exam 4 study guide

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101 Terms

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Biological Approach

Focuses on the role of genetics, brain chemistry, and physical health in mood disorders.

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Freud’s Psychoanalytic Approach

Emphasizes unconscious conflicts, early childhood experiences, and repressed emotions as causes of mood disorders.

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Beck’s Cognitive Distortion Model

Suggests that negative thought patterns (e.g., all-or-nothing thinking) contribute to depression.

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Lewinsohn’s Reinforcement Model

Proposes that a lack of positive reinforcement leads to withdrawal and depression; Behavioral Activation is a treatment that increases engagement with rewarding activities.

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Learned Helplessness

Explains that individuals learn to feel powerless after repeated exposure to uncontrollable events, leading to depression.

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Klerman and Weissman’s Interpersonal Approach

Focuses on improving interpersonal relationships and communication to reduce depressive symptoms.

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Victor Frankl’s Existential Theory

Suggests that meaninglessness in life can lead to depression; Logotherapy helps individuals find meaning in suffering.

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Principle of Reciprocal Determinism

Explains how behavior, environment, and personal factors interact and influence each other.

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Diathesis-Stress Models

Propose that psychological disorders arise from an interaction between genetic predispositions (diathesis) and stressful life events.

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Cup Analogy

Describes how individuals have a "cup" of stress, and when it overflows (due to stressors or vulnerabilities), mood disorders emerge.

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Akiskal’s Integrative Model of Depression

Combines biological, psychological, and social factors, acknowledging multiple causes for depression and treating it with diverse approaches.

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Akiskal’s Model Treatment Implications

No single therapy is universally best; effective treatments should consider various factors and tailor interventions accordingly.

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NIMH Multisite Study

Compared different psychotherapies for depression, emphasizing that a variety of treatments can be effective depending on the individual.

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Integrative Model of Mood Disorders (Textbook p. 244)

A holistic approach that integrates biological, psychological, and social factors in understanding mood disorders and their treatment.

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Positive Symptoms of Schizophrenia

Symptoms that reflect an excess or distortion of normal functioning, such as hallucinations and delusions.

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Negative Symptoms of Schizophrenia

Symptoms that reflect a reduction or loss of normal functioning, such as lack of motivation or emotional expression.

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Phases of Schizophrenia

Include the prodromal phase (early signs), active phase (full-blown symptoms), and residual phase (symptoms lessen but persist).

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Chronic Schizophrenia Phases

Patients with chronic schizophrenia often cycle between the active and residual phases, with fluctuating severity of symptoms.

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Schizophrenia Spectrum Personality Disorders

Include disorders like schizoid, schizotypal, and paranoid personality disorders, which share traits with schizophrenia, but are less severe.

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Schizophrenia Spectrum Disorders Definitions

Schizoid is characterized by social detachment, schizotypal by odd behaviors and beliefs, and paranoid by persistent distrust.

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Most Similar Schizophrenia Spectrum Disorder

Schizotypal personality disorder is most similar to schizophrenia due to shared symptoms like odd beliefs or magical thinking.

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Type I Schizophrenia

Characterized by positive symptoms such as hallucinations and delusions; considered less severe with better prognosis.

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Type II Schizophrenia

Characterized by negative symptoms like lack of motivation and emotional expression; considered more severe with a worse prognosis.

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Type I vs Type II Terminology

Historically, Type I and Type II were also referred to as "reactive" (Type I) and "process" (Type II) schizophrenia.

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Type I and II Differences

Type I typically responds better to antipsychotic medication, whereas Type II is more resistant to treatment.

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Shared Underlying Disorder of Type I and II

Despite differences, both types are seen as part of the same disorder due to common biological and genetic factors.

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Emil Kraepelin’s View of Schizophrenia

Kraepelin referred to schizophrenia as “dementia praecox” and viewed it as a progressive, degenerative brain disorder.

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Kraepelin’s Contributions

Kraepelin’s work established the foundation for understanding schizophrenia as a distinct mental illness, focusing on its long-term course and prognosis.

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Eugene Bleuler’s Contributions

Bleuler introduced the term "schizophrenia" and emphasized the importance of cognitive and emotional disintegration in the disorder.

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Bleuler’s Four As

The Four As (associative loosening, affective blunting, ambivalence, and autism) describe the core symptoms of schizophrenia.

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DSM-5 and Bleuler’s Influence

Bleuler’s Four As remain relevant in the DSM-5’s description of schizophrenia, highlighting the importance of cognitive and emotional disturbances.

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Kurt Schneider’s Contributions to Schizophrenia

Schneider contributed to understanding schizophrenia by identifying key symptoms that help diagnose the disorder, especially "first-rank symptoms" (e.g., auditory hallucinations, delusions of control).

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Schneider’s First Rank Symptoms

These symptoms are considered particularly diagnostic for schizophrenia, including auditory hallucinations and thoughts being controlled by external forces.

