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what diagnostic triage based on
clincal history and examination
is imaging approriate for a patient w/ LBP and no red flags
no
uncecessary imagning
increasd cost
not superior in pain, function, QOL
potential harms
potential harms of unecasry imaging
more fear
more surgeru
more injections
more opiods
more work absence
LBP w/ low-velocity trauma, osteoporosis, elderly individual or chronic steroid use
x-ray, MRI, CT
subacute or chronic LBP with thoughts of surgery or intervention
MRI
spine trauma where imaging that migth need imaging
bony or midline tenderness
neurologicl signs
intoxication
major distracting injury
spine trauma imaging
CT
inflammatory back pain
X ray
x ray of SI joint
axial spondyloarhtritis
insidious onset
night pain
get better when moving
stiffness
young
AP
frontal plane curvature
spinous processes
facet joints
pedicles
lateral
spinal “lines”
vert body heights
discs spaces
IV foramina
facets and Pars
spondylolisthesis
slippage
spondylolyisis
fracture
complete ankylosis
not joint line at SI joint
disc nomenclature
disc buldge
>25% not focal
annular fissure
disc herniation
< 25% focal
protrusion
extrusion
sequestration
location
MRI and pathology
poor correlation between most degenerative changes and symptoms
positive correlation
large extrusions
major neural compromise
intravertebral herniation
schmorl’s node
sup/inf herniation
through endplate
degenerative changes in vertebral endplates
modic type 1
type 2
type 3
modic type 1
edema, inflammatory & fibroplastic activty
hypo on T1, hyperintense on T2
modic type 2
red marrow replaced by yellow marrow
hyper in T2, less hyperintense or iso on T2
modic type 3
sclerosis of end-plates
hypo on T1 and T2
what is the purpose of surgery
stabilize unstable segments (fractures and slips)
eliminate painful motion
cure infections/tumors
decompress neurologic elements
realign mal-aligned segments
restore normal spinal balance to reduce muscular stress and load
absolte indications for surgery
fracture instability
neural compression leading to motor weakness
cauda equina syndrome
cord compression
significant progressive deformity
relative indications for surgery
leg pain due to root compression
LBP due to slip, IDR or facet disease. All w/ inability to do desired activities
neurogenic cladification
malaligned muscular back pain w/ poor adaptation mechanisms
contradindications
medically, can the patient tolerate the surgery? are there medically ameliorate issues?
what is the patient’s T score? (osteoporosis)
does the patient understand the expectations?
is the patient high risk? (DM type 1, CAD, age, stroke)
is the intensity of the surgical procedure and recovery greater than the benefits of the expected surgical success?
for fusion patients, do they smoke or drink alcohol?
lumbar hnp
motor weakness (except in EHL but not L5)
pain intolerable
failure of conservative treatment w/ continued impairment
coming up on 3-6 months of sx w/o relief
cauda eqina
surgical emergency
should be operated within 12 hours of symptom onset
saddle paraethesia
progress after microdisectomy
numbness typically fades away within 6 months. the patch of numbness will contract over time
not unusual that some ache in the leg returns w/ increased function. if the pain draws attention a short course of an oral steriod can be helpful
if the pain is greater than VAS 5-6, a new MRI could be helpful (serome or recurrent herniation). seroma can be aspirated
athletes don’t participate in competitive activities fro 8-12 wks
annular tears decline degenerative discs
continued leg pain after microdiscetectomy
wrong diagnosis
wrong level
recurrent disc HNP
epidural fibrosis
seroma/hemotoma
incomplete decompression
collapse of foramen at surgical decompression site
new onset compression at different level
chronic nerve injury
recurrent disc hernation
w/ HNP, annulus by definition has tear through all 30 rings
disc is avascular so tear can’t heal
same process that induced nucleus to initially desiccate/separate continues
recorrance risk is 10-15% w/ or w/o surgery
central laminotomy and varants
central laminectomy
central laminotomy
bilateral hemilaminotomy
lateral reess decompression
foraminal decompression
otomy
half removal
-ectomy
full removal
fusion purpose
join 2 or more vertebra together w/ a living bridge of bone to stop painful motion of disc, facets, eliminate instability or restore alignment due to collapse of the foramen or progressive scoliosis/kphosis
fusion requirements
must stablize to prevent motion
must have graft material to induce bone growth
