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History of caries lesion and information for non/invasive treatment
Reason for interest in clinical appearance
Understanding Disease Process
What is provided by histology of caries
Shape of Lesion
Reflects where biofilm has been allowed to grow
Developmental Pits and Fissures, SES, Root Surfaces
3 clinical sites for caries initiation
Developmental Pits and Fissures
Site most susceptible to caries
Smooth Enamel Surface
Site sheltering cariogenic biofilm
Enamel Prism/Rods/Prism Borders/Striations , Band of Retzius and Surface zone
Basic structures of normal enamel
White Spot
Earliest evidence of caries on SES
Can
Non cavitated caries can or cannot be remineralized?
Retains most of original crystalline framework of enamel rods
What occurs on enamel rods in non cavitated caries
Retained Crystalline Framework of Enamel Rods
Serves as nucleating agents of remineralization
Calcium and Phosphate Ions
Ions from saliva that penetrate enamel surface and precipitate on highly reactive crystalline surfaces of enamel lesions
Fluoride Ions
Enhances precipitation of Calcium and Phosphate Ions in remineralization
More Resistant
Effect of Fluoride ions on remineralized enamel
Fluorapatite
Acid-resistant substance that makes remineralized enamel more resistant
4 Microscopic Zones of Caries
Translucent Zone, Dark Zone, Body of Lesion, Surface of Lesion
Translucent Zone
Innermost zone and the advancing front of caries
Dark Zone
Immediately above Translucent Zone
6% Mineral Loss
Demineralization percentage exhibited in Dark Zone
Body of Lesion
Area of maximum demineralization occupying major portion of the lesion
Striae of Retzius
Structure that is enhanced by Body of Lesion
20-100 um
Thickness of Surface Lesion
Inactive lesion
Where surface lesion is thicker
Active Lesion
Where surface lesion is thinner
10% Mineral Loss
Percentage of demineralization in surface of lesion
Prism Sheaths
This structure is broadened bu the Surface of Lesion making it its characteristic feature