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Schneider’s Influence on DSM-5

Schneider’s first-rank symptoms remain influential in how schizophrenia is diagnosed in the DSM-5.

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Genetic Contribution to Schizophrenia

Genetics play a significant role in schizophrenia, with a higher risk for those with family history.

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Prevalence of Schizophrenia in Western Culture

Schizophrenia occurs in about 1% of the general population in Western cultures.

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Concordance Rate in Dizygotic Twins

The concordance rate for schizophrenia in dizygotic twins is about 17%, meaning if one twin has schizophrenia, the other twin has a 17% chance of developing it.

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Brain Abnormality in Positive Symptoms

Enlarged ventricles and dopamine dysregulation are associated with positive symptoms of schizophrenia.

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Dopamine and Positive Symptoms

High dopamine levels in certain brain areas are linked to positive symptoms like delusions and hallucinations.

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Parkinson’s Disease and Schizophrenia

Parkinson’s disease is relevant because its treatment (dopamine-blocking drugs) can induce symptoms similar to schizophrenia.

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Amphetamines and Schizophrenia

Amphetamines, which increase dopamine activity, can induce or worsen positive symptoms of schizophrenia.

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Brain Abnormality in Negative Symptoms

Smaller hippocampus and prefrontal cortex volumes are more linked to negative symptoms of schizophrenia.

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Children-at-Risk Studies

These studies look at children who show early signs of developing schizophrenia, identifying impairments such as attention and motor skills.

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Neurointegrative Deficit

The neurointegrative deficit is a concept that refers to early cognitive and motor impairments in children who develop schizophrenia, proposed by researchers like Gochros.

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Double-Bind Hypothesis

Proposed by Bateson, it suggests that contradictory communication patterns from parents contribute to schizophrenia.

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Schizophrenogenic Mother Hypothesis

Fromm-Reichmann proposed that overprotective or rejecting mothers were responsible for causing schizophrenia in children.

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Validity of Double-Bind and Schizophrenogenic Mother

These hypotheses are largely discredited as primary causes of schizophrenia, though they contributed to understanding family dynamics.

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Expressed Emotion

Refers to the negative emotions expressed by family members, such as criticism or hostility, and it predicts relapse in patients with schizophrenia.

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Research on Expressed Emotion

Studies show that high expressed emotion in families significantly increases the risk of relapse in schizophrenia patients.

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Social Selection Theory

Suggests that people with schizophrenia may end up in lower socio-economic status due to their illness.

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Increased Stress Theory

Proposes that stressful social environments can trigger or exacerbate schizophrenia symptoms.

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Social Selection vs Increased Stress

These theories are not mutually exclusive, as both social factors and stressors can contribute to the development and progression of schizophrenia.

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Labeling Theory and Social Stigma

Labeling theory suggests that societal labels can reinforce mental illness, while social stigma makes it harder for people with schizophrenia to function in society.

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First Integrative (Biopsychosocial) Model of Schizophrenia

The first biopsychosocial model, developed by researchers like Gochros, integrates biological, psychological, and social factors to explain schizophrenia.

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Neurointegrative Deficit in the Integrative Model

In this model, the neurointegrative deficit explains early cognitive and motor impairments seen in individuals later diagnosed with schizophrenia.

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Schizophrenia-Spectrum Personality Disorders

These disorders are included in the biopsychosocial model as they share traits with schizophrenia, such as odd beliefs or social withdrawal.

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Antipsychotic Medications and Symptom Relief

Antipsychotic medications are most effective for treating positive symptoms, such as delusions and hallucinations, due to their dopamine-blocking effects.

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Major Side Effects of Antipsychotics

Common side effects include weight gain, sedation, and movement disorders like tremors.

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Tardive Dyskinesia

A serious side effect of long-term antipsychotic use, characterized by involuntary, repetitive movements.

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Antipsychotic Medications: Necessary and Sufficient?

Antipsychotic medications are necessary for managing schizophrenia but not sufficient alone; psychosocial treatments are also important.

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Major Psychosocial Treatments for Schizophrenia

Include Cognitive Behavioral Therapy (CBT), social skills training, and family therapy.

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Recovery vs Cure

Recovery refers to managing symptoms and improving quality of life, whereas cure implies complete elimination of symptoms.

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Elements of Recovery

Key elements include improving functioning, enhancing social integration, and managing symptoms, rather than a full cure.

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Who First Described Autism

Leo Kanner first described autism in 1943 as a unique developmental disorder.

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Autism as Childhood Version of Schizophrenia

Autism was initially thought to be a childhood version of schizophrenia, though this view has since been revised.

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Refrigerator Mother Hypothesis

This hypothesis suggested that cold, unloving mothers caused autism, similar to the "Schizophrenogenic Mother" theory for schizophrenia.

