscle sarcoplasm
Kreb’s cycle
Acetyl CoA + oxaloacetic acid → citric acid → Co2 + oxaloacetic acid + hydrogen+ + 2 ATP yield
takes place in the matrix of the mitochondria
Electron Transport Chain (ETC)
H+ (carried by NADH → FADH) → cristae folds of mitochondria → H2O + 34 ATP Yield
About ATP-PC system
exothermic, anaerobic, ATP yield = 1:1, high intensity (e.g. long jump), ATP is only usable form of energy to allow us to do work, duration of system = 2 - 10 secs
5 strengths of ATP-PC system
no fatiguing byproducts
PC stored in muscles so readily available to be broken down
not reliant on o2 being present - fast reaction chain
allows performer to do explosive work
anaerobic reaction
2 weaknesses of ATP-PC system
low yield compared to other energy systems
PC gets used up very quickly and stores are limited
About lactic acid system
exothermic, anaerobic, ATP yield = 1:2, high intensity (e.g. 200m running), duration of system = 10 secs - 3 mins
OBLA
onset blood lactate accumulation - lactic acid being produced quicker than it is being removed
4 strengths of lactic acid system
anaerobic
fast reaction chain (no o2)
glycogen is readily available in large quantity in muscles and liver
provides energy to work at high intensity for 10s - 3mins
2 weaknesses of lactic acid system
fatiguing byproduct (lactic acid) - decreases performance intensity
low yield compared to aerobic system
About aerobic system
aerobic, ATP yield = 1:38, low to moderate intensity, duration of system = 3 mins - 2 hrs
3 strengths of aerobic system
provies energy for longer duration
no fatiguing byproducts
high ATP yield
weakness of aerobic system
reliant on o2 - decrease in intensity of exercise
Energy continuum
the relative contribution of each energy system to overall energy production
4 factors that affect interplay of energy systems
intensity of exercise, duration of exercise, recovery periods, fitness levels
EPOC
excess post-exercise oxygen consumption (o2 debt)
4 stages of recovery
pre-exercise state
alactacid stage
lactacid stage
oxygen deficit
Fast alactacid component of recovery
10% of EPOC, 1-4 litres of oxygen required to return body to pre-exercise state, replenishes blood + muscles with O2, takes 3 mins, resynthesises ATP + PC stores, first minute of EPOC - o2 resaturates the blood stream, associating with haemoglobin, within 3 mins - restores oxymyoglobin link in muscles
Slow lactacid component of recovery
EPOC required 5-8 litres of o2 to return body to pre-exercise state, 1hr - up to 24 hrs, provides energy to maintain ventilation + circulation + body temp, removes lactic acid + replenishes glycogen
3 key processes of fast alactacid component
saturates haemoglobin with oxygen to form oxyhaemoglobin and transport it to muscles + restore oxyhaemoglobin in muscle cells
resynthesises ATP
restores PC using energy from aerobic system to put P+C together
5 key processes of slow lactacid component
ventilation (maintains BR, delivers o2, removes CO2)
circulation (carboaminohaemoglobin in plasma and blood carry Co2 back to lungs)
body temp (if body temp remains high, BR stays high to take in more o2)
lactate removal - LA converts into PA →CO2, H2O, ATP (50-75%), glycogen to store in muscles/liver (20%), glucose (5%)
glycogen (from lactate removal, taken in through eating complex carbs)
Warm up - implications for recovery
increases HR, respiratory rate, MR - accelerates use of aerobic system, decreases use of anaerobic system + reduce o2 deficit, limits amount of o2 needed to pay back during EPOC
Active recovery - implications for recovery
maintains respiratory rate + HR to flush muscles + capillaries w o2, requires 40-60% VO2 max = decreases time for lactate removal + decreases time for slow lactacid component
cooling aids - implications for recovery
used post event to lower muscle and blood temp to resting levels, speeds up lactic acid removal, decreases muscle damage, decreases DOMS, reduces MR and demand on slow lactacid component
Intensity of training - implications for recovery
monitor intensity of training using HR specifically to energy systems, high intensity = boosts efficiency of fast alactacid component, low intensity = delays OBLA using aerobic energy production
work:relief ratios - implications for recovery
explosive intensity - work:sleep ratio = 1:3+, high intensity = 1:2, endurance = 1:1/2:1
strategies and tactics - implications for recovery
set plays and low intensity = delays OBLA and fatigue, delay play and low intensity = no lactic acid and ATP+PC acceleration
nutrition - implications for recovery
maximise PC stores (creatine/phosphagen/protein = increased ATP-PC efficiency), maximise glucose + glycogen (complex carbs), LA eased by bicarbonate nitrate = lower o2 costs and enhances o2 w/ blood