must have “injured” surfaces to induce healing reaction
oral supplement ant metabolic impedence to fusion
NSAID (3 mos-inflammatory phase)
steroids
blood clotters and thinners (supplements beginnig w/ “G”)
diabetes, malnutrition and alcoholism
cigarette smoking
osteoporosis (Forteo)
benefit of TLIF
direct decompression of the exiting nerve root
can decompress the central canal if stenosis is present
removal of facet to open foramen
autogenous bone graft from facet
all performed trhough one posterior incsiion
indications for surgery for Isthmic spondylolisthesis
progressive slip grade 2 or larger
back pain-failed RX
radiculopathy - failed RX
motor weakness
oblique latersl interbody fusion/XLIF
lateral approach to the lumbar spine through the abdominal wall
approach through or anterior to the psoas muscle
normally needs backup posterior fixation
femoral nerve caution
lumbar artifical disc replacements
knee and hips joints are solo joints-replacement covers the entire motion segments
a motion segmen in the spine contains 3 joints
if the disc space has collapsed, stretcheing the diminished space to implant an ADR will put alterd forces on the facets
if the disc fails, removal can be life trheatening due to vena caval adhesions
metal on plastic causes wear depbris and osteolysis-loosening
ADRs have no shock absorption capability
stress concentration of metal endplate on bone endplate can cause overload fractures
interspinous kyphogenic device
patients w/ central stensis bend forward to reduce canal compression w/ forward flexion
this device is jammed between the spinous processes and forces flexion (kyphosis) of the motion
erosion causes pain
coflex interspinous devices
coflex device designed to be “shock absorption” device
as you can see this device causes severe kypohis between L3-4 and L4-5 causing a flat back deformity
vertebroplasty
Poly Methyl-methacrylate injected into fracture cavity, PMMA hardens into a cement space filler
placed through a pedicle cannula
cement can extrude through a fracture line or through basivertebral vein to cause canal compression
may be ok in elderly who have chronic pain after insufficiency fractures don’t heal
Foraminal Collapse
Full angular collapse with failure of facets and disc and asymmetric collapse of the vertebral segment
Nerve is crushed by loss of disc height, spur formation and rotary listhesis
Only way to relieve pain and reconstruct is with TLIF or XLIF
indications for Pars fracture repair
Painful uni or bilateral fractures
Failed 3+ months bracing with pain on return to activities
Recurrent fracture
Intact disc without significant slip
No significant degenerative disc
No severe atrophic fracture ends or significant gap of the ends
Younger patient
contraindications for surgical repair of pars fracture
• Any significant slip
• Degenerative disc DZ
• Fracture is distracted >3mm or bilaterally atrophic
• age?
Scheuermann’s Kyphosis
• Young male with severe lower thoracic pain and standing fatigue pain
• Failed all conservative care
• Normal kyphosis 20-40 degrees
• Kyphosis= 77 degrees
scoliosis
Chronic pain due to multiple angular collapses
surgical complications revision
Previously called “Failed Spine Surgery Syndrome” (FBSS) or “Post Laminectomy Syndrome” as failure from spine surgery was not uncommon 20 years ago and no one understood why patients had continued pain post-operatively
SI Fusion Implant Malposition
• Overused surgical technique
• Incidence of SI syndrome 3-4% with SI pain
• Need for surgery should be much lower than 2%
• Implant malposition can cause radiculopathy as noted here
• Patient here had new L5 radiculopathy post-op
• Removal of fixation device cured post thigh pain
when to start therapy
decompression/microdiscs; start 1-2 weeks post-op
work on “nerve flossing” gently not uncommon to have flair-up radiculopathy delayed 12-24 hrs
motor strengthening in the face of neurological weakness
no EMS w/ first 3 months-neurological weakness
isometric strengthening is best for 1st 4-6 wks
avoid the dreaded Bend, load, twist
when to start therapy for fusion
start therapy 6 wks post-op
fusion mass is quite soft at this point - heavy troque in therapy could losen hardware
no bend, load, twist for 3 months
cardio conditioning and isometrics
start Full ROM at 3 months
red flags in recovery
incision drainage, redness, significant tenderness
constitutional signs (fever, chills, lethargy)
progressive SLR - increased leg pain/weakness
increased lower back pain/spasm (infection)
ileus; swollen belly, reduced bowel sounds (discitis)
swollen foot/+ Homan’s sign (DVT)
SOB/chest pain (PE, Pneumonia)