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Behavior Modification for Autism

Developed and popularized by Ivar Lovaas, this treatment is based on behaviorism and uses reinforcement techniques to teach children with autism.

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Lovaas’ 1987 Study

The study showed that early, intensive behavior modification could lead to significant improvements in social and communication skills in children with autism.

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Criticisms of Lovaas Study

Criticisms include ethical concerns about the intensity of the treatment and questions about its long-term effectiveness.

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Controversies in Behavior Modification for Autism

Some controversies involve whether such treatment methods are too harsh or if they genuinely lead to lasting improvements.

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Lovaas’ Recommendations for Effective Treatment

Lovaas recommended intensive, early, individualized interventions with a focus on positive reinforcement and structured learning.

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Predictors of Favorable vs. Unfavorable Outcomes for Home-Based Behavior Modification for Autism

Factors that predict favorable outcomes include early intervention, intensity of treatment, and family involvement; unfavorable outcomes are linked to late intervention and lack of consistency.

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Critical Period in Autism

The critical period refers to the idea that early intervention is most effective during certain developmental windows in a child’s life.

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Anorexia Nervosa

An eating disorder characterized by extreme restriction of food intake, intense fear of gaining weight, and a distorted body image.

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Bulimia Nervosa

An eating disorder characterized by binge eating followed by compensatory behaviors like vomiting or excessive exercise to prevent weight gain.

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Binge Eating Disorder

An eating disorder characterized by recurrent binge eating episodes without compensatory behaviors (e.g., no purging or over-exercising).

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Treatment Goals for Eating Disorders

The main goals are to normalize eating patterns, address body image issues, and improve psychological well-being.

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Treatment Modes for Eating Disorders

Treatment can include individual therapy, family-based therapy, group therapy, and inpatient or outpatient care.

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Treatment Approaches for Eating Disorders

Common approaches include Cognitive Behavioral Therapy (CBT), family therapy, and nutritional counseling.

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Attention-Deficit Hyperactivity Disorder (ADHD)

A neurodevelopmental disorder characterized by symptoms of inattention, hyperactivity, and impulsivity.

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Specific Learning Disorder

A neurodevelopmental disorder characterized by difficulties in specific academic areas, such as reading, writing, or math, despite having average or above-average intelligence.

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Autism Spectrum Disorder (ASD)

A neurodevelopmental disorder marked by social communication challenges, repetitive behaviors, and restricted interests.

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Intellectual Disability

A neurodevelopmental disorder characterized by below-average intellectual functioning and deficits in adaptive behavior, starting before the age of 18.

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Sleep-Wake Disorders

Two major types: Dyssomnias (problems with the amount, quality, or timing of sleep) and Parasomnias (abnormal behaviors during sleep, such as sleepwalking or night terrors).

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Dyssomnias

Disorders like insomnia, narcolepsy, and sleep apnea, which involve issues with the quantity or quality of sleep.

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Parasomnias

Disorders like sleepwalking, night terrors, and REM sleep behavior disorder, involving abnormal activities during sleep.

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Sexual Dysfunctions

A category of disorders characterized by persistent difficulties in sexual response or desire, such as erectile dysfunction or premature ejaculation.

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Paraphilic Disorders

Involve sexual arousal to atypical objects, situations, or individuals, such as fetishistic disorder or pedophilic disorder.

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Gender Identity Dysphoria

A condition where an individual experiences a disconnect between their assigned gender at birth and their experienced or expressed gender.

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Major Neurocognitive Disorder

A disorder marked by significant cognitive decline in areas such as memory, attention, or reasoning, affecting daily functioning.

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Mild Neurocognitive Disorder

A less severe decline in cognitive functioning, where individuals may experience cognitive difficulties but can still maintain independence in daily life.

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Alzheimer’s Disease

The leading cause of major neurocognitive disorder, characterized by progressive memory loss, disorientation, and changes in behavior.

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Etiological Factors of Alzheimer’s Disease

These include genetics (e.g., APOE4 gene), aging, and environmental factors such as lifestyle choices.

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Treatment Focus for Neurocognitive Disorders

Since there is no cure for irreversible neurocognitive disorders, treatments typically focus on managing symptoms, improving quality of life, and slowing progression.

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Substance Use

The consumption of alcohol, drugs, or other substances in any amount, regardless of negative consequences.

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Intoxication

The physiological and psychological effects of substance use, which can impair judgment, coordination, and functioning.

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Substance Use Disorder

A condition characterized by compulsive use of a substance despite harmful consequences, and an inability to control use.

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Addiction

A severe form of substance use disorder, marked by the compulsive need for a substance, often with tolerance and withdrawal symptoms.

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Physiological Dependence

The state where the body adapts to a substance, requiring more of it to achieve the same effect, and withdrawal symptoms occur when the substance is not used.

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Tolerance

The need to use increasing amounts of a substance to achieve the same effect due to the body's adaptation.